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1 fusion reflects the macro- and microvascular coronary circulation.
2 nt insights into disease activity within the coronary circulation.
3 an extremely rare congenital anomaly of the coronary circulation.
4 sessing the anatomy and functionality of the coronary circulation.
5 oward managing functional alterations of the coronary circulation.
6 k of disease in both the cerebrovascular and coronary circulation.
7 not have to restore efficient blood flow in coronary circulation.
8 rdial and proepicardial contributions to the coronary circulation.
9 microvasculature but interrogates the entire coronary circulation.
10 er to the different flow dynamics typical of coronary circulation.
11 E14.5, concurrent with the establishment of coronary circulation.
12 n microvessels that are perfused by the host coronary circulation.
13 sociated with functional disturbances of the coronary circulation.
14 transplantation and integrates with the host coronary circulation.
15 vasodilation plays a vital role in the human coronary circulation.
16 d of myocytes and vessels connected with the coronary circulation.
17 of the mechanisms action of NE in the human coronary circulation.
18 in patch clamp recordings and in the intact coronary circulation.
19 diated responses are potentiated only in the coronary circulation.
20 ence of CMV in atheromatous plaques from the coronary circulation.
21 and reducing the vasodilatory reserve of the coronary circulation.
22 may have contributed to the evolution of the coronary circulation.
23 ell known to affect vascular function in the coronary circulation.
24 timates of microvascular shear stress in the coronary circulation.
25 sts that they may be important regulators of coronary circulation.
26 r acetylcholine in the atherosclerotic human coronary circulation.
27 constrictor properties of ET-1 in the canine coronary circulation.
28 tic material and soluble substances into the coronary circulation.
29 ng is a measure of the overall health of the coronary circulation.
30 gnificant elevations in NO production by the coronary circulation (46 +/- 23, 129 +/- 44, and 63 +/-
31 n microvessels anastomosed with the rat host coronary circulation and delivered blood to the grafts.
32 the suggestion that inhaled NO may reach the coronary circulation and have a negative inotropic effec
33 ental injection to directly access the fetal coronary circulation and injection at E12.5 when the mas
34 ial dysfunction, and atherothrombosis in the coronary circulation and may explain the greater efficac
35 (a) provide an overview of the physiology of coronary circulation and myocardial perfusion; (b) descr
36 methyl-L-thiocitrulline (SMTC), on the human coronary circulation and on flow-mediated dilatation in
37 al reference range for studies assessing the coronary circulation and post-stenotic coronary vasodila
38 ium, but their relative contributions to the coronary circulation and the molecular mechanisms regula
39 lator effects of testosterone in vivo in the coronary circulation and the potential mechanisms of its
40 diated vasodilation in either the femoral or coronary circulations and did not alter SNP responses in
41 based gene delivery is poorly adapted to the coronary circulation, and current gene transfer vectors
42 1, is involved in vascular reactivity in the coronary circulation, and has a protective role in ische
43 ved CNP is involved in the regulation of the coronary circulation, and NPR-C activation underlies the
44 new vessels were connected with the primary coronary circulation, and this increase in vascularizati
45 elaxation was impaired during hypoxia in the coronary circulation, and this resulted in greater cardi
46 atherosclerotic epicardial and microvascular coronary circulation; and 2) acetylcholine-induced coron
49 impairs microvascular dilation in the human coronary circulation as a result of reduced NO bioavaila
50 nctional and/or structural alteration of the coronary circulation associated with coronary risk facto
52 , and physiological assessment of the native coronary circulation before CABG may also reduce the inc
54 airs vascular function in the peripheral and coronary circulations, but its effects on cerebral arter
56 ina, synergistic changes in the systemic and coronary circulations combine to improve vascular-ventri
68 s interventions probably by offering reserve coronary circulation, if in-stent restenosis were to occ
69 scence to assess NO(x) production across the coronary circulation in chronically instrumented dogs du
70 beneficial effects of ACE inhibitors on the coronary circulation in DCM that are not shared by AT1 r
71 rcumflex arteries, suggesting a role for the coronary circulation in determining local drug depositio
72 sion during adenosine stress, reflecting the coronary circulation in patients during long-term follow
74 maging strategy, to define the 3-dimensional coronary circulation in the young and aged heart and its
75 icrovascular diameters and velocities in the coronary circulation in vivo and used these variables to
76 coronary artery stems, and thus a functional coronary circulation, in the embryos injected with VEGF-
80 he progression of atherosclerotic plaques in coronary circulation is modulated by several risk factor
85 oth cohorts), a culprit location in the left coronary circulation (left anterior descending or circum
87 tion generates a distinct compartment of the coronary circulation located within the inner half of th
88 riod, which refers to early-decompression of coronary circulation marked by pressure decay (dP/dt < 0
89 n of the interventional cardiologist and the coronary circulation, now in 2001 nearly every vascular
91 but not scrambled control peptide, into the coronary circulation of isolated perfused hearts rapidly
92 may reduce vasodilator responsiveness in the coronary circulation of subjects with coronary disease o
93 rmined the changes in NO production from the coronary circulation of the conscious dog during exercis
94 g of their effects on the myocardium and the coronary circulation, optimization of delivery technique
96 In contrast to any other vascular bed, the coronary circulation receives its perfusion mostly durin
98 lly digested via retrograde perfusion of the coronary circulation, resulting in rapid and extensive b
99 oglycerin causes changes in the systemic and coronary circulation that combine to reduce myocardial o
101 Whether the abnormal responses of the human coronary circulation to acetylcholine in patients with h
102 f preconditioning, has been infused into the coronary circulation to induce therapeutic preconditioni