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1 2 mug HGF and 2 to 8 mug IGF-1) 30 min after coronary reperfusion.
2 tegies that can enhance timely and effective coronary reperfusion.
3 resolution is an early noninvasive marker of coronary reperfusion.
4 in preserving microvascular perfusion after coronary reperfusion.
5 e of myocardial salvage and viability during coronary reperfusion.
6 by which time 89% of patients had undergone coronary reperfusion.
7 eared to be safe but did not improve pre-PCI coronary reperfusion.
9 tal administration of ticagrelor can improve coronary reperfusion and the clinical outcome is unknown
11 ic therapy within 1 hour, and use of overall coronary reperfusion by 11% to 12% for patients with inf
12 nloading the left ventricle despite delaying coronary reperfusion during an acute MI reduces myocardi
13 about the clinical significance of HF in the coronary reperfusion era and the impact of its timing on
14 e salvage-dependent from salvage-independent coronary reperfusion in acute myocardial infarction and
15 m)Tc-N-NOET imaging to assess the success of coronary reperfusion in patients with acute myocardial i
16 tricular (LV) workload (LV unloading) before coronary reperfusion is emerging as a potential approach
17 ery centrifugal bypass system while delaying coronary reperfusion limits myocardial injury in a model
20 infarcting hearts with alternating cycles of coronary reperfusion/occlusion attenuates infarction, bu
22 n A-antagonist administration at the time of coronary reperfusion preserves postischemic microvascula
23 n-specific thrombolytic regimens and improve coronary reperfusion rates in streptokinase-based regime
24 evidence demonstrating benefits from prompt coronary reperfusion, registries continue to show that m
26 increasing use and success of interventional coronary reperfusion strategies, morbidity and mortality
28 stablished before the introduction of modern coronary reperfusion therapy and secondary prevention st
30 essed their formation in patients undergoing coronary reperfusion via percutaneous transluminal coron
32 e, defined as the time from symptom onset to coronary reperfusion, was a pre-specified subgroup of in
33 ility as an index of oxidative stress during coronary reperfusion, we measured urinary levels by gas