ライフサイエンスコーパス: corpora lutea

1 normal distribution of ovarian follicles and corpora lutea.

2 educed numbers of large antral follicles and corpora lutea.

3 rrested folliculogenesis and failure to form corpora lutea.

4 and atrophic reproductive tracts with absent corpora lutea.

5 egenerating antral follicles, and absence of corpora lutea.

6 xhibited a marked reduction in the number of corpora lutea.

7 produced fewer oocytes and also showed fewer corpora lutea.

8 ntation-promoting activity has been found in corpora lutea.

9 e, whereas they undergo differentiation into corpora lutea.

10 ary to VEGF expression in both follicles and corpora lutea.

11 tions of follicles, they ovulate and develop corpora lutea.

12 wing preovulatory follicles as well as early corpora lutea.

13 was weak in atretic follicles and regressing corpora lutea.

14 o ovulate and develop numerous inappropriate corpora lutea.

15 ulti-oocyte follicles and an accumulation of corpora lutea.

16 ndations were adherent in most instances for corpora lutea (88%), simple cysts (56%), and cysts sugge

17 C/EBP beta-deficient ovaries lack corpora lutea and fail to down-regulate expression of th

18 le infertility with abnormal ovaries lacking corpora lutea and increase in luteinizing hormone levels

19 ing washed mitochondria isolated from bovine corpora lutea and purified recombinant His-tag StAR prot

20 ed imaging of the ovary revealed cyst walls, corpora lutea, and ovarian medulla.

21 Mutant mice also failed to develop corpora lutea, as evident by the lack of luteal marker g

22 zyme 20alpha-hydroxysteroid dehydrogenase in corpora lutea (CL) inactivated of Lgr5.

23 f ischemic necrosis were demonstrated in the corpora lutea (CLs) of treated animals.

24 in wild type recipients, significantly more corpora lutea containing un-ovulated oocytes were presen

25 nsplants displayed normal estrous cycles and corpora lutea, despite DHT treatment, implying extraovar

26 her percentage of antral follicles and fewer corpora lutea; follicles progressed to the antral stage

27 aphase II oocytes can be retrieved from, and corpora lutea form in, ovaries of aged Bax-/- females fo

28 a lack of follicular development and lack of corpora lutea formation, as well as lack of secretory ch

29 f preovulatory follicles and luteal cells of corpora lutea had no effect on ovarian morphology and fe

30 ning, we identified individual follicles and corpora lutea in intact ovaries.

31 contrast, progesterone, which is secreted by corpora lutea, increased SLA and beta(2)m in luminal epi

32 n mice on lower P:C (1:8), and the number of corpora lutea, indicative of recent ovulations, was grea

33 increase in NUR77 mRNA was observed in mice corpora lutea just before parturition at a time when 20a

34 In normally developing corpora lutea, macrophages were intimately juxtaposed wi

35 ncreased fertility based on ovulated egg and corpora lutea numbers.

36 Analysis of PKC delta activity in corpora lutea obtained during pregnancy by both the IC k

37 sessed the activation status of PKC delta in corpora lutea obtained when the corpus luteum is exposed

38 ithelial and granulosa cells and also in the corpora lutea of GREKO(-/-) mice.

39 munohistochemical analyses demonstrated that corpora lutea of p27(Kip1), p21(Cip1) double-null mice s

40 osis of granulosa cells, decreased number of corpora lutea, reduced ovarian size, and subfertility in

41 ibit defects in luteinization, with numerous corpora lutea, some of which contain central trapped, fu

42 Similar to corpora lutea, the lesions were highly vascularized, alt

43 support development and maintain functional corpora lutea through the production of estrogen.

44 ssion was evaluated in ovarian follicles and corpora lutea utilizing immunohistochemical techniques a

45 70, inhibition of endometrial maturation and corpora lutea was observed.

46 o found to be severely subfertile, and fewer corpora lutea were found to form in response to exogenou

47 Corpora lutea were present in all groups.

48 No corpora lutea were present.

49 In the ovaries of macrophage-depleted mice, corpora lutea were profoundly abnormal, with elevated Pt

50 turation of secondary ovarian follicles into corpora lutea, which express high levels of p27, was mar

51 of follicle maturation, most notably around corpora lutea, without significantly affecting follicula