ライフサイエンスコーパス: corpus callosum

1 nd white matter of cerebrum, cerebellum, and corpus callosum).

2 frontal-occipital fasciculus, cingulum, and corpus callosum).

3 pallidus, ponto-cerebellar atrophy, and thin corpus callosum.

4 ciency was associated with hypoplasia of the corpus callosum.

5 ty recovered by 21 d post MCAO in KI mice in corpus callosum.

6 ior dorsal cingulum bundle above genu of the corpus callosum.

7 tructural connectivity in anterior/posterior corpus callosum.

8 ite matter organization, particularly in the corpus callosum.

9 trics (FA, MD, RD, and AD), primarily in the corpus callosum.

10 which appeared progressive, and a prominent corpus callosum.

11 mispheric commissural bridges traversing the corpus callosum.

12 as frontal white matter and the genu of the corpus callosum.

13 also failed to cross the midline to form the corpus callosum.

14 rected P < .05) in posterior portions of the corpus callosum.

15 ng an increase in unmyelinated fibers in the corpus callosum.

16 ased in the superior cerebellar peduncle and corpus callosum.

17 myelination of the axonal projections in the corpus callosum.

18 ct orientation within white matter, e.g. the corpus callosum.

19 cross-hemispheric communication through the corpus callosum.

20 cerebellar vermis and lobules V and VI, and corpus callosum.

21 ith cuprizone, and OPCs were sorted from the corpus callosum.

22 ls of PEDF was capable of infiltration along corpus callosum.

23 f genes related to myelination and increased corpus callosum.

24 rrhages predominantly in the splenium of the corpus callosum.

25 m aberrant axonal bundles within the rostral corpus callosum.

26 decreased numbers of myelinated axons in the corpus callosum.

27 tal retardation (MR) and malformation of the corpus callosum.

28 he interhemispheric connecting fibers of the corpus callosum.

29 evere mental retardation and agenesis of the corpus callosum.

30 asciculus, as well as in the splenium of the corpus callosum.

31 ossibly associated with the evolution of the corpus callosum.

32 wing demyelinating injury to the adult mouse corpus callosum.

33 ivo pathology of the sensorimotor cortex and corpus callosum.

34 orks as well as in the frontal aspect of the corpus callosum.

35 culus, internal capsule, and splenium of the corpus callosum.

36 nd one patient had a cytotoxic lesion of the corpus callosum.

37 ed with thinning of the myelin sheath in the corpus callosum.

38 tural brain anomalies, including agenesis of corpus callosum.

39 s on oligodendrocyte lineage cells (OLCs) in corpus callosum.

40 TBI rats had abnormalities in the cortex and corpus callosum.

41 the corpus callosum, and fiber strain of the corpus callosum.

42 missure: 0.05 ug . g(-1) +/- 0.01, P = .002; corpus callosum: 0.05 ug . g(-1) +/- 0.02, P = .001; cra

43 ar pathology and with a predilection for the corpus callosum (23 of 39; 59%; 95% CI: 42, 74) and juxt

44 sum consistent with cytotoxic lesions of the corpus callosum (4.1%).

45 as achieved with FA index measurement of the corpus callosum (51%).

46 alities such as ventriculomegaly and a small corpus callosum (67%).

47 Key features on neuroimaging include a thin corpus callosum (90%), ventriculomegaly (65%) often with

48 SATB2 specifies neuronal projections via the corpus callosum, a large axon tract connecting the two n

49 lities included intracranial calcifications, corpus callosum abnormalities, abnormal cortical formati

50 ic individuals with isolated agenesis of the corpus callosum (ACC) without intellectual disability.

51 bility in the structural organization of the corpus callosum accounts for these differences.

52 precursor protein aggregates in axons of the corpus callosum after traumatic brain injury as compared

53 tion in 25 participants with agenesis of the corpus callosum (AgCC) and 21 matched neurotypical indiv

54 c dominance in patients with agenesis of the corpus callosum (AgCC) and found reduced laterality (i.e

55 e investigated the effect of agenesis of the corpus callosum (AgCC), one of the most common brain mal

56 sociated with brain malformations, including corpus callosum agenesis (ACC) and microcephaly.

57 mental delay and/or intellectual disability, corpus callosum agenesis or hypoplasia, flexion contract

58 sability, variable axon pathfinding defects (corpus callosum agenesis or hypoplasia, mirror movements

59 isorders, such as autism spectrum disorders, corpus callosum agenesis, Joubert syndrome, Kallmann syn

60 ctrum disorder, intellectual disability, and corpus callosum agenesis.

61 and all cases exhibited partial or complete corpus callosum agenesis.

62 re present in frontal and temporal lobes and corpus callosum (all p values <0.01).

63 However, it is unclear whether corpus callosum alterations are related to the underlyin

64 Except for the genu of the corpus callosum, an increase in AD values was also found

65 diffusion anisotropy of the splenium of the corpus callosum and adjacent parietal white matter (P <

66 osomal recessive inheritance pattern of thin corpus callosum and axonal Charcot-Marie-Tooth disease i

67 sive hereditary spastic paraplegia with thin corpus callosum and axonal peripheral neuropathy (SPG7/P

68 RD, and MD in more extended portions of the corpus callosum and beyond (eg, corona radiata and infer

69 nction leads to insufficient angiogenesis in corpus callosum and catastrophic axon loss.

70 midal neurons, but strongly expressed in the corpus callosum and caudate axon fibers.

71 ed lower FD in numerous tracts including the corpus callosum and corona radiata compared to mature-bo

72 The largest differences were observed in the corpus callosum and corona radiata.

73 volumes, and 3) fractional anisotropy in the corpus callosum and corona radiata.

74 TGFbeta1 was significantly decreased in the corpus callosum and cortex of Cav1.2 knock-out mice thro

75 y childhood onset cognitive impairment, thin corpus callosum and enlarged ventricles.

76 eurofilament NF200, the reduced thickness of corpus callosum and external capsule, and decline of mat

77 tant rats, which were most pronounced in the corpus callosum and external capsule.

78 ng, white matter injury was prominent in the corpus callosum and internal capsule on day 3 and then p

79 We examined the corpus callosum and its subregion volumes and their rela

80 a reduced number of myelinated axons in the corpus callosum and optic nerves.

81 cific damage to the genu and splenium of the corpus callosum and parahippocampal tract bilaterally (P

82 sive hereditary spastic paraplegia with thin corpus callosum and peripheral axonal neuropathy, and ac

83 es and attenuated white matter growth of the corpus callosum and pons relative to nondrinkers.

84 onal anisotropy is reduced in Plp-Nf1 (fl/+) corpus callosum and that interhemispheric functional con

85 yelination of the graft-derived axons in the corpus callosum and that their terminals form excitatory

86 ad lower mean diffusivity in the genu of the corpus callosum and the anterior thalamic tracts.

87 hted that altered WM connectivity within the corpus callosum and the cingulum are strongly associated

88 ced in the extensive regions of the CST, the corpus callosum and the right PCG.

89 of proteins regulating the formation of the corpus callosum and their respective developmental funct

90 OLs, and improved overall myelination in the corpus callosum and white matter tracts of the spinal co

91 lizes to white matter tracts, especially the corpus callosum, and at a low level in neurons, while PI

92 ions in the hippocampus, degeneration of the corpus callosum, and ataxia and seizures.

93 /- but not Nova1-/- mice had agenesis of the corpus callosum, and axonal outgrowth defects specific t

94 tly present with spastic paraparesis, a thin corpus callosum, and cognitive impairment.

95 plegia, developmental delay, agenesis of the corpus callosum, and enlargement of the third cerebral v

96 strain (MPS) of the whole brain, MPS of the corpus callosum, and fiber strain of the corpus callosum

97 late gyrus, cortex of the temporal lobes and corpus callosum, and fractional anisotropy (FA) index me

98 encephalic commissures (anterior commissure, corpus callosum, and hippocampal commissure) along with

99 Small telencephalic commissures (anterior, corpus callosum, and hippocampal), an enlarged posterior

100 nifest stage around the striatum, within the corpus callosum, and in posterior white matter tracts.

101 ficantly higher in the medial cortex, in the corpus callosum, and in the thalamus than in the corresp

102 interconnect auditory and motor structures, corpus callosum, and in tracts interconnecting cortical

103 sion abnormalities, cytotoxic lesions of the corpus callosum, and intensive care unit-related complic

104 to OPCs residing in secondary motor cortex, corpus callosum, and primary somatosensory cortex of adu

105 rmalities in astrocytes, particularly in the corpus callosum, and provide support for hypotheses that

106 p the mutant BTBR mouse brain, which lacks a corpus callosum, and recapitulated its known connectopat

107 otonia, developmental delay, thinning of the corpus callosum, and seizures.

108 rain malformation (microcephaly, agenesis of corpus callosum, and simplified gyration), and severe en

109 ith the nodes of Ranvier in the optic nerve, corpus callosum, and spinal cord of young adult mice or

110 ghout the CNS, including in the optic nerve, corpus callosum, and the spinal cord.

111 ampal dentate gyrus, partial agenesis of the corpus callosum, and ventriculomegaly.

112 vous system (white matter disturbances, thin corpus callosum, and widened ventricles); global delay w

113 ultiple individuals included agenesis of the corpus callosum, ano-rectal malformations, seizures, and

114 small normally proportioned cerebellum, and corpus callosum anomalies.

115 DystoniaNet identified clusters in corpus callosum, anterior and posterior thalamic radiati

116 regions included genu, body and splenium of corpus callosum, anterior and superior corona radiata, s

117 neurons in the neocortex results in lack of corpus callosum, anterior commissure, and corticospinal

118 aining and MPF in all anatomical structures (corpus callosum, anterior commissure, internal capsule,

119 ntry of new astrocytes from the SVZ into the corpus callosum appears to be balanced by astroglial apo

120 Brain malformations involving the corpus callosum are common in children with developmenta

121 We found significantly increased corpus callosum area and thickness in children with auti

122 e., fractional anisotropy alterations in the corpus callosum) as a shared feature of ASD, ADHD, and O

123 uding hippocampus hypoplasia and agenesis of corpus callosum, as well as neuromuscular and behavioral

124 enetic fate-mapping, we demonstrate that new corpus callosum astrocytes are continuously generated fr

125 These nestin fate-mapped corpus callosum astrocytes are uniformly postmitotic, ex

126 oglial apoptosis, because overall numbers of corpus callosum astrocytes remain constant during normal

127 ces were particularly robust in the anterior corpus callosum at the 6 and 12 month time points.

128 at could be named ACOG syndrome (agenesis of corpus callosum, axon pathfinding, cardiac, ocular, and

129 )/stria terminalis, genu and splenium of the corpus callosum, bilateral anterior and posterior limbs

130 , as did white matter areas (splenium of the corpus callosum, bilateral superior-parietal lobe, bilat

131 Corpus callosum bisections demonstrated that premotor co

132 in the diet induces rapid loss of OL in the corpus callosum by 2 d, accompanied by expression of sev

133 ruited during the remyelination phase to the corpus callosum (CC) and are capable of forming new olig

134 Lesions in the corpus callosum (CC) are important radiological clues to

135 nts report white matter abnormalities of the corpus callosum (CC) as an important predictor of neurod

136 idepost cells essential for interhemispheric corpus callosum (CC) axon navigation.

137 The corpus callosum (cc) contains nitric oxide (NO)-producin

138 anual responses to these unseen stimuli, the corpus callosum (CC) dynamically recruited areas in the

139 Interhemispheric axons of the corpus callosum (CC) facilitate the higher order functio

140 While microstructural alterations in corpus callosum (CC) have been identified as a consisten

141 the right cingulum hippocampus and anterior corpus callosum (CC) in aMCI compared to HC.

142 The corpus callosum (CC) is one of the most commonly reporte

143 The corpus callosum (CC) is the largest fiber tract in the m

144 In this light, the corpus callosum (CC) may represent the main responsible

145 ptic strength from the intact cortex via the corpus callosum (CC) onto deep neurons in deprived prima

146 1 controls the formation of the layer II/III corpus callosum (CC) projections through the development

147 The corpus callosum (CC) represents a clinically-relevant lo

148 m have been often reported to have a smaller corpus callosum (CC) than control subjects.

149 the widths of the frontal horn (FH) and the corpus callosum (CC) were not significantly different be

150 TR), superior longitudinal fasciculus (SLF), corpus callosum (CC), and corticospinal tract (CST).

151 vity (MD) and radial diffusivity (RD) in the corpus callosum (CC), superior longitudinal fasciculus (

152 connect the two cerebral hemispheres via the corpus callosum (CC).

153 hite matter (WM) damage, particularly to the corpus callosum (CC).

154 entilation, brain atrophy, dysgenesis of the corpus callosum, cerebellar vermis hypoplasia, and facia

155 decreased volume and abnormal signal), thin corpus callosum, cerebellar vermis hypoplasia, optic ner

156 features of the brain, such as the amygdala, corpus callosum, cerebellum, and gyrnecephalic index, al

157 ignificant decrease in FA in the genu of the corpus callosum characterized both conditions.

158 psy showed white matter abnormalities in the corpus callosum, cingulum and external capsule, with dif

159 ll to medium effect sizes, especially in the corpus callosum, cingulum and external capsule.

160 tensor imaging abnormalities observed in the corpus callosum, cingulum, and temporal lobe likely cons

161 anisotropy (FA) in the tapetum region of the corpus callosum (Cohen's d = -0.11, p = 0.0055).

162 aly, white-matter abnormalities, hypoplastic corpus callosum, congenital heart defects, and central h

163 also evident in the anterior portions of the corpus callosum connecting left and right frontal lobes.

164 the increased white matter integrity in the corpus callosum connecting these regions, suggesting an

165 ee with restricted diffusion foci within the corpus callosum consistent with cytotoxic lesions of the

166 l and macrostructural variability within the corpus callosum, consistent with differential effects on

167 d reductions in fractional anisotropy in the corpus callosum, corona radiata and external capsule, an

168 2% of WM tracts, respectively, including the corpus callosum, corona radiata, superior longitudinal f

169 e in several brain structures, including the corpus callosum, cortex, and striatum, and the corpus ca

170 d the psALS group, encompassing parts of the corpus callosum, corticospinal tracts and superior longi

171 The anterior corona radiata (d=0.40) and corpus callosum (d=0.39), specifically its body (d=0.39)

172 Remarkably, all seven genes showed corpus callosum defects, including thicker (Atg16l1, Cor

173 ial-temporal lobe epilepsy, microcephaly and corpus callosum deficiency, and by postnatal Day 21, mic

174 e crossing of an axonal subpopulation of the corpus callosum derived from the anterior cingulate cort

175 The role that corpus callosum development has on the hemispheric speci

176 rce for identifying new proteins integral to corpus callosum development that will provide new insigh

177 previously-identified proteins in aspects of corpus callosum development, and identifies new candidat

178 econdary hypomyelination, microcephaly, thin corpus callosum, developmental delay, intellectual disab

179 in the lateral aspect of the truncus of the corpus callosum, due to greater increase in FA (standard

180 determine the translatome of OLCs in vivo in corpus callosum during the remyelination phase of a chro

181 ns of Ube3b (-/-) mice had hypoplasia of the corpus callosum, enlarged ventricles, and decreased thic

182 The human corpus callosum exhibits substantial atrophy in old age,

183 microstructural integrity of the body of the corpus callosum (FA, beta = 0.01 [P = .01]; RD, beta = -

184 bilateral atrophy of the dorsal cingulum and corpus callosum fibers, which we interpret as a conseque

185 interhemispheric connectivity by controlling corpus callosum formation remains unclear.

186 n of WM damage that involved the body of the corpus callosum, fornix, and main anterior-posterior pat

187 Thus, the accentuated decline of the corpus callosum found in aging humans is not a universal

188 In slices of corpus callosum from mice subjected to a demyelination p

189 dsagittal area and regional thickness of the corpus callosum from T1-weighted MRI data from 213 chimp

190 ces and corticospinal tracts, to include the corpus callosum; frontal, sensory, and premotor cortices

191 findings suggest novel dual mechanism of the corpus callosum function in spatial attention and have b

192 First, whereas FA in anterior corpus callosum (genu) correlated with word-matching BPA

193 The corpus callosum has been implicated in the pathogenesis

194 he module (dorsomedial) lying closest to the corpus callosum has the most complete set of commissural

195 These findings suggest that the corpus callosum helps to drive language lateralization.

196 nd dermal abnormalities and the absence of a corpus callosum; his immune deficit was fully corrected

197 aging showed subtle abnormalities, including corpus callosum hypoplasia and ventriculomegaly.

198 bility, simplification of cerebral gyration, corpus callosum hypoplasia, and dysmorphic facial featur

199 onia, movement disorders, behavior problems, corpus callosum hypoplasia, and epilepsy.

200 fections, respectively; abnormalities of the corpus callosum in 16 of 17 (94%) and 22 of 28 (78%) inf

201 ity before and after surgical section of the corpus callosum in 22 patients with medically refractory

202 but inattention scores were related to AD in corpus callosum in a cluster sized 716 voxels.

203 hese findings suggest a notable role for the corpus callosum in maintaining stable functional communi

204 allosal structure, supporting a role for the corpus callosum in mediating functional asymmetry.

205 ues in posterior thalamic radiation and left corpus callosum in patients (all p < 0.05).

206 against brain injury in the hippocampus and corpus callosum in rats with vascular dementia.

207 thesis, here we investigated the role of the corpus callosum in the cortical spreading of NREM slow w

208 Our results demonstrate a causal role of the corpus callosum in the cross-hemispheric traveling of sl

209 y of interhemispheric connections within the corpus callosum, in particular parieto-parietal connecti

210 PN) connect the cerebral hemispheres via the corpus callosum, integrating cortical information and pl

211 h as white matter (subcortical white matter, corpus callosum, internal capsule, anterior commissure),

212 g abnormalities of the cerebellum, cingulum, corpus callosum, internal capsule, thalamus, basal foreb

213 abnormalities with sparing of the U fibers, corpus callosum involvement with sparing of the outer bl

214 rous brain imaging studies indicate that the corpus callosum is smaller in older children and adults

215 icospinal tract is unique to mammals and the corpus callosum is unique to placental mammals (eutheria

216 NFIX) results in abnormal development of the corpus callosum, lateral ventricles, and hippocampus.

217 not in FA values, including the splenium of corpus callosum, left posterior corona radiate/posterior

218 The corpus callosum, longer WM tracts and areas that are mor

219 hat the interactions of glucose x FA in left corpus callosum, longitudinal fasciculus and corona radi

220 Section of the corpus callosum markedly reduced interhemispheric functi

221 en and adults, our findings suggest that the corpus callosum may be larger in infants who go on to de

222 Together, our findings suggest that the corpus callosum may have a dual inhibitory and excitator

223 stion by comparing age-related difference in corpus callosum morphology of chimpanzees and humans.

224 s within these hippocampal subfields and the corpus callosum, novel findings that would have been dif

225 laterally within the basal ganglia, thalami, corpus callosum, occipital, temporal, parietal and front

226 ctroscopy data from the anterior body of the corpus callosum of 13 patients with systemic lupus eryth

227 aphy images of white matter samples from the corpus callosum of a monkey brain reveal that blood vess

228 High focal uptake was found in the posterior corpus callosum of a TBI subject.

229 G-ratio of myelin, were also detected in the corpus callosum of adult HdhQ250 mice.

230 he remyelination phase of the CPZ model, the corpus callosum of Cav1.2(KO) animals presented a signif

231 rpus callosum, cortex, and striatum, and the corpus callosum of Cav1.2(KO) animals showed an importan

232 Furthermore, the corpus callosum of Fth KO animals presented a significan

233 he dentate nucleus, subthalamic nucleus, and corpus callosum of multiple system atrophy, and in all r

234 drocyte differentiation were impaired in the corpus callosum of Olig1-null mice, resulting in hypomye

235 staining of astrocytes, particularly in the corpus callosum of the Gnptab Ser321Gly homozygote mice

236 tensor imaging also detected deficits in the corpus callosum of the Gnptab Ser321Gly mice.

237 nificant loss of OLIG2-positive cells in the corpus callosum of Tmem106b-/- mice, which was present a

238 ecentral gyri, corticospinal tracts, and the corpus callosum) of participants with ALS (two-sample t

239 neuropathy, cognitive impairment and a thin corpus callosum on brain MRI.

240 ons surrounding the anterior crossing of the corpus callosum on E18 as well as the persistence of lar

241 hile the interaction of glucose x FA in left corpus callosum, or in longitudinal fasciculus was assoc

242 on, relevant to limited brain areas like the corpus callosum, or multiple orientations but without th

243 of basal ganglia (caudate nucleus, putamen, corpus callosum, posterior limb of internal capsule), le

244 arietal regions, and lower FA in the body of corpus callosum, posterior superior longitudinal fascicu

245 c functional connectivity in relation to the corpus callosum presents a case in point.

246 Evoked action potentials in the myelinated corpus callosum projections of Msh2-null mice were small

247 The volumetric assessment of the corpus callosum proved to be a useful tool in discrimina

248 d in some subcortical regions, including the corpus callosum, putamen, and cerebellum.

249 the highest effect sizes observed within the corpus callosum (R(2) = 0.041, P(corr) < 0.001) and cing

250 ction neurons of the corticospinal tract and corpus callosum, respectively.

251 d by 67.4% and 203.0% in the hippocampus and corpus callosum, respectively.

252 iculi (IFOF), genu (GCC) and splenium of the corpus callosum (SCC), posterior limbs of the internal c

253 crostructural differences in the body of the corpus callosum; schizophrenia and bipolar disorder feat

254 l neuropathy with or without agenesis of the corpus callosum (SLC12A6).

255 in a priori regions of interest: splenium of corpus callosum (SPCC) and posterior limb of internal ca

256 ated with word-matching BPA, FA in posterior corpus callosum (splenium-occipital) correlated with fac

257 corona radiata, right tapetum, and bilateral corpus callosum, statistically moderates whether sleep s

258 nce in cellular degeneration between cortex, corpus callosum, striatum, globus pallidus, and thalamus

259 trophy (by measuring the volume of thalamus, corpus callosum, subcortical nuclei, hippocampus) as par

260 n five brain areas, including left and right corpus callosum, superior longitudinal fasciculus, poste

261 cortico-thalamic fibers: the corona radiata, corpus callosum, superior longitudinal fasciculus, poste

262 Astrogliosis significantly increased in corpus callosum (TBI = 6.7 +/- 0.69, Sham = 2.5 +/- 0.38

263 es in white matter tracts of the genu of the corpus callosum that connect the two hemispheres of the

264 onal connectivity in individuals in whom the corpus callosum (the major commissure between the hemisp

265 ieved with the volumetric measurement of the corpus callosum - the values were 73% and 71%, respectiv

266 ismatch repair deficiency is agenesis of the corpus callosum, the cause of which has not been establi

267 e fewer axons than in wild-type mice and, in corpus callosum, the myelin is thinner than in controls.

268 capsule (RLIC), the body and splenium of the corpus callosum, the superior and posterior corona radia

269 sister taxon of eutherians but do not have a corpus callosum; their intercortical commissural neurons

270 Reduced corpus callosum thickness confirmed trend-level observat

271 Glial cells in the cortex and corpus callosum underwent delayed FJB staining from d7 t

272 nal anisotropy (FA) index measurement of the corpus callosum using diffusion tensor imaging.

273 ips, as well as the areas and volumes of the corpus callosum, ventricular system, hippocampus, amygda

274 deficiency may contribute to agenesis of the corpus callosum via reduction in CUX1(+) neurons.

275 linkage; a QTL on chromosome 1p influencing corpus callosum volume and a region on chromosome 7p lin

276 ated with alcohol use behavior and posterior corpus callosum volume, both in a subset of COGA and in

277 pus, on average, but lower right caudate and corpus callosum volume, relative to 22q-del carriers.

278 emispheric intrinsic connectivity and larger corpus callosum volume.

279 tional anisotropy within the splenium of the corpus callosum was found in each NDD group, compared wi

280 silesional corticospinal tract and bilateral corpus callosum was increased but sensorimotor CBF was d

281 nal outcome were significantly improved, the corpus callosum was intact, whereas this was not the cas

282 Interestingly, the corpus callosum was markedly thinner, a characteristic w

283 HD symptoms and structure of the genu of the corpus callosum was negative in youths with TBI and posi

284 Absence of the corpus callosum was noted at screening prenatal head ult

285 Luxol fast blue staining of the corpus callosum was significantly greater in the BCCAO r

286 erc1), thinner (Kif21b and Wdr89), or absent corpus callosum (Wdr47), revealing a common role for WDR

287 distribution volume (VT), determined in the corpus callosum, we calculated the binding potential (re

288 or cerebellar atrophy, and hypoplasia of the corpus callosum were consistent among individuals harbor

289 ied gyral pattern of the cortex and abnormal corpus callosum were noted on MRI of three individuals,

290 necting ipsilateral cortical regions and the corpus callosum were significantly heritable, ranging fr

291 Dissociations were found in the genu of corpus callosum which accounted for short-term memory bi

292 ite matter tracts, including the body of the corpus callosum, which are most commonly affected by dif

293 c cerebral palsy and partial agenesis of the corpus callosum, while histochemical and biochemical ana

294 a patient with a left splenium lesion of the corpus callosum who perceives the right side of faces as

295 particularly significant with regard to the corpus callosum, whose development undergoes several dyn

296 In WT mice, microglia expanded in the corpus callosum with age, whereas aged Trem2(-/-) mice h

297 Dysplasia of corpus callosum with focal thinning of the posterior par

298 n brain imaging ranging from agenesis of the corpus callosum with hydrocephalus to cystic formations,

299 ite matter with sparing of the U fibers, the corpus callosum with sparing of the outer blades, the ba

300 ight frontal cortex to 0.46 mL cm(-3) in the corpus callosum, with intermediate VT values in subcorti