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1 e was studied in vitro using isolated rabbit corpus cavernosum.
2 PDE5, which causes cGMP to accumulate in the corpus cavernosum.
3 oth muscle and endothelial subcluster in the corpus cavernosum.
4 genous antioxidant genes and proteins in the corpus cavernosum.
5 rotein was present in the endothelium of the corpus cavernosum.
6 ived nitric oxide is present in the diabetic corpus cavernosum.
7 nhibitor rosuvastatin (RSV) were examined on corpus cavernosum and aorta from streptozotocin-induced
8  monophosphate hydrolysis [corrected] in the corpus cavernosum and therefore increases the penile res
9 Since NO synthase is found in human clitoral corpus cavernosum and vagina, we hypothesized that human
10 -) currents underlie detumescent tone in the corpus cavernosum, and that modulation of this mechanism
11        Many tonic SMCs (vascular, airway and corpus cavernosum) are driven by a cytosolic Ca2+ oscill
12             A Rho-kinase inhibitor increases corpus cavernosum (CC) pressure in an in vivo rat model
13 tal internal pudendal artery (dIPA), and the corpus cavernosum (CC).
14                                           In corpus cavernosum defects in rabbits and pigs, implantat
15 - mice, and this effect was reproduced in WT corpus cavernosum exposed to NOS inhibitors.
16  excess reactive oxygen species (ROS) in the corpus cavernosum have been implicated as a causative fa
17 xamination and a blood-gas analysis from the corpus cavernosum helps to distinguish between ischaemic
18 Specifically, we 3D printed a hydrogel-based corpus cavernosum incorporating a strain-limiting tunica
19                             In rabbit penile corpus cavernosum, intracellular cGMP was determined to
20                                          The corpus cavernosum is the most important structure for pe
21 Our study provides an insight into the human corpus cavernosum microenvironment and a reference for p
22  tissues, including the smooth muscle of the corpus cavernosum of the penis.
23 ression of heme oxygenase-1 increased in the corpus cavernosum of the PHZ group, but PDE5 protein exp
24 osine, and 4-hydroxynonenal increased in the corpus cavernosum of the PHZ group, suggesting a state o
25 increased blood flow into the penis, raising corpus cavernosum pressure to culminate in penile erecti
26 ntagonism of Rho-kinase results in increased corpus cavernosum pressure, initiating the erectile resp
27                                    Increased corpus cavernosum relaxant responses to acetylcholine an
28 d electrical field stimulation (EFS)-induced corpus cavernosum relaxations in vitro were obtained.
29                   Relaxation of arterial and corpus cavernosum smooth muscle (CCSM) is necessary to i
30 o study membrane currents in isolated rabbit corpus cavernosum smooth muscle cells.
31          Therefore, we aimed to evaluate the corpus cavernosum smooth muscle relaxant function in a m
32 t intravascular hemolysis promotes increased corpus cavernosum smooth muscle relaxation associated wi
33 oavailability to NO synthase in human penile corpus cavernosum smooth muscle, the inhibition of human
34                                              Corpus cavernosum strips were dissected free and placed
35 ent smooth muscle relaxation in human penile corpus cavernosum tissue that is required for erection.
36 ate that arginase is present in human penile corpus cavernosum tissue, and that the arginase inhibito
37 ed the functional role of BK channels in the corpus cavernosum utilizing a knock-out mouse lacking th
38  structures; and an implantable model of the corpus cavernosum, whose complex vascular network is cri