ライフサイエンスコーパス: corrodens

1 possibly lysine decarboxylase from Eikenella corrodens.

2 periodontitis harbored multiple clones of E. corrodens.

3 ly, is responsible for phase variation in E. corrodens.

4 strains of Streptococcus mitis and Eikenella corrodens.

5 P. gingivalis (59%), P. micros (51%), and E. corrodens (37%), at levels 2+/-2, 5+/-4, 9+/-6, 4+/-5, a

6 evotella intermedia (58%/54%), and Eikenella corrodens (90%/82%) was higher with MT4 than MSP.

7 Fusobacterium nucleatum, Eikenella corrodens, Actinobacillus actinomycetemcomitans, and Cam

8 volved in mediating the toxic activity in E. corrodens and plaque extracts.

9 trarily primed PCR for clonal analysis of E. corrodens and the multiclonal colonization of E. corrode

10 P. gingivalis, A. actinomycetemcomitans, E. corrodens, and F. nucleatum was determined using an immu

11 emcomitans, Prevotella intermedia, Eikenella corrodens, and Fusobacterium nucleatum were determined b

12 egatibacter actinomycetemcomitans, Eikenella corrodens, and Fusobacterium nucleatum/periodonticum wer

13 comitans, Cardiobacterium hominis, Eikenella corrodens, and Kingella kingae (HACEK) clinical isolates

14 s ratios for Campylobacter rectus, Eikenella corrodens, and Porphyromonas gingivalis were less than 2

15 parvula, Capnocytophaga sputigena, Eikenella corrodens, and Prevotella intermedia-like species than g

16 such as P. nigrescens, T. forsythia, and E. corrodens, as well as C. concisus, C. gingivalis, and D.

17 ermedia, Campylobacter rectus, and Eikenella corrodens at 6 months.

18 m nucleatum, Campylobacter rectus, Eikenella corrodens, Bacteroides forsythus, and Treponema denticol

19 PCR detection showed C. rectus and Eikenella corrodens both to occur in 93% of the study subjects and

20 Actinomyces gerencseriae, C. gingivalis, E. corrodens, C. concisus, Prevotella nigrescens, T. forsyt

21 arvula, Capnocytophaga gingivalis, Eikenella corrodens, Campylobacter concisus, Porphyromonas gingiva

22 raised in goats to LDC-rich extracts from E. corrodens cell surfaces were used to inhibit Ecor-LDC an

23 ganism from the sites) of the subgingival E. corrodens clonal types between the baseline and the foll

24 The mean numbers of distinct E. corrodens clones harbored by nondiseased subjects and su

25 The numbers of distinct E. corrodens clones increased significantly (Mann-Whitney r

26 Comparison of E. corrodens clones recovered at the baseline and those rec

27 at the follow-up examination showed that E. corrodens colonization was not stable.

28 Antibodies to LDC from Eikenella corrodens (Ecor-LDC) inhibit LDC activity and retard gin

29 leatum, Fusobacterium polymorphum, Eikenella corrodens, Eubacterium nodatum, Campylobacter gracilis,

30 The human pathogen Eikenella corrodens expresses type IV pili and exhibits a phase va

31 zing MAb 3hE5 blocked the toxic effect of E. corrodens extract S. mitis extracts contained a single,

32 E. corrodens extracts contained a number of antigens detect

33 tatus and higher levels of P. intermedia, E. corrodens, F. nucleatum, and IL-1beta than non-users.

34 s forsythus, Campylobacter curvus, Eikenella corrodens, Fusobacterium nucleatum, Porphyromonas gingiv

35 tophaga spp., Cardiobacterium sp., Eikenella corrodens, Fusobacterium spp., Gemella haemoylsans, and

36 odens and the multiclonal colonization of E. corrodens in the oral cavity.

37 . mucosa, Veillonella parvula, and Eikenella corrodens increased in both groups, but later in samples

38 Eikenella corrodens is a commensal subgingival bacterium commonly

39 Whether E. corrodens is the major source of LDC in dental biofilms

40 In addition, multiple E. corrodens isolates from each sample were recovered for c

41 The genetic diversity of 205 Eikenella corrodens isolates recovered from dental plaque, mucosal

42 ophila, L. decolor, L. paeta, L. brunnea, L. corrodens, L. mendax, L. rufa, L. pearmani, and L. trico

43 ni, L. rufa, L.mendax, L. bostrychophila, L. corrodens, L. paeta, and L. tricolor) were collected fro

44 comitans, Cardiobacterium hominis, Eikenella corrodens, or Kingella species) gram-negative bacilli is

45 al diversity and stability of subgingival E. corrodens over time.

46 ans, C. rectus, P. intermedia/nigrescens, E. corrodens, P. micros, Capnocytophaga and Fusobacterium s

47 Campylobacter rectus, Eikenella corrodens, Porphyromonas gingivalis, and pathogen-relate

48 ree putative periodontal pathogens-Eikenella corrodens, Porphyromonas gingivalis, and Prevotella inte

49 to identify Campylobacter rectus, Eikenella corrodens, Porphyromonas gingivalis, pathogen-related or

50 excess (by weight) of the purified IgG to E. corrodens protein specifically cross-precipitated an 80-

51 The human pathogen Eikenella corrodens synthesizes type IV pili and exhibits a phase

52 omitans, Porphyromonas gingivalis, Eikenella corrodens, Tannerella forsythensis, Prevotella intermedi

53 media, oral Campylobacter species, Eikenella corrodens, Treponema denticola, Gemella haemolysans, Gra

54 ture and function in the clinical isolate E. corrodens VA1.

55 s phase variation in the clinical isolate E. corrodens VA1.

56 Eikenella corrodens was detected by microbial testing.

57 Eikenella corrodens was present equally in subjects with and witho

58 sociated with DG, whereas, high levels of E. corrodens were associated with 13-fold increased odds fo

59 wever, in the ADP group, higher levels of E. corrodens were correlated with higher levels of IL-1beta

60 intermedia, Parvimonas micra, and Eikenella corrodens were identified at the 1-month reevaluation af

61 ermedia, Campylobacter rectus, and Eikenella corrodens were significantly reduced.

62 The extracts of E. corrodens were toxic to HL60 cells, whereas similar extr

63 noprecipitate by rabbit IgG antibodies to E. corrodens whole cells.