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1 suring physical parameters of the actomyosin cortical layer.
2 re characteristic of pyramidal cells in each cortical layer.
3 rons are generally localized within the same cortical layer.
4  radial glia fibers to reach the appropriate cortical layer.
5 clei and cytoskeleton form a two-dimensional cortical layer.
6 * nAChR currents depended on neuron type and cortical layer.
7 ated by currents and firing in supragranular cortical layers.
8  apical dendrites extending into superficial cortical layers.
9 at runs perpendicular to the pia into deeper cortical layers.
10 seek to assign FP/LFP recordings to specific cortical layers.
11 s mediated by diffusion of potassium to deep cortical layers.
12 on with genes that are expressed in specific cortical layers.
13 al modulation varies by cell type and across cortical layers.
14 nsity of channelrhodopsin2 expression across cortical layers.
15 yonic brain displayed altered development of cortical layers.
16  a lesser extent, in V1 neurons in the other cortical layers.
17  efferent connections terminate in different cortical layers.
18 ng six with increased expression in the deep cortical layers.
19 ein distributions across synapse classes and cortical layers.
20 how this effect varies across cell types and cortical layers.
21 duction of proliferative zones and disrupted cortical layers.
22 e enhancement (DCS-LTP) was recorded in deep cortical layers.
23 le levels of GAD67 mRNA is ~30% lower across cortical layers.
24 and faint (Off) spiking synchronously across cortical layers.
25 those species, is observed only in the upper cortical layers.
26 which give rise to projection neurons of all cortical layers.
27 ncy at eye opening, which are similar across cortical layers.
28 rogeneity, and their responses varied across cortical layers.
29 eurons that project expansively to the upper cortical layers.
30 nd a pronounced reactive gliosis in the deep cortical layers.
31 lowing to discriminate between the different cortical layers.
32 and examine the involvement of the different cortical layers.
33 f input partially overlapped and spanned all cortical layers.
34 ng of neurons in the superficial and deepest cortical layers.
35 trol of the flow of sensory responses across cortical layers.
36 ables simultaneous, long-term imaging of all cortical layers.
37 n a gradient from the pial surface to deeper cortical layers.
38 nsity of neurons and conservation of the six cortical layers.
39 ons, many of which populated the superficial cortical layers.
40  the most pronounced loss observed in deeper cortical layers.
41 nts of spasms are initiated in infragranular cortical layers.
42 amma band phase synchrony within and between cortical layers.
43 integrating synaptic inputs across different cortical layers.
44  neurogenesis of projection neurons in outer cortical layers.
45 igate the way these signals flow through the cortical layers.
46 head movements trigger excitation across all cortical layers.
47 ively measure neural activity from different cortical layers.
48 panzees do not display astrogliosis in other cortical layers.
49 erstood, pathway emanating from two distinct cortical layers.
50 ed activation of different cell types across cortical layers.
51 ature astrocytes localized primarily in deep cortical layers.
52 t has severe limitations for studying deeper cortical layers.
53 the spontaneous, ongoing LFP recorded across cortical layers.
54  more prominent impact on those of the lower cortical layers.
55 r in amygdala, striatum or cortex and in all cortical layers.
56  vessels followed by vessels penetrating the cortical layers.
57 icient mice are postnatal viable with normal cortical layering.
58                                              Cortical layer 1 (L1) contains a sparse and molecularly
59                                              Cortical layer 1 (L1) interneurons have been proposed as
60     Here, we investigated how INs located in cortical layer 1 (L1) of rat barrel cortex affect whiske
61 veloping GABAergic projection from the ZI to cortical layer 1 that is essential for proper developmen
62 ons form a GABAergic axon plexus in neonatal cortical layer 1, making synapses with neurons in both d
63 sly unknown interneuronal circuits that link cortical layer 1-3 (L1-3) interneurons and L5 pyramidal
64 re higher in striatum but not as dense as in cortical Layer 1.
65 ctions, each of which terminates in distinct cortical layers [1-3].
66                                   Neurons in cortical layer 2/3 differ significantly from those in th
67 l-autonomous requirements for Bdnf in visual cortical layer 2/3 neurons.
68 ic acid (GABA) release on dendrites of mouse cortical layer 2/3 pyramidal neurons could induce gephyr
69  synaptogenesis and mature synapse number on cortical layer 2/3 pyramidal neurons in vivo.
70 t time and found that parenchymal vessels in cortical layer 2/3 were orientation selective.
71     Parasubicular neurons span the height of cortical layers 2 and 3, and we observed no obvious asso
72 oth PV+ and SST+ interneurons selectively in cortical layers 2-4 without numerically changing the tot
73 erived from two radial glia on average, span cortical layers 2-6, and are composed of a random sampli
74 puts to single excitatory neurons throughout cortical layers 2/3-6 in the mouse primary visual cortex
75 rm potentiation into long-term depression at cortical layer 3/5 synapses.
76 S) approach to quantify over 200 proteins in cortical layers 3 and 5 of two cohorts of human subjects
77 ified protein patterns that differed between cortical layers 3 and 5.
78           Long-term depression (LTD) between cortical layer 4 spiny stellate cells and layer 2/3 pyra
79  biophysicochemical model of a somatosensory cortical layer 4 to layer 2/3 synapse to study the role
80 s revealed by cytochrome-oxidase-staining of cortical layer 4.
81 a model assuming thalamic projections to two cortical layer-4 cell populations: one excitatory (putat
82 ure with simultaneous synaptic activation of cortical layer-4 neurons, mimicking the effect of a sing
83                                              Cortical layer 5 (L5) pyramidal neurons integrate inputs
84                           It is specifically cortical layer 5 (L5) that is thought to underlie these
85  of a spike and wave discharge originated in cortical layer 5.
86                           Pyramidal cells in cortical Layers 5 and 6 are the only cells in the cerebr
87 and heterogeneous, with axons emanating from cortical layers 5 and 6, and plays a key role in directi
88 rate successful OGB-1 AM loading of cells in cortical layers 5-6 and subsequent three-photon imaging
89  64-channel silicon microelectrode arrays in cortical layers 5/6 of the primary visual cortex of chro
90                                   Neurons in cortical layer 5B (L5B) connect the cortex to numerous s
91 t to the thalamus originates from neurons in cortical layer 6 (L6); however, the function of this cor
92 e neurons, located in the white matter below cortical layer 6 and characterized here for the first ti
93 ice that develop without a cortex or without cortical layer 6 axonal projections, and find that RGC a
94 t to the dLGN in mice that specifically lack cortical layer 6 projections to the dLGN.
95 eurons leads to mislocalized cells in deeper cortical layers, abnormal positioning of the centrosome-
96            Microarray analysis of individual cortical layers across sensorimotor and association cort
97 r processing schemes change as a function of cortical layer and cell type in awake mice.SIGNIFICANCE
98 onal partition of plasticity within a single cortical layer and reveal the LII/III to LV connection a
99 motif of a core thalamic input to the middle cortical layer and that thalamocortical synapses form a
100 e for Gata4 activity in morphogenesis of the cortical layer and the preservation of normal cardiac fu
101 -up modulations were strongest in the middle cortical layer and weaker in deep and superficial layers
102 of migratory cells contributes to defects in cortical layering and hypocellularity in the ventral LGN
103 GF-1 receptor failed to migrate to the upper cortical layers and accumulated at the ventricular/subve
104 d subunit composition of AMPARs differ among cortical layers and among cell types.
105                                              Cortical layers and areas acquire adult-like molecular p
106 specific molecular signatures for individual cortical layers and areas, prominently involving genes a
107 fects of muscarinic signaling differ between cortical layers and between brain areas.
108 grating anatomical data at the resolution of cortical layers and borders, we know little about the mo
109 n identified but the contribution of each to cortical layers and cell types through specific lineages
110 ever, it is unclear how neurons in different cortical layers and circuits contribute.
111  ACh that directly excite neurons throughout cortical layers and contribute to attention.
112 s that specific neuronal connections, across cortical layers and even within individual neurons, resp
113 level, ASD genes are enriched in superficial cortical layers and glutamatergic projection neurons.
114 uit excitatory and inhibitory neurons across cortical layers and how brain state modulates laminar in
115  dynamics of several thousand neurons across cortical layers and in the hippocampus of awake behaving
116  interneuron subtypes, residing in different cortical layers and innervating complementary laminar do
117 ntain neurons from both deep and superficial cortical layers and map transcriptionally to in vivo fet
118 n neurons that project focally to the middle cortical layers and more "matrix" calbindin neurons that
119 obe tissue reveals fine structures including cortical layers and myelinated axons.
120 oral response dynamics of PMd neurons across cortical layers and show stronger and earlier decision-r
121  stimulate identified sets of neurons across cortical layers and simultaneously record the produced s
122 ases, such as amyloid plaques and changes in cortical layers and subcortical nuclei.
123 neously recorded neuron firing in prefrontal cortical layers and the caudate-putamen of rhesus monkey
124  requiring the integration of signals across cortical layers and the selection of executive variables
125 f 1980 spiking neurons distributed among six cortical layers and two thalamic nuclei.
126 n and observed that the degree to which deep cortical layers and white matter are incorporated into t
127 idered the avian homologue of mammalian deep cortical layers and/or amygdalar subdivisions, but one-t
128  or CBSH3+ DNAs, migrated to the appropriate cortical layer, and became integrated in cortical circui
129 g depth of 2PM within the mouse brain to the cortical layer, and imaging subcortical structures curre
130 cDCS reliably induced DCS-LTD in superficial cortical layers, and a long-term potentiation (LTP)-like
131  activate layer 4 first and sequentially all cortical layers, and MMN is elicited independent of the
132 s." Different models have suggested distinct cortical layers, and rhythms implement predictive coding
133                                              Cortical layers appear after a highly regulated migratio
134 imination reveals white matter fascicles and cortical layer architecture in brains of live mice.
135 4.5) and at birth-after neurons from all six cortical layers are born.
136  in midgestation (E70-E81), when superficial cortical layers are generated.
137               These results suggest that all cortical layers are influenced by sensory stimulation du
138 electrical stimulation across cell types and cortical layers are largely driven by their different ax
139 cification and differential integration into cortical layers are largely unknown.
140   We found that neurons in thalamus and deep cortical layers are most sensitive to changes in conscio
141 stem exists in which distinct mPFC areas and cortical layers are targeted depending on the location o
142 ormed by local neural populations, such as a cortical layer, are typically inferred from anatomical c
143 various vessel compartments and at different cortical layers as well as transient ischemic events wer
144  majority of SST-expressing cells across all cortical layers, as well as some PV-expressing cells in
145 ficantly upregulated PAI-1 expression in all cortical layers assessed (p < 0.05) and reduced neuronal
146 d by protrusions of neurons beyond the first cortical layer at the pial surface of the brain.
147  uniform within the neocortex, even within a cortical layer, but are specialized within subcircuits.
148 ore likely to be connected vertically across cortical layers, but not laterally within the same layer
149 tal deafness between supragranular and other cortical layers, but similar dystrophic effects in all i
150 ons mediate widespread inhibition across all cortical layers by recruiting fast-spiking inhibitory ne
151 7(KIP1) control neuronal output for distinct cortical layers by regulating different stages of precur
152 twork trained on data recorded at a specific cortical layer can be used to accurately segment active
153 d immunofluorescent labelling in AD in every cortical layer compared to CNT and neocortical LBD.
154 d with the frontal ERP, concentrated in deep cortical layers corresponding to the zone of BF input, w
155                                     Although cortical layering, cytoarchitecture, and proteome were f
156  bodies with end plates loss restriction and cortical layer discontinuity was observed.
157 vide an adhesive code for the development of cortical layers, due to their homophilic interactions an
158      Contrary to expectation that the output cortical layers encode stimulus information most accurat
159  FLVCR2 mutations exhibited reduced cerebral cortical layers, enlargement of the cerebral ventricles,
160 ctions to area 17 was largely in superficial cortical layers, especially layer 1.
161               Both germinal and post-mitotic cortical layers exhibit fronto-temporal gradients, with
162 ate that establishes distinct upper and deep cortical layers for neurons and astrocytes, resembling t
163 eocortex, which are known to be critical for cortical layer formation and are hypothesized to be impo
164 ressed proliferation, and affected embryonic cortical layer formation.
165 hm, which segments the bones and removes the cortical layer from the images.
166 quantification of proteins within individual cortical layers from human postmortem brain tissue, prov
167 chanism across sensory systems and (2) which cortical layers generate alpha oscillations.
168 over the more fundamental questions of which cortical layers generate alpha rhythms and whether the g
169 Ca2+ imaging of neuronal populations in deep cortical layers has remained a major challenge, as the r
170        This is crucial because the different cortical layers have distinct intra- and interregional c
171      These rhythms, as well as the different cortical layers, have also been closely related to feedf
172 secting hippocampal Schaffer collaterals and cortical layer I axons.
173 lly characterized interneuron types of adult cortical layer I, suggesting a fairly broad expression a
174 ce have long apical dendrites extending into cortical layer I.
175 h effective, may not be able to disambiguate cortical layer identity in all cells.
176 ions recapitulate the development of in vivo cortical layer identity.
177 tions from neurons in ipsilateral entorhinal cortical layer II as well as from bilateral dorsal CA2 a
178 ny other species and their linear density in cortical layer II generally increased with brain size.
179 ource of immature neurons in adult brains is cortical layer II.
180  and resulted in the reduction of neurons in cortical layer II/III.
181 est in corticostriatal projection neurons in cortical layer II/III.
182 r with the accumulation of tau inclusions in cortical layers II and III, distinguishes CTE from Alzhe
183 r neocortical areas, GINs were only found in cortical layers II and III.
184 umber of GADD45b-stained cells in prefrontal cortical layers II, III, and V in psychotic patients.
185 er are concentrated in excitatory neurons in cortical layers II-III, IV, and V, as well as the dentat
186 creased cortical neuron number deriving from cortical layers II-IV is undetermined.
187 x and differentiate into Satb2(+) neurons in cortical layers II-IV.
188 l properties resemble those of the mammalian cortical layers II/III.
189 ed, suggesting a generalizable role for this cortical layer in influencing motor commands and cogniti
190              The subplate layer, the deepest cortical layer in mammals, has important roles in cerebr
191  the SVZ produce excitatory neurons for each cortical layer in the neocortex.
192 ermine physical parameters of the actomyosin cortical layer in vivo directly from laser ablation expe
193 tal cortex, concomitant with an inversion of cortical layering in the rostral cortex.
194 nally targeted two-photon imaging across all cortical layers in awake mice using a microprism attachm
195 xpression of synaptic AMPARs across multiple cortical layers in awake mice using two-photon imaging.
196 eural network to segment, automatically, the cortical layers in both hemispheres.
197 ei in birds, cortical areas in reptiles, and cortical layers in mammals.
198 d power (30-80 Hz) in the middle-superficial cortical layers in regions surrounding the activated whi
199 d light on the different functional roles of cortical layers in spatial integration and on how L3 dyn
200  results suggest a division of labor between cortical layers in the coding of visual input and visual
201 t how this process is implemented across the cortical layers in the frontoparietal saccade network re
202 y of the spindle activity in the superficial cortical layers in the model.
203 pyramidal neurons (PNs) from rat superficial cortical layers in vivo and in vitro using 2-photon imag
204 tials (EPSPs) in different cell-types across cortical layers in wS1.
205 ons originate from different combinations of cortical layers, include an inhibitory component, and fo
206 ts in the subarachnoid space and superficial cortical layers, indicative of chronic bleeding events o
207                                              Cortical layering is normal, but neurons are smaller tha
208 se 2 disparate signals are integrated across cortical layers is poorly understood.
209 h synaptic contacts terminating primarily in cortical layer IV.
210 al populations, including pyramidal cells in cortical layer IV.
211 ound beta-amyloid staining, predominantly in Cortical Layers IV and VI in 27 of the 32 cats used in t
212 of astrocytes, abundant in thalamo-recipient cortical layers ("kissing" astrocytes), overlap markedly
213 on's birth, and certain populations, such as cortical layer (L) 4 excitatory neurons of the primary s
214 rcuitry of V1 by deleting separately in each cortical layer (L) a gene required to close the critical
215                                              Cortical layers (L) 5 and 6 are populated by intermingle
216 ivo 2-photon imaging of pyramidal neurons in cortical layers L4 and L2/3 of awake mouse primary audit
217 ought to propagate primarily from the middle cortical layer (layer 4, L4) up to L2/3 and down to the
218  between single-neuron morphology, mesoscale cortical layering, macroscopic cortical thickness, and,
219 signed to a laminar identity using canonical cortical layer marker genes.
220  co-expressed canonical fetal deep and upper cortical layer markers.
221 changes in the terminal distribution between cortical layers may also play a role.
222               Molecular innovations of upper cortical layers may be an important component in the evo
223 arpened angular tuning of vS1 neurons in all cortical layers measured.
224 urons to passive vibrissa stimulation in all cortical layers measured.
225  sharpen the frequency tuning in superficial cortical layers more than in middle or deep layers.
226                          In Satb1-null mice, cortical layer morphology was normal.
227 led to activity in both superficial and deep cortical layers, motor imagery engaged only superficial
228 progenitor cells can generate deep and upper cortical layer neurons and form functional neuronal netw
229 l (NPC) apoptosis, which led to reduction in cortical layer neurons.
230 hape changes are insured by a thin, dynamic, cortical layer of cytoskeleton underneath the plasma mem
231 grade tracing studies in rats to examine the cortical layer of origin, the sizes of parent axons, and
232 sh heart regeneration, cardiomyocytes in the cortical layer of the ventricle induce the transcription
233 depth and dendritic path lengths within each cortical layer of vS1, as well as spiking patterns durin
234 tensive neural circuits that span across all cortical layers of a V1 column, and reflects both feedfo
235 ions of signals originating from the various cortical layers of the areas of a given network.
236 d in the migration of young neurons into the cortical layers of the brain during early human developm
237 like layer of cuticle between the median and cortical layers of the inflation.
238  using simultaneous recordings from multiple cortical layers of the premotor cortex of monkeys perfor
239 fic combinations of signals from the various cortical layers of their input areas, possibly differing
240 re most clearly defined in the supragranular cortical layers of V1, particularly at middle levels of
241 ed movements evoked responses in superficial cortical layers only.
242 expression of genes previously implicated in cortical layer or phenotypic identity in individual cell
243 hether attention acts non-selectively across cortical layers or whether it engages the laminar circui
244 ions and expanded these analyses to localize cortical layer-preferential projections.
245         Drawing from various cortical areas, cortical layers, recording methodologies, and species, w
246 ntegration is dynamically coordinated across cortical layers remains poorly understood.
247  theories proposing that neurons in the deep cortical layers represent perceptual hypotheses and ther
248 to iron-positive deposits in the superficial cortical layers, representing the chronic manifestation
249                Unit recordings in the middle cortical layers revealed a precise tonotopic organizatio
250 tent of interneuron recruitment in different cortical layers.SIGNIFICANCE STATEMENT This study identi
251 ultaneously from V1 neurons spanning all six cortical layers so that we could characterize the lamina
252                         Experiments identify cortical layer specific effects during induced arousal f
253                                              Cortical layer-specific dynamics were apparent, as emerg
254             Thus, the ability to investigate cortical layer-specific protein levels in human postmort
255 onal cortical growth rates while maintaining cortical layer structure.
256 ginning of gliogenesis, and later within the cortical layers, suggesting a mechanism by which astrocy
257 estricted to, or more pronounced in, certain cortical layers, suggesting that genetic vulnerabilities
258 , these neurons localize at more superficial cortical layers than their control counterparts.
259 riations across different cortical areas and cortical layers that appear species-specific, and expres
260 ogenesis was persistently impaired in deeper cortical layers that experienced higher gliosis.
261 S directly activates fibers within the upper cortical layers that leads to the activation of dendrite
262            Neurons with fbRFs are located in cortical layers that receive strong feedback projections
263 teractions are clustered non-randomly across cortical layers to form synergy and redundancy hubs.
264 lary blood flow and oxygen distribute across cortical layers to meet the local metabolic demand is in
265 al neurons process information from multiple cortical layers to provide a major output of cortex.
266 protein expression varies across regions and cortical layers to provide insights into the differences
267  CUX2-expressing projection neurons in upper-cortical layers underlying meningeal inflammation; such
268 A functions as a specific marker of the deep cortical layers until the first postnatal week.
269 icrolesions were easily identified in deeper cortical layers using the neuronal marker NeuN, showed a
270 progressive neurodegeneration prominently in cortical layer V and spinal ventral horn, motor dysfunct
271 enia heritability with maximal enrichment in cortical layer V excitatory neurons.
272  in pyramidal neurons of the hippocampal and Cortical layer V of the Epm2a or Nhlrc1 knockout mice br
273 ase, there is selective destruction of motor cortical layer V pyramidal neurons and degeneration of t
274        These astroglia are enriched in mouse cortical layer V; express distinct molecular markers, in
275 ibuting to the GABAergic interneuron pool of cortical layers V and VI.
276               This was due to differences in cortical layers V-VI, but not layers I-IV.
277 targets neuronal nuclei, specifically of the cortical layers V/VI.
278 ured spheroids had high levels of TBR1 (deep cortical layer VI) and Nkx2.1 (ventral cells), and matri
279 cells being most numerous and dense close to cortical layer VI, decreasing significantly in density w
280                                           No cortical layer was uniformly spared, with the clearest s
281 nergic input, as well as input from specific cortical layers, was similar onto both pathways.
282 e inputs from presynaptic cells in different cortical layers, we investigated whether AMPAR-mediated
283 ogical atlas of cortical thickness and which cortical layers were contributing to these gradients.
284                                Because upper cortical layers were expanded during primate evolution,
285  a narrowing of the frontal cortex, although cortical layers were normally organized.
286 igns of astrogliosis and a compaction of the cortical layers were observed at 90 days postinfection.
287 s, feed-forward information enters at middle cortical layers whereas feedback information arrives at
288  such that early-born neurons settle in deep cortical layers whereas late-born neurons populate more
289 ity is predominantly observed in superficial cortical layers, whereas alpha- and beta-band activity i
290 d thalamic mediodorsal nucleus to the middle cortical layers, which are thought to be highly efficien
291 cDCS aftereffects are not uniform throughout cortical layers, which may explain the incomplete cDCS c
292 class of SPNs receives inputs from only deep cortical layers, while the second class of SPNs receives
293 nstituted the majority of neurons across all cortical layers whose responses dominated the net spikin
294 ium (AId), an avian analog of mammalian deep cortical layers with involvement in motor function.
295 rated that microstimulation of infragranular cortical layers with patterns of microcurrents derived f
296                 Bulk-loading cells in deeper cortical layers with synthetic calcium indicators could
297 alpha generators were present in each of the cortical layers, with a strong source in superficial lay
298 y to improvements in discriminability across cortical layers, with changes in firing rates most impor
299  causality, were also dominant between these cortical layers within a time window when the contour si
300 tory neurons whose cell body resides in deep cortical layers yet whose axons arborize throughout all

 
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