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1 al, lateral parietal, and posterior temporal cortices).
2 as well as in the entorhinal and perirhinal cortices.
3 poral, medial prefrontal/frontal and insular cortices.
4 h pain processing, the insular and cingulate cortices.
5 lly between the primary and secondary visual cortices.
6 spontaneous activity patterns across sensory cortices.
7 ls and are much more abundant in early-stage cortices.
8 n less laterality than primary sensory/motor cortices.
9 te pulvinar, projects extensively to sensory cortices.
10 ischemic infarct was territorial in cerebral cortices.
11 ly diffuse and extensive beyond sensorimotor cortices.
12 zation of attended representations in visual cortices.
13 = 1,189) in primary motor and primary visual cortices.
14 been classified as one of several olfactory cortices.
15 zed regions of the entorhinal and perirhinal cortices.
16 thickness from motor to frontal association cortices.
17 visual, somatosensory, motor, and prefrontal cortices.
18 long bilateral middle and posterior temporal cortices.
19 tal affiliations of RSP neurons with sensory cortices.
20 re more functionally connected to prefrontal cortices.
21 ectivity between LGd and extrastriate visual cortices.
22 activation of sensorimotor representational cortices.
23 minance than those from motor and prefrontal cortices.
24 ncreases in the primary motor and cerebellar cortices.
25 n of excitatory synaptic strength in sensory cortices.
26 to maintain inhibition of irrelevant sensory cortices.
27 ncluding visual, parietal, and retrosplenial cortices.
28 tory speech representation in early auditory cortices.
29 ally in heteromodal and unimodal association cortices.
30 by unimodal and then transmodal associative cortices.
31 lasticity in perilesional motor and premotor cortices.
32 s and with lower GWC in pre- and postcentral cortices.
33 areas, as well as in sensorimotor and visual cortices.
34 posteromedial entorhinal and parahippocampal cortices.
35 ntral anterior insular (i.e., frontoinsular) cortices.
36 y visual and dorsolateral prefrontal (DLPFC) cortices.
37 task-set reconfigurations in frontoparietal cortices.
38 ents with strokes in the left frontoparietal cortices.
39 m and posterior temporo-occipital junctional cortices.
40 inferior frontal, supramarginal and insular cortices.
41 to primary somatosensory and vibrissal motor cortices.
42 ased NAA in posterior cingulate and parietal cortices.
43 nses in visual and 'higher-order' prefrontal cortices.
44 emporal (p<0.0001), and occipital (p=0.0053) cortices.
45 pocampal connectivity with motor and sensory cortices.
46 , anterior cingulate and supplementary motor cortices.
47 ch structurally connects the bilateral motor cortices.
48 treamlines leave the prefrontal and temporal cortices.
49 oint-angles (intrinsic coordinates) in motor cortices.
50 ctional connectivity between primary sensory cortices.
51 r bone volume and poorly defined metaphyseal cortices.
52 hemisphere in both the insular and temporal cortices.
53 es in superior temporal and inferior frontal cortices.
54 ipsilaterally in the entorhinal and piriform cortices.
55 active smokers) human postmortem prefrontal cortices.
56 250 electrodes distributed over peri-Sylvian cortices.
57 nal interaction of auditory and speech motor cortices.
58 ents with tumors near ipsilateral hand motor cortices.
59 activation bilaterally in the primary motor cortices.
60 transcallosal projections between two visual cortices.
61 d with amygdala and insula coupling to motor cortices.
62 us (GPi) and glutamatergic inputs from motor cortices.
63 n-related effects on the primary and sensory cortices.
64 interactions in mammalian sensory and motor cortices.
65 tion in the posterior and anterior cingulate cortices.
66 showed abnormal thinning in inferior frontal cortices.
67 es in parietal, frontal and medial occipital cortices, (3) with higher UPDRS-III scores in the putame
70 We observed a hierarchical gradient: sensory cortices aligned most quickly, followed by mid-level reg
73 in the ventral prefrontal and orbitofrontal cortices among youth reporting STBs, and there is reduce
75 cation between the early folding postcentral cortices and associative temporal cortices, folding late
76 n the rostral and ventral anterior cingulate cortices and bilateral thalamus/caudate, as well as the
77 zation in somatosensory-auditory/associative cortices and dorsal thalamus, suggesting the presence of
78 in pyramidal cells of visual and prefrontal cortices and hippocampal CA1, synaptic inhibition also c
80 al activation in the left and right temporal cortices and insulae and enhanced activation in the post
81 ives inputs from frontal, sensory, and motor cortices and interconnected thalamic areas that provide
82 refrontal, primary motor, and primary visual cortices and investigated with a Golgi stain and compute
85 septum, thalamic reticular nucleus, certain cortices and other limbic structures, as well as in some
87 increased in Huntington's disease (HD) mouse cortices and striata and in human postmortem caudate.
88 e subnetworks anchored in the frontoparietal cortices and subcortical regions (including the thalamus
90 howed abnormal cortical thinning of temporal cortices and thickness increases in visual/somatosensory
91 tuation in the amygdala and relevant sensory cortices and to maintain inhibition of irrelevant sensor
92 ate, lateral temporal, and superior parietal cortices and ventrolateral thalamic, and medial amygdalo
93 ated with higher GWC in insula and cingulate cortices and with lower GWC in pre- and postcentral cort
94 with reduced connectivity within the primary cortices and within the executive network, but increased
95 lvinar, which is closely connected to visual cortices and would thus have been expected to reflect co
97 frontal, posterior cingulate, and precuneus cortices) and relative increases in metabolism (sensorim
98 es both cortical (e.g., frontal and parietal cortices) and subcortical (e.g., the superior colliculus
99 al targets (extrinsic coordinates) in visual cortices, and across movements with equivalent joint-ang
100 led that activity of the thalamus, cingulate cortices, and angular gyri are fundamental for human con
101 vity between supratemporal and orbitofrontal cortices, and between orbitofrontal and nucleus accumben
102 (NAA) in the anterior cingulate and insular cortices, and decreased NAA in posterior cingulate and p
103 e biased in prefrontal and superior parietal cortices, and male biased in ventral occipitotemporal, a
104 for synaptophysin in occipital and temporal cortices, and no changes for SNAP-25, PSD-95, VAMP, and
105 with electric fields strengths in prefrontal cortices, and no correlation was found on the scalp.
106 ganglia, thalamus, operculum, frontoparietal cortices, and sensory cortices relative to the HCs.
108 tex, most pronounced in frontal and parietal cortices; and (c) a significant disruption of the functi
109 refrontal cortex and frontopolar and sensory cortices; and decreased connectivity of both regions wit
110 ccipital (P = 0.024), and insula (P < 0.001) cortices; and in the subcortical regions of caudate (P <
111 fied, which included the thalamus, cingulate cortices (anterior, mid- and posterior), caudate nucleus
112 included the bilateral insulae/orbitofrontal cortices, anterior cingulate/paracingulate gyri, and inf
113 ial temporal lobe (entorhinal and perirhinal cortices, anterior hippocampus, and amygdala), where 36%
114 ral striatum, prefrontal and secondary motor cortices are greater when executing consolidated sequenc
115 vibrissal somatosensory (S1) and motor (MC) cortices are key links in the brain neuronal network tha
116 that such cross-modal influences in sensory cortices are mediated by the synchronisation of ongoing
120 orrelations (SCCs) are ubiquitous in sensory cortices, are characterized by rich structure, and arise
121 amate and the subgenual/perigenual cingulate cortices (areas 25/32), the causal involvement of these
123 in premotor, primary motor and somatosensory cortices as monkeys performed a reaching task, for up to
124 oth anterior cingulate and medial prefrontal cortices as well as at basolateral amygdala inputs and s
125 consisting of the primary motor and premotor cortices as well as the anterior intraparietal sulcus, b
126 aseline in the anterior and medial cingulate cortices, as well as in the prefrontal cortex only after
127 parahippocampal, retrosplenial and parietal cortices, as well as the hippocampal formation and stria
128 te/paracingulate gyri, and inferior parietal cortices, as well as the left middle temporal gyrus.
130 modulates neuronal activity in early visual cortices at frequencies that match articulatory lip move
132 increase in CD36 protein with age in several cortices, basal ganglia, hippocampus, and midbrain, a de
133 imer's degeneration starts in the entorhinal cortices, before spreading to the temporoparietal cortex
134 is mediated by both left and right auditory cortices but with differential sensitivity to specific a
136 processes have been described across sensory cortices, but direct comparisons of these processes have
137 ip-reading in silence activates the auditory cortices, but it is not known whether such activation re
138 omolog of the LEC in rodents) and perirhinal cortices, but not in the posteromedial entorhinal and pa
139 ial effects on the primary motor and sensory cortices by using transcranial magnetic stimulation tech
140 sory processing in even the earliest sensory cortices can be systematically modified by various exter
142 us, anterior, middle and posterior cingulate cortices, caudate nucleus and nucleus accumbens (correct
144 ontal cortex, putamen, temporal and parietal cortices, cerebellum, and thalamus (from lowest to highe
145 gray matter (globus pallidus, thalamus), and cortices (cingulate, motor, somatosensory, entorhinal).
147 al orbitofrontal and dorsolateral prefrontal cortices compared with healthy men and female patients.
148 ypes over effector-specific regions of motor cortices compared with typically developing individuals
150 the dorsal striatum and prefrontal and motor cortices correlated with more severe positive PLEs.
153 eq in hippocampi and dorsolateral prefrontal cortices (DLPFCs) from 551 individuals (286 with schizop
154 rd neural activity in auditory and olfactory cortices during an auditory-olfactory matching task.
156 temporal correlations) of the frontocentral cortices during rest and follow-up neural and behavioral
157 und widespread iEEG responses in association cortices during wakefulness, which were attenuated and r
158 effect size: -0.36, p = 0.04), and cingulate cortices (effect size: -0.54, p = 0.02), but no signific
159 ding in silence still activates the auditory cortices, even when participants do not know what the ab
161 ostcentral cortices and associative temporal cortices, folding later during neurodevelopment, reveale
162 g in the second half of gestation in primary cortices, followed by unimodal and then transmodal assoc
164 eural activity over dorsal occipito-parietal cortices for pseudowords, when compared to irregular wor
166 that MD in the right somatosensory and motor cortices from arm to hand were positively correlated wit
169 In the absence of diabetes, AD parietal cortices had decreased mtDNA, reduced MAP2 (neuronal) an
171 udies in visual, auditory, and somatosensory cortices have revealed that different cell types as well
172 ssociated with those regulations (prefrontal cortices, hippocampus, para hippocampus, amygdala, insul
174 ears over the roles of frontal and posterior cortices in mediating consciousness and unconsciousness.
175 ially funnels information to lateral frontal cortices in mice becomes predominant in the massively ex
177 tical role of the piriform and orbitofrontal cortices in relapse to opioid seeking after voluntary ab
178 lower in the frontal and anterior cingulate cortices in schizophrenia with large effect sizes (Cohen
180 n (H3K9ac) mark in 669 aged human prefrontal cortices; in contrast with amyloid-beta, tau protein bur
181 cantly larger FC between the RN and temporal cortices including the middle temporal gyrus (MTG), para
182 rimary somatosensory cortex (S1) and frontal cortices, including both the whisker region of primary m
183 oral, lateral parietal, and inferior frontal cortices, including tracts important for language proces
185 as a model for studying how primary sensory cortices integrate sensory, affective, and cognitive sig
186 Not all was normal in the reduced model cortices: intracellular dynamics acquired a character di
187 c premotor, parietal, and posterior temporal cortices is activated even under subliminal perceptual c
188 neurons in the monkey visual and prefrontal cortices is comparable with or better than that of state
189 berrant gyrification of temporal associative cortices is critical for impaired cognitive performance
192 rocessing in the primary and secondary taste cortices, located in the insula and orbitofrontal cortex
193 eatures; this information is fed to auditory cortices, most likely facilitating speech parsing.SIGNIF
194 the rostral and posterior anterior cingulate cortices must be considered when examining circuits that
195 neural activity in the corresponding sensory cortices, neglecting potential activity-silent mechanism
196 ual anterior cingulate (pgACC) and occipital cortices (OCC) and quantified peripartum plasma NAS.
198 drives synchronized activity across "visual" cortices of blind, more so than sighted, individuals.
200 y OPCs in culture isolated from neonatal rat cortices of both sexes and young male and female mice wi
201 ar signaling centers pattern the much larger cortices of carnivore and primate species, however, is u
204 , we demonstrate in neurons dissociated from cortices of male and female mice that the shift in mEPSC
205 s in the visual, somatosensory, and auditory cortices of male and female mice to discrete, punctate s
207 haemodynamic response in the frontotemporal cortices of patients with major depressive disorder (MDD
208 aningful auditory stimuli synchronize visual cortices of people born blind.SIGNIFICANCE STATEMENT Nat
209 he prevalence of alpha-syn aggregates in the cortices of sporadic disease cases emphasizes the need t
210 s reduced after practice in the sensorimotor cortices of the adolescents, but was stronger in the adu
213 fibroblasts, and in both the hippocampi and cortices of young (age 15-40 years old) and aged (40-65
215 cation abnormalities in associative temporal cortices on adult IQ is influenced itself by gyrificatio
216 ns in the prefrontal, temporal and cingulate cortices or the underlying white matter might affect cog
217 Together with findings from other sensory cortices, our results provide evidence of a common mecha
218 control from the prelimbic and orbitofrontal cortices over striatal activity through distinct thalamo
219 2-back) in the lateral and medial prefrontal cortices (PFC) during task anticipation and performance
220 Dorsal premotor (PMd) and primary motor (M1) cortices play a central role in mapping sensation to mov
221 Regions of the prefrontal and cingulate cortices play important roles in the regulation of behav
222 CE STATEMENT The dorsal and ventral premotor cortices (PMd and PMv, respectively) are two specialized
224 synchrony between primary motor and premotor cortices precedes motor high beta bursts, suggesting a p
227 rimary (S1) and secondary (S2) somatosensory cortices process stimuli depending on recent experiences
228 ision-making have often assumed that sensory cortices provide noisy but otherwise veridical sensory i
229 of these results to those from other sensory cortices provides evidence of common mechanisms across t
230 beta (12-30 Hz) suppression in sensorimotor cortices related to performance during speech and hand m
232 y aspects of how the cerebral and cerebellar cortices remain thin, expand in surface area, and acquir
233 ies that require tissue removal near elegant cortices, require patients to remain awake and responsiv
235 updating across the posterior to prefrontal cortices, resulting in dysfunctional integration of new
236 rostrolateral visual, and medial entorhinal cortices send projections only to the ipsilateral claust
237 Here, simultaneous recordings from motor cortices show that increases in network beta synchrony a
240 egions in the parietal, frontal, and insular cortices shows increases in 2-4 Hz power during scalp EE
241 Neural activity in the premotor and motor cortices shows prominent structure in the beta frequency
242 ations and in audiovisual and frontoparietal cortices signaling a prediction error when presented at
243 rain from sensory, behavioral, and executive cortices.SIGNIFICANCE STATEMENT Making sense of what we
244 compared with the same cell type in sensory cortices.SIGNIFICANCE STATEMENT Understanding cortical c
245 tion sites and medial and lateral prefrontal cortices significantly predicted clinical improvement.
246 , engaging prefrontal and anterior cingulate cortices similarly to many types of effortful task switc
247 econd, increased AMC of temporal associative cortices specifically contributed to the association bet
248 the centromedial amygdala and the prefrontal cortices, striatum, occipital cortex, and thalamus (all
249 ost prominently in the frontal and cingulate cortices, striatum, thalamus, deep white matter, and cer
250 tubercle, and frontal and temporal piriform cortices, suggesting dissociable whole-brain networks fo
251 minability in lateral occipital and fusiform cortices, suggesting that activation patterns within the
253 given its interconnectivity with association cortices that encode spatial relationships and its proje
254 c theta oscillations in frontal and parietal cortices that provide a clocking mechanism for two alter
255 y called Mphosph1, Mpp1 or KRMP1) have small cortices that show elevated apoptosis and defects in mat
258 hinal, retrosplenial, and anterior cingulate cortices, the subicular complex, and the dorsal, medial
261 mporal parietal junction, and medial frontal cortices, there were large differences in neural respons
262 tions established in sensory and association cortices, thereby framing multisensory integration in th
263 he primary somatosensory (S1) and motor (M1) cortices, these terminals have been the main focus of re
265 trans-thalamic circuits from purely sensory cortices to a sensorimotor cortical circuit (i.e., prima
266 r, the relative contribution of sensorimotor cortices to action comprehension may vary as a function
267 terns across cortical depth in early sensory cortices to auditory, visual and audiovisual stimuli und
268 establish its presence beyond purely sensory cortices to determine whether there is a trans-thalamic
269 ings reveal a strong contribution of sensory cortices to hemispheric specialization in action control
270 d by the recruitment of posterior perceptual cortices to maintain information in VWM(6-9), which caus
271 tion between intact and deprived somatomotor cortices to recruit deprived cortex in response to intac
272 red 6920 synapses in mouse motor and sensory cortices using three-dimensional electron microscopy.
275 GBC changes in the prefrontal and cingulate cortices, warranting further investigations of GBC as a
276 ilability in the cingulate and orbitofrontal cortices was associated with the rate of social laughter
277 luster, 20.5% +/- 0.5%) in the primary motor cortices was observed in ALS subjects for (11)C-PBR28 SU
278 714 uptake (17.0% +/- 5.6%) in primary motor cortices was observed in ALS subjects, as measured by bo
279 uctural and functional plasticity in sensory cortices, we examined the acute effect of 17alpha-Estrad
280 rn classification analyses in ventral visual cortices, we found that signals in a region early in the
282 contributions from auditory and sensorimotor cortices were apparent after isochronous beats with anti
284 cally, multisensory interactions in auditory cortices were stronger in deeper laminae, while attentio
286 sured by MRI in sensory and motor processing cortices, were present in a histological atlas of cortic
287 as a consequence of sharing the early visual cortices, what you see and what you are holding in mind
288 evidence for a specific role of sensorimotor cortices when healthy participants judge the meaning of
289 pronounced in the entorhinal and perirhinal cortices, where it could be observed in one of three neu
290 ally overlapped in the premotor and parietal cortices, whereas individual movements were uniquely rep
291 creased connectivity between M1 and premotor cortices, whereas the bilateral CST group showed higher
292 cted in right inferior temporal and fusiform cortices, which correlated negatively with CGG repeat le
293 entorhinal and (in humans) medial prefrontal cortices, which maintain their co-activation structure a
294 rew in our dataset, have diminutive cerebral cortices, which makes the cerebellum appear relatively l
295 ted across temporal, parietal and prefrontal cortices, which sequentially generate five levels of rep
296 to reach higher order "extrastriate" visual cortices, which surround the VISp on its medial and late
297 disrupts auditory processing in association cortices while relatively sparing responses in PAC, open
298 in anterior cingulate and lateral prefrontal cortices while they view socially relevant videos of mar
299 stent with semantic embodiment, sensorimotor cortices will rapidly become active while healthy partic
300 cingulate, and subcortical-posterior insular cortices, with hubs in medial prefrontal, but not poster