ライフサイエンスコーパス: corticocortical

1 specific cellular and network mechanisms of corticocortical activity state coupling.

2 ements; this may be the result of increasing corticocortical activity to compensate for striatal dysf

3 In the present study, we investigated the corticocortical afferent projections of one of these sub

4 sensory systems, central olfactory pathways, corticocortical and commissural connections, and pathway

5 rm two major types of long-range connections-corticocortical and cortico-subcortical.

6 ene expression leads to aberrantly motorized corticocortical and corticofugal output connectivity.

7 gyrus, and they suggest an important role of corticocortical and corticolimbic forward connections in

8 f the marmoset PFC and found two contrasting corticocortical and corticostriatal projection patterns:

9 orphologically diverse cell groups that have corticocortical and corticosubcortical projections.

10 describes the ipsilateral and contralateral corticocortical and corticothalamic connectivity of the

11 describes the ipsilateral and contralateral corticocortical and corticothalamic connectivity of the

12 d diminished functional connectivity (FC) in corticocortical and frontostriatal connections.

13 hypoconnectivity dominated, particularly for corticocortical and interhemispheric functional connecti

14 ory areas is strongly influenced by nonlocal corticocortical and neuromodulatory inputs.

15 sory and motor properties, likely reflecting corticocortical and neuromodulatory inputs.

16 We propose that corticocortical and pulvinocortical connections are invo

17 affecting computations involving long-range corticocortical and subcortical inputs, yet their respon

18 ocortical layer 1 (L1) is the main target of corticocortical and subcortical projections that mediate

19 of both ascending modulatory and fast-acting corticocortical and subcortical-cortical neural pathways

20 These corticocortical and thalamocortical changes in functiona

21 nce of different communication relayed among corticocortical and thalamocortical circuitry for the ab

22 might be a general feature of long-distance corticocortical and thalamocortical circuits.

23 thalamic areas that are themselves linked by corticocortical and thalamocortical connections.

24 was used to assess the relationship between corticocortical and thalamocortical connectivity and aty

25 role that layer 3 pyramidal neurons play in corticocortical and thalamocortical connectivity, we hyp

26 ovide novel insight by showing that specific corticocortical and thalamocortical functional connectiv

27 ese results highlight the importance of both corticocortical and thalamocortical interactions in rewa

28 order to determine the origin and extent of corticocortical and thalamocortical projections to layer

29 Both corticocortical and trans-thalamic pathways mainly signa

30 dense synaptic zone consisting of horizontal corticocortical and widespread layer VII projections, in

31 l extrinsic projection systems (commissural, corticocortical, and thalamocortical) in all AC areas.

32 ynapses suggests the involvement of DISC1 in corticocortical as well as thalamocortical connections.

33 We found lower membrane excitability of the corticocortical axons and normal intracortical gamma-ami

34 rior part of secondary motor cortex receives corticocortical axons from the rostral retrosplenial cor

35 amine, basal forebrain, thalamocortical, and corticocortical axons is mainly responsible for this reg

36 hand use and are densely interconnected via corticocortical axons, lacking a sharp demarcating borde

37 connections tend to be larger than those of corticocortical, but the projection foci are less dense.

38 tightly interlinked with local cortical and corticocortical (CC) circuits, forming extended chains o

39 ur classes of efferent neurons were studied: corticocortical (CC) neurons with ipsilateral projection

40 Corticocortical (CC) projections in the visual system fa

41 We focused on thalamocortical (TC) and corticocortical (CC) synapses along the apical-basal axi

42 lamocortical (TC) synapses are stronger than corticocortical (CC) synapses.

43 mouse AC, defined by their output as either corticocortical (CCort) or corticocollicular (CCol).

44 ese input areas show that excitatory Layer 6 corticocortical cells (L6 CCs) are a major projection pa

45 dicating that corticothalamic and interareal corticocortical cells in the subgranular layers represen

46 erlap between corticothalamic and interareal corticocortical cells in the subgranular layers.

47 We uncover a corticocortical circuit in primary somatosensory cortex

48 he structural and functional properties of a corticocortical circuit that could enable movement-relat

49 se findings establish an excitatory RSC-->M2 corticocortical circuit that engages diverse types of ex

50 , we demonstrate that distinct intrinsic and corticocortical circuitries arise from barrel and septal

51 Our work in a rodent model of corticocortical circuitry demonstrates that feedback pat

52 nt with abnormalities in thalamocortical and corticocortical circuitry, suggesting that disruption of

53 y a central role in both thalamocortical and corticocortical circuitry.

54 a model, we characterized the intrinsic and corticocortical circuits arising in the major propriocep

55 ct amyloid deposition suggests that specific corticocortical circuits express selective, but late, vu

56 gement of parallel direct and trans-thalamic corticocortical circuits may be present as a general fea

57 ract tracing analyses of the organization of corticocortical circuits of identified layers of primary

58 tion of the ipsilesional corticothalamic and corticocortical circuits, and the extent of activation w

59 uce differential patterns of intra-areal and corticocortical circuits.

60 This suggests that corticocortical communication frequencies in the PMv-M1

61 By influencing network state, corticocortical communication from motor cortex may ensu

62 wS2 by whisker stimuli and the emergence of corticocortical communication from wS1 to wS2.

63 dulatory functions can be expected to act on corticocortical communication in addition to their actio

64 esults emphasize the importance of recurrent corticocortical communication in the maintenance of cons

65 The efficacy of spiking synchrony in corticocortical communication is poorly understood.

66 Long-range corticocortical communication may have important roles i

67 Historically, corticocortical communication was thought to occur exclu

68 or and implications of having two routes for corticocortical communication with an emphasis on unique

69 relays seem especially important to general corticocortical communication, and this challenges and e

70 halamo-cortical pathway that is critical for corticocortical communication.

71 der thalamic relays), thus serving a role in corticocortical communication.

72 cortex, reflecting a transthalamic route for corticocortical communication.

73 ting a role for the higher-order thalamus in corticocortical communication.

74 This suggests a generalized arrangement for corticocortical communication.

75 her primates, is well positioned to regulate corticocortical communication.

76 cy range (30-100 Hz) are thought to subserve corticocortical communication.

77 from corticosubcortical to more predominant corticocortical communications in the human brain.

78 We corroborated our findings using a corticocortical computational model representing perturb

79 ignificant because PPC and AGm are linked by corticocortical connections and are both critical compon

80 eaved auditory processing modules related to corticocortical connections and embedded in the isofrequ

81 us may provide a substrate for plasticity in corticocortical connections and Schaffer collateral syna

82 to simple cells, and others have argued that corticocortical connections are likely to be important i

83 Direct corticocortical connections are often paralleled by tran

84 oanatomical studies have long indicated that corticocortical connections are organized in networks th

85 The corticocortical connections between RSC and secondary mo

86 hat the 3D branching structure of macroscale corticocortical connections follows the same organizatio

87 Ascending corticocortical connections from subgriseal neurons were

88 These data demonstrate that corticocortical connections in cat somatosensory cortex

89 studies over the last 50 years, the role of corticocortical connections in motor control and the pri

90 it arises with the evolutionary expansion of corticocortical connections in primates, crossing the th

91 essor of Ctip2 and regulatory determinant of corticocortical connections in the developing cerebral c

92 Knowledge of AC corticocortical connections is modest other than in the

93 We studied the corticocortical connections of architectonically defined

94 ration of neurons that furnish glutamatergic corticocortical connections that subserve cognition.

95 siderable information is available about the corticocortical connections to the IPL, much less is kno

96 system thalamocortical and interhemispheric corticocortical connections was estimated using probabil

97 Most corticocortical connections were patchy, in a manner sug

98 ralized principles of feedback applicable to corticocortical connections will also be considered.

99 udal AGm parallel their known differences in corticocortical connections, and represent another basis

100 that primarily affects intrahemispheric and corticocortical connections, and that places these two d

101 between cortical areas in parallel to direct corticocortical connections, but their specific role in

102 Despite the importance of corticocortical connections, few published studies have

103 In addition to this cascade of corticocortical connections, the auditory cortex receive

104 Because these cortical areas are linked by corticocortical connections, the present findings indica

105 the L4SS cell from excitatory and inhibitory corticocortical connections, which were simulated (both

106 idate strengthened several corticolimbic and corticocortical connections.

107 thalamus and other ipsilateral and callosal corticocortical connections.

108 w that such communication is based on direct corticocortical connections.

109 eurodegenerative diseases that proceed along corticocortical connections.

110 s also a progressive shift in the pattern of corticocortical connections.

111 primate visual cortex have different sets of corticocortical connections.

112 where it is thought to arise primarily from corticocortical connections.

113 o support function through enabling targeted corticocortical connections.

114 complexes, but differed in their patterns of corticocortical connections.

115 -subcortical connectivity and short-distance corticocortical connections.

116 y by controlling the refinement of recurrent corticocortical connections.SIGNIFICANCE STATEMENT The f

117 rence in the proportion of cortex devoted to corticocortical connectivity across these orders.

118 owed that control subjects exhibited greater corticocortical connectivity among middle cingulate, pos

119 oncerning the existence of neural areas, for corticocortical connectivity among neural areas, and for

120 her association areas, with modifications of corticocortical connectivity and gene expression, althou

121 We first determined the extent of intrinsic corticocortical connectivity between the hand and the fa

122 se was accompanied by a decrease in backward corticocortical connectivity from frontal to parietal co

123 ons of tractography for analyzing interareal corticocortical connectivity in nonhuman primates and a

124 ming earlier reports of the patchy nature of corticocortical connectivity in the adult cat somatosens

125 on pattern to the functional organization of corticocortical connectivity in the mouse cortex.

126 rticipate in the refinement of the recurrent corticocortical connectivity in those layers.

127 To investigate, we analyzed corticocortical connectivity patterns from electroenceph

128 seline compared with controls, whereas their corticocortical connectivity remained intact.

129 rain magnetic resonance imaging, large-scale corticocortical connectivity was mapped from ages 12 to

130 strated column-scale precision of reciprocal corticocortical connectivity, suggesting that PFC contai

131 erved a breakdown of subcortico-cortical and corticocortical connectivity.

132 ea-to-area serial or hierarchical pattern of corticocortical connectivity.

133 the size of layer 3 neurons-and whole-brain corticocortical connectivity.

134 that deficits in thalamocortical, as well as corticocortical, connectivity contribute to auditory dys

135 results are consistent with abnormalities in corticocortical, corticobasal ganglia, mesocortical dopa

136 tem cell-grafted rats demonstrated increased corticocortical, corticostriatal, corticothalamic and co

137 ontains subnetworks corresponding to classic corticocortical, corticosubcortical, and subcortico-subc

138 ygdalocortical communication and distributed corticocortical coupling across multiple functional brai

139 ith theoretical perspectives positing that a corticocortical "disconnection" partly explains cognitiv

140 ce of simulated external thalamocortical and corticocortical drives can produce the FC-ERP, similar t

141 However, the dynamics of corticocortical effective connectivity has never been di

142 Corticocortical evoked potential analysis revealed that

143 ith single-pulse electrical stimulation, and corticocortical evoked potential responses were measured

144 Reliable corticocortical evoked potentials (CCEPs) were measured

145 Using two measures of network connectivity, corticocortical evoked potentials (indexing effective co

146 ng rest predict the pattern and magnitude of corticocortical evoked potentials elicited within 500 ms

147 Corticocortical evoked potentials of unique shape and hi

148 Emerging evidence suggests that corticocortical-evoked potentials (CCEPs) resulting from

149 an be made for impairment of hippocampal and corticocortical excitatory pathways, but in general the

150 ping of the dendritic distributions of these corticocortical excitatory synapses onto CSPs in both ar

151 t, regular-spiking neurons, which are mainly corticocortical, exhibited sharp frequency tuning simila

152 Corticocortical FA was significantly reduced only in whi

153 matically clustered manner, possibly through corticocortical feedback and horizontal connections.

154 tex by specifically altering the strength of corticocortical feedback in a layer-dependent manner.

155 We studied the influence of corticocortical feedback in alert monkeys using cortical

156 ts demonstrate the involvement of the direct corticocortical feedback pathway, providing temporally p

157 eep-layer cortical neurons that provide both corticocortical feedforward inputs (to area MT) and cort

158 ity to study the human cortical networks and corticocortical functional connectivity mediating arbitr

159 activity to reveal the cortical networks and corticocortical functional connectivity mediating visuom

160 ion of amygdala impacts amygdalocortical and corticocortical functional connectivity.

161 nt of the underlying cognitive processes and corticocortical functional connectivity.

162 ings inform our understanding of large-scale corticocortical influence as well as the interpretation

163 i may then provide a complementary route for corticocortical information flow.

164 simulations, reveal that the nonlinearity of corticocortical inhibition cancels the nonlinear excitat

165 ether this localised hyperactivity is due to corticocortical inhibition or excess activity of inhibit

166 tatory synaptic drive and actively processes corticocortical input during behavior.

167 work reveals patterns of thalamocortical and corticocortical input unique to the auditory cortex.

168 erlaps PLST, indicating that PLST receives a corticocortical input, either directly or indirectly, fr

169 idal neurons, and relatively weak long-range corticocortical inputs and outputs.

170 works that receive topographically organized corticocortical inputs from distant sensorimotor areas,

171 have studied the distribution of reciprocal corticocortical inputs to pyramidal cells and gamma-amin

172 These reciprocal corticocortical inputs to SI were concentrated in layer

173 lanted organoids receive thalamocortical and corticocortical inputs, and in vivo recordings of neural

174 been suggested as one possible mechanism for corticocortical interaction.

175 We conclude that interneuron-mediated S2-S1 corticocortical interactions are critical for efficient

176 ral sentence reading and analyzed long-range corticocortical interactions between local neural activa

177 r, little is known about the dynamics of the corticocortical interactions implementing these rapid an

178 This suggests that corticocortical interactions, both within a hemisphere (

179 such networks have been explored in terms of corticocortical interactions, subcortical regions are li

180 s depends on large-scale thalamocortical and corticocortical interactions.

181 activity is mediated by mutually inhibitory corticocortical interactions.

182 at state transitions are coordinated through corticocortical interactions.

183 oss layers and projection classes, including corticocortical/intratelencephalic neurons (reciprocally

184 Corticocortical labeling with 1, 1''-dioleyl-3, 3, 3'',

185 In the corticocortical leg, S1 M1 connections from L2/3 and L5A

186 y motor cortex (M2), suggesting a functional corticocortical link from the RSC to M2 and thus a bridg

187 However, whether such corticocortical loops exist remains unclear.

188 In humans, the study of corticocortical motor networks is currently based on MRI

189 might represent specific settings within the corticocortical network and provide meaningful informati

190 s neurochemical changes in the supragranular corticocortical network to which these areas belong.

191 nctional connectivity of thalamocortical and corticocortical networks, particularly those involved in

192 ation by prolonged recruitment of long-range corticocortical networks.

193 tic transport of APP, we used a microfluidic corticocortical neuronal network-on-a-chip to examine AP

194 Corticostriatal neurons in layer 5A and corticocortical neurons (callosal projection neurons sim

195 om M2 monosynaptically excited M2-projecting corticocortical neurons in the RSC, especially in the su

196 regulates visual processing not only through corticocortical neurons projecting to the visual cortex

197 probability of feed-forward connections from corticocortical neurons to corticotectal neurons is appr

198 of layer V (CF5s), those of layer VI (CF6s), corticocortical neurons with ipsilateral projection (CCI

199 how that Cre-positive neurons are CT and not corticocortical or corticoclaustral types.

200 robability of monosynaptic connections among corticocortical or corticotectal cells.

201 l neurons is significantly higher than among corticocortical or corticotectal pyramidal neurons.

202 be implemented via top-down projections from corticocortical or neuromodulatory pathways.

203 cause of this hypoactivity may be defective corticocortical or thalamocortical connections.

204 ly outnumbered white-dominant neurons in the corticocortical output layers 2/3, but the numbers of bl

205 channeling information toward supragranular corticocortical output layers.

206 We used a combination of corticocortical paired associative stimulation (ccPAS) t

207 Here, by use of corticocortical paired associative transcranial magnetic

208 e that opposite STDP-like effects induced by corticocortical PAS are associated with increased commun

209 s connecting frontal and parietal regions: a corticocortical pathway and a subcortical pathway.

210 unction, underscoring its role as a critical corticocortical pathway linking the medial prefrontal, c

211 When the direct corticocortical pathway was interrupted, secondary somat

212 ways, particularly the lemnisco-cortical and corticocortical pathways carrying tactile signals via th

213 imates, and concomitant with an expansion in corticocortical pathways compared with mice in adulthood

214 Corticocortical pathways interconnect cortical areas ext

215 e understanding of the role of transthalamic corticocortical pathways is to determine the nature of t

216 pathways, which ubiquitously parallel direct corticocortical pathways, but their role in sensory proc

217 ception by modulating corticosubcortical and corticocortical pathways.

218 messages carried through the well-documented corticocortical pathways.

219 inate multiple levels of thalamocortical and corticocortical processing to rapidly learn, and stably

220 frequency range consistent with distributed corticocortical processing, whereas responses to standar

221 al projection neurons (cCStrPNs) and crossed-corticocortical projection neurons (cCCPNs).

222 2 or Ctip2 can alter the axonal targeting of corticocortical projection neurons and cause them to pro

223 ies, underscoring the vulnerability of these corticocortical projection neurons during the onset and

224 e monkey neocortical pyramidal neurons: long corticocortical projection neurons from superior tempora

225 the distribution of neurofilament protein in corticocortical projection neurons in areas V1, V2, V3,

226 more subtle abnormalities, with the largest corticocortical projection neurons of layer IIIc express

227 ex can be classified into two major classes: corticocortical projection neurons, which are concentrat

228 ted to other distinctive properties of these corticocortical projection neurons.

229 s from V1; furthermore, any effect on direct corticocortical projections and local V1 circuitry would

230 by modulating network oscillations in S1 via corticocortical projections and subcortical feedforward

231 paradigm to characterize the organization of corticocortical projections from primary somatosensory (

232 Corticocortical projections from the dorsal and ventral

233 supports the view that the release of latent corticocortical projections from tonic inhibition throug

234 sis (ALS), and the preferential loss of long corticocortical projections in Alzheimer's disease (AD).

235 They also support the hypothesis that direct corticocortical projections in the brain are paralleled

236 esis, altered neuronal identity and aberrant corticocortical projections in the developing mouse brai

237 Corticocortical projections mainly terminated in the dor

238 mmediate neighboring barrel columns, whereas corticocortical projections reach the second somatosenso

239 Corticocortical projections targeted areas projected to

240 Septal corticocortical projections terminate in the dysgranular

241 may be a more important unifying feature for corticocortical projections than morphology.

242 Long-range corticocortical projections thus act through local micro

243 Corticocortical projections to the caudal and rostral ar

244 th the order observed in thalamocortical and corticocortical projections, and which characterizes all

245 understanding of these two components of the corticocortical projections, I studied the distribution

246 y impaired performance despite intact direct corticocortical projections, thus challenging the purely

247 drive multisensory functions as strongly as corticocortical projections.

248 ant component in the evolution of long-range corticocortical projections.

249 an SI and subsequently via intrahemispheric (corticocortical) projections to the SI hand region.

250 pathways to the primary visual cortex (V1): corticocortical, pulvinocortical, and cholinergic.

251 We examined the corticocortical receptive field organization of resting-

252 connectivity following deafness may reflect corticocortical rewiring affording acoustically deprived

253 to interact with each other by multisynaptic corticocortical routes in strategy implementation.

254 e population has been suggested to determine corticocortical signaling efficacy, but others have argu

255 gamma remains unclear, and the influence on corticocortical signaling largely untested.

256 ough multiple downstream networks as in some corticocortical signaling schemes.

257 indirect neural network connections such as corticocortical, subcortical, or intrinsic spinal circui

258 including vestibular activity, is a typical corticocortical substrate of body motion.

259 Do thalamocortical and corticocortical synapses differ in their plasticity and

260 se is required at thalamocortical but not at corticocortical synapses for building the whisker to bar

261 e explained either by synaptic depression in corticocortical synapses or by an inhibition-mediated su

262 rward thalamocortical gain while suppressing corticocortical synapses.

263 sting a possible causal relationship between corticocortical synchrony and localized increases in bet

264 Notably, increased corticocortical synchrony between primary motor and prem

265 dent experiments to offer a viewpoint on how corticocortical systems contribute to learning and produ

266 The labeled corticocortical terminals in the primary motor (MI) and

267 mediodorsal thalamus (MDT) is a higher-order corticocortical thalamic nucleus involved in cognition a

268 of generalized anatomical rules to describe corticocortical, thalamocortical and corticothalamic pro

269 emporal to basal forebrain structures versus corticocortical tract disconnection.

270 rum imaging to compare anterior-to-posterior corticocortical tracts between primates and other mammal

271 ell as an expansion of anterior-to-posterior corticocortical tracts compared with other mammals.

272 increased bursting may be more important in corticocortical transmission than in transmission of pri

273 entially reflecting a greater sensitivity of corticocortical versus thalamocortical projections to th

274 functional connectivity is likely driven by corticocortical white matter connections but with comple

275 was used to construct white matter trees of corticocortical wiring.