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1 acquisitions (0.91 for precontrast, 0.92 for corticomedullary, 0.91 for early nephrographic, and 0.93
2  was greater than that of oncocytomas in the corticomedullary (125 HU vs 106 HU, P = .045) and excret
3 s greater than that of papillary RCCs in the corticomedullary (125 HU vs 54 HU, P < .001), nephrograp
4 greater than that of chromophobe RCCs in the corticomedullary (125 HU vs 74 HU, P < .001) and excreto
5                                  On both the corticomedullary and nephrographic phase images, clear c
6   The tumor-to-cortex enhancement indexes at corticomedullary and nephrographic phases were largest f
7 nical MR protocol was used before and after (corticomedullary and nephrographic phases) intravenous a
8 ctal properties, resulting in a considerable corticomedullary area.
9 cted the spatial ordering of cells along the corticomedullary axis and quantitated expression levels
10 diction of spatial gene expression along the corticomedullary axis and quantitation of osmotically re
11 , outer, and inner medulla, to represent the corticomedullary axis, was performed.
12 y localized to renal tubules situated at the corticomedullary boundary, consistent with the region of
13 n of Bowman's capsule followed temporally by corticomedullary cysts involving collecting ducts, proxi
14 tained before transplantation in donors, the corticomedullary difference was reduced in allografts (P
15  and bone bark), metaphyseal undulation, and corticomedullary differences were evaluated qualitativel
16 c-SCID who had died 3.5 mo after HSCT showed corticomedullary differentiation in the thymus, T cell z
17  ureteral dilatation, pelvic dilatation, and corticomedullary differentiation were analyzed and compa
18 ely assessed by two radiologists in terms of corticomedullary differentiation, subcalvarial space, sk
19 ion in thymus size, cellularity, and lack of corticomedullary differentiation.
20 ing four phases each, including precontrast, corticomedullary, early nephrographic, and nephrographic
21 sition of kidney tubule cells and to predict corticomedullary gene expression.
22 on Functional sodium MRI accurately depicted corticomedullary gradient (CMG) dynamic changes in healt
23 ly tool currently available to assess kidney corticomedullary gradient (CMG).
24 osmo-responsive genes follow the physiologic corticomedullary gradient of tissue osmolality.
25 ases were then generated using the synthetic corticomedullary images as input.
26 stnatal sheep the irregularly shaped adrenal corticomedullary interface is innervated by corticotropi
27 eys in one fetus showed tubular cysts at the corticomedullary junction and diffuse effacement of the
28 itic cells and macrophages were found at the corticomedullary junction and in foci in the thymic cort
29  of the peritubular capillary network in the corticomedullary junction and increased numbers of bone
30 usion-weighted imaging hyperintensity in the corticomedullary junction and skin biopsy revealed intra
31 ng cells were found in the cortex and at the corticomedullary junction in both mouse strains.
32  revealed increased apoptotic cells near the corticomedullary junction in lymphoid follicles and in t
33  is a major cause of tubular necrosis in the corticomedullary junction of isolated perfused kidneys,
34 ed predominantly in intestinal villi and the corticomedullary junction of the kidney, whereas guanyli
35                             Pericytes at the corticomedullary junction produce the S1P egress signal,
36      Most affected is the outer medullary or corticomedullary junction region where blood flow remain
37 , in situ apoptosis in the cortex and at the corticomedullary junction was responsible for a synchron
38 d kidney size, cysts are concentrated at the corticomedullary junction, and tubulointerstitial fibros
39  situ by thymic epithelium in the cortex and corticomedullary junction, two locations at which VCAM-1
40 ce revealed extensive ischemic injury at the corticomedullary junction, whereas kidneys from sham HO-
41 ure thymocytes exit via blood vessels at the corticomedullary junction.
42  in the cortex and is most abundant near the corticomedullary junction.
43 n the degree of interstitial fibrosis in the corticomedullary junction.
44 ed in tubular cells of the outer medulla and corticomedullary junction.
45 ed kidneys and on active reabsorption in the corticomedullary junction.
46 rring in 10.3 +/- 9.5% and 32.3 +/- 17.8% of corticomedullary-junction tubules by conventional light
47 the boundary between DP and SP thymocytes at corticomedullary junctions was disrupted and medullary s
48 the initiation of cytotoxic gene expression, corticomedullary migration, or thymus exit.
49 hasic protocol, which included non-contrast, corticomedullary, nephrographic and urographic phases.
50 tember 2007, the prenephrectomy multiphasic (corticomedullary, nephrographic, and excretory phases),
51 ar positions on images from the precontrast, corticomedullary, nephrographic, and excretory phases.
52 MCECT imaging across 4 phases (non-contrast, corticomedullary, nephrographic, and excretory) was perf
53  CT with as many as four phases (unenhanced, corticomedullary, nephrographic, and excretory).
54 Ls by approximately 60%, abolished the renal corticomedullary NH(4) (+) gradient, reduced the capacit
55 nized along a radial axis highlighted by the corticomedullary organization and regional patterning of
56  revisiting the complex and poorly described corticomedullary organization of the thymus.
57                                An adult-like corticomedullary organization underlying kidney function
58 , Osp94 mRNA expression paralleled the known corticomedullary osmolality gradient showing highest exp
59                                          The corticomedullary osmotic gradient between renal cortex a
60 ephrographic phase compared with that in the corticomedullary phase (P = .0002 and P < .0001, respect
61 an in clear cell carcinomas, but only in the corticomedullary phase (P = .0231).
62 nal fat-suppressed T1-weighted gradient-echo corticomedullary phase acquisitions, obtained before and
63  was demonstrated in 11 neoplasms during the corticomedullary phase and in all neoplasms in the nephr
64                  Signal intensity changes on corticomedullary phase images were the most effective pa
65                   For Bosniak IIF cysts, the corticomedullary phase SI provided an AUC of 0.902, with
66                                   During the corticomedullary phase, ccRCCs with the loss of chromoso
67 orresponded to "cyst attenuation" during the corticomedullary phase.
68 ypovascular tumors analyzed during the early corticomedullary phase.
69  relative contrast enhancement values in the corticomedullary phase.
70 tions and 27 MCE MRI acquisitions during the corticomedullary phase.
71 86%, 86-88% and 74-78%, respectively, in the corticomedullary phase.
72 lear cell RCCs and oncocytomas peaked in the corticomedullary phase; mean enhancement of papillary an
73 ode), during (helical mode, 25-second delay, corticomedullary-phase images), and after (axial mode, 1
74    We reveal that CTLA-4 is expressed in the corticomedullary region of the thymus.
75  improved injury scores in both cortical and corticomedullary regions.
76 generate anatomically coregistered synthetic corticomedullary style images using T2-weighted images a
77 lated in the perivascular channels of thymic corticomedullary venules.