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1 h LG offspring, implicating a membrane-bound corticosteroid receptor.
2 n hippocampal neurons densely populated with corticosteroid receptors.
3 dicating the involvement of membrane-located corticosteroid receptors.
4  noradrenergic system influences hippocampal corticosteroid receptors.
5                   These long-term changes in corticosteroid receptor activation, evident 24 h after I
6 lucocorticoids, allowing local regulation of corticosteroid receptor activation.
7                        Aldosterone, a type I corticosteroid receptor agonist (10(-9) M), significantl
8                                  The type II corticosteroid receptor agonist, Ru28362 (10(-8) M), als
9                               The effects of corticosteroid receptor agonists on the expression of 5-
10  results lend support to the hypothesis that corticosteroid receptors, and in particular the Type I r
11 age of the retrosplenial cortex and RU38486 (corticosteroid receptor antagonist) as a similar subpopu
12 oid activity by dexamethasone suppression or corticosteroid receptor antagonists caused a substantial
13 ally occupy the mineralocorticoid subtype of corticosteroid receptor) at least 45 min (median 132.5)
14                  Antagonists of type I or II corticosteroid receptors blocked the effect of DEX on mI
15 e, lampreys express only a single, ancestral corticosteroid receptor (CR).
16 idepressants to decrease as well as increase corticosteroid receptor expression and feedback sensitiv
17 ne) and TCA (imipramine) treatment on neural corticosteroid receptor gene expression in adrenalectomi
18      Our results suggest an expansion of the corticosteroid receptor hypothesis of depression to incl
19 ystem, contains the highest concentration of corticosteroid receptors in the brain and may play an im
20 iated endocrine systems were affected (e.g., corticosteroid receptors), including some that have not
21 erved following IS exposure produced greater corticosteroid receptor occupancy in the brain and pitui
22 ts was not blocked by antagonists of nuclear corticosteroid receptors, or by inhibitors of gene trans
23        IS exposure had very little effect on corticosteroid receptor protein expression as determined
24 tion is the hippocampus, a structure rich in corticosteroid receptors, which has been hypothesized to