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1 nhibitory effects of GCs on the secretion of corticotrophin (ACTH), and used molecular, genetic, and
4 ibitum exhibited increased basal but reduced corticotrophin and corticosterone responses to restraint
5 es of the paraventricular nucleus to release corticotrophin releasing factor (CRF) into the hypothala
7 (CART), glucagon-like peptide-1 (GLP-1) and corticotrophin releasing factor (CRF) might be mediated
8 memory formation, potentially by activating corticotrophin releasing factor (CRF) neurons in the ant
9 es have demonstrated that antagonists of the corticotrophin releasing factor (CRF) receptor markedly
10 inputs originating from the LH and PAG, and corticotrophin releasing factor (CRF) selectively inhibi
16 c neurons projecting to vasopressinergic and corticotrophin releasing factor neurons in the paraventr
17 ediated through beta-arrestin involvement in Corticotrophin Releasing Factor receptor signalling path
19 density of serotonergic, noradrenergic, and corticotrophin releasing factor-positive fibers increase
22 endogenous interleukin-1beta (IL-1beta) and corticotrophin releasing hormone (CRH) after a periphera
23 ne-related transcript (CART), oxytocin (OX), corticotrophin releasing hormone (CRH) and calcitonin ge
25 In situ hybridization revealed increased corticotrophin releasing hormone (CRH) mRNA expression i
26 mplicated in anxiety-related behaviours, and corticotrophin releasing hormone (crh) neurons, key regu
27 ower values of paraventricular nucleus (PVN) corticotrophin releasing hormone (CRH), arginine vasopre
28 leus (-24%) and increased levels of mRNA for corticotrophin releasing hormone (CRH, an inhibitor of f
29 ll lines (CHO) transfected with either human corticotrophin releasing hormone 1 (hCRH1) receptors and
30 dexamethasone suppression test followed by a corticotrophin releasing hormone challenge at 10 and 15
31 reduced (P < 0.05) cortisol response to the corticotrophin releasing hormone challenge versus contro
33 ch region extends centromerically beyond the corticotrophin releasing hormone receptor 1 gene (CRHR1)
34 ress gating of BDNF signaling is mediated by corticotrophin-releasing factor (CRF) acting in the NAc.
35 s of addiction also posit a central role for corticotrophin-releasing factor (CRF) and an interaction
36 icular thalamic nucleus (aPVT) which express corticotrophin-releasing factor (CRF) and are preferenti
38 avior are influenced by the highly conserved corticotrophin-releasing factor (CRF) family of peptides
39 gated the existence of somatostatin (SS) and corticotrophin-releasing factor (CRF) in forebrain neuro
40 ollowing dexamethasone (DEX) inhibition, and corticotrophin-releasing factor (CRF) induced activation
44 ion center in the human dorsal pons contains corticotrophin-releasing factor (CRF) neurons, and wheth
45 leptin, insulin, MC-3/4R agonist (MTII), and corticotrophin-releasing factor (CRF) on RSNA in leptin
46 d oxytocin (OT), arginine-vasopressin (AVP), corticotrophin-releasing factor (CRF) or tyrosine hydrox
47 NOS, beta-NGF, proenkephalin B and orphanin, corticotrophin-releasing factor (CRF) R, estrogen R, PAI
49 s modulates GnRH secretion by activating the corticotrophin-releasing factor (CRF) system and sympath
50 the central amygdala (CeA), ethanol acts via corticotrophin-releasing factor (CRF) type 1 receptors t
51 neurons, exhibited different sensitivity to corticotrophin-releasing factor (CRF), a stress hormone
52 lesioned monkeys had decreased levels of CSF corticotrophin-releasing factor (CRF), and both lesioned
53 ts excitatory and inhibitory transmission in corticotrophin-releasing factor (CRF)-expressing dorsal-
55 nucleus (PVN) contains neurons that release corticotrophin-releasing factor (CRH) and thus provide t
56 n the paraventricular nucleus (PVN), rich in corticotrophin-releasing factor (CRH) and thyrotrophin-r
57 armacological blockade of the stress hormone corticotrophin-releasing factor or of peripheral GC acti
58 es report that application of urocortin I (a corticotrophin-releasing factor receptor-2 agonist) to t
60 A model of the peptide antagonist, cyclic corticotrophin-releasing factor residues 30-41 (cCRF(30-
61 is early aversive effect is mediated via the corticotrophin-releasing factor type 1 receptor (CRF1R,
63 l structures of this region of receptors for corticotrophin-releasing factor, pituitary adenylate cyc
65 ha-MSH, and this was enhanced in response to corticotrophin-releasing hormone (CRH) acting primarily
66 0.001) and augmenting ACTH responsiveness to corticotrophin-releasing hormone (CRH) action (4-fold, P
67 e input from two hypothalamic secretagogues, corticotrophin-releasing hormone (CRH) and arginine vaso
68 e input from two hypothalamic secretagogues, corticotrophin-releasing hormone (CRH) and arginine vaso
70 xpression and responsiveness of hypothalamic corticotrophin-releasing hormone (CRH) and arginine vaso
71 orticotrophic hormone (ACTH) by hypothalamic corticotrophin-releasing hormone (CRH) and then stimulat
74 one, adrenocorticotrophic hormone (ACTH), or corticotrophin-releasing hormone (CRH) mirroring the glu
75 lar nucleus of the hypothalamus that produce corticotrophin-releasing hormone (CRH) reverse social im
78 The principle regulator of the HPA axis is corticotrophin-releasing hormone (CRH), which is made in
79 s (BNST) that otherwise restrain activity of corticotrophin-releasing hormone (CRH)-expressing neuron
80 ium channels that are critical for promoting corticotrophin-releasing hormone (CRH)-induced bursting
82 Testosterone significantly suppressed the corticotrophin-releasing hormone and arginine vasopressi
83 rexin in the lateral hypothalamus and in the corticotrophin-releasing hormone and oxytocin in the PVH
84 eletion decreased hypothalamic expression of corticotrophin-releasing hormone and oxytocin, neuropept
85 corticoid resistance, resulting in increased corticotrophin-releasing hormone expression, chronic hyp
86 sin, arginine vasopressin V1a receptors, and corticotrophin-releasing hormone mRNA was measured follo
87 Furthermore, these neurons largely express corticotrophin-releasing hormone or thyrotropin-releasin
88 m, identified eight intronic SNPs within the corticotrophin-releasing hormone receptor 1 or CRHR1 loc
89 S (but not unstressed) corticotrophs display corticotrophin-releasing hormone-induced bursting even w