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1 nhibitory effects of GCs on the secretion of corticotrophin (ACTH), and used molecular, genetic, and
2 hromosome 12p13 region were present in human corticotrophin (ACTH)-secreting pituitary tumours.
3               After an overnight fast, basal corticotrophin and corticosteroid levels are elevated, a
4 ibitum exhibited increased basal but reduced corticotrophin and corticosterone responses to restraint
5 es of the paraventricular nucleus to release corticotrophin releasing factor (CRF) into the hypothala
6                             The neuropeptide corticotrophin releasing factor (CRF) is released during
7  (CART), glucagon-like peptide-1 (GLP-1) and corticotrophin releasing factor (CRF) might be mediated
8  memory formation, potentially by activating corticotrophin releasing factor (CRF) neurons in the ant
9 es have demonstrated that antagonists of the corticotrophin releasing factor (CRF) receptor markedly
10  inputs originating from the LH and PAG, and corticotrophin releasing factor (CRF) selectively inhibi
11                          We investigated how corticotrophin releasing factor (CRF), a critical stress
12                                              Corticotrophin releasing factor (CRF), a stress-related
13                                              Corticotrophin releasing factor (CRF), enkephalin (ENK),
14 mber of c-Fos immunoreactive neurons express corticotrophin releasing factor (CRF).
15                                        While corticotrophin releasing factor is the prototypic member
16 c neurons projecting to vasopressinergic and corticotrophin releasing factor neurons in the paraventr
17 ediated through beta-arrestin involvement in Corticotrophin Releasing Factor receptor signalling path
18                                     However, corticotrophin releasing factor-like-stimulated transpor
19  density of serotonergic, noradrenergic, and corticotrophin releasing factor-positive fibers increase
20 d 50% of cells were depolarised by exogenous corticotrophin releasing factor.
21 ent of neuromodulation by both dynorphin and corticotrophin releasing factor.
22  endogenous interleukin-1beta (IL-1beta) and corticotrophin releasing hormone (CRH) after a periphera
23 ne-related transcript (CART), oxytocin (OX), corticotrophin releasing hormone (CRH) and calcitonin ge
24 icosteroid feedback and its interaction with corticotrophin releasing hormone (CRH) drive.
25     In situ hybridization revealed increased corticotrophin releasing hormone (CRH) mRNA expression i
26 mplicated in anxiety-related behaviours, and corticotrophin releasing hormone (crh) neurons, key regu
27 ower values of paraventricular nucleus (PVN) corticotrophin releasing hormone (CRH), arginine vasopre
28 leus (-24%) and increased levels of mRNA for corticotrophin releasing hormone (CRH, an inhibitor of f
29 ll lines (CHO) transfected with either human corticotrophin releasing hormone 1 (hCRH1) receptors and
30 dexamethasone suppression test followed by a corticotrophin releasing hormone challenge at 10 and 15
31  reduced (P < 0.05) cortisol response to the corticotrophin releasing hormone challenge versus contro
32 5 versus 10-week-old pigs in response to the corticotrophin releasing hormone challenge.
33 ch region extends centromerically beyond the corticotrophin releasing hormone receptor 1 gene (CRHR1)
34 ress gating of BDNF signaling is mediated by corticotrophin-releasing factor (CRF) acting in the NAc.
35 s of addiction also posit a central role for corticotrophin-releasing factor (CRF) and an interaction
36 icular thalamic nucleus (aPVT) which express corticotrophin-releasing factor (CRF) and are preferenti
37                                          The corticotrophin-releasing factor (CRF) family of neuropep
38 avior are influenced by the highly conserved corticotrophin-releasing factor (CRF) family of peptides
39 gated the existence of somatostatin (SS) and corticotrophin-releasing factor (CRF) in forebrain neuro
40 ollowing dexamethasone (DEX) inhibition, and corticotrophin-releasing factor (CRF) induced activation
41                                              Corticotrophin-releasing factor (CRF) is expressed in th
42          Endogenous neuropeptide Y (NPY) and corticotrophin-releasing factor (CRF) modulate the respo
43                                              Corticotrophin-releasing factor (CRF) modulates the infl
44 ion center in the human dorsal pons contains corticotrophin-releasing factor (CRF) neurons, and wheth
45 leptin, insulin, MC-3/4R agonist (MTII), and corticotrophin-releasing factor (CRF) on RSNA in leptin
46 d oxytocin (OT), arginine-vasopressin (AVP), corticotrophin-releasing factor (CRF) or tyrosine hydrox
47 NOS, beta-NGF, proenkephalin B and orphanin, corticotrophin-releasing factor (CRF) R, estrogen R, PAI
48                  Natural peptide agonists of corticotrophin-releasing factor (CRF) receptors bind to
49 s modulates GnRH secretion by activating the corticotrophin-releasing factor (CRF) system and sympath
50 the central amygdala (CeA), ethanol acts via corticotrophin-releasing factor (CRF) type 1 receptors t
51  neurons, exhibited different sensitivity to corticotrophin-releasing factor (CRF), a stress hormone
52 lesioned monkeys had decreased levels of CSF corticotrophin-releasing factor (CRF), and both lesioned
53 ts excitatory and inhibitory transmission in corticotrophin-releasing factor (CRF)-expressing dorsal-
54                    Here, we demonstrate that corticotrophin-releasing factor (CRF)-expressing neurons
55  nucleus (PVN) contains neurons that release corticotrophin-releasing factor (CRH) and thus provide t
56 n the paraventricular nucleus (PVN), rich in corticotrophin-releasing factor (CRH) and thyrotrophin-r
57 armacological blockade of the stress hormone corticotrophin-releasing factor or of peripheral GC acti
58 es report that application of urocortin I (a corticotrophin-releasing factor receptor-2 agonist) to t
59                     Urocortin 3 (Ucn 3) is a corticotrophin-releasing factor related neuropeptide hig
60    A model of the peptide antagonist, cyclic corticotrophin-releasing factor residues 30-41 (cCRF(30-
61 is early aversive effect is mediated via the corticotrophin-releasing factor type 1 receptor (CRF1R,
62                        Sympathoactivation to corticotrophin-releasing factor, however, was not affect
63 l structures of this region of receptors for corticotrophin-releasing factor, pituitary adenylate cyc
64 und this core cluster express a reporter for corticotrophin-releasing hormone (Bar(CRH) ).
65 ha-MSH, and this was enhanced in response to corticotrophin-releasing hormone (CRH) acting primarily
66 0.001) and augmenting ACTH responsiveness to corticotrophin-releasing hormone (CRH) action (4-fold, P
67 e input from two hypothalamic secretagogues, corticotrophin-releasing hormone (CRH) and arginine vaso
68 e input from two hypothalamic secretagogues, corticotrophin-releasing hormone (CRH) and arginine vaso
69              Physiological concentrations of corticotrophin-releasing hormone (CRH) and arginine vaso
70 xpression and responsiveness of hypothalamic corticotrophin-releasing hormone (CRH) and arginine vaso
71 orticotrophic hormone (ACTH) by hypothalamic corticotrophin-releasing hormone (CRH) and then stimulat
72             We show that a constant level of corticotrophin-releasing hormone (CRH) can activate a dy
73                                          The corticotrophin-releasing hormone (CRH) family of peptide
74 one, adrenocorticotrophic hormone (ACTH), or corticotrophin-releasing hormone (CRH) mirroring the glu
75 lar nucleus of the hypothalamus that produce corticotrophin-releasing hormone (CRH) reverse social im
76      However, in the infant rat hypothalamic corticotrophin-releasing hormone (CRH) stress responses
77                                          The corticotrophin-releasing hormone (CRH) system integrates
78   The principle regulator of the HPA axis is corticotrophin-releasing hormone (CRH), which is made in
79 s (BNST) that otherwise restrain activity of corticotrophin-releasing hormone (CRH)-expressing neuron
80 ium channels that are critical for promoting corticotrophin-releasing hormone (CRH)-induced bursting
81 ion of numerous genes; one such gene encodes corticotrophin-releasing hormone (CRH).
82    Testosterone significantly suppressed the corticotrophin-releasing hormone and arginine vasopressi
83 rexin in the lateral hypothalamus and in the corticotrophin-releasing hormone and oxytocin in the PVH
84 eletion decreased hypothalamic expression of corticotrophin-releasing hormone and oxytocin, neuropept
85 corticoid resistance, resulting in increased corticotrophin-releasing hormone expression, chronic hyp
86 sin, arginine vasopressin V1a receptors, and corticotrophin-releasing hormone mRNA was measured follo
87   Furthermore, these neurons largely express corticotrophin-releasing hormone or thyrotropin-releasin
88 m, identified eight intronic SNPs within the corticotrophin-releasing hormone receptor 1 or CRHR1 loc
89 S (but not unstressed) corticotrophs display corticotrophin-releasing hormone-induced bursting even w
90                        After completion of a corticotrophin stimulation test, serum cortisol and 11be