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1  peptides melanocyte-stimulating hormone and corticotropin.
2 FSH)], with adrenocorticomelanotropic cells [corticotropin (ACTH) and alpha-melanotropin (alpha-MSH)]
3 in level and lack of stress- and cue-induced corticotropin and cortisol responses, higher anxiety, an
4                            The production of corticotropin and cortisol was assessed in 11 specimens
5 ects of cortisol metabolism: daily levels of corticotropin and cortisol; plasma cortisol clearance, m
6 retion of cortisol suppresses the release of corticotropin by pituitary corticotrophs, which results
7 cronodular adrenal hyperplasia appears to be corticotropin-dependent.
8 ticotropin precursor proopiomelanocortin and corticotropin expression were assessed by means of a pol
9 rease <9 mug/dL after 250 mug of intravenous corticotropin from basal values <35 mug/dL.
10 lls: inositol polyphosphate-5-phosphatase A, corticotropin hormone precursor, ribosome biogenesis reg
11                 A semiquantitative score for corticotropin immunostaining in the samples correlated w
12       We examined the abnormal production of corticotropin in these hyperplastic adrenal glands.
13                                              Corticotropin-independent Cushing's syndrome is caused b
14                                Some cases of corticotropin-independent macronodular adrenal hyperplas
15        Transcriptome-based classification of corticotropin-independent macronodular adrenal hyperplas
16            Thus, the disease has been termed corticotropin-independent macronodular adrenal hyperplas
17                                              Corticotropin-independent macronodular adrenal hyperplas
18 and tumor DNA obtained from 33 patients with corticotropin-independent macronodular adrenal hyperplas
19 A axis dysregulation, marked by higher basal corticotropin level and lack of stress- and cue-induced
20       Serum and salivary cortisol and plasma corticotropin levels decreased, and clinical signs and s
21                                 However, low corticotropin levels have also been reported in critical
22                                      Adrenal corticotropin levels were higher in adrenal venous blood
23 er in the patients than in controls, whereas corticotropin levels were lower (P<0.001 for both compar
24                                              Corticotropin levels were measured in adrenal and periph
25 y corticotrophs, which results in low plasma corticotropin levels.
26                                          The corticotropin precursor proopiomelanocortin and corticot
27 d greater neutral, relaxed-state cortisol to corticotropin ratio (adrenal sensitivity) were each pred
28                                              Corticotropin-receptor antagonists significantly inhibit
29 gh an interaction between the stress hormone corticotropin releasing factor (CRF) and glutamate relea
30                      The stress neuropeptide corticotropin releasing factor (CRF) and its receptors (
31                                              Corticotropin releasing factor (CRF) coordinates the bra
32                      The stress neuropeptide corticotropin releasing factor (CRF) is anxiogenic and i
33      Here we demonstrate that, in the vBNST, corticotropin releasing factor (CRF) is expressed in neu
34                                              Corticotropin releasing factor (CRF) is the primary medi
35 port that the stress-associated neuropeptide corticotropin releasing factor (CRF) produces a profound
36 icotine withdrawal was mediated by increased corticotropin releasing factor (CRF) receptor-1 expressi
37                                              Corticotropin releasing factor (CRF) regulates physiolog
38                                              Corticotropin releasing factor (CRF) signaling and CRF n
39                                          The corticotropin releasing factor (CRF) system in the centr
40                                          The corticotropin releasing factor (CRF) system in the centr
41                                              Corticotropin releasing factor (CRF) within the ventral
42 ontains a large number of neurons expressing corticotropin releasing factor (CRF), a neuropeptide tha
43  until the corticotropes are stimulated with corticotropin releasing factor (CRF), whereupon SSTR2 ex
44 ine seeking by the stress-sensitive peptide, corticotropin releasing factor (CRF).
45 presence of the prototypical stress hormone, corticotropin releasing factor (CRF).
46 of action for the anxiogenic neuromodulator, corticotropin releasing factor (CRF).
47 he transmembrane domains of the glucagon and corticotropin releasing factor 1 (CRF1) receptors to dev
48                                              Corticotropin releasing factor binding protein (CRF-BP)
49 oupled receptors, which bind peptides of the corticotropin releasing factor family and are key mediat
50 technique to a member of a class B GPCR, the corticotropin releasing factor receptor 1 (CRF1R).
51  analysis showed significant upregulation of corticotropin releasing factor receptor 2 (CrfR2) in the
52       The current study examines the role of corticotropin releasing factor receptor subtypes 1 and 2
53 nvestigated here the interaction between the corticotropin releasing factor receptor type 1 (CRF1R) a
54                                              Corticotropin releasing factor type 1 (CRF1) is a key co
55 fering the neural stress response induced by corticotropin releasing factor, and promoting stress res
56 in stress and appetite regulation, including corticotropin releasing factor, pro-opiomelanocortin B,
57 88-induced c-Fos activation were observed in corticotropin releasing factor-containing neurons of the
58 eus of the hypothalamus and primarily in non-corticotropin releasing factor-containing neurons of the
59 rom the basolateral amygdala (BLA) away from corticotropin releasing factor-expressing (CRF(+)) centr
60  excitability induced by the stress hormone, corticotropin releasing factor.
61 ORs that colocalize with the stress hormone, corticotropin releasing factor.
62 on, and its glutamatergic neurons expressing corticotropin releasing hormone (Bar(Crh/Vglut2)) are im
63 eloped for the detection of the neuropeptide Corticotropin Releasing Hormone (CRH) based on the immob
64 ional interaction between macroautophagy and Corticotropin Releasing Hormone (Crh) in the gut.
65                                              Corticotropin releasing hormone (CRH) is disrupted in in
66 al requires Nmu receptor 2 and signaling via corticotropin releasing hormone (Crh) receptor 1.
67                          Glucocorticoids and corticotropin releasing hormone (CRH) underlie the physi
68 ethylation of the cortisol-regulating genes, corticotropin releasing hormone (CRH; P=0.05) and glucoc
69 icosterone receptors, their regulator Fkbp5, corticotropin releasing hormone and its receptor, oxytoc
70  from the nucleus tractus solitarii (NTS) to corticotropin releasing hormone neurons in the paraventr
71 erone levels), and alleviated by antalarmin (corticotropin releasing hormone receptor 1 antagonist).
72 cluding the membrane progestin receptor, the corticotropin releasing hormone receptor, and the 5HT1a
73                                      Using a corticotropin releasing hormone-cre driver X tdTomato re
74 rons express dynorphin, somatostatin, and/or corticotropin releasing hormone.
75                               We report that corticotropin-releasing factor (CRF) acts in the ventral
76         Although it has long been known that corticotropin-releasing factor (CRF) and CRF receptors a
77  mRNA was found to partially colocalize with corticotropin-releasing factor (CRF) and growth hormone-
78                                              Corticotropin-releasing factor (CRF) and nociceptin/orph
79                 Release of the neuropeptides corticotropin-releasing factor (CRF) and orexin-A in the
80                                              Corticotropin-releasing factor (CRF) and the three relat
81         Here we report the identification of corticotropin-releasing factor (CRF) as a critical compo
82                               To investigate corticotropin-releasing factor (CRF) contributions to fe
83 neurons that release the stress neuropeptide corticotropin-releasing factor (CRF) drive anxiety-like
84  regulation of the central extrahypothalamic corticotropin-releasing factor (CRF) expression is assoc
85                                          The corticotropin-releasing factor (CRF) family of peptides
86         Urocortin 2 (Ucn2), a peptide of the corticotropin-releasing factor (CRF) family, binds with
87  hypersecretion of the stress neuromediator, corticotropin-releasing factor (CRF) has been implicated
88 rofiling of relevant PI cells identified the corticotropin-releasing factor (CRF) homolog, DH44, as a
89 ike ShA cocaine self-administration, reduced corticotropin-releasing factor (CRF) immunodensity in th
90 ed body of work indicates a crucial role for corticotropin-releasing factor (CRF) in neurobiological
91 termined the role of the stress neurohormone corticotropin-releasing factor (CRF) in stress-induced b
92 ed on previous work hypothesizing a role for corticotropin-releasing factor (CRF) in the IC during cr
93       We recently described the existence of corticotropin-releasing factor (CRF) in the mouse cochle
94 ine was compared following overexpression of corticotropin-releasing factor (CRF) in the NAc of femal
95  literature suggests that catecholamines and corticotropin-releasing factor (CRF) interact in a seria
96                                              Corticotropin-releasing factor (CRF) is an important lin
97                                              Corticotropin-releasing factor (CRF) is critical for the
98                                              Corticotropin-releasing factor (CRF) is expressed in the
99                                              Corticotropin-releasing factor (CRF) mediates anxiogenic
100  from chronic alcohol exposure contains ~80% corticotropin-releasing factor (CRF) neurons and that th
101         Although it has long-been known that corticotropin-releasing factor (CRF) neurons are promine
102 and anxiety and activates a subpopulation of corticotropin-releasing factor (CRF) neurons in the bed
103 cohol intake specifically recruited GABA and corticotropin-releasing factor (CRF) neurons in the mPFC
104  the relationship between corticosterone and corticotropin-releasing factor (CRF) on both beta-amyloi
105 ed the effect of an intravenous injection of corticotropin-releasing factor (CRF) on fructose malabso
106 and the influence of the stress neuropeptide corticotropin-releasing factor (CRF) on these responses.
107 nced fear memory but did not increase either corticotropin-releasing factor (CRF) or corticosterone.
108                                              Corticotropin-releasing factor (CRF) orchestrates the st
109                                          The corticotropin-releasing factor (CRF) plays a key role in
110          Considerable evidence suggests that corticotropin-releasing factor (CRF) plays an important
111                                          The corticotropin-releasing factor (CRF) receptor 1 (CRFR1)
112 duction and Abeta elevation are dependent on corticotropin-releasing factor (CRF) receptor 1 signalin
113                                              Corticotropin-releasing factor (CRF) receptors are found
114                                      Because corticotropin-releasing factor (CRF) receptors are impli
115  We recently demonstrated that activation of corticotropin-releasing factor (CRF) receptors in the ca
116                                              Corticotropin-releasing factor (CRF) receptors-which bin
117             The stress-related neuropeptide, corticotropin-releasing factor (CRF) regulates the dorsa
118 induced relapse through alterations in brain corticotropin-releasing factor (CRF) regulation of neuro
119                                              Corticotropin-releasing factor (CRF) signaling at the CR
120                               Alterations in corticotropin-releasing factor (CRF) signaling pathways
121 eviously demonstrated that activation of the corticotropin-releasing factor (CRF) system potentiates
122                                          The corticotropin-releasing factor (CRF) system, which is in
123 f neonatal amygdala (Neo-A) lesions on brain corticotropin-releasing factor (CRF) systems and hypotha
124                                              Corticotropin-releasing factor (CRF) that is released fr
125                                          The corticotropin-releasing factor (CRF) type 1 receptor (CR
126                         Systemic blockade of corticotropin-releasing factor (CRF) type 1 receptors (C
127 clase-activating peptide, PAC1 receptor, and corticotropin-releasing factor (CRF), (CRF1) receptor.
128                          Here we report that corticotropin-releasing factor (CRF), a neuropeptide rel
129 nsistent with this, the CEA highly expresses corticotropin-releasing factor (CRF), an important modul
130 (Dh44), a neuropeptide related to vertebrate corticotropin-releasing factor (CRF), and its receptor,
131 to the BNSTDL, is thought to communicate via corticotropin-releasing factor (CRF), but studies have y
132                            The neuropeptide, corticotropin-releasing factor (CRF), coordinates the ph
133 ological studies indicate the involvement of corticotropin-releasing factor (CRF), noradrenaline, dop
134 es in receptors for the stress neuropeptide, corticotropin-releasing factor (CRF), that render the lo
135                                              Corticotropin-releasing factor (CRF), the major stress p
136             The focus is on the receptor for corticotropin-releasing factor (CRF), the orchestrator o
137                                              Corticotropin-releasing factor (CRF), the stress-related
138 ways expressing the stress-sensitive peptide corticotropin-releasing factor (CRF), which has been ide
139 ral amygdala noradrenergic substrates [via a corticotropin-releasing factor (CRF)-dependent mechanism
140 allenge has been shown previously to cause a corticotropin-releasing factor (CRF)-mediated increase i
141                                              Corticotropin-releasing factor (CRF)-mediated mechanisms
142 de transmitter in the brain that counteracts corticotropin-releasing factor (CRF)-mediated stress and
143 und that GluN2D is functionally expressed on corticotropin-releasing factor (CRF)-positive BNST cells
144 d reward, including neuropeptide Y (NPY) and corticotropin-releasing factor (CRF).
145 vation of glucocorticoid receptors (GRs) and corticotropin-releasing factor (CRF).
146 ation of VTA dopaminergic neurons expressing corticotropin-releasing factor (CRF).
147 ) system by the stress-related neuropeptide, corticotropin-releasing factor (CRF).
148 nity monoclonal antibody, CTRND05, targeting corticotropin-releasing factor (CRF).
149 amygdala (CeA) that produce the neuropeptide corticotropin-releasing factor (CRF).
150    This study tested the hypothesis that the corticotropin-releasing factor (CRF1) antagonist GSK5616
151 ist (eticlopride), D2R agonist (quinpirole), corticotropin-releasing factor 1 (CRF1) antagonist (anta
152 t of brain stress neurotransmitters, such as corticotropin-releasing factor and dynorphin, in the neu
153  alcohol drinking by increased expression of corticotropin-releasing factor and its feedback regulati
154  that promote stress and resilience, such as corticotropin-releasing factor and nociceptin, has been
155 nsatory mechanism in the brain to counteract corticotropin-releasing factor and/or stress.
156 cuses on the HPA axis-based interventions of corticotropin-releasing factor antagonists and the gluco
157                                              Corticotropin-releasing factor binds with high affinity
158            The neuropeptides vasopressin and corticotropin-releasing factor facilitate, while seroton
159 ephalin, thyrothropin-releasing hormone, and corticotropin-releasing factor immunoreactive cells in t
160                                              Corticotropin-releasing factor infusions into LA impair
161                                              Corticotropin-releasing factor infusions into the centra
162 s in the anterior hypothalamus that may gate corticotropin-releasing factor output from the amygdala
163 ss, the physiological consequence of central corticotropin-releasing factor receptor (CRF-R) activati
164 viously reported differential involvement of corticotropin-releasing factor receptor (CRFR) 1 and 2 i
165  We aimed to characterize the effects of the corticotropin-releasing factor receptor 1 (CRF-R1) antag
166  noradrenergic (NE) receptors (alpha1) via a corticotropin-releasing factor receptor 1 (CRF-R1)-depen
167 rial evaluating the efficacy of GSK561679, a corticotropin-releasing factor receptor 1 (CRF1 receptor
168                                  Blockade of corticotropin-releasing factor receptor 1 (CRF1) suppres
169  Similarly to what has been observed for the corticotropin-releasing factor receptor 1 (CRFR1), SAP97
170 me proliferator-activated receptor gamma and corticotropin-releasing factor receptor 1 were notable e
171  mutants with constitutive activation of the corticotropin-releasing factor receptor family homologue
172 anaphylaxis and psychological stress through corticotropin-releasing factor receptor subtype 1 (CRF(1
173    In this study we investigated the role of corticotropin-releasing factor receptor subtype 2 (CRF(2
174 d, we took the CRF(2(a))R and the homologous corticotropin-releasing factor receptor type 1 (CRF(1)R)
175 esent study investigated whether blockade of corticotropin-releasing factor receptor type 1 (CRF-R1)
176 n reflect reductions in anandamide driven by corticotropin-releasing factor receptor type 1 (CRF1) po
177  we investigated interactions of the class B corticotropin-releasing factor receptor type 1 (CRF1R) w
178                                              Corticotropin-releasing factor receptor type 1 (CRFR1) i
179 ular membrane compartments, we show that the corticotropin-releasing factor receptor type 1 has a spe
180 ure of the transmembrane domain of the human corticotropin-releasing factor receptor type 1 in comple
181                                          The corticotropin-releasing factor receptor type 1 is a clas
182 here this equilibrium is established for the corticotropin-releasing factor receptor type 1.
183 vation of the central stress response, while corticotropin-releasing factor receptor type 2 (CRFR2) h
184                                              Corticotropin-releasing factor receptor type 2 (CRFR2) h
185 eviously, we observed abnormal expression of corticotropin-releasing factor receptor type 2 (CRFR2) t
186                                          The corticotropin-releasing factor receptor type 2a (CRF(2(a
187                                              Corticotropin-releasing factor receptor-1 (CRF(1)) media
188 n in the BNST is unaffected by alpha1-AR and corticotropin-releasing factor receptor-1 (CRFR(1)) anta
189  Effects on attention were attenuated by the corticotropin-releasing factor receptor-1 antagonist ant
190                                              Corticotropin-Releasing Factor Receptors (CRFRs) are cla
191                                              Corticotropin-releasing factor receptors (CRFRs), class
192 alysis studies it has been shown to increase corticotropin-releasing factor release in extrahypothala
193                       Alterations in central corticotropin-releasing factor signaling pathways have b
194 ry-adrenal axis), (4) the (gastrointestinal) corticotropin-releasing factor system, and (5) the intes
195 is function was probed using a dexamethasone/corticotropin-releasing factor test.
196                                Activation of corticotropin-releasing factor type 1 (CRF1) receptors i
197 icated that repeated social stress decreased corticotropin-releasing factor type 1 receptor and incre
198                                We found that corticotropin-releasing factor type 1 receptor within th
199 disrupts this LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s)
200 disrupts this LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s)
201                                          The corticotropin-releasing factor type 2 receptor (CRFR2) i
202 demonstrated that the mechanism involved the corticotropin-releasing factor type 2 receptor, cAMP ele
203 cial behavior (especially neuropeptide Y and corticotropin-releasing factor) are modulated by alcohol
204                                              Corticotropin-releasing factor, a stress-related neurope
205 including hypocretin/orexin, norepinephrine, corticotropin-releasing factor, and cytokines.
206 -system recruitment of brain stress systems (corticotropin-releasing factor, dynorphin, norepinephrin
207 tuitary-adrenal axis, including signaling by corticotropin-releasing factor, in the pathophysiology o
208 w stress interacts with the neuromodulators, corticotropin-releasing factor, norepinephrine, dopamine
209 were identified (i.e., serotonin, dynorphin, corticotropin-releasing factor, oxytocin).
210 ess effect by counteracting the functions of corticotropin-releasing factor, the primary stress-media
211 peptides (ghrelin, nesfatin-1, somatostatin, corticotropin-releasing factor, thyrotropin-releasing ho
212 rmittent access to palatable food results in corticotropin-releasing factor-1 (CRF1) receptor antagon
213 e diuretic hormone 44 (DH44), an ortholog of corticotropin-releasing factor.
214       They further suggest that an activated corticotropin-releasing factor/hypothalamic-pituitary-ad
215 luding those that encode the stress hormones corticotropin-releasing hormone (CRH) and adrenocorticot
216                                          The corticotropin-releasing hormone (CRH) and its type 1 rec
217 n-33 (IL-33), and stress molecules including corticotropin-releasing hormone (CRH) and neurotensin (N
218 larval zebrafish with transgenically labeled corticotropin-releasing hormone (CRH) cells, which repre
219  employed viral-genetic approaches to reduce corticotropin-releasing hormone (Crh) expression in the
220 axis initiates the production and release of corticotropin-releasing hormone (CRH) from the paraventr
221 activity for the suppression of hypothalamic corticotropin-releasing hormone (CRH) gene expression an
222 nstrate that a cluster of neurons expressing corticotropin-releasing hormone (Crh) in the pontine mic
223                                              Corticotropin-releasing hormone (CRH) is a central integ
224                                              Corticotropin-releasing hormone (CRH) is a major regulat
225                                              Corticotropin-releasing hormone (CRH) is an essential, e
226                                              Corticotropin-releasing hormone (CRH) is secreted under
227                     Here we demonstrate that corticotropin-releasing hormone (CRH) neurons are modula
228 tin, thyrotropin-releasing hormone (TRH) and corticotropin-releasing hormone (CRH) neurons expressed
229 synaptic metaplasticity in stress-responsive corticotropin-releasing hormone (CRH) neurons in female
230  affects excitatory and inhibitory inputs to corticotropin-releasing hormone (CRH) neurons in the hyp
231 naptic plasticity at glutamate synapses onto corticotropin-releasing hormone (CRH) neurons in the par
232 tical areas transmit signals to hypothalamic corticotropin-releasing hormone (CRH) neurons, which con
233 augments excitatory synaptic strength in PVN corticotropin-releasing hormone (CRH) neurons, with GLP-
234 el synergistic actions of corticosterone and corticotropin-releasing hormone (CRH) on synaptic physio
235 ing hormone, oxytocin, arginine vasopressin, corticotropin-releasing hormone (CRH) or thyrotropin-rel
236 ress, adrenal corticosterone and hippocampal corticotropin-releasing hormone (CRH) permeate memory-fo
237 g-standing paradigm posits that hypothalamic corticotropin-releasing hormone (CRH) regulates neuroend
238                                              Corticotropin-releasing hormone (CRH) released from the
239                                          The corticotropin-releasing hormone (CRH) system coordinates
240     In addition, increased activation of the corticotropin-releasing hormone (CRH) system within the
241 working memory (WM) deficits; changes to the corticotropin-releasing hormone (CRH) system; and struct
242 rtisol concentrations and gene expression of corticotropin-releasing hormone (CRH) were not affected
243 e production and release of the neuropeptide corticotropin-releasing hormone (CRH) within the hippoca
244                      Activation of amygdalar corticotropin-releasing hormone (CRH)+ neurons abolished
245         Extensive preclinical data implicate corticotropin-releasing hormone (CRH), acting through it
246                                     Although corticotropin-releasing hormone (CRH), produced by parvo
247 nals were collected and processed to measure corticotropin-releasing hormone (CRH), urocortin (Ucn),
248  activities by stimulating the expression of corticotropin-releasing hormone (CRH), urocortin, proopi
249  multilabeled for vasotocin, mesotocin (MT), corticotropin-releasing hormone (CRH), vasoactive intest
250 ormone 44 (Dh44), a homolog of the mammalian corticotropin-releasing hormone (CRH), were specifically
251 ticoids and the stress-released neuropeptide corticotropin-releasing hormone (CRH), which influence t
252 evious studies have described the effects of corticotropin-releasing hormone (CRH), which is released
253 he adult brain, we have virally traced local corticotropin-releasing hormone (CRH)-expressing inhibit
254 nced vertebrate genomes for genes resembling corticotropin-releasing hormone (CRH).
255 ed subset of OXT neurons that co-express the corticotropin-releasing hormone (CRH).
256 al stress physiology, specifically placental corticotropin-releasing hormone (pCRH).
257 found that the rs28365143 variant within the corticotropin-releasing hormone binding protein (CRHBP)
258                                          The corticotropin-releasing hormone family mediates function
259                       Neurons expressing the corticotropin-releasing hormone gene (Crh) in central am
260 MNL-CMR-associated PTD showed higher CRP and corticotropin-releasing hormone levels.
261 loss-of-function mice, either with global or corticotropin-releasing hormone neuron-specific deletion
262 ds of aberrant membrane receptors but not by corticotropin-releasing hormone or dexamethasone.
263                                   The type 1 corticotropin-releasing hormone receptor (CRH-R1) influe
264 gated expression of stress-response receptor corticotropin-releasing hormone receptor (CRHR) in bladd
265 e other ligand-binding site defined--for the corticotropin-releasing hormone receptor 1 (CRF1R)--whic
266                                              Corticotropin-releasing hormone receptor 1 (CRHR1) activ
267                            Variations in the corticotropin-releasing hormone receptor 1 (CRHR1) gene
268                          In addition, GR and corticotropin-releasing hormone receptor 1 (CRHR1) genot
269 51 [FKBP5], glucocorticoid receptor [NR3C1], corticotropin-releasing hormone receptor 1 [CRHR1]) in i
270 elch-like protein 2)), chromosome 17 (CRHR1 (corticotropin-releasing hormone receptor 1) and MAPT (mi
271 7, rs77804065 (p = 1.5 x 10(-12)), at CRHR1 (corticotropin-releasing hormone receptor 1); the protein
272 opeptide urocortin 2 (UCN2) and its receptor corticotropin-releasing hormone receptor 2 (CRHR2) are h
273                                Activation of corticotropin-releasing hormone receptor 2 (CRHR2) in th
274 5-2 muM) for 6 hours significantly increases corticotropin-releasing hormone receptor-1 (CRHR-1) mRNA
275 found a significant three-way interaction on corticotropin-releasing hormone receptor-1 (Crhr1) gene
276 bellar Purkinje cells, and co-localized with corticotropin-releasing hormone receptors in the latter.
277 stress-coping responses by binding to type 2 corticotropin-releasing hormone receptors.
278 in both the initial and replication samples: corticotropin-releasing hormone signaling, cardiac beta-
279 l enrichment analyses revealed enrichment of corticotropin-releasing hormone signaling, GNRH signalin
280                      Furthermore, endogenous corticotropin-releasing hormone(+) (CRH(+)) LC inputs fr
281 way in rat paraventricular hypothalamic CRH (corticotropin-releasing hormone) neuroendocrine neurons
282                                         CRH (corticotropin-releasing hormone), a peptide released fro
283 ernal blood (i.e., C-reactive protein (CRP), corticotropin-releasing hormone, and cytokines) were com
284 at several peptide markers (cholecystokinin, corticotropin-releasing hormone, and tachykinin 1) label
285  not GABAergic, and do not express oxytocin, corticotropin-releasing hormone, vasopressin, or prodyno
286 mic activation of the pituitary changes from corticotropin-releasing hormone-dominant to arginine vas
287              In addition, we discovered that corticotropin-releasing hormone-expressing cells contain
288 f inhibition by dynorphin, somatostatin, and corticotropin-releasing hormone-expressing neurons in th
289 al raphe-originating serotonergic control of corticotropin-releasing hormone-mediated excitation of t
290 refrontal cortex restrains the amygdala, the corticotropin-releasing hormone/hypothalamic-pituitary-a
291                          We demonstrate that corticotropin-releasing-hormone-binding protein (CRHBP),
292 ts potential use as a targeted treatment for corticotropin-secreting pituitary adenomas.
293    We evaluated adrenal function using short corticotropin stimulation test in 157 episodes of gastro
294    We evaluated adrenal function using short corticotropin stimulation test in patients with cirrhosi
295 correlated with a lower cortisol response to corticotropin stimulation.
296 , contributed to hypercortisolemia and hence corticotropin suppression.
297                                              Corticotropin was detected in steroidogenic cells arrang
298                     The cortisol response to corticotropin was inversely correlated with Model for En
299                       The release of adrenal corticotropin was stimulated by ligands of aberrant memb
300 ushing's syndrome appears to be regulated by corticotropin, which is produced by a subpopulation of s

 
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