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1 excitability induced by the stress hormone, corticotropin releasing factor.
2 ORs that colocalize with the stress hormone, corticotropin releasing factor.
3 e diuretic hormone 44 (DH44), an ortholog of corticotropin-releasing factor.
4 he transmembrane domains of the glucagon and corticotropin releasing factor 1 (CRF1) receptors to dev
7 ist (eticlopride), D2R agonist (quinpirole), corticotropin-releasing factor 1 (CRF1) antagonist (anta
8 urthermore, pharmacologic inhibition (with a corticotropin-releasing factor 1 receptor antagonist) of
9 s was identified as potent and orally active corticotropin-releasing factor-1 (CRF(1)) receptor antag
10 ctive withdrawal-like state characterized by corticotropin-releasing factor-1 (CRF(1)) receptor antag
12 ation of 8, an earlier lead pyrazinone-based corticotropin-releasing factor-1 (CRF(1)) receptor antag
13 rmittent access to palatable food results in corticotropin-releasing factor-1 (CRF1) receptor antagon
17 t of brain stress neurotransmitters, such as corticotropin-releasing factor and dynorphin, in the neu
18 alcohol drinking by increased expression of corticotropin-releasing factor and its feedback regulati
19 ween the N/OFQ and Hcrt systems in which the corticotropin-releasing factor and N/OFQ systems coordin
20 that promote stress and resilience, such as corticotropin-releasing factor and nociceptin, has been
23 fering the neural stress response induced by corticotropin releasing factor, and promoting stress res
25 by a variety of central (eg, neuropeptide Y, corticotropin-releasing factor, and neuromedin U) and pe
26 cuses on the HPA axis-based interventions of corticotropin-releasing factor antagonists and the gluco
27 cial behavior (especially neuropeptide Y and corticotropin-releasing factor) are modulated by alcohol
31 88-induced c-Fos activation were observed in corticotropin releasing factor-containing neurons of the
32 eus of the hypothalamus and primarily in non-corticotropin releasing factor-containing neurons of the
33 gh an interaction between the stress hormone corticotropin releasing factor (CRF) and glutamate relea
37 blocked by intravenous pretreatment with the corticotropin releasing factor (CRF) antagonist, astress
43 of this study was to examine the ability of corticotropin releasing factor (CRF) or antibody to insu
45 port that the stress-associated neuropeptide corticotropin releasing factor (CRF) produces a profound
47 icotine withdrawal was mediated by increased corticotropin releasing factor (CRF) receptor-1 expressi
50 lar nucleus (PVN) have been shown to inhibit corticotropin releasing factor (CRF) synthesis via GABA(
55 ontains a large number of neurons expressing corticotropin releasing factor (CRF), a neuropeptide tha
56 intracerebroventricular (icv) injections of corticotropin releasing factor (CRF), a putative anxioge
57 analyzed brain regional, CCK-8, substance P, corticotropin releasing factor (CRF), and neuropeptide Y
58 for 15 days and measured mRNA expression of corticotropin releasing factor (CRF), neuropeptide Y (NP
59 until the corticotropes are stimulated with corticotropin releasing factor (CRF), whereupon SSTR2 ex
66 were measured at baseline and in response to corticotropin-releasing factor (CRF) (0.5 microg kg(1))
67 c raphe nucleus, including a coordination of corticotropin-releasing factor (CRF) actions at both of
69 f chronic nicotine SA on the coexpression of corticotropin-releasing factor (CRF) and arginine vasopr
71 ocus of the present review is on the role of corticotropin-releasing factor (CRF) and CRF-related pep
73 mRNA was found to partially colocalize with corticotropin-releasing factor (CRF) and growth hormone-
74 ajor site of extrahypothalamic expression of corticotropin-releasing factor (CRF) and its G-protein-c
76 laced on the neuropharmacological actions of corticotropin-releasing factor (CRF) and norepinephrine
84 ng the endocrine arm of the stress response, corticotropin-releasing factor (CRF) can act in the brai
85 s-induced release of neuromodulators such as corticotropin-releasing factor (CRF) can drive drug-depe
87 ses and how the stress-related neuropeptide, corticotropin-releasing factor (CRF) directs this by a b
88 neurons that release the stress neuropeptide corticotropin-releasing factor (CRF) drive anxiety-like
89 regulation of the central extrahypothalamic corticotropin-releasing factor (CRF) expression is assoc
97 hypersecretion of the stress neuromediator, corticotropin-releasing factor (CRF) has been implicated
98 rofiling of relevant PI cells identified the corticotropin-releasing factor (CRF) homolog, DH44, as a
99 ike ShA cocaine self-administration, reduced corticotropin-releasing factor (CRF) immunodensity in th
100 gates the regulation of cytosolic calcium by corticotropin-releasing factor (CRF) in midbrain dopamin
101 ed body of work indicates a crucial role for corticotropin-releasing factor (CRF) in neurobiological
102 vioral studies support a modulatory role for corticotropin-releasing factor (CRF) in regulating the d
103 termined the role of the stress neurohormone corticotropin-releasing factor (CRF) in stress-induced b
104 was used to study immunoreactivity (IR) for corticotropin-releasing factor (CRF) in the guinea pig e
105 ed on previous work hypothesizing a role for corticotropin-releasing factor (CRF) in the IC during cr
106 lores the relationship between dynorphin and corticotropin-releasing factor (CRF) in the induction of
108 bout the distribution of the stress hormone, corticotropin-releasing factor (CRF) in the mouse brain.
110 ine was compared following overexpression of corticotropin-releasing factor (CRF) in the NAc of femal
111 dies have found that the stress neurohormone corticotropin-releasing factor (CRF) inhibits 5-HT neuro
112 literature suggests that catecholamines and corticotropin-releasing factor (CRF) interact in a seria
113 Stress induces the release of the peptide corticotropin-releasing factor (CRF) into the ventral te
121 The present experiments examined whether corticotropin-releasing factor (CRF) modulates memory co
122 rning of day 15, we measured AT(1) receptors corticotropin-releasing factor (CRF) mRNA and immunoreac
124 rsal mPFC enhanced restraint-induced Fos and corticotropin-releasing factor (CRF) mRNA expression in
125 re to elicit reliable increases in Fos-ir or corticotropin-releasing factor (CRF) mRNA in the PVH.
127 from chronic alcohol exposure contains ~80% corticotropin-releasing factor (CRF) neurons and that th
129 and anxiety and activates a subpopulation of corticotropin-releasing factor (CRF) neurons in the bed
130 cohol intake specifically recruited GABA and corticotropin-releasing factor (CRF) neurons in the mPFC
132 the relationship between corticosterone and corticotropin-releasing factor (CRF) on both beta-amyloi
133 ed the effect of an intravenous injection of corticotropin-releasing factor (CRF) on fructose malabso
134 and the influence of the stress neuropeptide corticotropin-releasing factor (CRF) on these responses.
135 nced fear memory but did not increase either corticotropin-releasing factor (CRF) or corticosterone.
143 duction and Abeta elevation are dependent on corticotropin-releasing factor (CRF) receptor 1 signalin
150 We recently demonstrated that activation of corticotropin-releasing factor (CRF) receptors in the ca
151 ife social isolation increases the levels of corticotropin-releasing factor (CRF) receptors in the se
154 induced relapse through alterations in brain corticotropin-releasing factor (CRF) regulation of neuro
159 o negative reinforcement is a recruitment of corticotropin-releasing factor (CRF) signaling within th
160 Previous studies have implicated the brain corticotropin-releasing factor (CRF) stress systems in m
161 likely resulting from dysregulation of brain corticotropin-releasing factor (CRF) stress systems.
162 ment of the extrahypothalamic stress peptide corticotropin-releasing factor (CRF) system and activati
165 eviously demonstrated that activation of the corticotropin-releasing factor (CRF) system potentiates
166 ess systems, including the extrahypothalamic corticotropin-releasing factor (CRF) system, following l
168 f neonatal amygdala (Neo-A) lesions on brain corticotropin-releasing factor (CRF) systems and hypotha
172 authors investigated the effect of blocking corticotropin-releasing factor (CRF) Type I and Type II
173 clase-activating peptide, PAC1 receptor, and corticotropin-releasing factor (CRF), (CRF1) receptor.
177 nsistent with this, the CEA highly expresses corticotropin-releasing factor (CRF), an important modul
178 he LC include excitatory amino acids (EAAs), corticotropin-releasing factor (CRF), and endogenous opi
179 (Dh44), a neuropeptide related to vertebrate corticotropin-releasing factor (CRF), and its receptor,
180 vitro and their modulation by dopamine (DA), corticotropin-releasing factor (CRF), and their combinat
182 to the BNSTDL, is thought to communicate via corticotropin-releasing factor (CRF), but studies have y
185 -protein-coupled receptor B1 family includes corticotropin-releasing factor (CRF), growth hormone-rel
186 -adrenal (HPA) axis-activating neuropeptide, corticotropin-releasing factor (CRF), may be the keyston
187 ological studies indicate the involvement of corticotropin-releasing factor (CRF), noradrenaline, dop
189 es in receptors for the stress neuropeptide, corticotropin-releasing factor (CRF), that render the lo
193 ways expressing the stress-sensitive peptide corticotropin-releasing factor (CRF), which has been ide
194 ral amygdala noradrenergic substrates [via a corticotropin-releasing factor (CRF)-dependent mechanism
195 allenge has been shown previously to cause a corticotropin-releasing factor (CRF)-mediated increase i
197 de transmitter in the brain that counteracts corticotropin-releasing factor (CRF)-mediated stress and
198 und that GluN2D is functionally expressed on corticotropin-releasing factor (CRF)-positive BNST cells
212 known as maternal aggression) is impaired by corticotropin-releasing factor-(CRF) related peptides, b
213 ed neural cell density, stress neuropeptide (corticotropin releasing factor--CRF) levels, and plasma
214 This study tested the hypothesis that the corticotropin-releasing factor (CRF1) antagonist GSK5616
215 ers of the corticoliberin family include the corticotropin releasing factors (CRFs), sauvagine, the u
216 -system recruitment of brain stress systems (corticotropin-releasing factor, dynorphin, norepinephrin
217 rom the basolateral amygdala (BLA) away from corticotropin releasing factor-expressing (CRF(+)) centr
219 ed with brain region-specific alterations of corticotropin-releasing factor expression and promoter m
221 oupled receptors, which bind peptides of the corticotropin releasing factor family and are key mediat
223 ortin 2, a recently identified member of the corticotropin-releasing factor family, is expressed in d
225 ephalin, thyrothropin-releasing hormone, and corticotropin-releasing factor immunoreactive cells in t
226 CRFR1) mediates the physiological actions of corticotropin-releasing factor in the anterior pituitary
227 tuitary-adrenal axis, including signaling by corticotropin-releasing factor, in the pathophysiology o
231 ated increments in restraint-induced Fos and corticotropin-releasing factor mRNA expression in the ne
232 w stress interacts with the neuromodulators, corticotropin-releasing factor, norepinephrine, dopamine
233 w explores the role of brain stress systems (corticotropin-releasing factor, norepinephrine, orexin [
234 s in the anterior hypothalamus that may gate corticotropin-releasing factor output from the amygdala
235 s an activation of the brain stress system's corticotropin-releasing factor outside of the hypothalam
237 in stress and appetite regulation, including corticotropin releasing factor, pro-opiomelanocortin B,
239 analysis showed significant upregulation of corticotropin releasing factor receptor 2 (CrfR2) in the
241 nvestigated here the interaction between the corticotropin releasing factor receptor type 1 (CRF1R) a
242 tuted pyridyl)pyrazolo[1,5-a]-1,3,5-triazine corticotropin releasing factor receptor-1 (CRF(1)) recep
243 methoxyphenyl)pyrazolo[1,5-a]-1,3,5-triazine corticotropin releasing factor receptor-1 (CRF(1)) recep
244 cyclic imidazo[4,5-b]pyridin-2-ones as human corticotropin-releasing factor receptor (CRF(1)) antagon
245 ss, the physiological consequence of central corticotropin-releasing factor receptor (CRF-R) activati
246 viously reported differential involvement of corticotropin-releasing factor receptor (CRFR) 1 and 2 i
249 In addition, we examined the role of the corticotropin-releasing factor receptor 1 (CRF(1)) in th
250 We aimed to characterize the effects of the corticotropin-releasing factor receptor 1 (CRF-R1) antag
251 noradrenergic (NE) receptors (alpha1) via a corticotropin-releasing factor receptor 1 (CRF-R1)-depen
252 rial evaluating the efficacy of GSK561679, a corticotropin-releasing factor receptor 1 (CRF1 receptor
256 Similarly to what has been observed for the corticotropin-releasing factor receptor 1 (CRFR1), SAP97
257 me proliferator-activated receptor gamma and corticotropin-releasing factor receptor 1 were notable e
258 ecifically activated by either neurokinin I, corticotropin-releasing factor receptor 1, or dopamine D
260 mutants with constitutive activation of the corticotropin-releasing factor receptor family homologue
261 anaphylaxis and psychological stress through corticotropin-releasing factor receptor subtype 1 (CRF(1
262 In this study we investigated the role of corticotropin-releasing factor receptor subtype 2 (CRF(2
263 d, we took the CRF(2(a))R and the homologous corticotropin-releasing factor receptor type 1 (CRF(1)R)
264 esent study investigated whether blockade of corticotropin-releasing factor receptor type 1 (CRF-R1)
265 n reflect reductions in anandamide driven by corticotropin-releasing factor receptor type 1 (CRF1) po
266 we investigated interactions of the class B corticotropin-releasing factor receptor type 1 (CRF1R) w
268 ular membrane compartments, we show that the corticotropin-releasing factor receptor type 1 has a spe
269 ure of the transmembrane domain of the human corticotropin-releasing factor receptor type 1 in comple
272 vation of the central stress response, while corticotropin-releasing factor receptor type 2 (CRFR2) h
274 eviously, we observed abnormal expression of corticotropin-releasing factor receptor type 2 (CRFR2) t
277 n in the BNST is unaffected by alpha1-AR and corticotropin-releasing factor receptor-1 (CRFR(1)) anta
278 Effects on attention were attenuated by the corticotropin-releasing factor receptor-1 antagonist ant
282 alysis studies it has been shown to increase corticotropin-releasing factor release in extrahypothala
284 ry-adrenal axis), (4) the (gastrointestinal) corticotropin-releasing factor system, and (5) the intes
287 ess effect by counteracting the functions of corticotropin-releasing factor, the primary stress-media
288 peptides (ghrelin, nesfatin-1, somatostatin, corticotropin-releasing factor, thyrotropin-releasing ho
290 This hypothesis was investigated for the corticotropin-releasing factor type 1 (CRF(1)) receptor
291 hich oxytocin (OTR), vasopressin (V1aR), and corticotropin-releasing factor type 1 (CRF1) or type 2 r
293 icated that repeated social stress decreased corticotropin-releasing factor type 1 receptor and incre
295 disrupts this LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s)
296 disrupts this LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s)
297 re sensitive to central anorectic effects of corticotropin-releasing factor type 2 (CRF(2)) receptor
299 demonstrated that the mechanism involved the corticotropin-releasing factor type 2 receptor, cAMP ele
300 n the amygdala, which required activation of corticotropin-releasing factor type-1 (CRF-R1) receptors