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1  excitability induced by the stress hormone, corticotropin releasing factor.
2 ORs that colocalize with the stress hormone, corticotropin releasing factor.
3 e diuretic hormone 44 (DH44), an ortholog of corticotropin-releasing factor.
4 he transmembrane domains of the glucagon and corticotropin releasing factor 1 (CRF1) receptors to dev
5       An orally active clinical candidate of corticotropin-releasing factor 1 (CRF 1) antagonist 1 sh
6 ective, and orally active antagonists of the corticotropin-releasing factor 1 (CRF 1) receptor.
7 ist (eticlopride), D2R agonist (quinpirole), corticotropin-releasing factor 1 (CRF1) antagonist (anta
8 urthermore, pharmacologic inhibition (with a corticotropin-releasing factor 1 receptor antagonist) of
9 s was identified as potent and orally active corticotropin-releasing factor-1 (CRF(1)) receptor antag
10 ctive withdrawal-like state characterized by corticotropin-releasing factor-1 (CRF(1)) receptor antag
11                       Evidence suggests that corticotropin-releasing factor-1 (CRF(1)) receptor antag
12 ation of 8, an earlier lead pyrazinone-based corticotropin-releasing factor-1 (CRF(1)) receptor antag
13 rmittent access to palatable food results in corticotropin-releasing factor-1 (CRF1) receptor antagon
14                                              Corticotropin-releasing factor, a stress-related neurope
15 amine and opioid peptide function, increased corticotropin-releasing factor activity).
16 hine exposure sensitizes the LC-NE system to corticotropin releasing factor and stress.
17 t of brain stress neurotransmitters, such as corticotropin-releasing factor and dynorphin, in the neu
18  alcohol drinking by increased expression of corticotropin-releasing factor and its feedback regulati
19 ween the N/OFQ and Hcrt systems in which the corticotropin-releasing factor and N/OFQ systems coordin
20  that promote stress and resilience, such as corticotropin-releasing factor and nociceptin, has been
21           This parallels a similar effect of corticotropin-releasing factor and suggests a form of ne
22 nsatory mechanism in the brain to counteract corticotropin-releasing factor and/or stress.
23 fering the neural stress response induced by corticotropin releasing factor, and promoting stress res
24 including hypocretin/orexin, norepinephrine, corticotropin-releasing factor, and cytokines.
25 by a variety of central (eg, neuropeptide Y, corticotropin-releasing factor, and neuromedin U) and pe
26 cuses on the HPA axis-based interventions of corticotropin-releasing factor antagonists and the gluco
27 cial behavior (especially neuropeptide Y and corticotropin-releasing factor) are modulated by alcohol
28                                              Corticotropin releasing factor binding protein (CRF-BP)
29                                              Corticotropin releasing factor-binding protein (CRF-BP)
30                                              Corticotropin-releasing factor binds with high affinity
31 88-induced c-Fos activation were observed in corticotropin releasing factor-containing neurons of the
32 eus of the hypothalamus and primarily in non-corticotropin releasing factor-containing neurons of the
33 gh an interaction between the stress hormone corticotropin releasing factor (CRF) and glutamate relea
34                      The stress neuropeptide corticotropin releasing factor (CRF) and its receptors (
35                    The circadian activity of corticotropin releasing factor (CRF) and the hypothalami
36                                          The corticotropin releasing factor (CRF) antagonist, alpha-h
37 blocked by intravenous pretreatment with the corticotropin releasing factor (CRF) antagonist, astress
38                                              Corticotropin releasing factor (CRF) coordinates the bra
39                                          The corticotropin releasing factor (CRF) family of ligands a
40                      The stress neuropeptide corticotropin releasing factor (CRF) is anxiogenic and i
41      Here we demonstrate that, in the vBNST, corticotropin releasing factor (CRF) is expressed in neu
42                                              Corticotropin releasing factor (CRF) is the primary medi
43  of this study was to examine the ability of corticotropin releasing factor (CRF) or antibody to insu
44                                              Corticotropin releasing factor (CRF) plays a major role
45 port that the stress-associated neuropeptide corticotropin releasing factor (CRF) produces a profound
46                                      Reduced corticotropin releasing factor (CRF) receptor activity a
47 icotine withdrawal was mediated by increased corticotropin releasing factor (CRF) receptor-1 expressi
48                                              Corticotropin releasing factor (CRF) regulates physiolog
49                                              Corticotropin releasing factor (CRF) signaling and CRF n
50 lar nucleus (PVN) have been shown to inhibit corticotropin releasing factor (CRF) synthesis via GABA(
51                                          The corticotropin releasing factor (CRF) system in the centr
52                                          The corticotropin releasing factor (CRF) system in the centr
53                                          The corticotropin releasing factor (CRF) type 1 receptor (CR
54                                              Corticotropin releasing factor (CRF) within the ventral
55 ontains a large number of neurons expressing corticotropin releasing factor (CRF), a neuropeptide tha
56  intracerebroventricular (icv) injections of corticotropin releasing factor (CRF), a putative anxioge
57 analyzed brain regional, CCK-8, substance P, corticotropin releasing factor (CRF), and neuropeptide Y
58  for 15 days and measured mRNA expression of corticotropin releasing factor (CRF), neuropeptide Y (NP
59  until the corticotropes are stimulated with corticotropin releasing factor (CRF), whereupon SSTR2 ex
60         Urocortin 1 (Ucn 1) is an endogenous corticotropin releasing factor (CRF)-related peptide.
61 presence of the prototypical stress hormone, corticotropin releasing factor (CRF).
62 of action for the anxiogenic neuromodulator, corticotropin releasing factor (CRF).
63 ine seeking by the stress-sensitive peptide, corticotropin releasing factor (CRF).
64                                The mammalian corticotropin releasing factor (CRF)/urocortin (Ucn) pep
65           Two new classes of tricyclic-based corticotropin-releasing factor (CRF(1)) receptor-1 antag
66 were measured at baseline and in response to corticotropin-releasing factor (CRF) (0.5 microg kg(1))
67 c raphe nucleus, including a coordination of corticotropin-releasing factor (CRF) actions at both of
68                               We report that corticotropin-releasing factor (CRF) acts in the ventral
69 f chronic nicotine SA on the coexpression of corticotropin-releasing factor (CRF) and arginine vasopr
70         Although it has long been known that corticotropin-releasing factor (CRF) and CRF receptors a
71 ocus of the present review is on the role of corticotropin-releasing factor (CRF) and CRF-related pep
72             Long-term alterations in central corticotropin-releasing factor (CRF) and glucocorticoid
73  mRNA was found to partially colocalize with corticotropin-releasing factor (CRF) and growth hormone-
74 ajor site of extrahypothalamic expression of corticotropin-releasing factor (CRF) and its G-protein-c
75                                              Corticotropin-releasing factor (CRF) and nociceptin/orph
76 laced on the neuropharmacological actions of corticotropin-releasing factor (CRF) and norepinephrine
77                 Release of the neuropeptides corticotropin-releasing factor (CRF) and orexin-A in the
78                                              Corticotropin-releasing factor (CRF) and related peptide
79                                              Corticotropin-releasing factor (CRF) and the closely rel
80                                              Corticotropin-releasing factor (CRF) and the three relat
81                                              Corticotropin-releasing factor (CRF) and urocortin (Ucn
82         Here we report the identification of corticotropin-releasing factor (CRF) as a critical compo
83                                    Exogenous corticotropin-releasing factor (CRF) but not corticoster
84 ng the endocrine arm of the stress response, corticotropin-releasing factor (CRF) can act in the brai
85 s-induced release of neuromodulators such as corticotropin-releasing factor (CRF) can drive drug-depe
86                               To investigate corticotropin-releasing factor (CRF) contributions to fe
87 ses and how the stress-related neuropeptide, corticotropin-releasing factor (CRF) directs this by a b
88 neurons that release the stress neuropeptide corticotropin-releasing factor (CRF) drive anxiety-like
89  regulation of the central extrahypothalamic corticotropin-releasing factor (CRF) expression is assoc
90 -term effects of an acute colitis on central corticotropin-releasing factor (CRF) expression.
91                Urocortins are members of the corticotropin-releasing factor (CRF) family of peptides
92                                          The corticotropin-releasing factor (CRF) family of peptides
93         Urocortin 3 (Ucn 3), a member of the corticotropin-releasing factor (CRF) family of peptides,
94                              Peptides of the corticotropin-releasing factor (CRF) family signal throu
95         Urocortin 2 (Ucn 2), a member of the corticotropin-releasing factor (CRF) family, and its cog
96         Urocortin 2 (Ucn2), a peptide of the corticotropin-releasing factor (CRF) family, binds with
97  hypersecretion of the stress neuromediator, corticotropin-releasing factor (CRF) has been implicated
98 rofiling of relevant PI cells identified the corticotropin-releasing factor (CRF) homolog, DH44, as a
99 ike ShA cocaine self-administration, reduced corticotropin-releasing factor (CRF) immunodensity in th
100 gates the regulation of cytosolic calcium by corticotropin-releasing factor (CRF) in midbrain dopamin
101 ed body of work indicates a crucial role for corticotropin-releasing factor (CRF) in neurobiological
102 vioral studies support a modulatory role for corticotropin-releasing factor (CRF) in regulating the d
103 termined the role of the stress neurohormone corticotropin-releasing factor (CRF) in stress-induced b
104  was used to study immunoreactivity (IR) for corticotropin-releasing factor (CRF) in the guinea pig e
105 ed on previous work hypothesizing a role for corticotropin-releasing factor (CRF) in the IC during cr
106 lores the relationship between dynorphin and corticotropin-releasing factor (CRF) in the induction of
107 halin (ENK)] with the stress-related peptide corticotropin-releasing factor (CRF) in the LC.
108 bout the distribution of the stress hormone, corticotropin-releasing factor (CRF) in the mouse brain.
109       We recently described the existence of corticotropin-releasing factor (CRF) in the mouse cochle
110 ine was compared following overexpression of corticotropin-releasing factor (CRF) in the NAc of femal
111 dies have found that the stress neurohormone corticotropin-releasing factor (CRF) inhibits 5-HT neuro
112  literature suggests that catecholamines and corticotropin-releasing factor (CRF) interact in a seria
113    Stress induces the release of the peptide corticotropin-releasing factor (CRF) into the ventral te
114                                              Corticotropin-releasing factor (CRF) is a key mediator o
115                                              Corticotropin-releasing factor (CRF) is also known as a
116                                              Corticotropin-releasing factor (CRF) is an important lin
117                             The neuropeptide corticotropin-releasing factor (CRF) is believed to play
118                                              Corticotropin-releasing factor (CRF) is critical for the
119                                              Corticotropin-releasing factor (CRF) is expressed in the
120                                              Corticotropin-releasing factor (CRF) mediates anxiogenic
121     The present experiments examined whether corticotropin-releasing factor (CRF) modulates memory co
122 rning of day 15, we measured AT(1) receptors corticotropin-releasing factor (CRF) mRNA and immunoreac
123                                              Corticotropin-releasing factor (CRF) mRNA expression in
124 rsal mPFC enhanced restraint-induced Fos and corticotropin-releasing factor (CRF) mRNA expression in
125 re to elicit reliable increases in Fos-ir or corticotropin-releasing factor (CRF) mRNA in the PVH.
126 ysis of Fos induction and relative levels of corticotropin-releasing factor (CRF) mRNA.
127  from chronic alcohol exposure contains ~80% corticotropin-releasing factor (CRF) neurons and that th
128         Although it has long-been known that corticotropin-releasing factor (CRF) neurons are promine
129 and anxiety and activates a subpopulation of corticotropin-releasing factor (CRF) neurons in the bed
130 cohol intake specifically recruited GABA and corticotropin-releasing factor (CRF) neurons in the mPFC
131                           Antagonists of the corticotropin-releasing factor (CRF) neuropeptide should
132  the relationship between corticosterone and corticotropin-releasing factor (CRF) on both beta-amyloi
133 ed the effect of an intravenous injection of corticotropin-releasing factor (CRF) on fructose malabso
134 and the influence of the stress neuropeptide corticotropin-releasing factor (CRF) on these responses.
135 nced fear memory but did not increase either corticotropin-releasing factor (CRF) or corticosterone.
136                                 Injection of corticotropin-releasing factor (CRF) or urocortin III, k
137                                              Corticotropin-releasing factor (CRF) orchestrates the st
138                                          The corticotropin-releasing factor (CRF) peptide hormone fam
139                                          The corticotropin-releasing factor (CRF) plays a key role in
140                                      Because corticotropin-releasing factor (CRF) plays a role in str
141          Considerable evidence suggests that corticotropin-releasing factor (CRF) plays an important
142                                          The corticotropin-releasing factor (CRF) receptor 1 (CRFR1)
143 duction and Abeta elevation are dependent on corticotropin-releasing factor (CRF) receptor 1 signalin
144                                              Corticotropin-releasing factor (CRF) receptor activation
145                                          The corticotropin-releasing factor (CRF) receptor CRFR2 is e
146                                              Corticotropin-releasing factor (CRF) receptor subtypes 1
147                                              Corticotropin-releasing factor (CRF) receptors are found
148                                      Because corticotropin-releasing factor (CRF) receptors are impli
149                                              Corticotropin-releasing factor (CRF) receptors have been
150  We recently demonstrated that activation of corticotropin-releasing factor (CRF) receptors in the ca
151 ife social isolation increases the levels of corticotropin-releasing factor (CRF) receptors in the se
152                                              Corticotropin-releasing factor (CRF) receptors-which bin
153             The stress-related neuropeptide, corticotropin-releasing factor (CRF) regulates the dorsa
154 induced relapse through alterations in brain corticotropin-releasing factor (CRF) regulation of neuro
155                                              Corticotropin-releasing factor (CRF) signaling at the CR
156                               Alterations in corticotropin-releasing factor (CRF) signaling pathways
157                                Activation of corticotropin-releasing factor (CRF) signaling pathways
158                   Over the past few decades, corticotropin-releasing factor (CRF) signaling pathways
159 o negative reinforcement is a recruitment of corticotropin-releasing factor (CRF) signaling within th
160   Previous studies have implicated the brain corticotropin-releasing factor (CRF) stress systems in m
161 likely resulting from dysregulation of brain corticotropin-releasing factor (CRF) stress systems.
162 ment of the extrahypothalamic stress peptide corticotropin-releasing factor (CRF) system and activati
163                         Dysregulation of the corticotropin-releasing factor (CRF) system has been imp
164                                          The corticotropin-releasing factor (CRF) system is the prima
165 eviously demonstrated that activation of the corticotropin-releasing factor (CRF) system potentiates
166 ess systems, including the extrahypothalamic corticotropin-releasing factor (CRF) system, following l
167                                          The corticotropin-releasing factor (CRF) system, which is in
168 f neonatal amygdala (Neo-A) lesions on brain corticotropin-releasing factor (CRF) systems and hypotha
169                                              Corticotropin-releasing factor (CRF) that is released fr
170                                          The corticotropin-releasing factor (CRF) type 1 receptor (CR
171                         Systemic blockade of corticotropin-releasing factor (CRF) type 1 receptors (C
172  authors investigated the effect of blocking corticotropin-releasing factor (CRF) Type I and Type II
173 clase-activating peptide, PAC1 receptor, and corticotropin-releasing factor (CRF), (CRF1) receptor.
174                                              Corticotropin-releasing factor (CRF), a 41 amino acid pe
175                          Here we report that corticotropin-releasing factor (CRF), a neuropeptide rel
176          The bimolecular interaction between corticotropin-releasing factor (CRF), a neuropeptide, an
177 nsistent with this, the CEA highly expresses corticotropin-releasing factor (CRF), an important modul
178 he LC include excitatory amino acids (EAAs), corticotropin-releasing factor (CRF), and endogenous opi
179 (Dh44), a neuropeptide related to vertebrate corticotropin-releasing factor (CRF), and its receptor,
180 vitro and their modulation by dopamine (DA), corticotropin-releasing factor (CRF), and their combinat
181                   The neuroactive substances corticotropin-releasing factor (CRF), arginine-vasopress
182 to the BNSTDL, is thought to communicate via corticotropin-releasing factor (CRF), but studies have y
183                            The neuropeptide, corticotropin-releasing factor (CRF), coordinates the ph
184                                              Corticotropin-releasing factor (CRF), encoded by the CRH
185 -protein-coupled receptor B1 family includes corticotropin-releasing factor (CRF), growth hormone-rel
186 -adrenal (HPA) axis-activating neuropeptide, corticotropin-releasing factor (CRF), may be the keyston
187 ological studies indicate the involvement of corticotropin-releasing factor (CRF), noradrenaline, dop
188                                              Corticotropin-releasing factor (CRF), originally charact
189 es in receptors for the stress neuropeptide, corticotropin-releasing factor (CRF), that render the lo
190                                              Corticotropin-releasing factor (CRF), the major stress p
191             The focus is on the receptor for corticotropin-releasing factor (CRF), the orchestrator o
192                                              Corticotropin-releasing factor (CRF), the stress-related
193 ways expressing the stress-sensitive peptide corticotropin-releasing factor (CRF), which has been ide
194 ral amygdala noradrenergic substrates [via a corticotropin-releasing factor (CRF)-dependent mechanism
195 allenge has been shown previously to cause a corticotropin-releasing factor (CRF)-mediated increase i
196                                              Corticotropin-releasing factor (CRF)-mediated mechanisms
197 de transmitter in the brain that counteracts corticotropin-releasing factor (CRF)-mediated stress and
198 und that GluN2D is functionally expressed on corticotropin-releasing factor (CRF)-positive BNST cells
199                                              Corticotropin-releasing factor (CRF)-related peptides ca
200                                              Corticotropin-releasing factor (CRF)-related peptides mo
201 ) system by the stress-related neuropeptide, corticotropin-releasing factor (CRF).
202 agonists for the stress-related neurohormone corticotropin-releasing factor (CRF).
203    A major determinate of these responses is corticotropin-releasing factor (CRF).
204 n the lateral parabrachial nucleus coexpress corticotropin-releasing factor (CRF).
205 ntracerebroventricular infusion of saline or corticotropin-releasing factor (CRF).
206 action of the stress-associated neurohormone corticotropin-releasing factor (CRF).
207 nity monoclonal antibody, CTRND05, targeting corticotropin-releasing factor (CRF).
208 amygdala (CeA) that produce the neuropeptide corticotropin-releasing factor (CRF).
209 d reward, including neuropeptide Y (NPY) and corticotropin-releasing factor (CRF).
210 vation of glucocorticoid receptors (GRs) and corticotropin-releasing factor (CRF).
211 ation of VTA dopaminergic neurons expressing corticotropin-releasing factor (CRF).
212 known as maternal aggression) is impaired by corticotropin-releasing factor-(CRF) related peptides, b
213 ed neural cell density, stress neuropeptide (corticotropin releasing factor--CRF) levels, and plasma
214    This study tested the hypothesis that the corticotropin-releasing factor (CRF1) antagonist GSK5616
215 ers of the corticoliberin family include the corticotropin releasing factors (CRFs), sauvagine, the u
216 -system recruitment of brain stress systems (corticotropin-releasing factor, dynorphin, norepinephrin
217 rom the basolateral amygdala (BLA) away from corticotropin releasing factor-expressing (CRF(+)) centr
218 rmones and is transcriptionally activated in corticotropin releasing factor-expressing cells.
219 ed with brain region-specific alterations of corticotropin-releasing factor expression and promoter m
220            The neuropeptides vasopressin and corticotropin-releasing factor facilitate, while seroton
221 oupled receptors, which bind peptides of the corticotropin releasing factor family and are key mediat
222  fashion both NGF and the hormone urotensin (corticotropin-releasing factor family ligand).
223 ortin 2, a recently identified member of the corticotropin-releasing factor family, is expressed in d
224       They further suggest that an activated corticotropin-releasing factor/hypothalamic-pituitary-ad
225 ephalin, thyrothropin-releasing hormone, and corticotropin-releasing factor immunoreactive cells in t
226 CRFR1) mediates the physiological actions of corticotropin-releasing factor in the anterior pituitary
227 tuitary-adrenal axis, including signaling by corticotropin-releasing factor, in the pathophysiology o
228                                              Corticotropin-releasing factor infusions into LA impair
229                                              Corticotropin-releasing factor infusions into the centra
230 in abnormal cerebrospinal fluid cortisol and corticotropin-releasing factor levels.
231 ated increments in restraint-induced Fos and corticotropin-releasing factor mRNA expression in the ne
232 w stress interacts with the neuromodulators, corticotropin-releasing factor, norepinephrine, dopamine
233 w explores the role of brain stress systems (corticotropin-releasing factor, norepinephrine, orexin [
234 s in the anterior hypothalamus that may gate corticotropin-releasing factor output from the amygdala
235 s an activation of the brain stress system's corticotropin-releasing factor outside of the hypothalam
236 were identified (i.e., serotonin, dynorphin, corticotropin-releasing factor, oxytocin).
237 in stress and appetite regulation, including corticotropin releasing factor, pro-opiomelanocortin B,
238 technique to a member of a class B GPCR, the corticotropin releasing factor receptor 1 (CRF1R).
239  analysis showed significant upregulation of corticotropin releasing factor receptor 2 (CrfR2) in the
240       The current study examines the role of corticotropin releasing factor receptor subtypes 1 and 2
241 nvestigated here the interaction between the corticotropin releasing factor receptor type 1 (CRF1R) a
242 tuted pyridyl)pyrazolo[1,5-a]-1,3,5-triazine corticotropin releasing factor receptor-1 (CRF(1)) recep
243 methoxyphenyl)pyrazolo[1,5-a]-1,3,5-triazine corticotropin releasing factor receptor-1 (CRF(1)) recep
244 cyclic imidazo[4,5-b]pyridin-2-ones as human corticotropin-releasing factor receptor (CRF(1)) antagon
245 ss, the physiological consequence of central corticotropin-releasing factor receptor (CRF-R) activati
246 viously reported differential involvement of corticotropin-releasing factor receptor (CRFR) 1 and 2 i
247 c disruption of signaling through the type 1 corticotropin-releasing factor receptor (CRFR1).
248              Administration of the selective corticotropin-releasing factor receptor 1 (CRF(1)) antag
249     In addition, we examined the role of the corticotropin-releasing factor receptor 1 (CRF(1)) in th
250  We aimed to characterize the effects of the corticotropin-releasing factor receptor 1 (CRF-R1) antag
251  noradrenergic (NE) receptors (alpha1) via a corticotropin-releasing factor receptor 1 (CRF-R1)-depen
252 rial evaluating the efficacy of GSK561679, a corticotropin-releasing factor receptor 1 (CRF1 receptor
253                          We describe a novel corticotropin-releasing factor receptor 1 (CRF1) antagon
254                                  Blockade of corticotropin-releasing factor receptor 1 (CRF1) suppres
255                                              Corticotropin-releasing factor receptor 1 (CRFR1) mediat
256  Similarly to what has been observed for the corticotropin-releasing factor receptor 1 (CRFR1), SAP97
257 me proliferator-activated receptor gamma and corticotropin-releasing factor receptor 1 were notable e
258 ecifically activated by either neurokinin I, corticotropin-releasing factor receptor 1, or dopamine D
259                            Mice deficient in corticotropin-releasing factor receptor 2 (CRF2) (C57BL/
260  mutants with constitutive activation of the corticotropin-releasing factor receptor family homologue
261 anaphylaxis and psychological stress through corticotropin-releasing factor receptor subtype 1 (CRF(1
262    In this study we investigated the role of corticotropin-releasing factor receptor subtype 2 (CRF(2
263 d, we took the CRF(2(a))R and the homologous corticotropin-releasing factor receptor type 1 (CRF(1)R)
264 esent study investigated whether blockade of corticotropin-releasing factor receptor type 1 (CRF-R1)
265 n reflect reductions in anandamide driven by corticotropin-releasing factor receptor type 1 (CRF1) po
266  we investigated interactions of the class B corticotropin-releasing factor receptor type 1 (CRF1R) w
267                                              Corticotropin-releasing factor receptor type 1 (CRFR1) i
268 ular membrane compartments, we show that the corticotropin-releasing factor receptor type 1 has a spe
269 ure of the transmembrane domain of the human corticotropin-releasing factor receptor type 1 in comple
270                                          The corticotropin-releasing factor receptor type 1 is a clas
271 here this equilibrium is established for the corticotropin-releasing factor receptor type 1.
272 vation of the central stress response, while corticotropin-releasing factor receptor type 2 (CRFR2) h
273                                              Corticotropin-releasing factor receptor type 2 (CRFR2) h
274 eviously, we observed abnormal expression of corticotropin-releasing factor receptor type 2 (CRFR2) t
275                                          The corticotropin-releasing factor receptor type 2a (CRF(2(a
276                                              Corticotropin-releasing factor receptor-1 (CRF(1)) media
277 n in the BNST is unaffected by alpha1-AR and corticotropin-releasing factor receptor-1 (CRFR(1)) anta
278  Effects on attention were attenuated by the corticotropin-releasing factor receptor-1 antagonist ant
279 NMR structure of the analogous domain of the corticotropin-releasing factor receptor.
280                                              Corticotropin-Releasing Factor Receptors (CRFRs) are cla
281                                              Corticotropin-releasing factor receptors (CRFRs), class
282 alysis studies it has been shown to increase corticotropin-releasing factor release in extrahypothala
283                       Alterations in central corticotropin-releasing factor signaling pathways have b
284 ry-adrenal axis), (4) the (gastrointestinal) corticotropin-releasing factor system, and (5) the intes
285                                 In addition, corticotropin-releasing factor systems were shown to fac
286 is function was probed using a dexamethasone/corticotropin-releasing factor test.
287 ess effect by counteracting the functions of corticotropin-releasing factor, the primary stress-media
288 peptides (ghrelin, nesfatin-1, somatostatin, corticotropin-releasing factor, thyrotropin-releasing ho
289                                              Corticotropin releasing factor type 1 (CRF1) is a key co
290     This hypothesis was investigated for the corticotropin-releasing factor type 1 (CRF(1)) receptor
291 hich oxytocin (OTR), vasopressin (V1aR), and corticotropin-releasing factor type 1 (CRF1) or type 2 r
292                                Activation of corticotropin-releasing factor type 1 (CRF1) receptors i
293 icated that repeated social stress decreased corticotropin-releasing factor type 1 receptor and incre
294                                We found that corticotropin-releasing factor type 1 receptor within th
295 disrupts this LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s)
296 disrupts this LTCC-based mechanism; instead, corticotropin-releasing factor type 1 receptors (CRF1s)
297 re sensitive to central anorectic effects of corticotropin-releasing factor type 2 (CRF(2)) receptor
298                                          The corticotropin-releasing factor type 2 receptor (CRFR2) i
299 demonstrated that the mechanism involved the corticotropin-releasing factor type 2 receptor, cAMP ele
300 n the amygdala, which required activation of corticotropin-releasing factor type-1 (CRF-R1) receptors

 
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