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1 n and mass reduction would facilitate timely counteraction.
2 e multifunctional proteins to coordinate the counteractions.
3 ly in host defense prior to the onset of IFN counteraction and the acquired immune response.
4 e results provide evidence in support of the counteraction aspect of the urea:TMAO paradigm linking s
5                               The functional counteraction between COUP-TFII and SMAD4 is reinforced
6                   Intriguingly, altering the counteraction between Par3 and Lgl1/2 induces cell-cell
7 tterning in vertebrates is determined by the counteraction between the Sonic Hedgehog (Shh) and the G
8  in APOBEC3F contributes to species-specific counteraction by HIV-2 and SIVsmm Vifs.
9 change in Vif contribute to species-specific counteraction by HIV-2 and SIVsmm.
10  antagonizes human tetherin and suggest that counteraction by O-Nefs may be suboptimal.IMPORTANCE Pre
11 bitory and stabilizing effects of DA and its counteraction by S40-phosphorylation, key regulatory mec
12 e signaling pathways involved; and 6) mutual counteraction by urea and methylamines.
13                          The extent to which counteraction can occur is seen to depend heavily on the
14 orster resonance energy transfer to test the counteraction hypothesis of counterbalancing effects bet
15  it is used here to test the validity of the counteraction hypothesis.
16 etic parameters, supporting the premise that counteraction is a property of the solvent system and is
17  suggests that Mip130 activity without DmMyb counteraction may be responsible for the Dm-myb mutant l
18 tional standpoint, our understanding of this counteraction mechanism is hampered by the fact that exi
19  A3G-Vif interaction, the key event of HIV's counteraction mechanism to evade antiviral innate immune
20 xamined factors that might determine whether counteraction occurs, namely different combinations of a
21  intact vif genes, highlighting the need for counteraction of APOBEC3 proteins.
22 ulting in non-essential immune responses and counteraction of bacterial protective immune responses w
23  those found in M-Vpus and mediate efficient counteraction of both the long and short isoforms of thi
24 iction of smooth muscle and antiinflammatory counteraction of chronic injury.
25 fic activation of stress-inducible genes via counteraction of corepressors.
26 olymer collapse and swelling transitions and counteraction of denaturant-induced protein unfolding.
27 er therapy efficacy, by acting to reduce the counteraction of ferroptosis by tumour cells by means of
28 vivo findings reveal inhibition of MyD88 via counteraction of IL-1-mediated proteoglycan depletion.
29 MinD binding to the surface is controlled by counteraction of initiation and dissociation complexes;
30 ation of BST-2 from the cell surface and the counteraction of restricted virion release.
31 entified as an additional mechanism of viral counteraction of SG formation.
32 o block both IL10 and PD-1 to strengthen the counteraction of T-cell immunosuppression and to enhance
33               Downmodulation of CD4, but not counteraction of tetherin, by RBF206 Vpu was dependent o
34 a pivotal role in enhancing HIV infection by counteraction of Tetherin.
35 -activated protein kinases p38 and JNK-1, 3) counteraction of the effects of CHG on TNFalpha producti
36 ll of these effects are consistent with TMAO counteraction of the effects of urea on LDH kinetic para
37                                  The optimal counteraction of the excessive sympathetic activity is s
38 GDNF signaling in a feed-forward loop and/or counteraction of the inhibitory pathway regulated by BMP
39  infection in most cases, in part due to the counteraction of these host defenses by viral accessory
40 t baboon cultures, suggesting less efficient counteraction of these responses by viral proteins in ma
41 s with an expanded RNR functionality through counteraction of this antiviral enzyme.
42                                  Optogenetic counteraction of those changes in the VLS and VMS impair
43                             We conclude that counteraction of urea effects on enzymes by methylamines
44 otoxin-induced lethal shock suggest that the counteraction or neutralization of MIF may serve as an a
45 es of automatic response bias and a conflict counteraction parameter to individual subject behavioral
46 nd evolutionary origin of this innate immune counteraction strategy.
47                                           In counteraction, the host uses the SAFB reader complex to
48 ptors and transmission of signals for proper counteractions through adaptor and effector molecules.
49 n of multiple felids are responsible for the counteraction triggered by FIV Petaluma Vif.
50 d the ISO-stimulated NKCC activity, and this counteraction was sensitive to the p38 mitogen-activated
51 o determine the molecular basis for tetherin counteraction, we reconstituted the AP-2 complex with a