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1 nse to EAAC1-mediated Na+ cotransport and K+ countertransport.
2 l competition between similar molecules, and countertransport.
3  it with three sodium ions and a proton, and countertransporting a potassium ion via an elevator mech
4 ibrium exchange conditions while maintaining countertransport acceleration.
5  and they did not show the expected level of countertransport acceleration.
6 t NHA genes may contribute to sodium-lithium countertransport activity and salt homeostasis in humans
7        These are hallmarks of sodium-lithium countertransport activity, a highly heritable trait corr
8            These ion gradients drive co- and countertransport and are critical for establishing the m
9                                              Countertransport and reversibility studies demonstrate t
10 plasm is known to be indirectly linked to H+ countertransport and, hence, stromal pH and photosynthet
11         Uptake of 5-FTHF is characterized by countertransport because adding unlabeled 5-FTHF in the
12 wed that ORP5 and ORP8 could mediate PI4P/PS countertransport between the ER and the PM, thus deliver
13 ion and release as a consequence of the H(+) countertransport by VMAT that accompanies vesicular upta
14     A related question is whether P4-ATPases countertransport ions or other substrates in the opposit
15  the electrochemical gradient of the co- and countertransported ions.
16 current and the concentration of analyte and countertransporting ions and is found to correspond well
17 on flow beside the well-known inverse proton countertransport occurring in active Ca(2+) transport.
18 nosomes preloaded with tyrosine demonstrated countertransport of 10 microM [3H]tyrosine, indicating c
19 ort of a proton and three Na(+) ions and the countertransport of a K(+) ion.
20 e net flux and complete a transport cycle by countertransport of K+, ASCT-1 mediates only homo- and h
21 mic acid with Na+, the cycle is completed by countertransport of K+.
22 l glutamate transport is achieved by the co-/countertransport of Na(+) and other ions down their conc
23  exchangers catalyze the electrically silent countertransport of Na+ and H+, controlling the transmem
24 tamic acid with three Na(+) ions followed by countertransport of one K(+) ion.
25 ansporting three Na(+) and one H(+) with the countertransport of one K(+).
26  of three sodium ions and one proton and the countertransport of one potassium ion.
27 hree sodium ions and one proton, followed by countertransport of potassium.
28 atp1-mediated uptake is energized by efflux (countertransport) of intracellular reduced glutathione (
29 er affinity for the neurotransmitter and the countertransported potassium ion than outward- or inward
30 ontrols studies are consistent with an anion countertransport process using a relay mechanism and a k
31 e becomes competitive toward F and Britton's countertransport property disappears with these activato
32 he K(+) ion, which is believed to serve as a countertransport substrate for SERT, was the most effect
33         This behavior may reflect a limiting countertransported substrate needed to promote turnover
34 so showed that no charged substrate is being countertransported, thereby distinguishing the P4-ATPase