ライフサイエンスコーパス: cranial nerve

1 relation to branches of the vagus nerve (Xth cranial nerve).

2 function (III, IV, motor V, VI, VII, and XII cranial nerves).

3 as, benign tumors that arise from the eighth cranial nerve.

4 ed to generate glia that myelinate the VIIth cranial nerve.

5 rized by bilateral schwannomas of the eighth cranial nerve.

6 : only hoxb1a can properly pattern the VIIth cranial nerve.

7 staining indicates abnormalities in the IXth cranial nerve.

8 s show vascular loops compressing the eighth cranial nerve.

9 of both the cervical vertebrae and the IXth cranial nerve.

10 intenance of neurons that compose the VIIIth cranial nerve.

11 t visual obscuration, and palsy of the sixth cranial nerve.

12 ensory ganglia (nodose ganglia) of the tenth cranial nerve.

13 sory areas innervated by branches of the Xth cranial nerve.

14 ith heavy T(2) weighting in the plane of the cranial nerves.

15 d in his understanding of the anatomy of the cranial nerves.

16 rest cells paralleling patterning defects in cranial nerves.

17 crest, a source of cells contributing to the cranial nerves.

18 rgans innervated by the VIIth, IXth, and Xth cranial nerves.

19 ograde direction along the tongue-associated cranial nerves.

20 ral prion infection of the tongue-associated cranial nerves.

21 ng vascular compression and hyperactivity of cranial nerves.

22 autonomic nerves, usually beginning with the cranial nerves.

23 s, plasticin is expressed in a subset of the cranial nerves.

24 d a more variable morphology of the pre-otic cranial nerves.

25 uroendocrine circuits and in motor nuclei of cranial nerves.

26 first detailed description of the post-otic cranial nerves.

27 ructures, including aortic arch, thymus, and cranial nerves.

28 es formed from NCC such as outflow tract and cranial nerves.

29 ns of the somatic and visceral components of cranial nerves.

30 h, fifth, sixth, seventh, tenth, and twelfth cranial nerves.

31 specially in the hindbrain, spinal cord, and cranial nerves.

32 lanted stimulators placed in the brain or on cranial nerves.

33 buted to agenesis of the abducens and facial cranial nerves.

34 asia of extraocular muscles and intraorbital cranial nerves.

35 allosum: 0.05 ug . g(-1) +/- 0.02, P = .001; cranial nerve: 0.02 ug . g(-1) +/- 0.01, P = .004).

36 /trochlear (cranial nerve 3/4), hypoglossal (cranial nerve 12) and lateral motor column of the cervic

37 lei of the normal rat, oculomotor/trochlear (cranial nerve 3/4), hypoglossal (cranial nerve 12) and l

38 Furthermore, the cranial nerve 3/4-restricted genes insulin-like growth f

39 nd facial asymmetry, often caused by seventh cranial nerve abnormalities (36 of 56 in the mutation-po

40 esthesis, vertigo, presyncope, paresthesias, cranial nerve abnormalities, ataxia, dysautonomia, and s

41 malities in these cases, including pupil and cranial nerve abnormalities, frequent optic atrophy, aut

42 ge/22q11.2 deletion syndrome (22q11DS), have cranial nerve and craniofacial dysfunction as well as di

43 recapitulate latent gene expression in human cranial nerve and dorsal root ganglia.

44 wer levels of expression are seen in certain cranial nerve and spinal motoneurons and in small popula

45 otatable three-dimensional (3D) model of the cranial nerves and anterior-most spinal nerves of early

46 al role of the Brn3b transcription factor in cranial nerves and associated nuclei of the brainstem.

47 tial for proper formation of the cerebellum, cranial nerves and cranial neural crest.

48 underdeveloped skull bones, malformations of cranial nerves and hemorrhagic blood vessels in the head

49 As with other cranial nerves and many CNS neurons, primary auditory ne

50 athways carrying visceral sensation from the cranial nerves and may be considered in itself a compone

51 s necessary to initiate tumorigenesis in the cranial nerves and meninges with typical histological fe

52 an spread from the tongue to the brain along cranial nerves and neuroinvasion does not require agent

53 confirm earlier descriptions of the pre-otic cranial nerves and present the first detailed descriptio

54 he organization and fasciculation of several cranial nerves and spinal nerves.

55 eadache, Horner's syndrome, paralysis of the cranial nerves and subsequently cerebral and retinal isc

56 d men, we reviewed some fundamental ideas on cranial nerves and their paralysis as presented by autho

57 l resection of the fifth, sixth, and seventh cranial nerves and to discuss the possible contribution

58 s between lymphatic fluid channels along the cranial nerves and vascular structures and the cervical

59 neurons, which contribute axons to distinct cranial nerves and whose functions are essential to vert

60 e) in 16 patients, radiation necrosis of the cranial nerves and/or their pathways in two patients, ra

61 Assessment) and normal cerebellar, sensory, cranial nerve, and autonomic function.

62 verriding aorta, ventricular septal defects, cranial nerve, and craniofacial skeletal patterning defe

63 us (VZV) establishes latency in dorsal root, cranial nerve, and enteric ganglia and can reactivate to

64 acta, red nucleus, dorsal motor nucleus of X cranial nerve, and giant neurons of sensory motor neocor

65 ocus ceruleus, dorsal motor nucleus of the X cranial nerve, and nucleus basalis of Meynert.

66 tations are pleomorphic affecting the spine, cranial nerves, and cerebral hemispheres.

67 e three meningeal layers, the perineurium of cranial nerves, and meningeal projections into the brain

68 consists of reduced dorsal root ganglia and cranial nerves, and the entire gastrointestinal tract is

69 us, cortex and white matter, spinal cord, or cranial nerves; and second, as a consequence of thrombos

70 of pharyngeal glands, craniofacial skeleton, cranial nerves, aortic arch arteries, cardiac outflow tr

71 ated brainstem from the turtle, in which the cranial nerves are directly stimulated in place of using

72 pathy and ophthalmoplegia involving multiple cranial nerves are the hallmarks of an orbital apex synd

73 ant phenotype; however, these cells, and the cranial nerves, are misdirected.

74 Cranial nerve assessment revealed mild right abducens ne

75 We find that many cranial nerve-associated crest cells coexpress the pan-a

76 allow more drug to access the extracellular cranial nerve-associated pathways and therefore favor de

77 either to the brain via direct extracellular cranial nerve-associated pathways or to the periphery vi

78 glossopharyngeal, vagus, and/or hypoglossal cranial nerves at least 3 months after cancer therapy.

79 nal commissural axons and repelled trochlear cranial nerve axons in these experiments.

80 the respiratory outflow of the Xth and XIIth cranial nerves, both at rest and during chemoreceptor st

81 oxb1a is required for migration of the VIIth cranial nerve branchiomotor neurons from their point of

82 hat form the sensory ganglia associated with cranial nerves, but the molecular mechanisms of placodal

83 is focused around degeneration of the lower cranial nerves, can occur in children or adults and form

84 n the formation and maintenance of the VIIth cranial nerve circuitry.

85 til the end of 2010, death, or occurrence of cranial nerve (CN)3, CN4, or CN6 palsies.

86 ew diagnoses of neuro-ophthalmic conditions (cranial nerve [CN] III, IV, VI, and VII palsy; diplopia;

87 re typically small, with the abducens nerve (cranial nerve [CN]6) often nondetectable.

88 val of the gustatory branches of the seventh cranial nerve [CT and greater superficial petrosal (GSP)

89 lasty of the carotid artery include avoiding cranial nerve damage, wound hematoma, and general anesth

90 es include ocular coloboma, choanal atresia, cranial nerve defects, distinctive external and inner ea

91 Cranial nerve deficits (100% vs 26%), autonomic dysfunct

92 Occurrence of radiation-induced cranial nerve deficits and radiation-induced optic neuro

93 nally presenting with exclusively unilateral cranial nerve deficits following a puncture wound to the

94 plasma nicotine concentrations during fetal cranial nerve development.

95 y lead to an improved understanding of early cranial-nerve development.

96 entiation prefigure subsequent disruption of cranial nerve differentiation and oropharyngeal function

97 gic mechanisms also target oropharyngeal and cranial nerve differentiation.

98 r patterning associated with divergent LgDel cranial nerve differentiation.

99 VZV) and pseudorabies virus (PRV), may cause cranial nerve disorder and unbearable pain.

100 ysician who specializes in the management of cranial nerve disorders.

101 , schwannomas also commonly develop in other cranial nerves, dorsal root ganglia and peripheral nerve

102 of children with acute flaccid paralysis and cranial nerve dysfunction geographically and temporally

103 Improvement of cranial nerve dysfunction has been noted in three (30%)

104 Clusters of acute flaccid paralysis or cranial nerve dysfunction in children are uncommon.

105 fined cluster of acute flaccid paralysis and cranial nerve dysfunction in children associated with an

106 ting with gastroenteritis, symptoms of acute cranial nerve dysfunction including ptosis, dysphagia, b

107 ving mainly grey matter on imaging, or acute cranial nerve dysfunction with brainstem lesions on imag

108 IgG4-RP commonly presents with headaches and cranial nerve dysfunction, posing diagnostic challenges

109 f congenital hypomyelination associated with cranial nerve dysfunction, respiratory failure, and hype

110 s of persons with signs or symptoms of acute cranial nerve dysfunction.

111 The cranial nerve dysfunctions resolved in all patients.

112 Headache and cranial nerve dysfunctions were the most common neurolog

113 rentiation of the trigeminal nerve (CN V), a cranial nerve essential for suckling, feeding and swallo

114 mpathetic nervous system receives input from cranial nerves exclusively and the sympathetic nervous s

115 no detailed and complete description of the cranial nerves exists for this species.

116 ge-clamp recordings in an in vitro brainstem-cranial nerve explant preparation were used to assess th

117 xtensive soft-tissue preservation of brains, cranial nerves, eyes, and potential cardiovascular tissu

118 buds are induced by contact with developing cranial nerve fibers late in embryonic development, sinc

119 ed to have atypical findings (eg, at least 1 cranial nerve finding that was unilateral or ascending p

120 The current model for cranial nerve formation is based on the Vth nerve, in wh

121 Perturbations in hindbrain segmentation, cranial nerve formation, and otic vesicle development we

122 display alterations in palate formation and cranial nerve formation.

123 hyme in a dorsolateral stream and an ectopic cranial nerve forms between the trigeminal and facial ga

124 reatment of the aneurysm, a full recovery of cranial nerve function was achieved.

125 Over 24 hrs, remaining cranial nerve function was lost.

126 felong latent infection in human sensory and cranial nerve ganglia after primary infection.

127 ZV) establishes latency in human sensory and cranial nerve ganglia during primary infection (varicell

128 cbfb is also expressed in Rohon-Beard cells, cranial nerve ganglia, hindbrain, retina, branchial arch

129 atic duct and sac, and neurons of the eighth cranial nerve ganglion.

130 It was also expressed in the cranial nerves (gV, gVII, gIV and gX) and in primary sen

131 al nerve (cranial nerve V), along with other cranial nerves, has in recent years become a popular tar

132 e first to identify the olfactory nerve as a cranial nerve, his dissections showed him that contrary

133 d vascular relationships of the optic nerve (cranial nerve II) and of the diagnosis and treatment of

134 n of Brn3b in the somatosensory component of cranial nerves II, V, VII, and VIII and visceromotor nuc

135 of left-sided weakness, followed shortly by cranial nerve III and VI palsies.

136 g 109 patients enrolled in the study, 22 had cranial nerve III palsy, 25 had cranial nerve IV palsy,

137 nervation of the choroid is via the ciliary (cranial nerve III) and pterygopalatine (cranial nerve VI

138 scles innervated by the superior division of cranial nerve III.

139 ationships of the three ocular motor nerves (cranial nerves III, IV, and VI) and of the diagnosis and

140 ody size, brain size, and fibers in selected cranial nerves in shrews and moles.

141 pulsation artifacts, and the conspicuity of cranial nerves in the posterior fossa.

142 l venous sinuses in dorsal regions and along cranial nerves in the ventral regions in the human brain

143 s are observed in the development of several cranial nerves, in the initial central projections of sp

144 causing uveitis, the lacrimal gland, and the cranial nerves, including the optic nerve itself.

145 ma (1.7%), cardiac complications (0.5%), and cranial nerve injury (0.4%).

146 Cranial nerve injury (CNI) was 5.5% in the CEA group, wh

147 0.9%, RR (95%CI):0.53 (0.35-0.83), P =.005], cranial nerve injury [0.4% vs.2.7%, RR(95%CI):0.14(0.08-

148 k of postoperative myocardial infarction and cranial nerve injury after TCAR compared to CEA, with no

149 Cranial nerve injury is disabling for patients, and faci

150 ization in patients at high risk for stroke, cranial nerve injury, or major cardiac event.

151 ons, diabetes insipidus, hypopituitarism and cranial nerve injury.

152 On the other hand, cranial nerve-innervated muscle and sensory and cognitiv

153 movement of prions from the brain stem along cranial nerves into skeletal muscle as a model of axonal

154 y motor neuropathy (56%), multinevritis with cranial nerve involvement (16%), Guillain-Barre syndrome

155 In most cases, the relationship of cranial nerve involvement to CIDP remains unclear.

156 ore-frequent preceding respiratory symptoms, cranial nerve involvement, and a better outcome.

157 et and more severe neuropathy with prominent cranial nerve involvement.

158 l metastases manifest primarily or solely as cranial nerve involvement.

159 the development and maintenance of the VIIth cranial nerve is dependent upon Hoxb1, both in the neura

160 tudy, 22 had cranial nerve III palsy, 25 had cranial nerve IV palsy, and 62 had cranial nerve VI pals

161 categories, including greater dysfunction in cranial nerves IX and X (42% versus 8%) and more sensory

162 Furthermore, fusions of cranial nerves IX and X and defects in cranial nerve V w

163 display patterning anomalies of NCCs forming cranial nerves IX and X, which derive from rhombomeres 6

164 atRA to 50 microg/g diet led to the loss of cranial nerves IX, X, XI, and XII and associated sensory

165 indbrain, which included a loss of posterior cranial nerves (IX, X, XI, and XII) and postotic pharyng

166 mplex congenital strabismus characterized by cranial nerve misguidance.

167 Data on the cranial nerve morphology of tadpoles are scarce, and onl

168 Cranial nerve motoneurons and octavolateral efferent neu

169 gh levels of AChE enzymic activity including cranial nerve motor neurons and dopaminergic neurons of

170 ng in amyotrophic lateral sclerosis-affected cranial nerve motor nuclei but not in the relatively spa

171 Within the brainstem, neurons in all cranial nerve motor nuclei contained intense NP-NF immun

172 th (nucleus ambiguus, NA), and twelfth (XII) cranial nerve motor nuclei in the rat, those motor nucle

173 d alpha1A riboprobe in the fifth and seventh cranial nerve motor nuclei showed that the alpha1A mRNA

174 Strikingly, in the brainstem all cranial nerve motor nuclei were PIST-positive at varying

175 leus basalis, hypothalamus, cerebral cortex, cranial nerve motor nuclei, and central and peripheral c

176 ular reticular formation, vestibular nuclei, cranial nerve motor nuclei, sensory trigeminal nerve nuc

177 lei, brainstem reticular nuclei, and several cranial nerve motor nuclei.

178 and for axon pathfinding of trigeminal (Vth cranial nerve) motor axons.

179 in zebrafish, is required for facial (VIIth cranial nerve) motor neuron migration and for axon pathf

180 Abnormal cranial nerve, motor, and sensory examinations, tremor,

181 ) which consisted of level of consciousness, cranial nerve, motor-sensory function, and simple behavi

182 s detected in auditory hair cells and VIIIth cranial nerve neurons.

183 s with early-onset severe sensory, motor and cranial nerve neuropathy we confirmed the strong genetic

184 a phenotypic spectrum of motor, sensory, and cranial nerve neuropathy, often with ataxia, optic atrop

185 One patient died of treatment-related cranial nerve neuropathy.

186 n3b does not affect the anatomy of the other cranial nerves normally expressing this transcription fa

187 ound that the cholinergic cells and axons of cranial nerve nuclei 3, 4, 6, 7,10 and 12 colocalize NPN

188 eurons of the somatic motor and visceromotor cranial nerve nuclei and the ventral horn of the spinal

189 rates the oculomotor, trochlear and abducens cranial nerve nuclei as well as the parabigeminal nucleu

190 f spinal sensory proprioceptive neurons, but cranial nerve nuclei have received little attention.

191 All of the cranial nerve nuclei labeled intensely for NMDAR1 and to

192 ined the number and morphology of neurons in cranial nerve nuclei of young, adult, and aged heterozyg

193 on of motoneurons of the third through sixth cranial nerve nuclei, and (3) a low density of the recep

194 chat-expressing cells are prominent in motor cranial nerve nuclei, and some scattered cells lie in th

195 l neocortex, hippocampus, pretectum, tectum, cranial nerve nuclei, and spinal cord.

196 including severe neuronal loss in the lower cranial nerve nuclei, anterior horns and corresponding n

197 higher than in neurons of the red nucleus or cranial nerve nuclei, but similar values were obtained i

198 , in pons nuclei, in the red nucleus, in all cranial nerve nuclei, in the cerebellum, and in motor ne

199 e regions of the vestibular brainstem and in cranial nerve nuclei.

200 r (nIII), trochlear (nIV) and abducens (nVI) cranial nerve nuclei.

201 later exposures affected the VIth and IIIrd cranial nerve nuclei.

202 hat the initiating lesion includes the motor cranial nerve nuclei.

203 ebellar granule and Purkinje cells, efferent cranial nerve nuclei; alpha2, beta2, and beta3-mRNAs not

204 lar granule and Purkinje cells, all efferent cranial nerve nuclei; alpha8-discrete neuronal, deep cor

205 nstem auditory associated nuclei and several cranial nerves nuclei, as well as in cerebellar Purkinje

206 There is inclusion formation in cranial nerve nucleus XII and in autonomic neurons of th

207 shown to inhibit sodium responses in the 7th cranial nerve of the rat.

208 urolymphomatosis (NL) is the infiltration of cranial nerves or nerves and roots from the peripheral n

209 y not attributable to direct polarization of cranial nerves or ventral roots.

210 screening whole brain and imaging brainstem, cranial nerves, orbits, cerebral cortex, mesial temporal

211 .3%), conjunctival microvasculopathy (2.3%), cranial nerve palsies (2%), herpes zoster ophthalmicus (

212 There were more cranial nerve palsies (22 vs 0) in the endarterectomy gr

213 mours; abnormal gait and coordination (78%), cranial nerve palsies (52%), pyramidal signs (33%), head

214 elayed neurological complications, including cranial nerve palsies and a unique delayed-onset parkins

215 Botulism manifests with cranial nerve palsies and flaccid paralysis in children

216 By excluding patients with third cranial nerve palsies and those with GCA, the incidence

217 ly benign causes of third, fourth, and sixth cranial nerve palsies in children, but a study from a te

218 f other causes for isolated fourth and sixth cranial nerve palsies was 4.7% (3/64).

219 NS3 disease (CSF WBC >= 5/muL with blasts or cranial nerve palsies, brain/eye involvement, or hypotha

220 l manifestations vary widely and can include cranial nerve palsies, meningeal involvement, parenchyma

221 is, etiology, work-up and treatment of third cranial nerve palsies, while incorporating information f

222 tral nervous system disorders and peripheral cranial nerve palsies.

223 f current neuro-imaging guidelines for third cranial nerve palsies.

224 l symptoms were painful radiculitis (65.9%), cranial nerve palsy (43.4%), and headache (28.3%).

225 = 2), small cerebral infarction (n = 2), and cranial nerve palsy (n = 1).

226 cedural MI (OR: 0.45; 95% CI: 0.27 to 0.75); cranial nerve palsy (OR: 0.07; 95% CI: 0.04 to 0.14); an

227 lvement (lymphoma cells in the CSF [n = 23], cranial nerve palsy [n = 9], both features [n = 4]), rep

228 days and up to 3 years; clinical outcome of cranial nerve palsy after PED placement; angiographic ev

229 ially avoiding the surgical complications of cranial nerve palsy and hematoma.

230 e composite outcome of death, stroke, MI, or cranial nerve palsy during the periprocedural period (OR

231 ) had local side effects that included third cranial nerve palsy in 6 (40%), orbital edema in 3 (20%)

232 lower incidence of myocardial infarction and cranial nerve palsy in patients undergoing stenting.

233 There was one event of cranial nerve palsy in the stenting group compared with

234 ed with lower rates of periprocedural MI and cranial nerve palsy than CEA.

235 all SF-36 domains, but periprocedural MI or cranial nerve palsy were not.

236 93.8%) completely recovered from symptoms of cranial nerve palsy within a median of 3.5 months.

237 s in an atraumatic sample or the presence of cranial nerve palsy) status.

238 in a nontraumatic sample or the presence of cranial nerve palsy) was present.

239 but a higher risk of myocardial infarction, cranial nerve palsy, and access site haematoma with enda

240 and 1 (0.2%) with orbital involvement and a cranial nerve palsy.

241 nts with acute SAH and 2 patients with acute cranial nerve palsy.

242 Additional PTC central nervous system and cranial nerve problems included peripheral visual field

243 tial for the proper organization of specific cranial nerve projections.

244 rity Score), and TBI-specific variables (eg, cranial nerve reflexes and findings from computed tomogr

245 implicated in cardiovascular regulation and cranial nerve reflexes, and gives rise to mossy fibers i

246 atory demyelination within the brainstem and cranial nerve roots during the progression of MS.

247 edominantly located in the brainstem and the cranial nerve roots in addition to the spinal cord and s

248 etyltransferase or retrogradely labeled from cranial nerve roots.

249 Large neurons in cranial nerve sensory ganglia were also labeled.

250 chemia is a frequent cause of acute isolated cranial nerve six (CN VI) palsy.

251 Here we report brain and cranial nerve soft-tissue preservation in Coccocephalus

252 Despite this hypertrophied cranial nerve, star-nosed mole brains were not larger th

253 d dysarthria were the most commonly reported cranial nerve symptoms in children with botulism; genera

254 The terminal nerve is an anterior cranial nerve that innervates the lamina propria of the

255 , tongue, pharynx, and larynx as well as the cranial nerves that control these structures.

256 In E10.5 embryos, deficiencies in cranial nerves that normally innervate oral structures,

257 The terminalis nerve (TN) is in a class of cranial nerves that plays important roles in animal deve

258 ain stem neurons that give rise to the tenth cranial nerve (the vagus).

259 Here, we report that stimulation of another cranial nerve, the trigeminal nerve, can also cause cort

260 routes of outflow of CSF in mice occur along cranial nerves to extracranial lymphatic vessels.

261 agent along both somatosensory and gustatory cranial nerves to the tongue and suggests subsequent syn

262 ox/flox) mice develop spinal, peripheral and cranial nerve tumors histologically identical to human s

263 20; 15%), leptomeninges (three of 20; 15%), cranial nerves (two of 20; 10%), and spinal nerves (two

264 ns of cranial nerves IX and X and defects in cranial nerve V were apparent in some Lmo4(-/-) and Lmo4

265 The trigeminal nerve (cranial nerve V), along with other cranial nerves, has i

266 ular motor cortex, brainstem motor nuclei of cranial nerves V, VII, and X-XII, and spinal cord alpha-

267 The formation of branchiomeric nerves (cranial nerves V, VII, IX and X) from their sensory, mot

268 the medial reticular formation and nuclei of cranial nerves V, VII, VIII, IX, and X.

269 ally manifest low-grade tumors affecting the cranial nerves (vestibular schwannomas), meninges (menin

270 asymmetric n=7), bulbar weakness (n=6), and cranial nerve VI (n=3) and VII (n=2) dysfunction.

271 ement disorder characterized by a failure of cranial nerve VI (the abducens nerve) to develop normall

272 y, 25 had cranial nerve IV palsy, and 62 had cranial nerve VI palsy.

273 ibuted into: 53.11 %, 36.36 %, and 2.8 % for cranial nerves VI, III, IV palsies respectively.

274 rbital approach to the preganglionic part of cranial nerve VII and to reveal its peripheral terminals

275 The radix autonomica cranial nerve VII was accessed via the tympanic cavity a

276 ary (cranial nerve III) and pterygopalatine (cranial nerve VII) ganglia, the latter consisting of a c

277 r axons innervating the distal forelimb, and cranial nerve VII.

278 n regions where the upper cervical roots and cranial nerves VII, IX and X project into the trigeminal

279 Cranial nerves VII, IX and X provide both gustatory (tas

280 h will form the Schwann cells lining several cranial nerves (VII, VIII and X).

281 cular reflex consisting of the otic vesicle, cranial nerve VIII and vestibular ganglia.

282 with gadolinium demonstrated enhancement of cranial nerve VIII bilaterally.

283 In addition, the development of cranial nerve VIII tumors correlates with functional imp

284 icits in hearing and balance associated with cranial nerve VIII tumors, would allow systematic evalua

285 Bilateral vestibular schwannomas, tumors on cranial nerve VIII, are pathognomonic for NF2 disease.

286 ed age-related differences in the density of cranial nerve VIII, with lower density in older adults w

287 ction of the right fifth, sixth, and seventh cranial nerves was diagnosed with severe chronic periodo

288 No defects in the sphenoid bone or cranial nerves were apparent.

289 Cranial nerves were involved in 9 of 48 patients (19%).

290 e was no evidence of sensory impairment, and cranial nerves were normal.

291 avily T2-weighted images in the plane of the cranial nerves were obtained in four subjects.

292 Effects on selected cranial nerves were similar to those on the phrenic nerv

293 finding was involvement of leptomeninges and cranial nerves, which manifested as abnormal enhancement

294 The nervus terminalis (NT) is a vertebrate cranial nerve whose function in adults is unknown.

295 The terminal nerve is a ganglionated cranial nerve with peripheral processes that enter the n

296 anial sensory ganglia, which coordinates the cranial nerves with the segments that they innervate.

297 microl of 0.5%) into the root of the eighth cranial nerve within the cranium caused rapid effects on

298 nd/or the transport of the prion agent along cranial nerves without agent replication.

299 /-), Hgf(-/-), and Met(-/-) mice with common cranial nerve XII (CNXII) outgrowth and myoblast migrati

300 videnced by a loss of the hypoglossal nerve (cranial nerve XII) in embryos from the 125 microg atRA/g