ライフサイエンスコーパス: crassa

1 ion in single cell oscillators of Neurospora crassa.

2 lulolytic gene expression and activity in N. crassa.

3 y initiation in the fungal model, Neurospora crassa.

4 and acts at the step of bilayer fusion in N. crassa.

5 cognate codons are used for initiation in N. crassa.

6 n in the model filamentous fungus Neurospora crassa.

7 r of lignocellulolytic gene expression in N. crassa.

8 growth in the filamentous fungus Neurospora crassa.

9 een characterized in detail using Neurospora crassa.

10 ation in the multicellular fungus Neurospora crassa.

11 emic species, Leavenworthia alabamica and L. crassa.

12 ulation in the filamentous fungus Neurospora crassa.

13 of self-compatibility in L. alabamica and L. crassa.

14 tide PAF26 using the model fungus Neurospora crassa.

15 ol repetitive selfish elements in Neurospora crassa.

16 esponses in the filamentous fungi Neurospora crassa.

17 ging of genetically engineered strains of N. crassa.

18 ll RNAs in the filamentous fungus Neurospora crassa.

19 mplexes in the filamentous fungus Neurospora crassa.

20 tation of blue-light responses in Neurospora crassa.

21 49 SSRs of 963 SSR types in the genome of N. crassa.

22 r HP1 localization and DNA methylation in N. crassa.

23 molecular markers for genetic studies in N. crassa.

24 otein from the filamentous fungus Neurospora crassa.

25 pathway in the filamentous fungus Neurospora crassa.

26 ulation of the filamentous fungus Neurospora crassa.

27 nation in the filamentous fungus, Neurospora crassa.

28 se chromosomes between N. tetrasperma and N. crassa.

29 e common in the ascomycete mould, Neurospora crassa.

30 point mutation (RIP) of repetitive DNA in N. crassa.

31 tant that is used in circadian studies in N. crassa.

32 f4 against F. graminearum but not against N. crassa.

33 ponent of the circadian system in Neurospora crassa.

34 nance of regular hyphal growth in Neurospora crassa.

35 ation of nitroethane catalyzed by Neurospora crassa 2-nitropropane dioxygenase was investigated by me

36 Catalytic turnover of Neurospora crassa 2-nitropropane dioxygenase with nitroethane as su

37 ating functional variation of proteins in N. crassa, 3) there are different levels of evolutionary fo

38 In Neurospora crassa, a circadian rhythm of conidiation (asexual spore

39 ast, the histone modifications in Neurospora crassa, a convenient model organism for multicellular eu

40 In Neurospora crassa, a eukaryotic model system for studying blue-ligh

41 ication in the filamentous fungus Neurospora crassa, a simple and experimentally amenable model syste

42 In Neurospora crassa, a single H3K9 methyltransferase complex, called

43 In Neurospora crassa, a transcription factor, WCC, activates the trans

44 that the minimal functional domain of the N. crassa AAP corresponded closely to the region that was m

45 ur understanding of the light response in N. crassa, about which the most is known, and will then jux

46 NcLPMO9C from Neurospora crassa acts both on cellulose and on non-cellulose beta-

47 log in the filamentous ascomycete Neurospora crassa affects the circadian clock output, yielding a pa

48 show that the powerful tools available in N. crassa allow for a comprehensive system level understand

49 (Chromista), and the ascomycetes Neurospora crassa and Aspergillus nidulans (Fungi), and bring to li

50 -stage over the last few decades--Neurospora crassa and Aspergillus nidulans.

51 data, we identified regulatory factors in N. crassa and characterized one (PDR-2) associated with pec

52 ulans, Aspergillus fumigatus, and Neurospora crassa and expressed the genes as secreted proteins with

53 We show that N. crassa and F. graminearum respond differently to MtDef4

54 pecific expression and editing in Neurospora crassa and F. verticillioides Furthermore,F. graminearum

55 s growth of the ascomycete fungi, Neurospora crassa and Fusarium graminearum, at micromolar concentra

56 Two species of Fungiidae corals, Ctenactis crassa and Herpolitha limax, displaying YBD-like lesions

57 lencing in the filamentous fungus Neurospora crassa and identified a bromo-adjacent homology (BAH)-pl

58 3K27me3 in the filamentous fungus Neurospora crassa and in other Neurospora species.

59 changes in gating the photic response of N. crassa and indicate that LOV-LOV homo- or heterodimeriza

60 te fungi Fusarium graminearum and Neurospora crassa and induces accumulation of reactive oxygen speci

61 The DEAD-box proteins CYT-19 in Neurospora crassa and Mss116p in Saccharomyces cerevisiae are broad

62 The DEAD-box proteins CYT-19 in Neurospora crassa and Mss116p in Saccharomyces cerevisiae are gener

63 starch-active PMOs from the fungi Neurospora crassa and Myceliophthora thermophila, NcAA13 and MtAA13

64 e functional predictions of novel genes in N.crassa and other filamentous ascomycete species.

65 However, the means by which N. crassa and other filamentous fungi sense the presence of

66 In Neurospora crassa and other filamentous fungi, light-dependent-spec

67 equence information available for Neurospora crassa and other fungi has greatly facilitated evolution

68 owing the divergence of S. cerevisiae and N. crassa and provides insight into the evolution of kinase

69 In Neurospora crassa and Saccharomyces cerevisiae, efficient splicing

70 In Neurospora crassa and Saccharomyces cerevisiae, the latter function

71 We then analyzed N. crassa and Schizosaccharomyces pombe telomerase reconsti

72 Recent work in Neurospora crassa and Sclerotinia sclerotiorum has illuminated how

73 er (PTC) function was analyzed in Neurospora crassa and wheat germ translation extracts using the tra

74 lly inoculated with the mycelium (Neurospora crassa), and following the initial incubation period, th

75 ulator of protoperithecial development in N. crassa, and double mutants carrying deletions of both vi

76 witches in the filamentous fungus Neurospora crassa, and found that one activates and two repress gen

77 e role of MAP kinase signaling in Neurospora crassa, and to identify downstream target genes of the p

78 mately 35% of genes marked by H3K27me3 in N. crassa are also H3K27me3-marked in Neurospora discreta a

79 aromyces cerevisiae and CYT-19 of Neurospora crassa are ATP-dependent helicases that function as gene

80 st eukaryotes, the centromeric regions of N. crassa are rich in sequences that are related to transpo

81 Filamentous fungi, such as Neurospora crassa, are very efficient in deconstructing plant bioma

82 The Neurospora crassa arg-2 uORF encodes the 24-residue arginine attenu

83 iae GCN4, S. cerevisiae CPA1, and Neurospora crassa arg-2, regulation by uORFs controls expression in

84 ng an in vivo tethering system in Neurospora crassa Artificial recruitment of the H3K9 methyltransfer

85 Here, we use Neurospora crassa as a model filamentous fungus to interrogate the

86 We have developed N. crassa as a model system where we can dissect the comple

87 Using the cellulolytic fungus Neurospora crassa as a model, we identified a xylodextrin transport

88 show that the P. syringae is able to use N. crassa as a sole nutrient source.

89 f LAD from the filamentous fungus Neurospora crassa at 2.6 A resolution.

90 ierarchy of initiation at start codons in N. crassa (AUG >> CUG > GUG > ACG > AUA approximately UUG >

91 In the lowly bread mould, Neurospora crassa, biomolecular reactions involving the white-colla

92 ungi, such as the model eukaryote Neurospora crassa, but is absent from the genomes of baker's yeast

93 , is needed for entry of this defensin in N. crassa, but not in F. graminearum.

94 T80 pathway is not involved in meiosis in N. crassa, but rather regulates the formation of female rep

95 l for light-mediated responses in Neurospora crassa, but the molecular mechanisms underlying gene ind

96 issues in the microbial eukaryote Neurospora crassa by using a "reverse-ecology" population genomic a

97 ture of the ring of c subunits in Neurospora crassa by using data from the crystal structure of the h

98 strial scale enzymes in the model system, N. crassa, by removing the endogenous negative feedback reg

99 l timing of the robust circadian clock in N. crassa can be disrupted in the dark when maintained in a

100 ticivorans oxygenase 2 (NOV2) and Neurospora crassa carotenoid oxygenase 1 (CAO1), using piceatannol

101 titution of NSP ubiquitylation in Neurospora crassa cell extracts.

102 In the filamentous fungus Neurospora crassa, cell fusion occurs during asexual spore germinat

103 Reconstitution of the N. crassa cellodextrin transport system in Saccharomyces ce

104 These data suggest that the N. crassa cellodextrin transporters act as "transceptors" w

105 ntified, including 10 of the 23 predicted N. crassa cellulases.

106 Here we identified Neurospora crassa centromeric DNA by chromatin immunoprecipitation

107 K [Osmotically Sensitive-2 (OS-2)] by the N. crassa circadian clock allows anticipation and preparati

108 omprehensive dynamic model of the Neurospora crassa circadian clock that incorporates its key compone

109 In the Neurospora crassa circadian clock, a protein complex of frequency (

110 The Neurospora crassa circadian negative element FREQUENCY (FRQ) exempl

111 Predicted key features of the N. crassa clock system are a dynamically frustrated closed

112 ble RNA and protein profiling data on the N. crassa clock.

113 Neurospora crassa colonizes burnt grasslands and metabolizes both c

114 Neurospora crassa colonizes burnt grasslands in the wild and metabo

115 dentified in one of the double mutants of N. crassa conferred resistance to both bafilomycin and conc

116 In Neurospora crassa, constitutive heterochromatin is characterized by

117 ase (CDH) isolated from the fungi Neurospora crassa, Corynascus thermophilus, and Myriococcum thermop

118 The Neurospora crassa CYT-18 protein is a mitochondrial tyrosyl-tRNA sy

119 Comparisons with previous N. crassa CYT-18 structures and a structural model of the A

120 howed that one of these proteins, Neurospora crassa CYT-18, binds group I introns by using both its N

121 The Neurospora crassa DEAD-box protein CYT-19 is a mitochondrial RNA ch

122 up I intron ribozyme by CYT-19, a Neurospora crassa DEAD-box protein that functions as a general chap

123 have generated mutant strains of Neurospora crassa defective in six subunits, C, H, a, c, c', and c'

124 t-induced point (RIP) mutation in Neurospora crassa degrades transposable elements by targeting repea

125 e of MEI3, the only RAD51/DMC1 protein in N. crassa, demonstrating independence from the canonical ho

126 the amino terminus that was unique to the N. crassa DGAT2 protein.

127 Two forms of N. crassa DGAT2 were tested: the predicted full-length prot

128 ree eukaryotic NR from the fungus Neurospora crassa, documenting that Moco is necessary and sufficien

129 rom a favored carbon source to cellulose, N. crassa dramatically up-regulates expression and secretio

130 rbon source such as sucrose to cellulose, N. crassa dramatically upregulates expression and secretion

131 a tritici, Magnaporthe oryzae and Neurospora crassa, exhibited PAMP activity, inducing cell death in

132 ed from one of its natural hosts, Neurospora crassa, exists in a multimeric form and has the ability

133 Using N. crassa expressing the Ca(2+) reporter aequorin, MsDef1,

134 Notably, the only other NMO from Neurospora crassa for which biochemical evidence is available lacks

135 e hydrophobin EAS from the fungus Neurospora crassa forms functional amyloid fibrils called rodlets t

136 Mutation or deletion of the N. crassa gene encoding subunit c' did not completely elimi

137 The protein encoded by the N. crassa gene was longer than that of U. ramanniana.

138 nication and fusion in the fungus Neurospora crassa Genetically identical germinating spores of this

139 In the filamentous fungus Neurospora crassa, genetically identical asexual spores (germlings)

140 o initiation of this project, the Neurospora crassa genome assembly contained only 3 of the 14 telome

141 istributions of the SSRs in the sequenced N. crassa genome differ systematically between chromosomes

142 H3K27me3 covers 6.8% of the N. crassa genome, encompassing 223 domains, including 774 g

143 hich account for 71% of total SSRs in the N. crassa genome, using a Poisson log-linear model.

144 In N. crassa, germinating asexual spores (germlings) of identi

145 Thus, N. crassa germlings undergoing chemotropic interactions rap

146 as identified from expression analysis of N. crassa grown on pure cellulose.

147 ssion data, the secretome associated with N. crassa growth on Miscanthus and cellulose was determined

148 N. crassa H3K27me3-marked genes are less conserved than unm

149 In the fungus Neurospora crassa, H3K9me3 and 5mC are catalyzed, respectively, by

150 We found that L. alabamica and L. crassa had high species-level genetic diversity (H(e)=0.

151 Unlike S. cerevisiae, N. crassa has a single isoform of the a subunit.

152 Neurospora crassa has been a model organism for the study of circad

153 Neurospora crassa has been for decades a principal model for filame

154 The filamentous fungus Neurospora crassa has been shown to be missing homologs of a number

155 Neurospora crassa has been utilized as a model organism for studyin

156 The eukaryotic filamentous fungus Neurospora crassa has proven to be a dependable model system for th

157 Neurospora crassa has the highest mutation rate and mutational burd

158 l other than the model ascomycete Neurospora crassa--has been neglected, leaving this type of questio

159 nisms in Aspergillus nidulans and Neurospora crassa have been intensively studied, leading to importa

160 nd function of DNA methylation in Neurospora crassa have led to a greater understanding of heterochro

161 Genetics studies of Neurospora crassa have revealed that a DNA methyltransferase (DIM-2

162 pombe and the filamentous fungus Neurospora crassa have served as important model systems for RNAi r

163 ponent of the quelling pathway in Neurospora crassa, have rapidly diverged in evolution at the amino

164 ted mutants of each of the four classical N. crassa HDAC genes and tested their effect on histone ace

165 A locus DNA methylation (DLDM) in Neurospora crassa Here we show that the conserved exonuclease ERI-1

166 tion experiments, a heterocomplex between N. crassa HET-C1 and PhcA was associated with phcA-induced

167 In the filamentous fungus Neurospora crassa, HET-C regulates a conserved programmed cell deat

168 We further evaluated the role of the N. crassa homolog of IME2, a kinase involved in initiation

169 urth component, Neurospora protein 55 (an N. crassa homolog of p55/RbAp48), is critical for H3K27me3

170 24 proteins, whereas CBH-2 depends on the N. crassa homolog of yeast Erv29p.

171 s able to attach and extensively colonize N. crassa hyphae, while an Escherichia coli control showed

172 d time-lapse live-cell imaging of Neurospora crassa in microfluidic environments to show how constrai

173 stal structures of an enzyme from Neurospora crassa in the resting state and of a copper(II) dioxo in

174 ined by translating mRNAs in a homologous N. crassa in vitro translation system or in rabbit reticulo

175 on deletion of Puf4 in filamentous fungi (N. crassa) in contrast to the increase upon Puf3 deletion i

176 c screen of the ascomycete fungus Neurospora crassa, in which dynein is nonessential.

177 Ectopic expression of phcA in N. crassa induced HI and cell death that was dependent on t

178 We show that osmotic stress in N. crassa induced the phosphorylation of a eukaryotic elong

179 ning microscopy that a LPMO (from Neurospora crassa) introduces carboxyl groups primarily in surface-

180 The filamentous fungus Neurospora crassa is a model laboratory organism, but in nature is

181 The eukaryotic model organism Neurospora crassa is an excellent system to study evolution and bio

182 This hyphal type in Neurospora crassa is being used as a model for studies on hyphal se

183 The model filamentous fungus Neurospora crassa is capable of utilizing a variety of carbohydrate

184 Internalization of MtDef4 in N. crassa is energy-dependent and involves endocytosis.

185 romatin in the filamentous fungus Neurospora crassa is marked by cytosine methylation directed by tri

186 The fluffy (fl) gene of Neurospora crassa is required for asexual sporulation and encodes a

187 tion/translation feedback loop in Neurospora crassa is the protein FREQUENCY (FRQ), shown here shown

188 N. crassa is typically found on woody biomass and is common

189 A model filamentous fungus, Neurospora crassa, is a multinucleate system used to elucidate mole

190 -4 (PRD-4), the CHK-2 ortholog of Neurospora crassa, is part of a signaling pathway that is activated

191 Using a population of 110 wild N. crassa isolates, we investigated germling fusion between

192 ted a number of site-directed variants of N. crassa LAD that are capable of utilizing NADP(+) as cofa

193 ils of rosette leaves, has shown that the L. crassa LFY ortholog, LcrLFY, rescues most aspects of flo

194 In Neurospora crassa, light promotes the interaction of WCCs and their

195 The bifunctional Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (CYT-18 pro

196 cture of a C-terminally truncated Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (CYT-18 pro

197 The Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (mtTyrRS; C

198 One such protein, the Neurospora crassa mitochondrial tyrosyl-tRNA synthetase (TyrRS; CYT

199 A N. crassa mutant carrying deletions for both transporters i

200 Previously, we have shown that a N. crassa mutant carrying deletions of three beta-glucosida

201 Here, we show that an N. crassa mutant carrying deletions of two genes encoding e

202 the Schizosaccharomyces pombe and Neurospora crassa Nbp2p orthologues and the high conservation of th

203 rresponding dicer-like genes from Neurospora crassa [Ncdcl-1 (50.5%); Ncdcl-2 (38.0%)] and Magnaporth

204 ity of a fast fungal kinesin from Neurospora crassa (NcKin).

205 family AA9 LPMOs from the fungus Neurospora crassa, NcLPMO9A (NCU02240), NcLPMO9C (NCU02916), and Nc

206 e have characterized an LPMO from Neurospora crassa (NcLPMO9C; also known as NCU02916 and NcGH61-3).

207 We demonstrate that an LPMO from Neurospora crassa, NcLPMO9C, indeed degrades various hemicelluloses

208 In Neurospora crassa, negative feedback is executed by a complex of Fr

209 s, such as BarA and TorS; and the Neurospora crassa Nik-1 (Os-1) sensor that contains a tandem array

210 fied, which may implicate mitochondria in N. crassa nonself recognition and PCD.

211 CdCl2 was contacted with supernatants of N. crassa obtained after growth in urea-containing medium.

212 associated DNA (RAD) mapping for use with N. crassa oligonucleotide microarrays.

213 we performed transcriptional profiling of N. crassa on 40 different carbon sources, including plant b

214 In this study, we show that the Neurospora crassa osmosensing MAPK pathway, essential for osmotic s

215 ing the model filamentous fungus, Neurospora crassa, our microfluidic system enabled direct visualiza

216 In Neurospora crassa, pairing of homologous DNA segments is monitored

217 es demonstrated that PAS is not unique to N. crassa PAS homologs likely influence the distribution of

218 on factor, is clock controlled in Neurospora crassa, peaking during the subjective day.

219 small modules engineered from the Neurospora crassa photoreceptor Vivid by orthogonalizing the homodi

220 The Neurospora crassa photoreceptor Vivid tunes blue-light responses an

221 The filamentous fungus Neurospora crassa played a central role in the development of twent

222 the nonreducing end product formed by an N. crassa PMO is a 4-ketoaldose.

223 ymorphic and fall into two haplogroups in N. crassa populations.

224 alyses of purified QDE-1 polymerases from N. crassa (QDE-1(Ncr)) and related fungi, Thielavia terrest

225 ously determined apo structure of Neurospora crassa QDE2 revealed that the PIWI domain has two subdom

226 spores in the filamentous fungus Neurospora crassa rcd-1 alleles are highly polymorphic and fall int

227 hat the model cellulolytic fungus Neurospora crassa relies on a high-affinity cellodextrin transport

228 Using Neurospora crassa repeat-induced point mutation (RIP) as a model sy

229 how the model filamentous fungus Neurospora crassa responds to the three main cell wall polysacchari

230 the introns of the model organism Neurospora crassa revealed a different organization at the 3' end o

231 three eukaryotic model organisms, Neurospora crassa, Saccharomyces cerevisiae, and Candida albicans,

232 The excellent fit of the N. crassa sequence to the E. hirae structure and the degree

233 crystalline precipitates on the hyphae of N. crassa showed that the main elements present in the crys

234 mponent of the circadian clock in Neurospora crassa, shows daily cycles that are exquisitely sensitiv

235 Neurospora crassa sports features of heterochromatin found in highe

236 y in yeast, and we also characterized the N. crassa STE12 homolog pp-1.

237 cts derived from MacroD-deficient Neurospora crassa strain exhibit a major reduction in the ability t

238 isms (SNPs) between the reference Neurospora crassa strain Oak Ridge and the Mauriceville strain (FGS

239 ur algorithm on a real dataset of Neurospora crassa strains, using the genetic and geographic distanc

240 e restriction site polymorphisms from two N. crassa strains: Mauriceville and Oak Ridge.

241 erived from S. cerevisiae OXA1 or Neurospora crassa SU9, both coding for hydrophobic mitochondrial pr

242 d the related protein Cyt-19 from Neurospora crassa suggest that these proteins form a subclass of DE

243 Studies with the Neurospora crassa synthetase (CYT-18 protein) showed that splicing

244 analysis affirmed that the reconstituted N. crassa telomerase synthesizes TTAGGG repeats with high p

245 The unique N. crassa TER 5'-splice site sequence is evolutionarily con

246 the canonical 5'-splice site GUAUGU, the N. crassa TER intron contains a non-canonical 5'-splice sit

247 t molecular coevolution of LcrLFY and the L. crassa TFL1 ortholog, LcrTFL1, contributed to the evolut

248 al new PMO families in the fungus Neurospora crassa that are likely to be active on novel substrates.

249 ation by IME-2 of a cell death pathway in N. crassa that functions in concert with the VIB-1 cell dea

250 TER 3'-end cleavage mechanism in Neurospora crassa that is distinct from that found specifically in

251 mponent of the circadian clock of Neurospora crassa that regulates the abundance of its core transcri

252 In Neurospora crassa, the circadian clock generates daily rhythms in t

253 m graminicola, the model organism Neurospora crassa, the human pathogen Sporothrix schenckii, and the

254 In the filamentous fungus Neurospora crassa, the IME2 homolog (ime-2) is not required for mei

255 In Neurospora crassa, the interactions between products of the frequen

256 Among natural accessions of Neurospora crassa, there is significant variation in clock phenotyp

257 ated to transposable elements; however, in N crassa these sequences have been heavily mutated.

258 In Neurospora crassa, three allelic specificities at the het-c locus a

259 ombe, and one filamentous fungus, Neurospora crassa-three species that arguably are not representativ

260 h wild-type and mutant strains of Neurospora crassa to gain insight into the role of heterochromatin

261 used the model filamentous fungus Neurospora crassa to search for uncharacterized transcription facto

262 he NPS6 ortholog from the saprobe Neurospora crassa to the Deltanps6 strain of C. heterostrophus rest

263 and constitutive heterochromatin, Neurospora crassa, to explore possible interactions between element

264 A screen of a N. crassa transcription factor deletion collection identifi

265 methods refined our understanding of the N. crassa transcriptional response to cellulose and demonst

266 Analyses of the 5'-leader regions in the N. crassa transcriptome revealed examples of highly conserv

267 Here, we show that in N. crassa, two cellodextrin transporters, CDT-1 and CDT-2,

268 is suggests that the widespread and basal N. crassa-type spliceosomal cleavage mechanism is more ance

269 meiosis in the filamentous fungus Neurospora crassa, unpaired genes are identified and silenced by a

270 In Neurospora crassa, unpaired genes are silenced by a mechanism calle

271 ecular Cell, Lee et al. show that Neuropsora crassa uses several Dicer-dependent and -independent pat

272 his system, derived from genes in Neurospora crassa, uses the transcriptional activator QF to induce

273 We initially identified TER from Neurospora crassa using a novel deep-sequencing-based approach, and

274 have successfully applied BiFC in Neurospora crassa using two genes involved in meiotic silencing by

275 Neurospora crassa utilizes DNA methylation to inhibit transcription

276 In Neurospora crassa, VIVID (VVD), a small LOV domain containing blue-

277 r L. alabamica was 0.065 vs 0.206 and for L. crassa was 0.084 vs 0.189).

278 2 gene encoding for the enzyme in Neurospora crassa was cloned, expressed in Escherichia coli, and th

279 The complex from Neurospora crassa was composed of Tob55-Sam50, Tob38-Sam35, and Tob

280 s that showed expression differences when N. crassa was cultured on ground Miscanthus stems as a sole

281 heterologous expression method in Neurospora crassa was developed as a step toward connecting regiose

282 earch, the urease-positive fungus Neurospora crassa was investigated for the biomineralization of cal

283 eotide (nt) SSRs, the most common SSRs in N. crassa, was significantly biased in exons.

284 d glucose homeostatic process, in Neurospora crassa We find that glycogen synthase (gsn) mRNA, glycog

285 k in the circadian model organism Neurospora crassa We show that, in a ras2-deficient strain, the per

286 p, H3K9me, and DNA methylation in Neurospora crassa, we built and tested mutants of the putative H3S1

287 ic approach in the model organism Neurospora crassa, we identified two alleles of a gene, NCU04278, e

288 nt in the model ascomycete fungus Neurospora crassa, we show that genetic diversity is maintained by

289 Different PMOs isolated from Neurospora crassa were found to generate oxidized cellodextrins mod

290 merly Mortierella) ramanniana and Neurospora crassa were introduced into maize using an embryo-enhanc

291 while maintaining light responsiveness in N. crassa when held in a steady metabolic state using biore

292 ly active in the meiotic cells of Neurospora crassa, where they evaluate the mutual identity of homol

293 nctional genomics resources available for N. crassa, which include a near-full genome deletion strain

294 is study, we employed LPMO9C from Neurospora crassa, which is active toward cellulose and soluble bet

295 n by a fungal TPP riboswitch from Neurospora crassa, which is mostly located in a large intron separa

296 Previous research with Leavenworthia crassa, which produces solitary flowers in the axils of

297 he most is known, and will then juxtapose N. crassa with A. nidulans, which, as will be described bel

298 1 (HDA1) mutant (hda-1) strain of Neurospora crassa with inactivated histone deacetylase 1.

299 cells of the model fungal system, Neurospora crassa, with droplet microfluidics and the use of a fluo

300 quation for the Caribbean sponge Aiolochroia crassa yielded age estimates of 5.2-10.4 y for the large