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1 n the activity of creatine phosphokinase and creatine kinase-MB.
2 measured by ECG, echocardiography and plasma creatine kinase-MB.
3  models and included haptoglobin, IL-10, and creatine kinase-MB.
4 utcome information as a cutoff of 5x ULN for creatine kinase-MB.
5 43; P=0.003 and hazard ratio per doubling of creatine kinase-MB, 1.30; 95% confidence interval, 1.05-
6 ocardial infarction defined by enzymes (peak creatine kinase-MB 173+/-119 U/L) were scanned twice by
7 re defined: MMS-1 (all 3 markers) and MMS-2 (creatine kinase-MB and troponin I only).
8                    No significant changes in creatine kinase-MB and troponin-I were seen.
9        Men were more likely to have elevated creatine kinase-MB and troponins, whereas women were mor
10 function, and perfusion), injury biomarkers (creatine-kinase-MB and troponin I), and histopathologic
11                                   Myoglobin, creatine kinase-MB, and troponin I were measured at 0, 3
12                   Infarct size determined by creatine kinase-MB area under the curve was the primary
13                              Infarct size by creatine kinase-MB area under the curve, the primary out
14 rocedural MI, the primary MI definition used creatine kinase-MB as the preferred biomarker, whereas t
15 rocedural MI, the primary MI definition used creatine kinase-MB as the preferred biomarker, whereas t
16 activity but reduced neither infarct size by creatine kinase-MB assessment nor adverse clinical outco
17  similar or lower in HBOC than HEX pigs, but creatine kinase-MB (but not creatine kinase-MB/creatine
18 of serum levels of cardiac troponin T, serum creatine kinase MB (CK-MB) levels, and electrocardiograp
19  cell count (WBC), C-reactive protein (CRP), creatine kinase MB (CK-MB), and troponin I levels were m
20                                        Serum creatine kinase-MB (CK-MB) and cardiac troponin T (cTnT)
21                                              Creatine kinase-MB (CK-MB) and cardiac troponins (cTn) a
22                           Mild elevations in creatine kinase-MB (CK-MB) are common after successful p
23 cally evident complications to elevations of creatine kinase-MB (CK-MB) enzyme levels during percutan
24       The role of serum troponin T (TnT) and creatine kinase-MB (CK-MB) for the risk stratification o
25 nts with unstable angina were determined for creatine kinase-MB (CK-MB) subforms, myoglobin, total CK
26 ditis Treatment Trial and were compared with creatine kinase-MB (CK-MB) values measured in the same p
27 rs including troponin I or T (cTnI or cTnT), creatine kinase-MB (CK-MB), and myoglobin.
28 (TnI), B-type natriuretic peptide (BNP), and creatine kinase-MB (CK-MB), and TnI and BNP by CART.
29 MI (PMI(Prot)) required a large elevation of creatine kinase-MB (CK-MB), with identical threshold for
30 e diagnostic performance of serum myoglobin, creatine-kinase-MB (CK-MB) and cardiac troponin-I (cTnI)
31 an HEX pigs, but creatine kinase-MB (but not creatine kinase-MB/creatine kinase ratio) was higher wit
32                          Evidence of MI with creatine kinase-MB elevation after PCI was seen in 36% o
33 l-resistant patients had higher incidence of creatine kinase-MB elevation than the respective sensiti
34 revious 24 hours and ischemic ECG changes or creatine kinase-MB elevation were eligible for enrollmen
35             Slow flow was observed in 3% and creatine kinase-MB enzyme elevation >3x normal occurred
36 ity C-reactive protein (hs-CRP), Troponin-T, creatine kinase-MB, fibrinogen, and D-Dimer concentratio
37                           Elevation of serum creatine kinase MB fraction (CK-MB) after percutaneous c
38 sed with increasing levels of periprocedural creatine kinase MB fraction elevation.
39 ons, with no deaths, myocardial infarctions (creatine kinase, MB fraction > 50 IU/liter) or emergent
40 rdial infarction were identified by elevated creatine kinase, MB fraction (CK-MB) and one of the foll
41          Serum markers such as myoglobin and creatine kinase, MB fraction (CK-MB) are effective in de
42 d the predictive properties of P-selectin to creatine kinase, MB fraction (CK-MB) for detecting acute
43 easurements of cardiac troponin I (cTnI) and creatine kinase, MB fraction (CK-MB) levels before, imme
44 n acute coronary syndrome (ACS) and negative creatine kinase, MB fraction (CK-MB) levels.
45  were triaged to a chest pain unit, cTnT and creatine kinase, MB fraction (CK-MB) were evaluated > or
46                                              Creatine kinase, MB fraction had a higher specificity fo
47                                              Creatine kinase, MB fraction values were determined usin
48                                     cTnI and creatine kinase-MB fraction (CK-MB) mass concentration w
49 ungstate significantly decreased circulating creatine kinase-MB fraction activity after hepatoenteric
50                         Finally, circulating creatine kinase-MB fraction activity was significantly a
51  injury, manifested by increased circulating creatine kinase-MB fraction activity, was significantly
52                          Cardiac biomarkers (creatine kinase-MB fraction or troponin I) were elevated
53  integrated into clinical algorithms such as creatine kinase-MB fraction or troponin testing for acut
54 e cardiac events, post-PCI peak troponin and creatine kinase-MB fraction, and a longer length of stay
55 fore presentation, and elevation in baseline creatine kinase-MB fraction.
56 dural non-Q-wave myocardial infarction (MI) (creatine kinase-MB > or =5 times normal) was notably hig
57  graft surgery [2.8% versus 3.3%]), although creatine kinase-MB >3X normal was more common with DCA (
58 similar information as a value of 5x ULN for creatine kinase-MB (hazard ratio, 4.31; 99% confidence i
59       We measured troponin T, troponin I and creatine kinase MB in 51 patients presenting with suspec
60 00 fg/ml troponin T, creatine kinase MM, and creatine kinase MB in serum.
61                     Elevated serum levels of creatine kinase (MB isoenzyme), troponin I, and troponin
62 ved hemodynamics and decreased the levels of creatine kinase, MB isoenzyme of creatine kinase, blood
63 dy evaluated the incidence and predictors of creatine kinase-MB isoenzyme (CK-MB) elevation after suc
64  the impact of aggressive stent expansion on creatine kinase-MB isoenzyme (CK-MB) release and clinica
65                                              Creatine kinase-MB isoenzyme elevation occurred in 25.6%
66                           PMI was defined as creatine kinase-MB isoenzyme level > or = 10x upper limi
67 re hemodynamic deterioration, preangiography creatine kinase-MB isoenzyme rise >2 x normal, and time
68                                       Plasma creatine kinase (MB isoform), indicating myocardial inju
69                                              Creatine kinase MB isoform elevation after revasculariza
70 eroxidase levels, in contrast to troponin T, creatine kinase MB isoform, and C-reactive protein level
71  study was to assess the long-term impact of creatine kinase-MB isoform (CK-MB) elevation after percu
72                                              Creatine kinase-MB isoform elevation only above 10 x ULN
73 (cardiac troponin I level > or =0.7 ng/mL or creatine kinase-MB level > or =5.0 ng/mL and/or diagnost
74 rdial infarction was classified according to creatine kinase-MB level as type 1 (>1 but <3 times norm
75  images provided equal performance, and peak creatine kinase-MB levels correlated with MRI infarct si
76                                              Creatine kinase-MB levels were measured after the proced
77  combined with select clinical variables and creatine kinase-MB levels, enhances the noninvasive pred
78                      Postprocedural cTnT and creatine kinase-MB mass levels (ULN, 6.7 ng/mL in men an
79                                              Creatine kinase MB, MB isoforms, and troponins I and T w
80 tive model containing clinical variables and creatine kinase-MB measures, its contribution remained s
81                     There was no increase in creatine kinase-MB or troponin associated with the adven
82                     Fewer women had elevated creatine kinase-MB or troponins, whereas more had elevat
83  thickening score, was not predicted by peak creatine kinase-MB (P=0.66) or by total infarct size, as
84                               Troponin-T and creatine kinase-MB peaked at day 1 after procedure (both
85 re was a 40% reduction in myocardial injury (creatine kinase-MB release, P=0.05) in patients who rece
86                                   Myoglobin, creatine kinase-MB subfraction (CK-MB) and troponin I (c
87 and serial myocardial marker measurements of creatine kinase-MB, total creatine kinase activity, and
88                  Maximum creatine kinase and creatine kinase-MB values declined (5.2% and 7.6%; P<0.0
89 asures with models that include clinical and creatine kinase-MB variables.
90                           In 3 patients, the creatine kinase-MB was not measured during the hospital