ライフサイエンスコーパス: crescentic glomerulonephritis

1 e as the pre-emptive treatment in abrogating crescentic glomerulonephritis.

2 kade by nutlin-3a ameliorated all aspects of crescentic glomerulonephritis.

3 izations were needed to induce a necrotizing/crescentic glomerulonephritis.

4 autoimmune glomerulonephritis or necrotizing/crescentic glomerulonephritis.

5 corticoids, renal function in a rat model of crescentic glomerulonephritis.

6 sion of crescentic lesions in a patient with crescentic glomerulonephritis.

7 iquely susceptible to experimentally induced crescentic glomerulonephritis.

8 elerating glomerulosclerosis in experimental crescentic glomerulonephritis.

9 (PEC) are major constituents of crescents in crescentic glomerulonephritis.

10 oto (WKY) rat shows marked susceptibility to crescentic glomerulonephritis.

11 of renal injury in an experimental model of crescentic glomerulonephritis.

12 s approach is effective in the prevention of crescentic glomerulonephritis.

13 proaches may be worth investigating in human crescentic glomerulonephritis.

14 ng the kidneys, where they cause necrotizing crescentic glomerulonephritis.

15 amined the effect of an antibody to VLA-1 in crescentic glomerulonephritis.

16 ll type targeted in ANCA-induced necrotizing crescentic glomerulonephritis.

17 was associated with a poor outcome in human crescentic glomerulonephritis.

18 CR1 also promoted influx of DC precursors in crescentic glomerulonephritis, a DC-dependent aggressive

19 ronounced in females, which developed severe crescentic glomerulonephritis accompanied by heavy prote

20 microscopic polyangiitis or necrotizing and crescentic glomerulonephritis achieve remission with the

21 apsing glomerulopathy variant), pauci-immune crescentic glomerulonephritis, acute interstitial nephri

22 city of Th17 cells in a mouse model of acute crescentic glomerulonephritis and in a mouse chronic mod

23 dose of 1600 microg/kg induced pauci-immune crescentic glomerulonephritis and lung hemorrhage in all

24 400 microg/kg of myeloperoxidase resulted in crescentic glomerulonephritis and lung hemorrhage in all

25 Crescentic glomerulonephritis and lung hemorrhage were M

26 s model so that all animals reliably develop crescentic glomerulonephritis and lung hemorrhage.

27 f patients with pauci-immune necrotizing and crescentic glomerulonephritis and systemic small vessel

28 both glomerular and interstitial fibrosis in crescentic glomerulonephritis and that neutralization of

29 atment experiment, all rats developed florid crescentic glomerulonephritis, and control rats showed m

30 li were also noted in amyloidosis, diabetes, crescentic glomerulonephritis, and diffuse endocapillary

31 604Glyfs*2), recurrent bacterial infections, crescentic glomerulonephritis, and end-stage renal failu

32 ome, idiopathic pauci-immune necrotizing and crescentic glomerulonephritis, and related and overlappi

33 With the emergence of necrotizing/crescentic glomerulonephritis, approximately 0.15% of re

34 microscopic polyangiitis and necrotizing and crescentic glomerulonephritis associated with antineutro

35 l, illustrate that TLR4 stimulation triggers crescentic glomerulonephritis by effects on both the ada

36 the role of SPARC in mediating experimental crescentic glomerulonephritis by inducing passive nephro

37 We hypothesized that the MDM2 would drive crescentic glomerulonephritis by NF-kappaB-dependent glo

38 onstrated to be involved in animal models of crescentic glomerulonephritis (cGN) and are potential ta

39 ocal segmental glomerulosclerosis (FSGS) and crescentic glomerulonephritis (CGN) remain poorly unders

40 Crescentic glomerulonephritis (Crgn) is a complex diseas

41 Crescentic glomerulonephritis (Crgn) is a complex disord

42 ion is effective in reducing the severity of crescentic glomerulonephritis even when treatment is sta

43 Treatment of acute, crescentic glomerulonephritis (GN) consists of unspecifi

44 Crescentic glomerulonephritis (GN) is a devastating dise

45 and Th17 cells are also the main drivers of crescentic glomerulonephritis (GN), the most severe form

46 phages to progressive renal injury in murine crescentic glomerulonephritis (GN).

47 splenocytes developed severe necrotizing and crescentic glomerulonephritis, granulomatous inflammatio

48 Excluding pauci-immune necrotizing and crescentic glomerulonephritis, IgG4 immunohistochemistry

49 Moreover, in patients with crescentic glomerulonephritis, IL-26 is expressed by ren

50 early along the mouse GBM in vivo, eliciting crescentic glomerulonephritis in Fcgr2b(-/-) mice suscep

51 and the deterioration of kidney function in crescentic glomerulonephritis in mice.

52 ty of collagen-induced arthritis in mice and crescentic glomerulonephritis in rats, in part by decrea

53 everal susceptibility loci, Crgn1-Crgn7, for crescentic glomerulonephritis in the Wistar Kyoto (WKY)

54 ndance resulting from experimentally induced crescentic glomerulonephritis in these rats.

55 Inducing crescentic glomerulonephritis in two Pax2Cre reporter mo

56 The residual genetic susceptibility to crescentic glomerulonephritis in WKY.LCrgn1,2 rats assoc

57 ANCA-positive patients with necrotizing and crescentic glomerulonephritis, including 69 with evidenc

58 Crescentic glomerulonephritis is an important cause of h

59 Crescentic glomerulonephritis is characterized by vascul

60 he migration of leukocytes into glomeruli in crescentic glomerulonephritis is fundamental to pathogen

61 e's syndrome (GPS) develop severe autoimmune crescentic glomerulonephritis, kidney failure, and lung

62 murine anti-MPO Abs derived from Spontaneous Crescentic Glomerulonephritis/Kinjoh mice.

63 Concomitant crescentic glomerulonephritis led to an initial mistaken

64 These azotemic EGF-deficient mice developed crescentic glomerulonephritis linked to HB-EGF/EGFR hype

65 that ANCA cause pauci-immune necrotizing and crescentic glomerulonephritis (NCGN) and systemic small

66 evidence that MPO-ANCA cause necrotizing and crescentic glomerulonephritis (NCGN) and vasculitis.

67 Necrotizing and crescentic glomerulonephritis (NCGN) occurs in all syndr

68 elucidated ANCA-IgG induced necrotizing and crescentic glomerulonephritis (NCGN), pathogenesis of AN

69 development of pauci-immune necrotizing and crescentic glomerulonephritis (NCGN).

70 o ANCA-associated vasculitis and necrotizing crescentic glomerulonephritis (NCGN).

71 kidney and lung basement membranes, neither crescentic glomerulonephritis nor alveolitis ensued, lik

72 Pauci-immune crescentic glomerulonephritis (PICGN) is a major cause o

73 WKY) rat strain, we have identified multiple crescentic glomerulonephritis QTL (Crgn) and positionall

74 or and found increased life span, suppressed crescentic glomerulonephritis, reduced spleen size, and

75 ly progressive glomerulonephritis/idiopathic crescentic glomerulonephritis (RPGN/ICG), IgA nephropath

76 Kidney biopsy revealed necrotizing and crescentic glomerulonephritis, similar to primary ANCA-G

77 MC1R null-mutant mice developed more severe crescentic glomerulonephritis than wild-type mice, marke

78 plenocytes into immune-deficient mice caused crescentic glomerulonephritis that could be completely b

79 Ranging from autoimmune-induced crescentic glomerulonephritis to HIV infection-induced c

80 toplasmic antibody (ANCA) vasculitis-induced crescentic glomerulonephritis, to identify common diseas

81 In these mice, crescentic glomerulonephritis was induced with a previou

82 Renal biopsy demonstrated a pauci-immune crescentic glomerulonephritis with 14 cellular crescents

83 control IgG developed focal necrotizing and crescentic glomerulonephritis with a paucity of glomerul

84 inin-521 in female Wistar-Kyoto rats induced crescentic glomerulonephritis with linear IgG deposits a

85 ematopoietic stem cell disorders, attenuated crescentic glomerulonephritis with recovery of kidney fu