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1  EAL domain protein (CC3396 from Caulobacter crescentus).
2 bacter actinomycetemcomitans and Caulobacter crescentus.
3 ll division protein FtsZ in live Caulobacter crescentus.
4 e ParB/parS partition complex in Caulobacter crescentus.
5  focused on the curved bacterium Caulobacter crescentus.
6  asymmetrically is the bacterium Caulobacter crescentus.
7 ulons of CtrA and DnaA in S. meliloti and C. crescentus.
8 transcriptionally cell cycle-regulated in C. crescentus.
9 roteobacterial species closely related to C. crescentus.
10 absence of polar localization in Caulobacter crescentus.
11 efaciens are vertically-integrated, as in C. crescentus.
12  SocAB, an atypical TA system in Caulobacter crescentus.
13 al for cell cycle progression in Caulobacter crescentus.
14 alization in the model bacterium Caulobacter crescentus.
15 rates of the ClpP associated proteases in C. crescentus.
16 kinases called pdhS1 and pdhS2, absent in C. crescentus.
17 tpX, a stalk-specific protein in Caulobacter crescentus.
18 ly regulates Z ring formation in Caulobacter crescentus.
19 ght temporal control of the cell cycle in C. crescentus.
20 related diguanylate cyclase from Caulobacter crescentus.
21 ional investigation of PerB from Caulobacter crescentus.
22 oordinately regulate stress physiology in C. crescentus.
23 gainst its phylogenetic relative Caulobacter crescentus.
24 sis and cell cycle regulation in Caulobacter crescentus.
25 gent response in the oligotroph, Caulobacter crescentus.
26 symmetrically dividing bacterium Caulobacter crescentus.
27 located between lpxA and lpxB in Caulobacter crescentus.
28 ith cytokinesis in the bacterium Caulobacter crescentus.
29  that influence FtsZ function in Caulobacter crescentus.
30 les in the alpha-proteobacterium Caulobacter crescentus.
31  member of this family (DipM) in Caulobacter crescentus.
32 00 cell cycle-regulated genes in Caulobacter crescentus.
33  drive cell cycle progression in Caulobacter crescentus.
34 herichia coli based on PopZ from Caulobacter crescentus.
35 imorphic Gram-negative bacterium Caulobacter crescentus.
36 AimB with MreB conformational dynamics in C. crescentus.
37 stigated the function of ZapA in Caulobacter crescentus.
38 hia coli, Bacillus subtilis, and Caulobacter crescentus.
39  that control peptidoglycan remodeling in C. crescentus.
40 n adder, as has been proposed in Caulobacter crescentus.
41 Z curvature and efficient constriction in C. crescentus.
42 including chromosomal regions in Caulobacter crescentus.
43 hich infect freshwater bacterium Caulobacter crescentus.
44 n the asymmetric model bacterium Caulobacter crescentus.
45 hways lead to persister cell formation in C. crescentus.
46 ture of a bactofilin domain from Caulobacter crescentus.
47 chia coli, Bacillus subtilis, or Caulobacter crescentus.
48  develop a model of nutrient signaling in C. crescentus.
49 e we investigate CTL function in Caulobacter crescentus.
50 cle progression in the bacterium Caulobacter crescentus.
51 symmetrically dividing bacterium Caulobacter crescentus(4-8).
52             At the cell poles of Caulobacter crescentus, a 177-amino acid (aa) protein called PopZ se
53 tiple flagella and no prosthecae, whereas C. crescentus, a freshwater bacterium, has a single polar f
54  polarized alpha-proteobacterium Caulobacter crescentus, a model for cell cycle regulation and asymme
55 l and one regulatory paralog, in Caulobacter crescentus, a monopolarly flagellated alpha-proteobacter
56 on and apply it to the bacterium Caulobacter crescentus, a paradigm for cell-cycle control.
57 udy, we focus on the behavior of Caulobacter crescentus, a singly flagellated bacterium, at the air/w
58 observe the swimming patterns of Caulobacter crescentus, a uniflagellated bacterium, in a linear oxyg
59 nt bacteria Escherichia coli and Caulobacter crescentus achieve cell size homeostasis by growing, on
60 ial cell cycle regulator GcrA in Caulobacter crescentus activates the transcription of target genes i
61 ruginosa, Bacillus subtilis, and Caulobacter crescentus all provided various levels of, but functiona
62 we identify a small protein from Caulobacter crescentus, an assembly inhibitor of MreB (AimB).
63 ree living alpha-proteobacterium Caulobacter crescentus and an orthologous system from an obligate in
64  the dynamics of c-di-GMP and (p)ppGpp in C. crescentus and analyze how the guanine nucleotide-based
65 on, we worked with the bacterium Caulobacter crescentus and asked whether exposure to a moderate conc
66 roximately P/NepR interaction in Caulobacter crescentus and characterized the effect of aspartyl phos
67 lymorphic locus, zwf, between lab-adapted C. crescentus and clinical isolates of Pseudomonas aerugino
68 rrangements of FtsZ and FtsA filaments in C. crescentus and E. coli cells and inside constricting lip
69 ridization, we show here that in Caulobacter crescentus and Escherichia coli, chromosomally expressed
70  of the uniflagellated bacterium Caulobacter crescentus and have found that each cell displays two di
71 required for the curved shape of Caulobacter crescentus and localizes to the inner cell curvature.
72 ll division and polarization for Caulobacter crescentus and Pseudomonas aeruginosa.
73 ent using purified proteins from Caulobacter crescentus and show that CTP is required for spreading.
74  similarity between the division cycle of C. crescentus and that of A. tumefaciens, the functional co
75 introduced site-specific DSBs in Caulobacter crescentus and then used time-lapse microscopy to visual
76 y network that controls the cell cycle of C. crescentus and, presumably, of many other Alphaproteobac
77 richia coli, Myxococcus xanthus, Caulobacter crescentus, and Mycobacterium tuberculosis, respectively
78 erichia coli, Bacillus subtilis, Caulobacter crescentus, and Myxococcus xanthus.
79 a putative LytM endopeptidase in Caulobacter crescentus, and show that it plays a critical role in ma
80 those reported for the bacterium Caulobacter crescentus, and they are crucial for survival in the hos
81 f specific cell wall proteins in Caulobacter crescentus are sensitive to small external osmotic upshi
82 tended to provide a taste of the power of C. crescentus as a model system to explore a diverse range
83                      Here, using Caulobacter crescentus as a model, we exploit genome-wide experiment
84 flagellated alphaproteobacterium Caulobacter crescentus as an experimental model system.
85 y encoded ParD-ParE complex from Caulobacter crescentus at 2.6 A resolution.
86                                  Caulobacter crescentus attachment is mediated by the holdfast, a com
87 rom polystyrene microspheres and Caulobacter crescentus bacteria, to the trapping region.
88 orted CtrA consensus sequence in Caulobacter crescentus Bacterial one-hybrid experiments showed that
89 nce of morphological patterns in Caulobacter crescentus biofilms.
90 quencing (Hi-C), we show that in Caulobacter crescentus, both transcription rate and transcript lengt
91 ion of bactofilin filaments from Caulobacter crescentus by high-resolution solid-state NMR spectrosco
92 cal functionality of the genome design in C. crescentus by transposon mutagenesis.
93                                        In C. crescentus, c-di-GMP works as a major regulator of pole
94 tion in the asymmetric bacterium Caulobacter crescentus (Caulobacter) is triggered by the localizatio
95  first protein, Cc0300, was from Caulobacter crescentus CB-15 (Cc0300), while the second one (Sgx9355
96 e profiles for two proteins from Caulobacter crescentus CB15 (Cc2672 and Cc3125) and one protein (Sgx
97  of GDP-perosamine synthase from Caulobacter crescentus CB15 suggested that only two mutations would
98  assay the assembly of FtsZ from Caulobacter crescentus (CcFtsZ) and reported that assembly required
99       Genome sequencing has revealed many C. crescentus cell cycle genes are conserved in other Alpha
100       DNA adenine methylation by Caulobacter crescentus Cell Cycle Regulated Methyltransferase (CcrM)
101 M.EcoP15I from Escherichia coli, Caulobacter crescentus cell cycle-regulated DNA methyltransferase (C
102                              The Caulobacter crescentus cell cycle-regulated DNA methyltransferase (C
103 hromosome replication during the Caulobacter crescentus cell cycle.
104 ce of power-law statistics in the tail of C. crescentus cell-size distribution, although there is a d
105      In the Alphaproteobacterium Caulobacter crescentus, cell cycle progression is believed to be con
106     In the alpha-proteobacterium Caulobacter crescentus, cell cycle-regulated transcription plays an
107 tify the straightening of curved Caulobacter crescentus cells after disruption of the cell-curving cr
108 parse oligotrophic environments, Caulobacter crescentus cells divide asymmetrically, yielding a motil
109 e peptide side chains of PG isolated from C. crescentus cells grown in the complex laboratory medium
110 ind that the sizes of individual Caulobacter crescentus cells increase exponentially in time.
111 nd that reversible attachment of Caulobacter crescentus cells is mediated by motile cells bearing pil
112 ion of rod shape in lemon-shaped Caulobacter crescentus cells pretreated with MP265 or A22 under nont
113 olate synchronous populations of Caulobacter crescentus cells to investigate assembly of the divisome
114 of amines on the surface of live Caulobacter crescentus cells.
115 erial actin protein MreB in live Caulobacter crescentus cells.
116 rrays in cryotomograms of intact Caulobacter crescentus cells.
117 sualize such interactions inside Caulobacter crescentus cells.
118 hemoreceptor arrays in wild-type Caulobacter crescentus cells.
119 growth and shape data for single Caulobacter crescentus cells.
120 ern is due to the low ParA copy number in C. crescentus cells.
121                      However, in Caulobacter crescentus, cells lacking the primary SOS-regulated inhi
122 tomography, here we show that in Caulobacter crescentus, chemoreceptor arrays in cells grown in diffe
123 ymer modeling indicates that the Caulobacter crescentus chromosome consists of multiple, largely inde
124 required to anchor the separated Caulobacter crescentus chromosome origins at the cell poles, a funct
125 and a role in ClpX positioning similar to C. crescentus CpdR, suggesting a conserved function of CpdR
126                               In Caulobacter crescentus, CpdR controls the polar localization of the
127 sphatase homolog in a bacterium, Caulobacter crescentus CtpA.
128                            Paradoxically, C. crescentus curvature is robustly maintained in the wild
129           The principal components of the C. crescentus degradosome are the endoribonuclease RNase E,
130       The cell-division cycle of Caulobacter crescentus depends on periodic activation and deactivati
131                                  Caulobacter crescentus differentiates from a motile, foraging swarme
132 he logic of stringent response control in C. crescentus differs from E. coli, the global transcriptio
133 sistent with its ecological distribution, C. crescentus displays a narrow range of osmotolerance, wit
134            Using these data, we show that C. crescentus displays aerotactic behavior by extending the
135                                  Caulobacter crescentus divides asymmetrically into a swarmer cell an
136 cillus subtilis) and asymmetric (Caulobacter crescentus) division and reconstruct their lineages to c
137            Here, we show that in Caulobacter crescentus, DnaX isoforms are unexpectedly generated thr
138 2 from the alpha-proteobacterium Caulobacter crescentus does not need the extended -10 motif for high
139                    The bacterium Caulobacter crescentus employs a specialized dimorphic life cycle co
140 s in the Gram-negative bacterium Caulobacter crescentus, enabling long-time-scale protein tracking an
141                    The bacterium Caulobacter crescentus encodes a soluble LOV-histidine kinase, LovK,
142                    The genome of Caulobacter crescentus encodes at least 31 sRNAs, and 27 of these sR
143                     For example, Caulobacter crescentus encodes six glycosyltransferase paralogs of l
144 me of the alpha-proteobacterium, Caulobacter crescentus, encodes eight ParE/RelE-superfamily toxins t
145    We report that the S-layer of Caulobacter crescentus exhibits calcium-mediated structural plastici
146 e free-living aquatic bacterium, Caulobacter crescentus, exhibits two different morphologies during i
147        We used fluorescence microscopy of C. crescentus expressing green fluorescent protein to track
148                         We show here that C. crescentus extracellular DNA (eDNA) inhibits the ability
149 ays a role in the surface modification of C. crescentus, facilitating the uptake of nutrients during
150   The podJ gene, originally identified in C. crescentus for its role in polar organelle development,
151 engineered the aerobic bacterium Caulobacter crescentus for REE adsorption through high-density cell
152 for FtsZ in bacteria, however in Caulobacter crescentus, FtsA arrives at midcell after stable Z-ring
153 e describe a role for the CTL of Caulobacter crescentus FtsZ as an intrinsic regulator of lateral int
154 23, had no stabilizing effect on Caulobacter crescentus FtsZ filaments in vitro, which complements pr
155                  We show that in Caulobacter crescentus, FzlA must bind to FtsZ for division to occur
156 Z to the membrane and demonstrate that in C. crescentus, FzlC is one such membrane anchor.
157 e regulator LovR also function within the C. crescentus general stress pathway.
158 Here, we show that the bacterium Caulobacter crescentus generates a gradient of the active, phosphory
159 nsional (3D) architecture of the Caulobacter crescentus genome by combining genome-wide chromatin int
160 les unable to bind myo-inositol abolishes C. crescentus growth in medium containing myo-inositol as t
161 h-affinity ribose binding allele affected C. crescentus growth on D-ribose as a carbon source, provid
162          The dimorphic bacterium Caulobacter crescentus has evolved marked phenotypic changes during
163                    The bacterium Caulobacter crescentus has morphologically and functionally distinct
164  a U-specific stress response in Caulobacter crescentus has provided a foundation for studying the me
165                                  Studying C. crescentus has reformed our understanding of bacterial i
166                                Studies in C. crescentus have also deepened our knowledge of other top
167 oteins from Escherichia coli and Caulobacter crescentus have been shown to bind peptidoglycan.
168 ial regulator of constriction in Caulobacter crescentus, helps link FtsZ to PG synthesis to promote d
169                         We also show that C. crescentus Hfq has sRNA binding and RNA annealing activi
170 enesis at the predicted catalytic site of C. crescentus HfsH phenocopied the DeltahfsH mutant and abo
171             Here we show that recombinant C. crescentus HisRS allowed complete histidylation of a C.
172 e U20a, created a competent substrate for C. crescentus HisRS.
173 f G(-1) did not improve aminoacylation by C. crescentus HisRS.
174  divergent (based on sequence similarity) C. crescentus HisRS.
175 We report a crystal structure of Caulobacter crescentus IbpA bound to myo-inositol at 1.45 A resoluti
176 hematical modeling, our study in Caulobacter crescentus identifies a novel NAP (GapR) whose activity
177 mer cell cycles of the bacterium Caulobacter crescentus in a near-mechanical step-like fashion.
178 l the general stress response in Caulobacter crescentus, including sigma(T), its anti-sigma factor Ne
179                                  Caulobacter crescentus initiates a single round of DNA replication d
180  including Proteus mirabilis and Caulobacter crescentus, initiates asymmetrically, accompanied by asy
181                               In Caulobacter crescentus, intact cables of the actin homologue, MreB,
182                                  Caulobacter crescentus integrates phospho-signaling pathways and tra
183                 Cell division in Caulobacter crescentus involves constriction and fission of the inne
184                                  Caulobacter crescentus is a premier model organism for studying the
185                                  Caulobacter crescentus is a species that has met this need effective
186 e synchronizable model bacterium Caulobacter crescentus is cell cycle regulated and we unearth a bact
187                The cell cycle of Caulobacter crescentus is controlled by a complex signalling network
188 rization in the model prokaryote Caulobacter crescentus is precisely orchestrated through at least th
189  alpha-proteobacteria, including Caulobacter crescentus, lacks the critical G(-1) residue.
190            Here, we show that in Caulobacter crescentus Lon controls deoxyribonucleoside triphosphate
191               Lysates of E. coli in which C. crescentus LpxI (CcLpxI) is overexpressed display high l
192                     We show that Caulobacter crescentus makes use of and requires a dedicated mechani
193 cell division and development in Caulobacter crescentus, many of which are also conserved among diver
194  three proteins in the bacterium Caulobacter crescentus: McpA, PopZ, and SpmX.
195 he UzcRS two-component system in Caulobacter crescentus mediates widespread transcriptional activatio
196  and that this interaction is favored for C. crescentus MreB over Escherichia coli MreB because of a
197 lecular dynamics simulations for Caulobacter crescentus MreB to extract mechanical parameters for inp
198                                  Caulobacter crescentus mutants that lack the trans translation pathw
199 nd a xylonolactonase (xylC) from Caulobacter crescentus, native E. coli xylonate dehydratase (yjhG),
200 the singly flagellated bacterium Caulobacter crescentus near a glass surface with total internal refl
201 ne" to synchronize the bacterium Caulobacter crescentus on-chip and to move the synchronized populati
202 erentiating alphaproteobacterium Caulobacter crescentus, organelle synthesis at cell poles is critica
203                  We show that in Caulobacter crescentus, PBP3 accumulates at the new pole at the begi
204 oroughly characterized the composition of C. crescentus peptidoglycan by high-performance liquid chro
205    Remarkably, glycine incorporation into C. crescentus peptidoglycan occurred even in the presence o
206 roperty of the aquatic bacterium Caulobacter crescentus permits visualization of single cells in a li
207 litative and quantitative analysis of the C. crescentus PG by high-performance liquid chromatography
208  here that glycine incorporation into the C. crescentus PG depends on the presence of exogenous glyci
209                                Hence, the C. crescentus PG is able to retain its physiological functi
210 We present genetic evidence that Caulobacter crescentus PhyR is a phosphorylation-dependent stress re
211                    We visualized Caulobacter crescentus pili undergoing dynamic cycles of extension a
212                              For Caulobacter crescentus, polar stalk synthesis is tied to its dimorph
213                  Two proteins in Caulobacter crescentus, PopZ and TipN, provide directional cues at t
214        The alpha-proteobacterium Caulobacter crescentus produces a motile swarmer cell and a sessile
215                               In Caulobacter crescentus, progression through the cell cycle is govern
216                              We show that C. crescentus ProRS can readily form Cys- and Ala-tRNA(Pro)
217                               In Caulobacter crescentus, protein degradation by the ClpXP protease is
218 We identify nearly 300 localized Caulobacter crescentus proteins, up to 10-fold more than were previo
219 s of Cb13 and CbK phage-infected Caulobacter crescentus, provides insight into the mechanisms of infe
220                  Substrate specificity of C. crescentus ProXp-ala is determined, in part, by elements
221 FlbT, and FlaF, proteins that in Caulobacter crescentus regulate flagellum biosynthesis.
222     In the alpha-proteobacterium Caulobacter crescentus, regulated protein degradation is required fo
223 ssion in the dimorphic bacterium Caulobacter crescentus requires spatiotemporal regulation of gene ex
224 try in the predivisional cell of Caulobacter crescentus requires that the regulatory protein DivL loc
225 The maintenance of cell shape in Caulobacter crescentus requires the essential gene mreB, which encod
226               Here, we show that Caulobacter crescentus responds to DNA damage by coordinately induci
227                  Loss of PflI function in C. crescentus results in an abnormally high frequency of ce
228 xamination of the hfsH deletion mutant in C. crescentus revealed that this strain synthesizes holdfas
229 how that, unlike its E. coli counterpart, C. crescentus RhlB interacts directly with a segment of the
230                              Furthermore, C. crescentus RNase E appears associated with the DNA indep
231     The crystal structure of a portion of C. crescentus RNase E encompassing the helicase-binding reg
232                               In Caulobacter crescentus, RodZ is essential for viability and is invol
233  finding was that when using the Caulobacter crescentus rrn leader sequence, there was little effect
234 c simulations to clarify how the Caulobacter crescentus S-layer assembles on the lipopolysaccharides
235  cryomicroscopy structure of the Caulobacter crescentus S-layer bound to the O-antigen of lipopolysac
236 eria, Sinorhizobium meliloti and Caulobacter crescentus, serve as models for investigating the geneti
237                                        In C. crescentus, several CheY homologs regulate motor functio
238    Here, we demonstrate that the Caulobacter crescentus SLP readily crystallizes into sheets in vitro
239             Here, using purified Caulobacter crescentus' sole S-layer protein RsaA, we obtained a 2.7
240 hat a developmental regulator of Caulobacter crescentus, SpmX, is co-opted in the genus Asticcacaulis
241         We also used modified E. coli and C. crescentus ssrA tags to independently control the degrad
242    Based on the previous observation that C. crescentus stalks are lysozyme-resistant, we compared th
243 ty, and stationary-phase survival between C. crescentus strain CB15 and its derivative NA1000 is dete
244  demonstrate that cell curvature enhances C. crescentus surface colonization in flow.
245                               In Caulobacter crescentus, surface attachment and subsequent biofilm gr
246                          Forward swimming C. crescentus swarmer cells tend to get physically trapped
247      We analyzed the adaptive response of C. crescentus swarmer cells to carbon starvation and found
248 ous interloci contour lengths in Caulobacter crescentus swarmer cells to determine the in vivo config
249 tify a toxin-antitoxin system in Caulobacter crescentus that acts by a unique mechanism.
250 he morphogenetic protein MreB in Caulobacter crescentus that gives rise to cells with a variable-widt
251 cin in the alpha-proteobacterium Caulobacter crescentus that is retained on the surface of producer c
252          Here, we demonstrate in Caulobacter crescentus that proteotoxic stress induces a cell-cycle
253                 Here, we show in Caulobacter crescentus that the polarity factor TipN regulates the d
254 y diffusing mRNA, and provide evidence in C. crescentus that this mRNA localization restricts ribosom
255                                        In C. crescentus the CtrA response regulator serves as the mas
256                               In Caulobacter crescentus, the actin homologue MreB is critical for cel
257 led "ori-ter" and exemplified by Caulobacter crescentus, the chromosome arms lie side-by-side, with t
258                               In Caulobacter crescentus, the ClpXP protease is essential and drives c
259 LD-crosslinking or lysozyme resistance in C. crescentus, the correlation between these two properties
260                                        In C. crescentus, the directionality of the transport is set u
261 l cycle regulatory genes are essential in C. crescentus, the essential genes of two Alphaproteobacter
262                               In Caulobacter crescentus, the G1-S transition involves the degradation
263                               In Caulobacter crescentus, the HHK ShkA is essential for accurate timin
264        In the vibrioid bacterium Caulobacter crescentus, the intermediate filament-like protein cresc
265                               In Caulobacter crescentus, the Lon protease degrades DnaA to coordinate
266                               In Caulobacter crescentus, the PopZ polar scaffold protein supports asy
267                                     As in C. crescentus, the S. meliloti PodJ1 protein appears to act
268 tured by the dimorphic bacterium Caulobacter crescentus, the stalk, a cylindrical extension of all ce
269  A. tumefaciens pathway resembles that of C. crescentus there are specific differences including addi
270  rebuilt the essential genome of Caulobacter crescentus through the process of chemical synthesis rew
271                                           C. crescentus thus repurposes pilus retraction, typically u
272                               In Caulobacter crescentus, tmRNA was localized in a cell-cycle-dependen
273 q from the alpha-proteobacterium Caulobacter crescentus to 2.15- angstrom resolution, resolving the c
274 of single cells of the bacterium Caulobacter crescentus to a glass surface in a microfluidic device.
275 y alterations in pilus activity stimulate C. crescentus to bypass its developmentally programmed temp
276 ns, from cell stalk formation in Caulobacter crescentus to chromosome segregation and motility in Myx
277   Thus, the major recognition elements in C. crescentus tRNA(His) are the anticodon, the discriminato
278 HisRS allowed complete histidylation of a C. crescentus tRNA(His) transcript (lacking G(-1)).
279                        The PG of Caulobacter crescentus, unlike that of many other Gram-negative bact
280     A recent study suggests that Caulobacter crescentus uses a novel regulator, FzlA, to activate rin
281                    The bacterium Caulobacter crescentus uses a ParA-based partitioning system to segr
282                                  Caulobacter crescentus uses the dynamic interactions between ParA an
283                    The bacterium Caulobacter crescentus uses two-component phospho-signalling to regu
284 tigate the distribution of HU in Caulobacter crescentus using a combination of super-resolution fluor
285 ith the surface of the bacterium Caulobacter crescentus using a double-helix point spread function mi
286 s of the Gram-negative bacterium Caulobacter crescentus using cryo-electron tomography (CET) and stat
287                 We focus here on Caulobacter crescentus, Vibrio cholerae, Helicobacter pylori, and Ca
288  drives cell-cycle initiation in Caulobacter crescentus We identify the type IV pilin protein PilA as
289 d tracking the S-layer protein (SLP) from C. crescentus, we show that 2D protein self-assembly is suf
290 sed during a life cycle stage of Caulobacter crescentus when the regulator is activated by phosphoryl
291 icrodomains at the cell poles in Caulobacter crescentus, where it functions as a hub protein that rec
292  is GDP-perosamine synthase from Caulobacter crescentus, which catalyzes the final step in GDP-perosa
293                Here, we focus on Caulobacter crescentus, which encodes a ProRS with a truncated INS d
294  Focusing on the model bacterium Caulobacter crescentus, which generates two different types of daugh
295 y from the alpha-proteobacterium Caulobacter crescentus, which is a model organism for studying morph
296 y of CheY-like (Cle) proteins in Caulobacter crescentus, which tune flagellar activity in response to
297 ouples phosphorylation events in Caulobacter crescentus with the AAA+ protease ClpXP to provide punct
298 haproteobacterial model organism Caulobacter crescentus, with a specific focus on LytM-like endopepti
299 bed in the alpha-proteobacterium Caulobacter crescentus, with the interacting partners of RNase E ide
300 nfirmed that these species are cleared by C. crescentus YbaK and ProXp-ala, respectively.

 
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