ライフサイエンスコーパス: cresol

1 dependent hydroxylation of toluene to form p-cresol.

2 lfate (PCS) but no detectable unconjugated p-cresol.

3 ependent hydroxylation of toluene to yield p-cresol.

4 the hydroxylation of toluene to yield 96% p-cresol.

5 at the meta position, producing primarily m-cresol.

6 n comparison, produces 97% p-cresol and 3% m-cresol.

7 and produced methylhydroquinone (80%) from o-cresol.

8 0 microM and produced 90% p-cresol and 10% m-cresol.

9 eaction specificity in HDO of furfural and m-cresol.

10 erent with resorcinol than with phenol and m-cresol.

11 ylaniline, 3,4-dimethylphenol, and 2-amino-p-cresol.

12 dependent hydroxylation of toluene to form p-cresol.

13 up hydrogen from the reactor walls to form o-cresol.

14 iently prepared in five steps from 2-amino-m-cresol.

15 ters Ti(16) O(16) (OEt)(32) , linkers, and m-cresol.

16 se associated with aromatic compounds like p-cresol.

17 503 to the p-hydroxybenzyl radical to form p-cresol.

18 mples were higher in gamma-nonalactone and m-cresol.

19 , were 0.3 ug kg(-1) for phenol, o-cresol, p-cresol; 0.6 ug kg(-1) for eugenol, isoeugenol; and 1 ug

20 Derivatives of p-cresol 1-4 were synthesized, and their photochemical rea

21 for butyric acid, 1 x 10(-4) mug/L air for p-cresol, 1 x 10(-5) mug/L air for indole, and 1 x 10(-5)

22 er than the pK(a) of tyrosine (10.1) or of p-cresol (10.2).

23 mounts (in %) are 2-methyladenine (60.6%), p-cresol (16.3%), adenine (12.5%), 2-(methylthio)adenine (

24 ed 4 electron equiv/mol when titrated with p-cresol (2 electrons from p-cresol and 2 from 4-hydroxybe

25 talyzed the oxidation of catechol, 6-amino-m-cresol, 2-amino-m-cresol, and 2-amino-4-chlorophenol.

26 4-Chloro-m-cresol (4-CmC) is a clinically relevant activator of the

27 4-Chloro-m-cresol (4-CmC) is a potent and specific activator of the

28 nse to high [K(+)], caffeine, and 4-chloro-m-cresol (4-CMC), the maximal tensions generated in Stac3-

29 metric specific force followed by 4-chloro-m-cresol (4-CmC)-evoked maximal contracture force in singl

30 R3), are efficiently activated by 4-chloro-m-cresol (4-CmC).

31 sistent with this interpretation, 4-chloro-m-cresol (4-CMC; 100 microm) increases the rate of Ca(2+)

32 hen, diclofenac, carbamazepine, clozapine, p-cresol, 4-ethylphenol, and 3-methylindole in human liver

33 derived from phenolic compounds including p-cresol, 4-hydroxybenzoate and numerous lignin monomers,

34 In the reaction of NO3 radicals with para-cresol, 4-methyl-2-nitrophenol (4M2NP) and HNO3 were ide

35 gets (diazinon, propiconazole, 4,6-dinitro-o-cresol, 4-nitrobenzyl chloride).

36 ed that the RYR-stimulating agent 4-chloro-m-cresol (4CmC) induced Ca(2+) release and thereby confirm

37 ,6-Trichlorophenol (2) and 2,4,6-trichloro-m-cresol (5) react with calcium hypochlorite (Ca(OCl)(2))

38 udomonas mendocina KR1 oxidizes toluene to p-cresol (96%) and oxidizes benzene sequentially to phenol

39 stores, since the application of 4-chloro-m-cresol, a direct type 1 ryanodine receptor activator, el

40 p-Cresol, a microbial metabolite produced by the human mic

41 ostridioides difficile is known to produce p-cresol, a phenolic compound with selective antimicrobial

42 oaded CD19(+) B and DAKIKI cells, 4-chloro-m-cresol, a potent activator of Ca2+ release mediated by t

43 uld be restored by application of 4-chloro-m-cresol, a ryanodine receptor agonist, indicating that th

44 es that contain the bases-2-methyladenine, p-cresol, adenine, and 2-(methylthio)adenine.

45 infrared spectrum of 4-methylimidazole or p-cresol alone.

46 ol did not increase Tf saturation with Al. p-Cresol also increased Tf-Al uptake in Friend erythroleuk

47 d ryanodine but not for Ca(2+) or 4-chloro-m-cresol, although they all induced Ca(2+) release.

48 4-Chloro-m-cresol, an activator of the skeletal muscle ryanodine re

49 In the presence of tyrosine or p-cresol, an unusual tricyclo[4.3.3.0] adduct has been cha

50 nt Km value of 250 microM and produced 90% p-cresol and 10% m-cresol.

51 n titrated with p-cresol (2 electrons from p-cresol and 2 from 4-hydroxybenzyl alcohol), PchF(C) acce

52 xidation-labile aromatic compounds such as p-cresol and 2-naphthol with high yields, 95% and 85%, res

53 .e., alcohol dehydration and alkylation of m-cresol and 2-propanol in the liquid phase, at high tempe

54 T4MO, in comparison, produces 97% p-cresol and 3% m-cresol.

55 ng precursors 60 and 68 were prepared from m-cresol and 3-ethylphenol, respectively.

56 Odorants such as p-cresol and a sweet-character unknown component were corr

57 Examples of top prioritized compounds were p-cresol and chlorophene, based on human health end points

58 The second-order rate constants for p-cresol and ferrocyanide reduction of the mutant compound

59 Both 4-chloro-m-cresol and halothane caused adenosine accumulation in bl

60 erates myriad toxic metabolites, including p-cresol and indoxyl sulfate.

61 e conditions suggesting the involvement of p-cresol and its impact on the biochemical composition of

62 By using o-cresol and o-methoxyphenol as model substrates, regiospe

63 %) and methylhydroquinone (9%), to oxidize m-cresol and p-cresol to 4-methylcatechol (100%), and to o

64 onversely, the A107T variant produced >98% p-cresol and p-nitrophenol from toluene and nitrobenzene,

65 Interestingly, p-cresol and phenol, which are the lower ligand in Sporomu

66 ells was significantly altered by 4-chloro-m-cresol and ryanodine.

67 enzyme HydG lyses free tyrosine to produce p-cresol and the CO and CN(-) ligands of the [2Fe](H) clus

68 e electronic and redox properties of bound p-cresol and the covalently bound FAD.

69 Our studies suggest that p-cresol and uremic fractions 4 to 8, 12, 14, and 15 incre

70 nd Cl-BDA formation, but the chlorination of cresols and 2,3-dimethylphenol yielded methyl- and dimet

71 e higher acidity of the S1 states of these p-cresols and the ability for excited-state intramolecular

72 ion of catechol, 6-amino-m-cresol, 2-amino-m-cresol, and 2-amino-4-chlorophenol.

73 usion coefficients of benzene, anthracene, m-cresol, and p-nitrophenol in enhanced-fluidity liquid mi

74 thylbenzimidazole, 5-methoxybenzimidazole, p-cresol, and phenol were determined.

75 e are converted into the respective phenols, cresols, and methoxyphenols by fast gas-phase reaction w

76 phenylacetonitrile, cyclobutanone, phenol, p-cresol, aniline) to form ammonia and trans-(DMPE)(2)Ru(H

77 enic bacterium Sporomusa ovata, phenol and p-cresol are converted into alpha-ribotides, which are inc

78 pha-amino group (the aromatic hydrogens of p-cresol are far less subject to exchange) and by imidazol

79 on of biphenyl, naphthalene, m-xylene, and p-cresol are predicted to be distributed among 15 gene clu

80 The oxidative half-reaction of PHHY using m-cresol as a substrate is similarly affected by the mutat

81 nds studied in resorcinol >> phenol > or = m-cresol as determined from their overall free energies of

82 ally characterized cell permeable diformyl-p-cresol based receptor (HL) selectively senses the AsO3(3

83 ing addition and fragmentation reaction of p-cresol-based N-phenylbenzoxazine with aliphatic and arom

84 Phenolic ligands, e.g., phenol and m-cresol, bind to 2Zn(II)-insulin hexamers and induce a co

85 identified compounds including 2,6-dinitro-p-cresol, bisphenol S, clonixin, and triclopyr.

86 nctionalised with 2-(2'-benzothiazolylazo)-p-cresol (BTAC).

87 The degradation of the toxic phenol p-cresol by Pseudomonas bacteria occurs by way of the prot

88 steps and 78% overall yield starting from o-cresol by using a one-pot regiocontrolled dialkylation o

89 teady-state rate constant for oxidation of p-cresol by various forms of PCMH and PchF; both nu(m) and

90 Cresol (C(7)H(8)O) is used as a BB proxy to investigate

91 thylbenzene (C(9)H(12)), phenol (C(6)H(6)O), cresol (C(7)H(8)O), 2,6-dimethylphenol (C(8)H(10)O), and

92 This study examined the form in which p-cresol circulates and quantified its removal by hemodial

93 We conclude that p-cresol circulates in the form of its sulfate conjugate,

94 (Cl(ind)), p-cresol sulfate (Cl(pcs)), and p-cresol (Cl(pc)) averaged only 5 +/- 1, 4 +/- 1, and 14 +

95 they were much less sensitive to 4-chloro-m-cresol (CMC).

96 zation or RyR agonists (caffeine, 4-chloro-m-cresol) compared with wtRyR1.

97 The sensor probe is characterized for p-cresol concentration range from 0microM (reference sampl

98 showed increased Al uptake and toxicity at p-cresol concentrations of 3 mg/dl in culture media.

99 atase resulted in recovery of this peak as p-cresol, confirming its identity.

100 etabolites revealed reductions in indole and cresol derivatives, as well as trimethylamine N-oxide, i

101 p-Cresol did not increase Tf saturation with Al. p-Cresol

102 richlorobenzene, imidacloprid, 4,6-dinitro-o-cresol, ethylacrylate, malathion, chlorpyrifos, aldicarb

103 etermination of phenols (phenol, o-cresol, p-cresol, eugenol, isoeugenol and guaiacol) in smoked food

104 ur findings indicated that while perinatal p-Cresol exposure did not affect gestational outcomes, pos

105 future epidemiological studies to quantify p-Cresol exposure during pregnancy and early childhood, an

106 These results suggest that p-Cresol exposure selectively induces ASD-like behaviors,

107 BHA, creosol, isoeugenol and di-o-propenyl p-cresol, fewer radicals were trapped by a single phenol m

108 ately 5-fold increase in the percentage of m-cresol formation relative to that of the natural isoform

109 elded shifts of regiospecificity away from p-cresol formation, with F205I giving an approximately 5-f

110 07T produced methylhydroquinone (92%) from o-cresol fourfold faster than wild-type T4MO and there was

111 ake and cell toxicity were proportional to p-cresol from 1.5 mg/dl to 3 mg/dl in culture media.

112 para-monooxygenase variant that formed 75% m-cresol from toluene and 100% m-nitrophenol from nitroben

113 Furthermore, G103S/A107T formed 100% p-cresol from toluene; hence, a better para-hydroxylating

114 involved in the fermentative production of p-cresol from tyrosine in clostridia.

115 lacetyl glycine (from phenylalanine); (ii) p-cresol (from tyrosine) yielding p-cresol sulfate and p-c

116 yline, indole-3-acetic-acid-O-glucuronide, p-cresol glucuronide, and pregnanediol-3-glucuronide.

117 om tyrosine) yielding p-cresol sulfate and p-cresol glucuronide; (iii) 4-OH-phenyllactic acid (from t

118 re lower in women with GD (4-cresyl sulfate, cresol, glycine, P-cresol sulfate, phenylacetic acid, an

119 It was demonstrated that p-cresol has a charge-transfer interaction with FAD when b

120 Elevated levels of p-Cresol have been consistently observed in patients with

121 itiated oxidation of ortho-, meta-, and para-cresol have been performed in large-volume chamber syste

122 s applied and validated for PCMC (4-chloro-m-cresol), household derived antimicrobial agent with no k

123 reduced circulating p-cresol sulfate, and p-cresol impaired contractility and rhythm in human induce

124 the product distribution, and gave o- and p-cresol in a 1:1 ratio.

125 ia coli in coculture to assess the role of p-cresol in modulating interspecies dynamics.

126 lysis confirmed the structural identity of p-cresol in samples containing the product of hydroxylatio

127 p-Cresol increased Tf-associated Al uptake only because th

128 tudies of the reaction of PCMH[Y384F] with p-cresol indicated that the K(m) for this substrate was un

129 ximately 0.0001), suggesting that 4-chloro-m-cresol-induced adenosine could readily distinguish betwe

130 , and consequently, SOCE after 4-chloro-meta-cresol-induced store depletion was suppressed.

131 p-Cresol is a simple molecular model for the para phenolic

132 p-cresol is produced by the conversion of para-Hydroxyphen

133 including phenol, bisphenol A, catechol and cresols is reported.

134 experiments the gas-phase reaction of ortho-cresol isomer with NO3 yielded (11.5 +/- 0.8) % 6-methyl

135 s observed from the reaction of NO3 and with cresol isomers.

136 s dihydroxyacetone, alanine, xanthine, and p-cresol-key metabolites in independent pathways.

137 Metabolomic analysis identified increased cresol levels in these mice, and exposure of cultured ol

138 pecific oxidation of aromatics (e.g., from o-cresol, M180H forms 3-methylcatechol, methylhydroquinone

139 tanding how environmental factors, such as p-Cresol, may contribute to social impairments and neurode

140 xposure to environmental toxins, including p-Cresol, may disrupt essential physiological processes du

141 hF) of the alpha(2)beta(2) flavocytochrome p-cresol methylhydroxylase (PCMH) from Pseudomonas putida

142 The alpha(2)beta(2) flavocytochrome p-cresol methylhydroxylase (PCMH) from Pseudomonas putida

143 Each flavoprotein subunit (PchF) of p-cresol methylhydroxylase (PCMH) has flavin adenine dinuc

144 otein component (PchF) of flavocytochrome, p-cresol methylhydroxylase (PCMH), and cytochrome-free Pch

145 The first enzyme in this pathway, p-cresol methylhydroxylase (PCMH), is a flavocytochrome c.

146 ese proteins are (1) the flavocytochrome c p-cresol methylhydroxylase (rPCMH, 1.85 A resolution) and

147 number of oxidoreductases from the VAO/para-cresol methylhydroxylase flavoprotein family catalyze th

148 variants of the flavoprotein component of p-cresol methylhydroxylase that contain noncovalently or c

149 hexamer containing two zinc ions, with two m-cresol molecules bound at each dimer-dimer interface sta

150 xylene (m-X), o-xylene (o-X), styrene (S), o-cresol (o-C), phenol (PhAl), p-cresol (p-C), indole (ID)

151 , near-quantitative deuterium retention in m-cresol obtained from 4-(2)H(1)-toluene, and partial loss

152 ymphocytes incubated with 0-10 mM 4-chloro-m-cresol or 0-10.7 mM halothane.

153 Similarly, at 1 mM 4-chloro-m-cresol or 0.96 mM halothane, adenosine levels were signi

154 -benzenetricarbaldehyde building blocks in m-cresol or acetic acid, named RT-COF-1 or RT-COF-1Ac/RT-C

155 fter RyR activation (caffeine and 4-chloro-m-cresol) or beta-adrenergic stimulation (isoproterenol).

156 a ryanodine receptor agonist (4-chloro-meta-cresol) or depolarizing pulses were used.

157 eptor (10 mM caffeine, 200 microM 4-chloro-m-cresol, or 10 mM KCl).

158 enzyl alcohol, the intermediate product of p-cresol oxidation by PCMH, reduced PchF(NC) fairly quickl

159 lation was found between the efficiency of p-cresol oxidation by these proteins and E(CT), the energy

160 g of protein were obtained for o-, m-, and p-cresol oxidation by wild-type T4MO, which are comparable

161 also that 4-hydroxybenzaldehyde, the final p-cresol oxidation product, is an efficient competitive in

162 tyrene (S), o-cresol (o-C), phenol (PhAl), p-cresol (p-C), indole (ID), and skatole (SK)).

163 HPLC-FLD determination of phenols (phenol, o-cresol, p-cresol, eugenol, isoeugenol and guaiacol) in s

164 on 3sigma, were 0.3 ug kg(-1) for phenol, o-cresol, p-cresol; 0.6 ug kg(-1) for eugenol, isoeugenol;

165 ter at 25 degrees C for benzene, p-xylene, p-cresol, p-dicyanobenzene, and hydroquinone from statisti

166 PCS by decreasing intestinal production of p-cresol, prevented these metabolic derangements.

167 tors of HpdBCA decarboxylase, which reduce p-cresol production and render C. difficile less able to c

168 itor of HpdBCA decarboxylase, only reduced p-cresol production by 54.9 +/- 13.5%.

169 ound, 4-Hydroxyphenylacetonitrile, reduced p-cresol production by 99.0 +/- 0.4%, whereas 4-Hydroxyphe

170 This study has identified promising p-cresol production inhibitors whose development could lea

171 This work examines the use of purified meta-cresol purple (mCP) for direct spectrophotometric calibr

172 nd chemical characteristics of purified meta-Cresol Purple (mCP) pH indicator dye suitable for pH mea

173 scein at pH 6.5, phenol red at pH 7.5, and m-cresol purple at pH 8.5) which permitted separation of s

174 d spectrophotometrically using purified meta-Cresol Purple indicator dye offering high precision (<0.

175 (phenol red) and meta-cresolsulfonphthalein (cresol purple) were synthesized by electrophilic fluorin

176 In contrast, p-cresol rapidly reduced PchF with covalently bound FAD (P

177 c analyses showed areas under the 4-chloro-m-cresol receiver-operating characteristic curves near mor

178 Although 4-chloro-m-cresol receiver-operating characteristic curves revealed

179 ible B cells treated with 0.75 mM 4-chloro-m-cresol relative to controls.

180 nt coupling and high regiospecificity with p-cresol representing >96% of total products from toluene.

181 tical to that of the natural isoform, with p-cresol representing 90-95% of the total product distribu

182 and a decrease in regiospecificity so that p-cresol represents approximately 60% of total products.

183 Its response time is also better than the p-cresol sensor currently available in the market for the

184 ained as compared to the recently reported p-cresol sensor.

185 re, 4-methylimidazole covalently linked to p-cresol, show that a feature near 1540 cm(-1) is unique t

186 luene; for example, G103S/A107G formed 82% o-cresol, so saturation mutagenesis converted T4MO into an

187 usly, Siamwiza and co-workers investigated p-cresol solutions to identify Raman spectroscopic signatu

188 o selectively modify benzylic C-H bonds of o-cresol substrates.

189 16-fold), indoxyl sulfate (0.21-fold), and p-cresol sulfate (0.39-fold) were much lower than the rate

190 108-fold), indoxyl sulfate (116-fold), and p-cresol sulfate (41-fold) were much greater than the conc

191 s 2.3 0.6 for patients with CKD; P<0.001), p-cresol sulfate (8.6 2.6 for controls versus 4.1 1.5 for

192 Cresol sulfate (beta=-0.07; adjusted-p <0.001), hippuric

193 e protein-bound solutes indican (Cl(ind)), p-cresol sulfate (Cl(pcs)), and p-cresol (Cl(pc)) averaged

194 enriched in gut-derived bacterial solutes p-cresol sulfate (PCS) and indoxyl sulfate (IS) that both

195 k whose mobility corresponded to synthetic p-cresol sulfate (PCS) but no detectable unconjugated p-cr

196 MDMs (uMDMs) phenylacetylglutamine (PAG), p-cresol sulfate (PCS), and indoxyl sulfate (IS) accumulat

197 In cultured cells, indoxyl sulfate and p-cresol sulfate activated the EGF receptor and downstream

198 Although p-cresol sulfate and indoxyl sulfate are well studied exam

199 HPLC confirmed the colonic origin of p-cresol sulfate and indoxyl sulfate, but levels of hippur

200 e); (ii) p-cresol (from tyrosine) yielding p-cresol sulfate and p-cresol glucuronide; (iii) 4-OH-phen

201 robiome formation of uremic toxins such as p-cresol sulfate and thereby their body exposure and need

202 mized mice treated with indoxyl sulfate or p-cresol sulfate as study models.

203 Low clearance of hippurate or p-cresol sulfate associated with greater risk of death ind

204 results suggested that indoxyl sulfate and p-cresol sulfate dock on a putative interdomain pocket of

205 Indoxyl sulfate and p-cresol sulfate have been suggested to induce kidney tiss

206 In conclusion, indoxyl sulfate and p-cresol sulfate may induce kidney tissue remodeling throu

207 eatment: from no reduction in the level of p-cresol sulfate or asymmetric dimethylarginine to signifi

208 Treatment of mice with indoxyl sulfate or p-cresol sulfate significantly activated the renal EGF rec

209 lasma levels of these compounds (including p-cresol sulfate).

210 reted solute (hippurate, cinnamoylglycine, p-cresol sulfate, and indoxyl sulfate) clearance using liq

211 ine, 5-hydroxyindole, indoxyl glucuronide, p-cresol sulfate, and indoxyl sulfate.

212 s, the SGLT2i likewise reduced circulating p-cresol sulfate, and p-cresol impaired contractility and

213 phenylsulfate, oxindole, indolepropionate, p-cresol sulfate, catechol sulfate, and salicylate, are im

214 olites, including indole-3-propionic acid, p-cresol sulfate, hippuric acid, pyrocatechol sulfate, and

215 he secreted anions phenylacetyl glutamine, p-cresol sulfate, indoxyl sulfate, and hippurate confirmed

216 toxins and solutes (e.g., indoxyl sulfate, p-cresol sulfate, kynurenine, creatinine, urate) include t

217 We then narrowed down to five metabolites [p-cresol sulfate, linoleic acid, glycocholic acid, lysoPC(

218 recovery, and changes in indolelactate and p-cresol sulfate, metabolites that impact host inflammator

219 ith GD (4-cresyl sulfate, cresol, glycine, P-cresol sulfate, phenylacetic acid, and stearoylcarnitine

220 tometric absorption of indoxyl sulfate and p-cresol sulfate.

221 acid, pantothenic acid, hippuric acid, and p-cresol sulfate.

222 malian-microbial co-metabolites hippurate, 4-cresol sulphate, and formate were reduced in critical il

223 dentified metabolites of bacterial origin (p-cresol sulphate, indoxyl sulphate and N-phenylacetylglut

224 roup metabolites of bacterial origin (e.g. p-cresol sulphate, indoxyl sulphate and N-phenylacetylglut

225 e resonance (LMR) based sensor for urinary p-cresol testing on optical fiber substrate is developed.

226 s undergo deamination reactions, and for all cresols the formation of quinone methides (QMs) was obse

227 ylene, cyclobutanone, aniline, phenol, and p-cresol, the reaction was observed to proceed via ion pai

228 In this study T4MO was found to oxidize o-cresol to 3-methylcatechol (91%) and methylhydroquinone

229 PCMH oxidizes p-cresol to 4-hydroxybenzyl alcohol, which is oxidized sub

230 hydroquinone (9%), to oxidize m-cresol and p-cresol to 4-methylcatechol (100%), and to oxidize o-meth

231 he enzyme first catalyzes the oxidation of p-cresol to p-hydroxybenzyl alcohol, utilizing one atom of

232 A107S produced 3-methylcatechol (98%) from o-cresol twofold faster and produced 3-methoxycatechol (82

233 nterpret the Raman and infrared spectra of p-cresol vapor and extend the previous correlation to the

234 dy, we exposed pregnant mice (C57BL/6J) to p-Cresol via drinking water from mid-gestation, through la

235 ant Al uptake by MH was observed only when p-cresol was added together with Tf-Al.

236 ospecific oxidation of o-methoxyphenol and o-cresol was changed for significant synthesis of 3-methox

237 dine receptor Ca channels agonist 4-chloro-m-cresol was compared in blood lymphocytes from malignant

238 inhibited, whereas that of CYP1A2-specific o-cresol was increased, results consistent with the format

239 p-Cresol was not toxic to MH in the absence of Tf-Al in me

240 In the FVP of (o-CH(3)O)(2)-PPE, o-cresol was the dominant product.

241 N-o-Vanillidine-2-amino-p-cresol was used as a chelating ligand and 1-undecanol wa

242 It was found that p-cresol was virtually incapable of reducing PchF with non

243 n amino acid-derived microbial metabolite, p-cresol, was supplemented along with WSD-ETPi diet, prima

244 bstitution of 4-(fluoroethynyl)benzenes by p-cresol were determined by (1)H NMR spectroscopy, and the

245 ope distributions of 5'-deoxyadenosine and p-cresol were evaluated using deuterium-labeled tyrosine s

246 tion of CYP2B4-specific benzyl alcohol and p-cresol were inhibited, whereas that of CYP1A2-specific o

247 lorobenzene, imidacloprid, and 4,6-dinitro-o-cresol were not biotransformed.

248 st extensively studied of these solutes is p-cresol, which has been shown to be toxic in vitro.

249 ntrations of the antimicrobial compound para-cresol, which provides the bacterium with a competitive

250 -hexenol, acetic acid, benzyl alcohol, and m-cresol, while the addition of oxygen significantly influ

251 o probe the mechanism of the alkylation of m-cresol with isopropyl alcohol in scCO(2) using Nafion SA

252 s also show a clear preference for binding p-cresol with the hydroxyl group hydrated rather than insi

253 oton transfer to this 4OB(*) radical forms p-cresol, with the conversion of this dehydroglycine ligan

254 iously from these ultrafiltrate fractions (p-cresol, xanthine, tryptophan, hippuric acid, and o-hydro

255 n efficiency of phenol approached 100% while cresols, xylenols, and 4-ethylphenol were 97% or higher

256 eveloped for the characterization of phenol, cresols, xylenols, and alkyl phenols like 4-ethylphenol

257 The reaction of NO3 radicals with meta-cresol yielded (21.2 +/- 1.4) % 3-methyl-2-nitrophenol (

258 otinate mononucleotide (NaMN) to phenol or p-cresol, yielding alpha-O-glycosidic ribotides.