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1 rat, stained with FJ and counterstained with cresyl violet.
2 ally for the dendritic marker MAP-2, or with cresyl violet.
3 of vermis were sectioned and stained with 1% cresyl violet.
4 s of 10-micron coronal sections stained with cresyl violet.
5                 All brains were stained with Cresyl violet, a Nissl stain.
6  was assessed in brain sections stained with cresyl violet and animals with misplaced lesions were ex
7                                              Cresyl violet and dimethylaminoazobenzene are Raman labe
8 alog, PP3, were examined histologically with cresyl violet and iron stain to assess the degree of dam
9 P-32 staining) leading to neurodegeneration (cresyl violet and neuronal nuclei staining) associated w
10 , postfixed and examined histologically with cresyl violet and Perl's iron stain to assess the degree
11 al staining but extensive neurodegeneration (cresyl violet and silver staining) when evaluated 4 days
12         Cryosections of LG were stained with cresyl violet, and acinar cells and ductal epithelial ce
13 rometer-thick brain slices were stained with cresyl violet, and the neuronal density of the amygdala,
14 t on fixed tissues using stains for neurons (cresyl violet), astrocytes (GFAP), microglia (Iba1), glu
15 were stained with hematoxylin & eosin (H&E), Cresyl violet, Bielschowsky silver stain, or immunohisto
16       Brains were harvested and stained with cresyl violet, caspase-3, and TUNEL to detect morphologi
17 rin 343 (C343)-TiO(2) nanoparticles (NP) and Cresyl Violet (CV(+))-TiO(2) NP systems, using time-corr
18 y interaction of a benzo-phenoxazine ligand (Cresyl Violet, CV) with antiparallel and (3 + 1) hybrid
19 dded, hippocampal sections were stained with cresyl violet for Nissl substance and immunolabeled for
20 articles and the long wavelength fluorophore cresyl violet, has been used for the determination of co
21 pars reticulata (TH immunohistochemistry and Cresyl violet histochemistry) by 28 days after ischemia/
22  variety of methodologies: cytoarchitecture (cresyl violet), histochemistry (peanut agglutinin), immu
23 ime PCR and histological analysis, utilizing Cresyl Violet/Luxol Fast Blue staining and evaluating th
24 cts of these treatments were evaluated using cresyl violet (Nissl) staining.
25 g and nuclear morphology were assessed using Cresyl violet (Nissl) staining.
26 rfused, and brain sections were stained with cresyl violet or immunolabeled with NeuN (for neuronal c
27 nglion cells (flat preparations stained with cresyl violet or retrograde labeling with a neurotracer)
28 LC), cytochrome oxidase (CO) histochemistry, cresyl violet, or demonstration of TCAs by placement of
29 X-100 and CoQ10 causes the MLs lysis and the cresyl violet oxidation, obtaining a decrease in the flu
30 er sensitized trans-cis isomerization, using cresyl violet perchlorate as the sensitizer, also led to
31 ion technique using fiducial markers such as cresyl violet, Ponceau S, and bromophenol blue that poss
32                    Retinas were stained with cresyl violet, retinal cell-specific markers, and a huma
33                                              Cresyl violet stain demonstrated massive loss of neurons
34                                  Counting of cresyl violet stained sections showed an apparent increa
35 sing an image analysis system (BioQuant) and cresyl violet stained sequential sections from the foreb
36 ions were evaluated for area of tissue loss (Cresyl-violet stained sections) and the number of GFAP i
37 ts of two adult rat spinal cords on adjacent cresyl violet-stained and in situ hybridization sections
38          Molecular fingerprint comparison of cresyl violet-stained CA1 and CA3 pyramidal neurons micr
39 on cells were counted by light microscopy in cresyl violet-stained retina sections, and the percentag
40 es immediately after I/R injury and counting cresyl violet-stained retinal ganglion cell layer cells
41                      Using still images from cresyl-violet-stained material, we present an adaptation
42                        For both methods, Arc cresyl violet staining (cell density) and NPY and Y1 rec
43           Brain tissue loss determined using cresyl violet staining and astrocyte hypertrophy and pro
44 brains of Tg and Wt mice as assessed by both Cresyl violet staining and by TUNEL staining for DNA fra
45              Neuronal damage, as assessed by cresyl violet staining and DNA fragmentation analysis, w
46 -induced neurodegeneration, as visualized by cresyl violet staining and quantified in 20 serially sta
47 developed a modified alcohol-based, buffered cresyl violet staining protocol that provides reproducib
48 n the present study, immunohistochemical and Cresyl violet staining showed that the noradrenergic neu
49 neuronal death using silver impregnation and cresyl violet staining.
50  30 and later at > or =6 months of age using cresyl violet, Timm, and rapid Golgi staining and immuno
51                 Cells were also stained with cresyl violet to detect morphological changes characteri
52 d brain slices and subsequently stained with cresyl violet to enable high-resolution spatial analysis
53 mortem sections of the ERC were stained with cresyl violet to visualize neurons and immunostained wit