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1                                   The use of crevicular AST activity to monitor periodontal disease p
2 udy examined conditions under which gingival crevicular blood (GCB) could be used to obtain a useful
3 l diabetes screening approach using gingival crevicular blood (GCB) could be used to test for hemoglo
4 GCF), a marker of neutrophil influx into the crevicular environment, has previously been shown to be
5 talloproteinase-9 (MMP-9) levels in gingival crevicular fluid (GCF) across various periodontal health
6 ociation with plaque index (PI) and gingival crevicular fluid (GCF) after OHR for 1 week.
7 le is known about MMP reductions in gingival crevicular fluid (GCF) after therapy.
8 and local inflammatory mediators in gingival crevicular fluid (GCF) among patients with untreated chr
9  testing, histological examination, gingival crevicular fluid (GCF) analysis for the levels of C-telo
10 at in chronic periodontitis (CP) by gingival crevicular fluid (GCF) analysis of myeloperoxidase (MPO)
11 eri-implant crevicular fluid (PICF)/gingival crevicular fluid (GCF) and a selection of subgingival/su
12                      At each visit, gingival crevicular fluid (GCF) and blood were collected for dete
13 pecific FN fragments are present in gingival crevicular fluid (GCF) and can be used as markers for pe
14 ere to delineate cyto/chemokines in gingival crevicular fluid (GCF) and evaluate systemic levels of e
15 evels of stress-related hormones in gingival crevicular fluid (GCF) and extent/severity of periodonta
16     PAF levels were investigated in gingival crevicular fluid (GCF) and gingival tissue (GT) samples
17    LTB4 levels were investigated in gingival crevicular fluid (GCF) and gingival tissue (GT) samples
18                                 The gingival crevicular fluid (GCF) and monocytic secretion of prosta
19  surgery on growth factor levels in gingival crevicular fluid (GCF) and periodontal healing.
20 s, and levels of cyto/chemokines in gingival crevicular fluid (GCF) and peripheral blood response.
21 the present study is to investigate gingival crevicular fluid (GCF) and plasma acute-phase cytokines,
22 s study is to investigate levels of gingival crevicular fluid (GCF) and plasma TGM-2 and oxidative st
23 d subgingival bacteria collected in gingival crevicular fluid (GCF) and RA parameters.
24 This study aimed to investigate the gingival crevicular fluid (GCF) and salivary HIF-1alpha, VEGF, an
25 nitial periodontal treatment on the gingival crevicular fluid (GCF) and salivary levels of 8-hydroxy-
26 ng pregnancy on periodontal health, gingival crevicular fluid (GCF) and serum cytokines, and pregnanc
27     This study aimed to investigate gingival crevicular fluid (GCF) and serum levels of LRG, interleu
28     This study aimed to investigate gingival crevicular fluid (GCF) and serum levels of LRG, interleu
29 This study is performed to evaluate gingival crevicular fluid (GCF) and serum levels of soluble recep
30  evaluate YKL-40 and IL-6 levels in gingival crevicular fluid (GCF) and serum of patients with period
31 To evaluate the cytokine profile in gingival crevicular fluid (GCF) and serum of pediatric inflammato
32 nti-inflammatory cytokine levels in gingival crevicular fluid (GCF) and serum of rheumatoid arthritis
33 gical periodontal therapy (NSPT) on gingival crevicular fluid (GCF) and serum progranulin (PGRN) leve
34 mor necrosis factor (TNF)-alpha, in gingival crevicular fluid (GCF) and serum samples between women w
35 idative stress index (OSI) in their gingival crevicular fluid (GCF) and serum were recorded.
36                        At baseline, gingival crevicular fluid (GCF) and subgingival plaque were colle
37                                     Gingival crevicular fluid (GCF) and subgingival plaque were colle
38 growth factor-beta 1 (TGF-beta1) in gingival crevicular fluid (GCF) and the periodontal status of sub
39 ith apoptosis in serum, saliva, and gingival crevicular fluid (GCF) and their association with period
40 is-specific molecular signatures in gingival crevicular fluid (GCF) and to confirm clinical diagnoses
41  antibody responses in non-invasive gingival crevicular fluid (GCF) and unstimulated whole saliva to
42 s and RANKL/OPG ratios in serum and gingival crevicular fluid (GCF) are investigated in patients with
43           Volumetric alterations in gingival crevicular fluid (GCF) are widely accepted to be associa
44 implant crevicular fluid (PICF) and gingival crevicular fluid (GCF) as surrogate measures of immune f
45 tudy is to determine whether either gingival crevicular fluid (GCF) bacterial gingivain/gingipain or
46 his study were to measure levels of gingival crevicular fluid (GCF) biomarkers and subgingival bacter
47                                     Gingival crevicular fluid (GCF) biomarkers associated with bone r
48 n serum, synovial fluid, saliva and gingival crevicular fluid (GCF) by multiplex immunoassay.
49  study are to: 1) determine whether gingival crevicular fluid (GCF) chemerin is a novel predictive ma
50 n unbiased metabolomic profiling of gingival crevicular fluid (GCF) collected from healthy, gingiviti
51 Aa), Tannerella forsythia (Tf), and gingival crevicular fluid (GCF) concentration of interleukin (IL)
52 present with increased salivary and gingival crevicular fluid (GCF) concentrations of cytokines.
53 this study was to compare serum and gingival crevicular fluid (GCF) concentrations of sICAM-1 in smok
54 ue, suggesting that their levels in gingival crevicular fluid (GCF) could be increased at gingivitis
55 gival plaque microbial profiles and gingival crevicular fluid (GCF) cytokine levels were determined b
56     It may be hypothesized that the gingival crevicular fluid (GCF) cytokine profile in patients with
57 l periodontal therapy (NSPT) on the gingival crevicular fluid (GCF) cytokine profile in sites with st
58 rived growth factor-BB (PDGF-BB) in gingival crevicular fluid (GCF) during early healing period after
59 metalloproteinase (MMP)-8 and -9 in gingival crevicular fluid (GCF) during early pregnancy with the p
60 he study are to determine serum and gingival crevicular fluid (GCF) endocan levels in the pathogenesi
61 s, systemic priming of neutrophils, gingival crevicular fluid (GCF) expression of inflammatory marker
62                                     Gingival crevicular fluid (GCF) flow was significantly lower in t
63 F) samples from the new implant and gingival crevicular fluid (GCF) from adjacent teeth were sampled
64 ence of Th1-associated cytokines in gingival crevicular fluid (GCF) from LagP patients and the high l
65 -derived growth factor (PDGF-BB) in gingival crevicular fluid (GCF) from localized intrabony defects
66 f human herpesviruses in saliva and gingival crevicular fluid (GCF) from patients with CKD.
67 n-surgical periodontal treatment on gingival crevicular fluid (GCF) HIF-1alpha, VEGF, and TNF-alpha l
68 ingival fibroblasts in vitro and in gingival crevicular fluid (GCF) in a randomized controlled split-
69 mpare the levels of resistin in the gingival crevicular fluid (GCF) in health, chronic periodontitis
70  to determine levels of ADAMTS-1 in gingival crevicular fluid (GCF) in patients with advanced periodo
71 IL)-11 and IL-17 and their ratio in gingival crevicular fluid (GCF) in patients with AgP.
72 trix metalloproteinase-8 (MMP-8) in gingival crevicular fluid (GCF) in patients with periodontal dise
73 al parameters of disease and on the gingival crevicular fluid (GCF) inflammatory mediator interleukin
74  of eight oral pathogens, levels of gingival crevicular fluid (GCF) interleukin-1beta (IL-1beta), pro
75 clinical periodontal parameters and gingival crevicular fluid (GCF) levels of 15-epi-lipoxin A4 (15-e
76 IgG) titers to 17 bacteria, and the gingival crevicular fluid (GCF) levels of 16 inflammatory mediato
77        The current study determines gingival crevicular fluid (GCF) levels of fibrosis-related IL-6-t
78                       Comparing the gingival crevicular fluid (GCF) levels of galectin-3, peptidylarg
79                       Comparing the gingival crevicular fluid (GCF) levels of galectin-3, peptidylarg
80 e of this study was to test whether gingival crevicular fluid (GCF) levels of IL-1beta and tumor necr
81 ochemical profile by evaluating the gingival crevicular fluid (GCF) levels of interleukin (IL)-17, IL
82                                     Gingival crevicular fluid (GCF) levels of interleukin (IL)-1beta
83 clinical periodontal parameters and gingival crevicular fluid (GCF) levels of interleukin (IL)-1beta,
84                  Serum, saliva, and gingival crevicular fluid (GCF) levels of interleukin (IL)-6, IL-
85 her glycemic control was related to gingival crevicular fluid (GCF) levels of interleukin-1beta (IL-1
86  to determine the serum, saliva and gingival crevicular fluid (GCF) levels of MT in smokers (S) and n
87 gical periodontal therapy (NSPT) on gingival crevicular fluid (GCF) levels of semaphorin-4D (SEMA-4D)
88 tcome of a chairside test measuring gingival crevicular fluid (GCF) levels of the enzyme aspartate am
89                                     Gingival crevicular fluid (GCF) levels of the polymorphonuclear l
90                      Four different gingival crevicular fluid (GCF) markers from 14 patients were cha
91                           Different gingival crevicular fluid (GCF) matrix metalloproteinase (MMP)-8
92  this study is to explore different gingival crevicular fluid (GCF) matrix metalloproteinase-8 (MMP-8
93 al and microbiologic parameters and gingival crevicular fluid (GCF) matrix metalloproteinases-8 (MMP-
94 levels of immunoglobulin A (IgA) in gingival crevicular fluid (GCF) may be "protective", while increa
95       The analysis of biomarkers in gingival crevicular fluid (GCF) may be helpful in forecasting pat
96 e of inflammatory products found in gingival crevicular fluid (GCF) may be of value in evaluating bot
97  Biochemical markers harvested from gingival crevicular fluid (GCF) may be useful to identify and pre
98             Estimation of serum and gingival crevicular fluid (GCF) MCP levels could be a reliable in
99 tudy is to investigate salivary and gingival crevicular fluid (GCF) nitric oxide (NO) metabolism and
100 seline and 2 weeks postoperatively, gingival crevicular fluid (GCF) obtained at baseline, 2 weeks and
101  with their corresponding levels in gingival crevicular fluid (GCF) obtained from the same participan
102 nzyme-linked immunosorbent assay in gingival crevicular fluid (GCF) of 24 individuals with periodonti
103 agenase activity in the gingival tissues and crevicular fluid (GCF) of adult periodontitis subjects i
104 se-8, -9, and AIF were evaluated in gingival crevicular fluid (GCF) of all participants via enzyme-li
105 d products (AGEs) are higher in the gingival crevicular fluid (GCF) of chronic periodontitis (CP) pat
106 to determine visfatin levels in the gingival crevicular fluid (GCF) of healthy individuals and patien
107 is factor-alpha (TNF-alpha), in the gingival crevicular fluid (GCF) of individuals with periodontitis
108 nvestigate the levels of sTREM-1 in gingival crevicular fluid (GCF) of individuals without periodonti
109 , 2 microbiostatic proteins, in the gingival crevicular fluid (GCF) of normal children.
110 eviously demonstrated that IgG from gingival crevicular fluid (GCF) of patients with chronic periodon
111 dy was to evaluate MFN1 and MFN2 in gingival crevicular fluid (GCF) of patients with periodontal dise
112 hat ADAM8 levels are also raised in gingival crevicular fluid (GCF) of patients with periodontal dise
113 factor-kappaB ligand (RANKL) in the gingival crevicular fluid (GCF) of patients with periodontitis af
114 1 through 3 (HNP 1-3) levels in the gingival crevicular fluid (GCF) of patients with periodontitis.
115 th factor beta 1 (TGF-beta1) in the gingival crevicular fluid (GCF) of patients with periodontitis.
116  that TACE would be elevated in the gingival crevicular fluid (GCF) of persons with periodontitis.
117 radation and bone resorption in the gingival crevicular fluid (GCF) of the same vulnerable subjects.
118           This study aims to assess gingival crevicular fluid (GCF) PLAP-1, sclerostin, and TNF-alpha
119 c tromethamine mouthrinse (0.1%) on gingival crevicular fluid (GCF) prostaglandin E2 (PGE2) concentra
120 f alveolar bone, and measurement of gingival crevicular fluid (GCF) prostaglandin E2 (PGE2) levels we
121  and the differential expression of gingival crevicular fluid (GCF) proteins using sequential window
122 eome and differential expression of gingival crevicular fluid (GCF) proteins using the sequential win
123 ne naphthylamine [BANA] assay), and gingival crevicular fluid (GCF) pyridinoline cross-linked carboxy
124                                     Gingival crevicular fluid (GCF) samples and clinical parameters,
125 0 levels were assessed in serum and gingival crevicular fluid (GCF) samples at baseline and at variou
126 ollowing: 1) biochemical content in gingival crevicular fluid (GCF) samples from sites of gingival re
127 urements (PD, CAL, PI, BOP, GI) and gingival crevicular fluid (GCF) samples were collected at baselin
128 Clinical periodontal parameters and gingival crevicular fluid (GCF) samples were collected at baselin
129                  Serum, saliva, and gingival crevicular fluid (GCF) samples were collected to measure
130 ssment of periodontal measurements, gingival crevicular fluid (GCF) samples were taken at one incisor
131  multiple inflammatory mediators in gingival crevicular fluid (GCF) samples.
132 in reaction was performed using the gingival crevicular fluid (GCF) samples.
133 livered antibiotics on the level of gingival crevicular fluid (GCF) stromelysin (SL) and tissue inhib
134 he hypothesis to be tested was that gingival crevicular fluid (GCF) TGF-beta1 production was altered
135 in are higher and more sustained in gingival crevicular fluid (GCF) than in serum.
136 clines reach steady-state levels in gingival crevicular fluid (GCF) that are several-fold higher than
137                         Biofilm and gingival crevicular fluid (GCF) to assess the microbiome profile
138  Secondary purposes were to compare gingival crevicular fluid (GCF) volume and GCF concentrations of
139 ndex (PI), gingival index (GI), and gingival crevicular fluid (GCF) volume from days 0 to 21 were sig
140 GI), bleeding on probing (BOP), PD, gingival crevicular fluid (GCF) volume, and total amount and conc
141                                     Gingival crevicular fluid (GCF) was also collected to measure mat
142                                     Gingival crevicular fluid (GCF) was analyzed for the same cytokin
143 n biological profiles in saliva and gingival crevicular fluid (GCF) was assessed and compared with th
144 nical evaluation was performed, and gingival crevicular fluid (GCF) was collected for selected sites
145 on probing (BOP) were measured, and gingival crevicular fluid (GCF) was collected from five deepest p
146                                     Gingival crevicular fluid (GCF) was collected in a cohort of 44 A
147 e mean concentration of 0.5% CLM in gingival crevicular fluid (GCF) was estimated by reverse-phase hi
148                                     Gingival crevicular fluid (GCF) was obtained from 2 human studies
149                       The volume of gingival crevicular fluid (GCF) was quantified, and interleukin-1
150                                     Gingival crevicular fluid (GCF) was sampled for the analysis of m
151 mplant crevicular fluid (PICF), and gingival crevicular fluid (GCF) were collected from patients with
152  proinflammatory cytokines level in gingival crevicular fluid (GCF) were evaluated.
153     Levels of visfatin in serum and gingival crevicular fluid (GCF) were explored in patients with pe
154 B (TGF-B) and collagen-1 (Col-1) in gingival crevicular fluid (GCF) were measured using enzyme-linked
155                          Samples of gingival crevicular fluid (GCF) were obtained 1, 3, 7, 14, and 21
156 status and the enzymatic profile of gingival crevicular fluid (GCF) when given as adjunct to periodon
157       Beta-glucuronidase (betaG) in gingival crevicular fluid (GCF), a marker of neutrophil influx in
158                             Saliva, gingival crevicular fluid (GCF), and blood samples from 30 patien
159 rotic cells, has been identified in gingival crevicular fluid (GCF), and elevated levels are associat
160 sion patterns of ghrelin in saliva, gingival crevicular fluid (GCF), and gingival tissue, as well as
161 gingival temperature, flow rates of gingival crevicular fluid (GCF), and neutrophil emigration into G
162 he aim of this study is to evaluate gingival crevicular fluid (GCF), saliva, and plasma levels of IL-
163     This study investigates whether gingival crevicular fluid (GCF), saliva, and plasma levels of int
164 nd TIMP-1 levels were determined in gingival crevicular fluid (GCF), saliva, and serum by immunofluor
165 im of this study is to evaluate the gingival crevicular fluid (GCF), saliva, and serum concentrations
166                                     Gingival crevicular fluid (GCF), saliva, and serum levels of IL-6
167 macokinetic data were obtained from gingival crevicular fluid (GCF), saliva, and serum of adult perio
168 17A/F, and IL-17E (IL-25) levels in gingival crevicular fluid (GCF), saliva, and serum of non-obese f
169                                     Gingival crevicular fluid (GCF), serum, and saliva samples were o
170 as well as gingival tissue (GT) and gingival crevicular fluid (GCF), was obtained from a total of 35
171 th factor beta 1 (TGF-beta1) in the gingival crevicular fluid (GCF), which accumulates around the nec
172 ta on microbial plaque composition, gingival crevicular fluid (GCF)-interleukin (IL)-1beta levels, an
173 sis factor-alpha (TNF-alpha) in the gingival crevicular fluid (GCF).
174 strates are different in saliva vs. gingival crevicular fluid (GCF).
175 nti-PC and anti-oxLDL production in gingival crevicular fluid (GCF).
176 ponses and tissue remodeling in the gingival crevicular fluid (GCF).
177 ino acid, enters the oral cavity in gingival crevicular fluid (GCF).
178 dy are to comparatively analyze the gingival crevicular fluid (GCF)/peri-implant sulcus fluid (PISF)
179 9 days) were compared with adjacent gingival crevicular fluid (GCF; at baseline, 1, and 4 months).
180 1alpha, GPX4, and MDA levels in peri-implant crevicular fluid (PICF) across peri-implant health, muco
181 ytokine levels were measured in peri-implant crevicular fluid (PICF) and gingival crevicular fluid (G
182 ecific inflammatory cytokine in peri-implant crevicular fluid (PICF) compared with healthy patients;
183             The proteome of the peri-implant crevicular fluid (PICF) has not been systematically inve
184 NA-150-5p, and miRNA-26a-5p) in peri-implant crevicular fluid (PICF) of subjects with healthy, peri-i
185                                 Peri-implant crevicular fluid (PICF) samples from the new implant and
186       Clinical measurements and peri-implant crevicular fluid (PICF) were taken at 1-, 2-, 4-, and 6-
187                          Serum, peri-implant crevicular fluid (PICF), and gingival crevicular fluid (
188 udy assesses cytokine levels in peri-implant crevicular fluid (PICF)/gingival crevicular fluid (GCF)
189 uld enhance DOX availability in the gingival crevicular fluid after its release from the collagen mem
190 ilm as well as on biomarkers in the gingival crevicular fluid after SRP.
191 of host markers in serum/plasma and gingival crevicular fluid along with clinical parameter improveme
192 rails to reduce MMP activity in the gingival crevicular fluid and gingival tissues, with a resultant
193 iodontitis have type-1 cytokines in gingival crevicular fluid and high titers of IFN-gamma-dependent
194 IgG2 reactive with P. gingivalis in gingival crevicular fluid and serum.
195 and is detected at higher levels in gingival crevicular fluid and tissue specimens.
196           Measures of gingival inflammation, crevicular fluid aspartate aminotransferase (AST) levels
197          Bioassay of doxycycline in gingival crevicular fluid associated with presence of ABDS-D gave
198 ted whole-mouth salivary flow rate, gingival crevicular fluid biomarkers).
199 leeding on probing (BOP) as well as gingival crevicular fluid bone marker assessment (ICTP) and micro
200 e if levels of IL-1alpha protein in gingival crevicular fluid can be correlated to patient genotype.
201 performed a metabolomic analysis of gingival crevicular fluid collected from healthy, gingivitis, and
202                     The analysis of gingival crevicular fluid components offers great potential as a
203 dontal pockets, which are bathed in gingival crevicular fluid consisting of 70% of blood plasma, an a
204 s of gingipain activity detected in gingival crevicular fluid could implicate a role for gingipains i
205 be used for measuring ions from the gingival crevicular fluid directly into the peri-odontal pocket,
206 ics at concentrations detectable in gingival crevicular fluid do not dramatically reduce total bacter
207                             The reduction in crevicular fluid flow at control sites suggested that cl
208 ol sites, a significant decrease in gingival crevicular fluid flow rate was evident at the conclusion
209 galactose-deficient IgG in sera and gingival crevicular fluid from periodontal disease patients, comp
210  segments of FN and used to analyze gingival crevicular fluid from periodontal disease sites in syste
211 nd between DHA and control for both gingival crevicular fluid hsCRP (-5.3 ng/mL, standard error [SE]
212 s) and local inflammatory response (gingival crevicular fluid IL-1beta) and derive periodontal comple
213 rom real samples, such as saliva or gingival crevicular fluid in a short time as a point-of-care (POC
214 e the levels of these biomarkers in gingival crevicular fluid in periodontal health, gingivitis, and
215 nical parameters were recorded, and gingival crevicular fluid levels of biomarkers were analyzed by e
216 pth, clinical attachment level, and gingival crevicular fluid levels of matrix metalloproteinases (MM
217 e loss, which correlated with lower gingival crevicular fluid levels of proinflammatory mediators (e.
218 ponse to a major human salivary and gingival crevicular fluid LPA species, 18:1, and that they would
219 l parameters and cytokine levels in gingival crevicular fluid of dogs with experimental periodontitis
220 parameters and cytokine profiles in gingival crevicular fluid of patients with moderate-to-severe chr
221 nes were evaluated in the serum and gingival crevicular fluid of shallow and deep sites by enzyme-lin
222  depths, gingival inflammation, and gingival crevicular fluid RANKL and MMP-8 levels (p < 0.05).
223 uated clinically and biochemically (gingival crevicular fluid RANKL, OPG, MMP-8, and serum IL-1beta l
224 (MD) on clinical parameters and peri-implant crevicular fluid receptor activator of nuclear factor-ka
225        Total RNA was extracted from gingival crevicular fluid samples and relative gene expression of
226                                 Peri-implant crevicular fluid samples collected from 164 participants
227 ected the karilysin gene in >90% of gingival crevicular fluid samples containing T. forsythia obtaine
228 Detection of these gene products in gingival crevicular fluid samples from human disease may contribu
229  in the pathogenesis of periodontal disease, crevicular fluid samples from LAgP patients were found t
230                                     Gingival crevicular fluid samples from six healthy, six gingiviti
231 iagnostic efficacy was evaluated in gingival crevicular fluid samples from subjects with and without
232                                     Gingival crevicular fluid samples were analyzed for changes in hi
233 Marginal and subgingival plaque and gingival crevicular fluid samples were collected at baseline; aft
234         Clinical parameters and peri-implant crevicular fluid samples were collected baseline and 6 m
235 Marginal and subgingival plaque and gingival crevicular fluid samples were collected from 15 current
236                       Subgingival plaque and crevicular fluid samples were collected from 31 systemic
237                         Gingival tissues and crevicular fluid samples were collected.
238                                     Gingival crevicular fluid samples were used to monitor daily drug
239  examination; blood, subgingival plaque, and crevicular fluid specimen collection; and medical and de
240 ignificantly higher IL-1beta in the gingival crevicular fluid than females during health and periodon
241 oncentrations were estimated in peri-implant crevicular fluid using enzyme-linked immunosorbent assay
242 dex (PlI), gingival index (GI), and gingival crevicular fluid volume (GCF) were evaluated for teeth 6
243 ive attachment levels (RAL); and 5) gingival crevicular fluid volume (GCF).
244                        In addition, gingival crevicular fluid was analyzed for an inflammation index
245                                     Gingival crevicular fluid was collected before treatment, after 1
246     The mean concentration of MF in gingival crevicular fluid was estimated by reverse-phase high-per
247  Samples of subgingival biofilm and gingival crevicular fluid were collected at baseline and at 1-, 3
248                          Plaque and gingival crevicular fluid were collected for trypsin-like enzyme
249 ed hemoglobin, and immunological analysis of crevicular fluid were evaluated.
250 specific immunoglobulin G levels in gingival crevicular fluid were measured in primates immunized wit
251                          Samples of gingival crevicular fluid were obtained 1, 3, 7, 14, and 21 days
252                          Recipient (gingival crevicular fluid) and donor (wound fluid) site VEGF-A, H
253  and measured MMP-9 levels in their gingival crevicular fluid, a liquid that seeps out from the tiny
254 l and inflammatory cytokines in the gingival crevicular fluid, and a synergistic effect was observed,
255  are secreted--specifically saliva, gingival crevicular fluid, and airway surface fluid--differ great
256  effect on alveolar bone, effect on gingival crevicular fluid, and effect on salivary flow.
257 e examined whether IgG in serum and gingival crevicular fluid, and IgG locally produced by plasma cel
258            Saliva, salivary glands, gingival crevicular fluid, and supragingival biofilms may harbor
259                                     Gingival crevicular fluid, bleeding on probing (BOP), and interle
260 inase (MMP) activity in gingival tissues and crevicular fluid, in association with clinical efficacy,
261                             Saliva, gingival crevicular fluid, subgingival plaque, and blood samples
262 on probing [BOP]) and a sampling of gingival crevicular fluid, the composite restoration was performe
263 up to 70% of serum concentration in gingival crevicular fluid.
264  8 selected inflammatory markers measured in crevicular fluid.
265                                              Crevicular fluids from localized juvenile periodontitis
266 t level, and bleeding on probing, as well as crevicular levels of interleukin-1beta and tumor necrosi
267  of AP induces a downregulation of amplified crevicular neutrophil activity.
268 lysis of betaG in saliva may be a measure of crevicular neutrophil influx for the whole mouth.
269 rinse enhances the antibacterial function of crevicular neutrophils.
270 orrelation between gingival inflammation and crevicular pH, which reportedly varies from 6.5 to 8.5.
271                      Tissue sulfiredoxin and crevicular TOS levels are increased significantly in the
272 rrelate the gingival tissue sulfiredoxin and crevicular total oxidative stress (TOS) levels in genera

 
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