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1 ation by the same or another MRGPRX2 ligand (cross desensitization).
2 ation by the same or another MRGPRX2 ligand (cross desensitization).
3 so showed homologous desensitization but not cross desensitization.
4 d the receptor homologously but there was no cross-desensitization.
5  GXM's inhibitory effect appeared to involve cross-desensitization.
6 osinophil intracytoplasmic Ca2+ and complete cross-desensitization.
7 be achieved by using agents that reduce such cross-desensitization.
8           Moreover, the A2a receptor-induced cross-desensitization also reduced the susceptibility of
9                                              Cross-desensitization among receptors for peptide chemoa
10                        By promoting receptor cross-desensitization and cointernalization, such functi
11  Ca2+ release, similar dose-response curves, cross-desensitization, and partial inhibition of release
12 signals, including receptor internalization, cross-desensitization, and phospholipase D activation, a
13                       Because chemotaxis and cross-desensitization are exclusively mediated by G(i),
14 ermore, inhibition of p38 MAPK prevented the cross-desensitization as well as cointernalization of mi
15  to maintained stimuli and may contribute to cross desensitization between chemical and electrical st
16                                              Cross-desensitization between capsaicin and ATP was Ca2+
17 ndicate the existence of reciprocal receptor cross-desensitization between CD4 and CCR5 induced by tw
18     These experiments demonstrate reciprocal cross-desensitization between CRF and stress using LC el
19                                 There was no cross-desensitization between galectin-3 and any of the
20 h either DAMGO or clonidine induced a mutual cross-desensitization between micro and alpha2A receptor
21                                   To examine cross-desensitization between receptors for lipid and pe
22 gnaling mechanisms, we evaluated patterns of cross-desensitization between SAA and other leukocyte ch
23                              We have studied cross-desensitization between these two agents in rat is
24                  mPAFR was also resistant to cross-desensitization by peptide chemoattractants at the
25 riments were designed to investigate whether cross-desensitization develops between CRF and stress.
26            Mediator release showed a similar cross-desensitization effect.
27 ggested that a hierarchy of sensitivities to cross-desensitization exists for the chemoattractant GPC
28                                              Cross-desensitization experiments using THP-1 cells sugg
29                                           In cross-desensitization experiments, among the numerous ch
30 responses to ATP (10 microM) suggesting that cross-desensitization had occurred between capsaicin and
31                A peripheral site of nicotine cross-desensitization is suggested via a nAChR-mediated
32 tle mu receptor endocytosis, induced neither cross-desensitization nor internalization of alpha2A rec
33 r to that induced by serotonin, and complete cross-desensitization occurred between the InsP3 and 5-H
34 ated equivalent PLCbeta3 phosphorylation and cross-desensitization of Ca2+ mobilization by FR, C5aR,
35               Furthermore, we found that the cross-desensitization of CCR1 by FPR1 was associated wit
36  and found that the mechanism of MOR-induced cross-desensitization of CCR5 involves the activation of
37  or dominant negative mutants suppresses the cross-desensitization of CCR5.
38 Further, CCL1 and CCL18 induced heterologous cross-desensitization of CCR8-transfected cells and huma
39 tudy the role of receptor phosphorylation in cross-desensitization of chemoattractant receptors, M2CX
40 okine receptor regulation further we studied cross-desensitization of chemokine receptors in normal P
41 acetylcholine receptors (nAChRs) in nicotine cross-desensitization of chemonociceptive responses of t
42 ) mobilization, suggesting that NMDA induces cross-desensitization of CXCR2.
43                                         This cross-desensitization of CXCR4 was manifested by the inh
44 porting the idea that TCR signaling leads to cross-desensitization of CXCR4.
45 on(-/-)) also showed decreased CCR5-mediated cross-desensitization of G protein activation and Ca(2+)
46 ide hydrolysis and cross-phosphorylation and cross-desensitization of IL-8RA.
47 horylation and cross-phosphorylation but not cross-desensitization of its Ca2+ mobilization by fMLP o
48 /G protein uncoupling, its susceptibility to cross-desensitization of its Ca2+ response by fMLP and C
49                 The data herein suggest that cross-desensitization of PAFR by peptide chemoattractant
50 pioid receptor (MOR) is capable of mediating cross-desensitization of several chemokine receptors inc
51                             The magnitude of cross-desensitization of the Akt or extracellular signal
52 urosporine blocked cross-phosphorylation and cross-desensitization of the PAFR by peptide chemoattrac
53 TrkA-expressing neurons to NGF resulted in a cross-desensitization of turning responses induced by a
54                           Chemokine receptor cross-desensitization provides an important mechanism to
55                                              Cross-desensitization studies and single-receptor transf
56 ive calcium flux in primary lymphocytes, and cross-desensitization studies indicate that MCP-2 acts v
57 sts revealed varying patterns of alpha4beta2 cross-desensitization that were predictive of the effect
58                   Finally, the micro-alpha2A cross-desensitization was absent in dorsal root ganglion
59                                          The cross-desensitization was closely associated with simult
60 rs, homologous desensitization was seen, but cross-desensitization was not observed.
61 kely a normal cellular process that leads to cross-desensitization, which is exploited by the B subun
62 ion, taurine-induced current showed complete cross-desensitization with glycine-activated currents bu
63 nsitization to capsaicin is due to selective cross-desensitization with the heteromeric P2X receptors
64 ous desensitization of FP and also exhibited cross-desensitization, with PGF2alpha resulting in a max