ライフサイエンスコーパス: crossbred

1 logical abnormalities on inbred (129/Ola) or crossbred (129/Ola x C57BL/6) genetic backgrounds.

2 We crossbred 5xFAD mice with APN(-/-) mice to generate APN-

3 milarity in function between ANK and NPP1 we crossbred Akp2(-/-) mice to ank/ank mice and found a par

4 A total of 119 cattle, comprising 104 RACS crossbreds and 15 Chinese Red Steppes cattle, were genot

5 is idea, Spast-KO and hSPAST-C448Y mice were crossbred, and the offspring were compared with the pare

6 The study utilized 116 samples from 29 crossbred Angus x Salers, across three muscle types.

7 The crossbred animals displayed axonal swellings as well as

8 to African environmental adaptations across crossbred animals showing an excess of taurine or indici

9 To do so, we crossbred ank/ank mice with mice lacking Vanin-1 panteth

10 Thus, we crossbred Apoe (-/-) with human apoprotein A1-transgenic

11 e our understanding of the genetic makeup of crossbred beef cattle and highlight their potential for

12 marbling score (CMAR) in a population of 493 crossbred beef cattle.

13 2) in crossbred beef females.

14 tion (AI) were conducted in 201 synchronized crossbred beef heifers.

15 antioxidants-added (controls) and Wagyu and crossbred beef pastes (14 days refrigerated storage).

16 ation study was conducted using a commercial crossbred broiler population (n = 1193).

17 praspinatus (SS) were obtained from ten Boer crossbred bucks (7-10 months old; 26.5 +/- 3.5 kg, BW).

18 Forty-four finishing crossbred bulls from two farms were examined.

19 ly linked to trypanotolerance and present in crossbred cattle living in trypanosomosis-infested areas

20 sis of Red Angus x Chinese Red Steppe (RACS) crossbred cattle, evaluating their genetic architecture,

21 es from Suino Nero Lucano (SNL) and a modern crossbred (CG) pig.

22 le from Suino Nero Lucano (SNL) and a modern crossbred (CG) pigs, before and after cooking and in vit

23 stein Friesian (Bos taurus) and Karan Fries (crossbred: cross of Bos indicus and Bos taurus) using (1

24 mmunostaining of cytoskeletal proteins and a crossbred D(1) receptor null:YFP transgenic reporter lin

25 Utilizing both purebred and crossbred data in animal genetics is widely recognized a

26 le purebred population and its corresponding crossbred descendant.

27 tered than mesocephalic, dolichocephalic and crossbred dogs.

28 educed odds of aural haematoma compared with crossbred dogs.

29 -cell lymphoma comprising 47 purebred and 13 crossbred dogs.

30 /-) and Rpe65(-/-) mice (n = 126) as well as crossbred Gnat1(-/-) mice lacking rod phototransduction

31 Retinas of Lrat(-/-), Rpe65(-/-), and crossbred Gnat1(-/-) mice tolerated prolonged high-dose

32 owing: parental lineage (purebred = 1 breed, crossbred >= 2 breeds), breed (n = 155), body size (larg

33 in-Friesian (HF), Holstein-Friesian x Jersey crossbreds (HF x J)) and season (spring, summer, autumn)

34 nal (small and large intestine) tissues from crossbred Holstein-Angus steers were analyzed.

35 rporation in spontaneous prostate tumors, we crossbred Id mutant mice with the transgenic adenocarcin

36 Linkage analysis of a crossbred informative pedigree showed five obligate reco

37 Importantly, when crossbred into a mouse genetic model of CHF (alpha-myosi

38 levels of orally administered tranilast, 36 crossbred juvenile pigs were randomized to placebo or tr

39 Crossbred Karan Fries (KF) cows, among the best yielders

40 Female crossbred Landrace/Yorkshire/Duroc pigs (27-32 kg).

41 In the current study, we have crossbred M83 mice on a DJ-1 null background (M83-DJnull

42 Steatohepatitis score of crossbred mice (ob/ob/ko) was similar to that of ob/ob m

43 Moreover, the crossbred mice are the best vertebrate model to date for

44 xidase system on activation of NF-kappaB, we crossbred mice deficient in p47(phox) with NF-kappaB rep

45 activation on amyloid pathology in vivo, we crossbred mice lacking CX3CR1 with the Alzheimer's mouse

46 ould increase survival in cardiomyopathy, we crossbred mice with Gq-associated cardiomyopathy and tho

47 e role of MYH9 deficiency in nephropathy, we crossbred Myh9-haploinsufficient mice (Myh9(+/-)) with H

48 esis in the absence of apoptosis in vivo, we crossbred NHEJ-deficient mice into a mutant p53R172P bac

49 among several purebred populations and their crossbred offspring populations limits the application o

50 O, RO and their nano-entities was noticed in crossbred pastes, while in Wagyu, nanoemulsions showed t

51 Several studies endeavor to predict the crossbred performance via the partial relationship, whic

52 nce models in a purebred Pietrain (PB) and a crossbred (Pietrain x Large White, CB) pig population wa

53 Crossbred piglets were assigned to three groups, intracr

54 thoracis muscle of 51 Apulo-Calabrese and 52 crossbred pigs differing in growth performances.

55 cass traits in 1039 individuals of a line of crossbred pigs.

56 principal component analysis (PCA), but the crossbred population showed the highest nucleotide diver

57 Seventy-six, crossbred, porcine reproductive and respiratory syndrome

58 ormation of intraneuronal tau inclusions, we crossbred previously described tau (T44) Tg mice overexp

59 A 2-month-old female crossbred puppy was submitted to necropsy with a history

60 The results revealed that the crossbred (RACS), Angus, and Red Angus breeds exhibited

61 igate the phenotype in a mammalian model, we crossbred SCA1 mice with mice over-expressing a molecula

62 Fifteen Brahman x Angus crossbred steers were fed one of three experimental diet

63 Eighteen juvenile male crossbred swine were included.

64 CAR at the surface of the airway epithelium, crossbred these mice with mice that were genetically dev

65 we generated L2-cyclin D1 (L2D1(+)) mice and crossbred these with p53-deficient mice.

66 participation of TLR7 in atherosclerosis, we crossbred TLR7-deficient (Tlr7 (-/-)) mice with apolipop

67 d PF4-driven tTA-viral protein 16 (VP16) was crossbred to a responder line.

68 ) ) and wild-type mice (WT, NOS3(+/+) ) were crossbred to generate homozygous NOS3(-/-) (KO), materna

69 e generated in the authors' laboratories and crossbred to generate Sur/SSTR5 KO mice.

70 The families are crossbred to maximize levels of heterozygosity and inclu

71 Single Ccl2- and Cx3cr1-deficient mice were crossbred to obtain Ccl2(-/-)/Cx3cr1(-/-) mice.

72 miR1-1 or miR1-2 knockout mice were crossbred to produce 75%-miR1-knockdown (75%KD; miR1-1(+

73 ay, PC(+/-)/FXI(-/-) mice were generated and crossbred to produce double-deficient progeny (PC(-/-)/F

74 Three erd-affected dogs were crossbred to three prcd-affected dogs, and their progeny

75 er facilitate bladder autoimmunity study, we crossbred URO-OVA mice with OVA-specific CD8(+) TCR Tg m

76 xidative stress reactions using ApoE-/- mice crossbred with 12/15LO-deficient (12/15LO-/-) mice (12/1

77 TSLP transgenic mice were crossbred with animals deficient for FcgammaRIIb on the

78 ically deficient in CD11c were generated and crossbred with apolipoprotein E (apoE)-/- mice to genera

79 Cystatin C-deficient mice (Cyst C-/-) were crossbred with apolipoprotein E-deficient mice (ApoE-/-)

80 ion were more dramatic in NEP2 knockout mice crossbred with APP transgenic mice.

81 epitopes in 12/15-lipoxygenase knockout mice crossbred with atherosclerosis-prone apo E-deficient mic

82 ptor-deficient (Tnsf10 or Tr(-/-)) mice were crossbred with ATP binding cassette subfamily B member 4

83 cle-specific PGC-1alpha transgenic mice were crossbred with cardiac-specific calsequestrin transgenic

84 Kit(+/copGFP) mice were crossbred with diabetic Lep(+/ob) mice to generate compo

85 A floxed synectin knockin mouse line was crossbred with endothelial-specific (Tie2, Cdh5, Pdgfb)

86 ls were counted from progeny of BDNF-OE mice crossbred with green fluorescent protein (GFP) (gustduci

87 Also, MIOX-KO were crossbred with Ins2 (Akita) to generate Ins2 (Akita)/KO

88 n of gangliosides, the GD3S mutant mice were crossbred with mice carrying a disrupted GalNAcT gene en

89 on of the disease, TSLP transgenic mice were crossbred with mice deficient for immunoglobulin-binding

90 When crossbred with mice lacking the other allele of tumor-su

91 shed cardiac function, Rab4 mutant mice were crossbred with mice overexpressing human beta2AR.

92 how (control) or high-fat diet (HFD) or were crossbred with mice that express human interleukin (IL)

93 heir wild-type littermates (CBS((+/+))) were crossbred with mice that overexpress GPx-1 [GPx-1((tg+))

94 irected expression of AC type VI (ACVI) were crossbred with mice with cardiomyopathy induced by cardi

95 oma formation with 30 - 40% penetrance, were crossbred with p27+/- mice for two successive generation

96 ntial, a strain of tal-1 transgenic mice was crossbred with p53-/- mice; the survival rate in these a

97 (+) T cells), and (ii) hk14-SCF Tg mice were crossbred with PMEL TCR transgenic mice without addition

98 admixture of the local Gudali breed and its crossbred with the Italian Simmental (Simgud) in three a

99 3 stabilize each other, SRC-3(-/-) mice were crossbred with the liver-specific transthyretin (TTR)-IG