ライフサイエンスコーパス: crus

1 zed structures called calcium release units (CRUs).

2 DHPRs) and RyRs within Ca(2+) release units (CRUs).

3 cial structures named calcium release units (CRUs).

4 ork of diffusively coupled Ca release units (CRUs).

5 t" (Ca sparks inducing Ca sparks in adjacent CRUs).

6 e full pressures, referenced to the anatomic crus.

7 e administered cisatracurium to paralyse the crus.

8 elate sphincteric pressure with the anatomic crus.

9 pproximately 3-fold lower than that of young CRUs.

10 (CRU) recruit new Ca sparks in neighbouring CRUs.

11 w intra-SR Ca diffusion and distance between CRUs.

12 ing of an ensemble of interacting stochastic CRUs.

13 circuit from cerebellar cortical areas Right crus 1 (Rcrus1) and posterior vermis through the cerebel

14 Greater Crus 1 and VIIIa volumes were also associated with highe

15 e cortex, caudate, orbitofrontal cortex, and crus 1 for bupropion.

16 ncluded the prefrontal cortex and cerebellar crus 1 for sertraline and the cingulate cortex, caudate,

17 positive interneurons to network dynamics in Crus 1 of mouse lateral cerebellar cortex during free wh

18 Purkinje cells (PCs) in Crus 1 represent whisker movement via linear changes in

19 a region in posterior cerebellum (lobule VII crus 1) was engaged specifically when a temporal-spatial

20 eria included three CRs, 12 unconfirmed CRs (CRus), 16 PRs, 26 with SD, four with PD, and 21 NA.

21 ribed morphological variations of cerebellar Crus 2 and pars triangularis allow to extend the traditi

22 associated with volumes in right cerebellar Crus 2 and pars triangularis of inferior frontal gyrus.

23 in the cerebellum (right lobule VI and left Crus 2) and neocortex (right medial OrbitoFrontal Cortex

24 o this end, multiple electrode recordings of crus 2a complex spike activity were obtained in awake ra

25 recordings of CSs and simple spikes (SSs) in crus 2a of anesthetized rats.

26 We used multiple electrode recording of crus 2a Purkinje cell complex spikes (CSs) in ketamine-x

27 x spike (CS) activity were obtained from 236 crus 2a Purkinje cells in anesthetized rats.

28 iple electrode recordings were obtained from crus 2a Purkinje cells, and carbenoxolone, a gap junctio

29 ); 2) stochastic Ca2+ release (or firing) of CRUs; 3) discrete, asymmetric distribution of CRUs along

30 the local SR Ca concentration of the firing CRUs above a critical level to sustain firing.

31 tracts involved in limbic circuitry (fornix crus [AD, beta = 0.02 (P = .046)] and cingulum [RD, beta

32 RUs; 3) discrete, asymmetric distribution of CRUs along the longitudinal (separation distance of 2 mi

33 ree semicircular canals; however, the common crus and ampullae housing the sensory tissue (crista) ar

34 regulators have on the intrinsic activity of CRUs and on the coupling between them.

35 dentate nucleus (DN), globus pallidus (GP), crus anterior of capsula interna (CA), and pons.

36 ccule, and the endolymphatic duct and common crus are invariably fused.

37 tion provides some isolation from neighbors, CRUs are not incommunicado.

38 Calcium release units (CRUs) are junctions between the sarcoplasmic reticulum (

39 Ca cycling model in which Ca release units (CRUs) are locally coupled by Ca diffusion throughout the

40 Logan graphical analysis with the cerebellum crus as a reference region.

41 a subsection of cerebellar gray matter (cere-crus) as well as a parametrically derived white matter-b

42 Rs) and triadin, two essential components of CRUs, but no RyRs (or feet).

43 r, and distance from the porus to the common crus (CC; P-CC) and posterior petrosal surface (PPS) to

44 reference regions (i.e., fusiform gyrus and crus-cerebellum) were significantly associated with symp

45 investigated motor fiber organization in the crus cerebri of the cerebral peduncle (ccCP) in the rhes

46 rona radiata (CR), internal capsule (IC) and crus cerebri of the cerebral peduncle (ccCP).

47 In skeletal muscle CRUs contain two isoforms of the sarcoplasmic reticulum

48 CRUs contain two proteins essential to e-c coupling: dih

49 Internal CRUs contained more RyRs in more clusters than CRUs on t

50 interior, respectively, although half of all CRUs contained only a single 'rogue' RyR.

51 pings of RyR clusters (Ca(2+) release units, CRUs), contained an average of 18 and 23 RyRs at the sur

52 re tightly packed along z-lines than surface CRUs, contained larger and more numerous RyR clusters, a

53 that expression of either isoform results in CRUs containing arrays of feet, indicating the ability o

54 A clusters of CRUs, each producing 10 pA simultaneously, can produce s

55 ing failure of endolymphatic duct and common crus formation, accompanied by epithelial dilatation and

56 ant weeds such as barnyardgrass [Echinochloa crus-galli (L.) P.

57 ng hexaploidization were observed between E. crus-galli and bread wheat.

58 three Echinochloa species (allohexaploid E. crus-galli and E. colona, and allotetraploid E. oryzicol

59 he wider row widths allowed for increased E. crus-galli densities.

60 Echinochloa crus-galli density was 120% greater in a 38-cm row width

61 As a result, methods that can alter E. crus-galli ecology are needed.

62 c and non-mimetic populations of Echinochloa crus-galli from the Yangtze River basin phenotypically a

63 hytoalexin momilactone A are found in the E. crus-galli genome, respectively.

64 rice cultivar and row widths on crop and E. crus-galli growth.

65 The hexaploid species Echinochloa crus-galli is one of the most detrimental weeds in crop

66 At the preharvest stage, E. crus-galli panicle counts were similar for the 13- and 1

67 Less E. crus-galli seed production occurred in competition with

68 tetraploid (E. oryzicola), and hexaploid (E. crus-galli) Echinochloa species.

69 Barnyardgrass (Echinochloa crus-galli) is a pernicious weed in agricultural fields

70 Barnyardgrass (Echinochloa crus-galli) is an invasive plant that is difficult to co

71 rice paddy weed, barnyard grass (Echinochloa crus-galli).

72 Overall, for E. crus-galli, as the row width increased, greater density,

73 genome sequence of the hexaploid species E. crus-galli, i.e., a 1.27 Gb assembly representing 90.7%

74 de the greatest ecological advantage over E. crus-galli.

75 etween the subgenomes in E. oryzicola and E. crus-galli.

76 ring capacity, and the spatial separation of CRUs help control the inherent instability of SR Ca2+ re

77 IS for all lobules in subregions VI and left crus I (p<0.05).

78 of multiple single units in cerebellar right Crus I (RCrus I) Purkinje cells (PCs) or dentate nucleus

79 ebellar abnormalities, particularly in Right Crus I (RCrusI), are consistently reported in autism spe

80 male youths, whereas white matter volumes in crus I and crus II and lobules VIIIA and VIIIB expanded

81 lum that were largely localized to bilateral Crus I and Crus II.

82 genetically younger parts of the cerebellum, crus I and crus II.

83 r cortical Golgi cells and Purkinje cells in Crus I and II of the posterior lobe cerebellar hemispher

84 een left PCC and right posterior cerebellum (Crus I and II) was reduced in both groups of children wi

85 ispheric lobule VI, and bilateral cerebellar crus I and II.

86 lysis provided evidence that left cerebellar Crus I and lobule VI contributed to ToM processing.

87 s of the cerebellar hemispheres bilaterally (Crus I and lobule VI).

88 erived primarily from reduced gray matter in crus I and lobules VI, VIIB, and VIIIA.

89 significant activation of cerebellar lobules Crus I and VI bilaterally related to the CS+ compared to

90 portantly, significant activation of lobules Crus I and VI was also present during the unexpected omi

91 bellar lesion volume, cerebellar Lobules VI, Crus I and VIIIa atrophy being independent predictors of

92 Greater Crus I and whole cerebellar volumes were associated with

93 able evidence implies that lobules IV-VI and Crus I are especially pertinent to pain processing, and

94 creased axon variability in lobule HV, while Crus I axons were unaffected.

95 high NEFH expression, while cognitive lobule Crus I contains PCs with low NEFH expression in postmort

96 on the effective connectivity of cerebellar crus I during visual attention.

97 The involvement of Crus I is consistent with an emerging picture in which i

98 that zone, consistent with the existence in crus I of olivo-cortico-nuclear microcomplexes.

99 tracers were microinjected into the folia of crus I of the cat cerebellum to investigate spatial loca

100 p = 0.003) and right (r = -0.71, p = 0.002) crus I of the cerebellum, and greater severity on the NP

101 Paravermal crus I sends the most connections to the VTA compared wi

102 n of the connections from V5 to PPC and from crus I to V5 by attention.

103 VIIIa, relative decrease of VIIIb and vermis Crus I volume) and white matter microstructure in the in

104 traction of total cerebellar, lobule IV, and Crus I volumes with additional X- or Y-chromosomes; X-sp

105 sensitivity of V5 populations to inputs from crus I was increased under attention.

106 in after tracer injection and the cerebellar crus I was used as the reference region.

107 motor vs. cognitive functions (lobule HV vs. Crus I) contain distinct PC populations characterized by

108 t superior cerebellum (hemispheric lobule VI/Crus I) impairs verbal working memory performance.

109 llum, specifically in hemispheric lobule VI, Crus I, and VIIb.

110 rdination were variably associated with HVI, Crus I, Crus II, HVII B and/or HIX.

111 es, including cognitive (lobules VI and VII, Crus I, frontoparietal and attention networks) and motor

112 nd three distinct regions of the cerebellum (Crus I, lobule VI, and lobules II/III) in male mice duri

113 functionally distinct cortical areas, within crus I, paraflocculus, and vermal regions IV/V and VI -

114 (p<0.001) and left lobule VI (p<0.01), left crus I, right VIIb and entire cerebellum (p<0.05 for eac

115 he cerebellar nuclei, lateral anterior lobe, crus I, rostral crus II, and lobule HVI ipsilateral to t

116 recordings were obtained from the D1 zone of crus I.

117 xist within each of the zones present within crus I.

118 obule HV were 2.2-fold thicker than those in Crus I.

119 e to the peripherally generated responses in Crus I.

120 w stimulation evokes a beam-like response in Crus I.

121 as primarily correlated with degeneration of Crus I.

122 erse kinematic transformation and sparing of Crus I.

123 ct cerebellar connectivity pattern, the left crus I/II appeared to be a common area with connectivity

124 S, anodal tDCS increased activation in right Crus I/II during semantic prediction and enhanced restin

125 l connectivity of executive function-related Crus I/II in the cerebellum was analysed.

126 Within the Crus I/II megacluster, specific cerebellar regions respo

127 ing activity in the human lateral cerebellar Crus I/II modulates the cerebral default mode network, w

128 al gyrus (z score = 4.31, P < .001), and the crus I/II of the cerebellum (z score = 3.77, P < .001),

129 tive sentences increased activation in right Crus I/II of the cerebellum.

130 t suppression of simple spike probability in crus I/II Purkinje cells.

131 deling, we hypothesize that right cerebellar Crus I/II supports prediction of upcoming sentence conte

132 A large megacluster extending across Crus I/II was consistently found with subregions linked

133 We found that activation within right Crus I/II was enhanced when semantic predictions were ma

134 y lower functional connectivity of the right Crus I/II with the left dorsolateral prefrontal cortex.

135 ght cerebellar lobules VI and VII (including Crus I/II) are engaged during a variety of language proc

136 erolateral portions of the cerebellum (right Crus I/II) contribute to language processing, but the na

137 owed marked atrophy in AD, ALS, FTD and PSP (Crus I/II), and MSA and PSP (lobules I-IV), respectively

138 ependent signal in the posterior cerebellum (Crus I/II).

139 ermis lobule VII or right lateral cerebellar Crus I/II, subregions that prominently couple to the dor

140 acking from parcels of cerebellar lobule VI, crus I/II, vermis, paravermis, and cerebrocerebellum.

141 nd T2 (r = 0.98) in the bilateral paravermal crus I/II.

142 onoyl glycerol in females only in cerebellar Crus I; and (3) increased dorsal hippocampus prostagland

143 Ca(2+) release units (CRUs), i.e. functional groupings of neighbouring RyR clu

144 indazole attenuated the vascular response to crus II activation in wild-type mice but not in D2-null

145 stimulation induces patch-like activation of Crus II and GABAA antagonists fail to convert this patch

146 , whereas white matter volumes in crus I and crus II and lobules VIIIA and VIIIB expanded faster in f

147 bing fibre responses were evoked in parts of crus II and paramedian lobule by stimulation of corticof

148 irs located mainly in the A2 and C1 zones in crus II and the paramedian lobule.

149 peripherally evoked patch-like responses in Crus II are aligned between parasagittal bands of EAAT4.

150 ce, upper lip stimulation increased BFcrb in crus II by 32 +/- 2%.

151 The field potentials evoked in crus II by upper lip stimulation did not differ between

152 put from Purkinje cells located primarily in Crus II of the ansiform lobule.

153 e show that the majority of granule cells in Crus II of the cerebrocerebellum receive sensory-evoked

154 tivity of neighboring individual MLIs in the Crus II region of awake female mice during two types of

155 Purkinje cells in Crus II showed continuous firing at relatively high rate

156 rily within the posterior lobe (lobule VIIB, crus II), vermis (VI, VIII), flocculonodular lobe (lobul

157 Crus II, a region of the cerebellar cortex that receives

158 clei, lateral anterior lobe, crus I, rostral crus II, and lobule HVI ipsilateral to the conditioned e

159 ffect the climbing fibre responses evoked in crus II, and produced a relatively small reduction of th

160 n were variably associated with HVI, Crus I, Crus II, HVII B and/or HIX.

161 hat gray matter volumes of lobules V and VI, crus II, lobule VIIB, and lobule X declined faster with

162 xpansion of total cerebellum, flocculus, and Crus II-lobule VIIIB volumes in males) and SCA (contract

163 or Y-chromosomes; X-specific contraction of Crus II-lobule VIIIB).

164 rea 46 project to granule cells primarily in Crus II.

165 re largely localized to bilateral Crus I and Crus II.

166 ers (EAATs) generates beam-like responses in Crus II.

167 younger parts of the cerebellum, crus I and crus II.

168 cell column, we recorded simultaneously from crus IIA areas and from left and right vermal lobule IX,

169 ranule cell layer (GCL) of cerebellar folium Crus IIa as freely moving rats engaged in a variety of n

170 of all four areas, i.e., the left and right crus IIA as well as the left and right lobule IX, can fi

171 ke synchrony observed between left and right crus IIA could indeed be mediated in part through couple

172 fractured tactile cerebellar map within the crus IIa folia of the cerebellar hemispheres reorganizes

173 the 1000-4500 Hz multiunit responses in the Crus IIa GCL of awake rats are correlated with tactile i

174 ossy fiber and climbing fiber projections to crus IIa in the lateral hemispheres of the rat cerebellu

175 mbing fiber activation of the left and right crus IIA in the rat can be explained by (1) bilateral in

176 mined in the tactilely responsive regions of crus IIa in the rat, the results show that SI influences

177 imultaneous recordings of the left and right crus IIA of the cerebellar cortex in the rat have demons

178 the tactile map in the granule cell layer of crus IIa reorganized, with representations of intact str

179 rents of the olivary regions that project to crus IIA was studied using Phaseolus vulgaris leucoagglu

180 First, more areas of crus IIa were nonresponsive in animals lesioned later in

181 e structural role of RyR3 in the assembly of CRUs in 1B5 cells independently expressing either RyR1 o

182 Special CRUs in cardiac muscle are constituted by SR domains bea

183 in three vineyards, representing different 'crus' in the cultivation areas of Barolo, Barbaresco and

184 cy of marrow competitive repopulation units (CRUs) increased approximately 2-fold from 2 months to 2

185 ayed afterdepolarizations, Ca release units (CRUs) interact with not just the immediately adjacent CR

186 The muscle fascicles of the right crus of diaphragm which form the esophageal hiatus are a

187 th achalasia had physical breaks in the left crus of the diaphragm CONCLUSIONS: Besides LES, the 3D p

188 Esophagus, LES, stomach, right and left crus of the diaphragm, and spine were segmented in each

189 tract-based spatial statistics suggested the crus of the fornix as a focus for this relationship.

190 ately 20 pA) through the Ca2+ release units (CRUs) of the sarcoplasmic reticulum (SR); 2) stochastic

191 Us contained more RyRs in more clusters than CRUs on the cell surface, and yielded longer duration Ca

192 alamus, right posterior cingulum, and fornix crus (seven studies; largest cluster, 980 voxels; z = 2.

193 fronto-occipital fasciculus, and left fornix crus (six studies; 323 voxels; z = 1.7; P = .001).

194 the canal pouch from the prospective common crus to a canal-like fate.

195 release and local recruitment of neighboring CRUs to fire more synchronously.

196 -SR Ca diffusion from neighboring non-firing CRUs to the firing CRUs, which helps to maintain the loc

197 anization and RyR2 orphaning (5000 of 20 000 CRUs) uncovered a local mechanism of delayed subcellular

198 ng, while the distal peak separated from the crus/upper-peak by 1.1 cm between FI and FE.

199 MH and NAWM were calculated using cerebellar crus uptake as a reference for both (11)C-PiB and (18)F-

200 ion-specific SUV ratios scaled to cerebellar crus uptake.

201 eract with not just the immediately adjacent CRUs via Ca diffusion, but also further CRUs via fast (

202 cent CRUs via Ca diffusion, but also further CRUs via fast ( approximately 0.1 ms) changes in Vm medi

203 ears of age, the BM homing efficiency of old CRUs was approximately 3-fold lower than that of young C

204 ceptible to RA treatment, whereas the common crus was particularly resistant, suggesting that the mol

205 One enzyme, CruS, was only distantly related to CrtD desaturases, wa

206 higher ratings for visualization of the left crus were associated with a decrease in hemorrhage rate

207 higher ratings for visualization of the left crus were associated with an increase in leak rate from

208 the spark frequency one would observe if the CRUs were incommunicado; 2), the coupling strength, whic

209 Internal CRUs were more tightly packed along z-lines than surface

210 ity, Ca(2+) sparks originating from internal CRUs were of longer duration than those at the surface.

211 a2+ currents through the Ca2+ release units (CRUs) were approximately 1-2 pA, producing sparks with p

212 oupling is governed by Ca(2+) release units (CRUs) whereby Ca(2+) influx via L-type Ca(2+) channels (

213 om neighboring non-firing CRUs to the firing CRUs, which helps to maintain the local SR Ca concentrat

214 diffusively coupled Ca(2)(+) release units (CRUs) with fixed refractory period.