1 ildren enrolled, 82 and 42 had rotavirus and cryptosporidial diarrhea, respectively.

2                          Among children with cryptosporidial diarrhea, those receiving LGG showed sig

3 nction following whole marrow HSCT may limit cryptosporidial disease, but survival was similar after

4 ovide a convenient and reproducible model of cryptosporidial disease, including its vicious cycle wit

5             However, the development of anti-cryptosporidial drugs is hindered by a lack of understan

6 cy in vivo to guide the optimization of anti-cryptosporidial drugs.

7 inal function in children with rotavirus and cryptosporidial gastroenteritis emphasizes the role of p

8  Differences in susceptibility to persistent cryptosporidial infection between two strains of adult a

9 erapeutics against the virtually untreatable cryptosporidial infection in immunocompromised patients.

10                             We conclude that cryptosporidial infection in protein-deficient mice can

11 alization or gene knockout, experience heavy cryptosporidial infection that may lead to death.

12                           In vitro models of cryptosporidial infection using a human biliary epitheli

13                           In vitro models of cryptosporidial infection using human biliary and intest

14 h CD4 T cells are required for recovery from cryptosporidial infection, mice with severe combined imm

15 ttled drinking water) could prevent or delay cryptosporidial infections among children residing in an

16 irst patients with IL-21R deficiency who had cryptosporidial infections associated with chronic chola

17 ed kindreds, with two patients each, who had cryptosporidial infections associated with chronic chola

18 nd clinical outcomes in Indian children with cryptosporidial or rotavirus diarrhea.

19 ng, branched microvilli clustered around the cryptosporidial vacuole.