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1  of an active form of XBP1 caused atrophy in cultured myotubes.
2 e inhibition of UPR causes severe atrophy in cultured myotubes.
3 gulating mitochondrial oxidative function in cultured myotubes.
4 vated AMPK and stimulated lipid oxidation in cultured myotubes.
5 e receptor (AChR) clusters on the surface of cultured myotubes.
6 pitulated by respiratory chain inhibition in cultured myotubes.
7 prevents lipid-induced insulin resistance in cultured myotubes.
8 ation and the dispersion of AChR clusters in cultured myotubes.
9 logic inhibition of PERK leads to atrophy in cultured myotubes.
10 can (rhBGN) increases utrophin expression in cultured myotubes.
11 on of the illuminated AChRs from clusters on cultured myotubes.
12 e and HDAC4 knockdown enhances glycolysis in cultured myotubes.
13 ay arise from the NMJ basement membrane with cultured myotubes.
14 and cardiac muscles, murine limb muscle, and cultured myotubes.
15 ression in mouse limb muscles in vivo and in cultured myotubes.
16 -4) inhibit agrin-induced AChR clustering on cultured myotubes.
17  calcium in agrin-induced AChR clustering on cultured myotubes.
18 on associated with AChR cluster dispersal in cultured myotubes.
19 s and triggers formation of AChR clusters on cultured myotubes.
20 te, but not PTH, induced FGF23 expression in cultured myotubes.
21 p62 aggregation or TDP-43 mislocalisation in cultured myotubes.
22                                           In cultured myotubes, Abl kinase activity was required for
23                                           In cultured myotubes, agrin stimulates the rapid phosphoryl
24  agrin and neuregulin-1 (Nrg-1) signaling in cultured myotubes and developing muscle fibers in vivo.
25 nduced reactive oxygen species production in cultured myotubes and improved insulin-stimulated glucos
26 mor-induced muscle catabolism and wasting in cultured myotubes and in mice.
27  endogenous AChR beta-subunit transcripts in cultured myotubes and in vivo, and this binding is incre
28  number of lipid-challenged models including cultured myotubes and isolated muscles strips incubated
29 ored whether MTX promotes AMPK activation in cultured myotubes and isolated skeletal muscle.
30                                              Cultured myotubes and primary brown adipocytes treated w
31 Knockdown (KD) of Mettl21e led to atrophy of cultured myotubes, and targeted mutation of Mettl21e in
32 tes was comparable between muscle tissue and cultured myotubes, and temporal lipid profiles correlate
33                                           In cultured myotubes, APP synergistically increases agrin-i
34 s increased in skeletal muscle tissue and in cultured myotubes basally and in response to insulin in
35 al muscle by hindlimb ischemia (HLI), and in cultured myotubes by hypoxia, suggesting a potential rol
36 e readily extracted from the cell surface of cultured myotubes by non-ionic detergent.
37                Addition of agonist scFv to a cultured myotube cell line induced AChR clustering and t
38                        Silencing of Perm1 in cultured myotubes compromises respiratory capacity and d
39                                           In cultured myotubes, CTRP9 specifically activates AMPK, Ak
40                                              Cultured myotubes derived from two patients with GFPT1 m
41 maging of acetylcholine receptors (AChRs) in cultured myotubes differentiated ex vivo from immortaliz
42 iber composition, show little resemblance to cultured myotube enhancers, and identify glycolytic and
43  exposure on glucose and lipid metabolism in cultured myotubes established from people with normal gl
44                                              Cultured myotubes exposed to high CO2 had reduced fiber
45 as supported by elevated oxidative stress in cultured myotubes exposed to palmitate in the presence o
46 mparisons with changes in gene expression in cultured myotubes following treatment with a non-damagin
47            Studies were conducted in primary cultured myotubes from beta1 knockout (KO), ryanodine re
48  the levels of GLUT4 ( approximately 50%) in cultured myotubes from C57BL6 mice.
49 ell as IL-6 secretion, was unaltered between cultured myotubes from normal glucose tolerant or type 2
50 f transcription 3 (STAT3) phosphorylation in cultured myotubes from normal glucose tolerant subjects.
51 athies, the relatively relaxed zebrafish and cultured myotubes from patients with RYR1-related myopat
52         Both in transfected COS cells and in cultured myotubes from rapsyn-negative and rapsyn-positi
53 tractility and Ca(2+) release from the SR of cultured myotubes from Stac3 mutant mice could be restor
54                                           In cultured myotubes, IL-6 promoted muscle degradation via
55 hen TrkB-mediated signaling was disrupted in cultured myotubes in the absence of motor nerve terminal
56 rs (AChRs) at laminin-associated clusters on cultured myotubes in the absence or presence of the nerv
57               Inhibition of ERK signaling in cultured myotubes increased slow-twitch fiber-specific p
58 y, forced expression of Gadd45a in muscle or cultured myotubes induces atrophy in the absence of upst
59                                           In cultured myotubes, inhibition of ERK, but not Jun NH2-te
60 e acetylcholine receptor (AChR) synthesis in cultured myotubes, is a member of the neuregulin family
61 te and PTH elevations on FGF23 expression in cultured myotubes isolated from mice and CKD patients.
62         Thus, in muscles in vivo, but not in cultured myotubes, neural agrin promotes the recycling o
63 Recombinant human IL-6 (rhIL-6) treatment of cultured myotubes only minimally increased SOCS3, howeve
64  was used to monitor Ca2+ signals in primary-cultured myotubes, prepared from forelimbs of wild-type
65             Agrin-induced AChR clustering in cultured myotubes proceeds via the initial formation of
66 conditioned medium collected from WT primary cultured myotubes promoted excess lipid accumulation in
67         Addition of soluble TWEAK protein to cultured myotubes reduced the mean myotube diameter and
68        Gain- and loss-of-function studies in cultured myotubes show that regulation of MuSK by PDZRN3
69                      Immunohistochemistry of cultured myotubes shows that not only is the beta 1 subu
70                                     Like the cultured myotubes, these tissues accumulated ceramides b
71                                           In cultured myotubes, Tid1 knockdown inhibited AChR cluster
72                                  Exposure of cultured myotubes to CKD serum resulted in myotube atrop
73                            Exposure of C2C12 cultured myotubes to either high glucose concentration,
74                        Here, we show that in cultured myotubes undergoing atrophy, the activity of th
75 hR density at agrin-induced AChR clusters in cultured myotubes via PI3 kinase acting through GSK3beta
76 ed aggregation of acetylcholine receptors on cultured myotubes was completely blocked by antibodies t
77                                           In cultured myotubes, we determined that alphaLNNd expressi
78  was used but not when nuclear extracts from cultured myotubes were used.
79 so coordinately regulated on the surfaces of cultured myotubes where MuSK and AChRs colocalize both i
80                                     Treating cultured myotubes with LDL containing ceramide promoted