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1 when in a low ratio (10%-20%) relative to co-cultured tumor cells.
2 optosis and suppressing the proliferation of cultured tumor cells.
3 with respect to EC20 was then determined in cultured tumor cells.
4 duced under hypoxic conditions in tumors and cultured tumor cells.
5 only approximately 2% of expressed genes in cultured tumor cells.
6 d displayed on oil drops induce apoptosis of cultured tumor cells.
7 alous circadian function in tumor models and cultured tumor cells.
8 eral breast cancer cell lines and in ex vivo cultured tumor cells.
9 ncharacterized mutations present only in the cultured tumor cells, a subset of which has been reporte
11 duces proteasome inhibition and apoptosis in cultured tumor cells and cancer cells from acute myeloid
12 factor-1 expression and promoter activity in cultured tumor cells and HaCaT keratinocytes, as well as
16 eported that overexpression of the enzyme in cultured tumor cells and mice activates metabolic flux t
17 hamide potentiated oncolytic effects against cultured tumor cells and subcutaneous tumor xenografts e
18 w that p300 is susceptible to acetylation in cultured tumor cells and that its acetylation status is
19 l spread at later stages, as demonstrated in cultured tumor cells, and in A549 and PC-3 solid tumor x
20 hat testisin was localized on the surface of cultured tumor cells as a glycosyl-phosphatidylinositol-
22 proliferation, viability and invasiveness of cultured tumor cells, as well as the growth rate and met
23 ted anticancer prodrug whose toxicity toward cultured tumor cells can be potentiated up to 100-fold b
25 expression between the solid tumors and the cultured tumor cells correlate with the expression of ga
28 results from the prior Hip1 mutant mice, we cultured tumor cells from homozygous Delta 3-5 allele-be
29 1 or PV mainly in the nuclear compartment of cultured tumor cells from TRbeta(PV/PV) mice, but cytopl
30 by quantitative reverse transcriptase PCR in cultured tumor cells in vitro and in a mouse xenograft m
32 6His protein did not affect the viability of cultured tumor cells, indicating that the antitumor effe
34 Free UTO was significantly more toxic to cultured tumor cell lines than the clinically used anthr
36 erties of 99mTc-HYNIC-folate were studied in cultured tumor cells that overexpress the folate recepto
38 th rate of rise (dB/dt) in the msec range to cultured tumor cells to assess whether this affects memb
39 vo, and they raise concerns about the use of cultured tumor cells to test the efficacy of Shh pathway
41 tor-mediated uptake of 99mTc-HYNIC-folate by cultured tumor cells was approximately 300 times higher