ライフサイエンスコーパス: cupric

1 emonstrated that this peak is insensitive to cupric A(x) and A(y) hyperfine interaction.

2 in the presence of either palladium acetate/cupric acetate catalytic system under oxygen atmosphere

3 se experiment, levels of (64)Cu from [(64)Cu]cupric acetate decreased from 4 to 24 h postadministrati

4 to 4 and then manganic triacetate dihydrate/cupric acetate induced radical cyclization, gave 1-subst

5 Cupric acetate is the copper source, and triethylamine b

6 A cupric acetate solution was used to produce the colorime

7 different from that for (64)Cu from [(64)Cu]cupric acetate.

8 ance (ENDOR) of protons at Type 2 and Type 1 cupric active sites correlates with the enzymatic pH dep

9 se (PHM) with two coppers that cycle through cupric and cuprous oxidation states.

10 were diazotized with tert-butyl nitrite and cupric chloride to furnish the isomerically pure 5-chlor

11 ble form of copper such as cupric sulfate or cupric chloride.

12 The metal-catalyzed oxidation (ascorbate/cupric chloride/oxygen) of recombinant human relaxin (rh

13 arged, coordinatively unsaturated trinuclear cupric cluster.

14 tone H3 cysteine 110 (H3C110) contributes to cupric (Cu(2+)) ion binding and its reduction to the cup

15 be controlled in vivo to avoid formation of cupric CzrA.

16 binuclear center in the "as-isolated" ferric/cupric enzyme is sluggish and without linkage to proton

17 y oxidants leading to the elimination of the cupric EPR signal consistent with formation of an antife

18 erizations revealed the presence of cuprous, cupric, ferrous, and ferric ions in the CP mineral.

19 zotization in the presence of an appropriate cupric halide.

20 reminiscent of plastocyanin, but the Type 1 cupric HOMO ground-state electronic g value and copper h

21 t charge at the adjacent metal site from +1 (cupric hydroxide) in wild-type enzyme to +2 (cobaltous H

22 or contains a benzenesulfonamide prong and a cupric iminodiacetate (IDA-Cu(2+)) prong separated by li

23 ed by oxidation at Calpha by the neighboring cupric ion and cleavage of the Calpha-C(O) bond to give

24 y the presence of chelators, suggesting that cupric ion had previously bound to these LPSs.

25 The participation of cupric ion in the oxidation process was demonstrated in

26 gnals are attributed to type 2 Cu2+ in which cupric ion is bound to four (less likely three) nitrogen

27 Cupric ion may also be taken up, but those processes are

28 cavenging, lipid peroxidation inhibition and cupric ion reducing activities of different fractions we

29 ntent (TPC), ascorbic acid (AA) content, and cupric ion reducing antioxidant capacity (CUPRAC) were d

30 ng from differences in quenching rate by the cupric ion.

31 ciated with the axial position of the Type-1 cupric ion.

32 It was found that cupric ions (Cu(2+)) were drastically reduced at pH > 7.

33 Dosed cupric ions acted as a disinfectant in stratified stagna

34 It was shown that ferric and cupric ions are effective oxidants of TEMPONE-H.

35 Our findings bring forth the role of cupric ions as an essential cofactor in selective TRAAK-

36 s and their respective primary antibodies by cupric ions at high pH.

37 However, cupric ions did not impair CzrA-DNA complex formation bu

38 ed to a linker sequence at the N-terminus to cupric ions embedded in a polyethylene-glycol-coated gla

39 imately proportional to the concentration of cupric ions in the medium, but increased more rapidly in

40 trophotometric method, based on reduction of cupric ions in the presence of cuproine complex, with a

41 ve inhibition of the proton transfer step by cupric ions in wild-type CA XII.

42 During distillation, cupric ions may catalyse the conversion of cyanide to EC

43 When the pattern of cupric ions on a complex matches the surface pattern of

44 Here, we discover cupric ions selectively modulate the binding of phosphat

45 unction of p97 in the cytoplasm by releasing cupric ions under oxidative conditions, which disrupt th

46 cytosolic NADPH to extracellular ferric and cupric ions via a FAD and heme-dependent pathway.

47 Ferrous, ferric, and cupric ions were also effective catalysts, indicating th

48 d to influence the redox potential of Type-1 cupric ions.

49 han does presence of copper dosed as soluble cupric ions.

50 e moroidin bicyclic motif in the presence of cupric ions.

51 The vast majority of flavonoids reduced cupric ions; their behaviour ranged from progressive gra

52 The activity was stimulated by ferric and cupric metal ions in addition to the cytochrome b-specif

53 disulfide bond formation in the presence of cupric orthophenanthroline.

54 Cupric oxide (CuO) and nickel oxide (NiO) showed signifi

55 attice exchange in the improper multiferroic cupric oxide (CuO) creates electromagnons at substantial

56 etallic copper to cuprous oxide (Cu(2)O) and cupric oxide (CuO), in addition to the formation of othe

57 bromide-containing waters in the presence of cupric oxide (CuO).

58 re an electromagnon excitation in monoclinic cupric oxide (CuO).

59 (SPH) relationships are established for nano-cupric oxide (n-CuO) as a function of shape, including n

60 We used cupric oxide ligninolysis coupled with gas chromatograph

61 tudy demonstrates the intrinsic abilities of cupric oxide nanoparticles (CuO-NP) towards arsenic adso

62 Dual-functional cupric oxide nanorods (CuONRs) as peroxidase mimics are

63 mg beta-carotene, 80 mg zinc oxide, and 2 mg cupric oxide per day was instituted on study day 2.

64 orms silicon carbide compounds in the heated cupric oxide reactor, rather than forming silicon dioxid

65 C, vitamin E, beta-carotene, and zinc (with cupric oxide) is recommended for AMD but not cataract.

66 no copper, 40% contained the poorly absorbed cupric oxide, and < 30% contained a highly bioavailable

67 Metallic copper (Cu), cupric oxide/hydroxide (Cuox), and copper sulfide (CuxS)

68 nce and for cross-linking in the presence of cupric-phenanthroline by SDS-PAGE and Western blot analy

69 study, Au nanoneedles are impregnated into a cupric porphyrin-based metal-organic framework by exploi

70 in either mercury-polyvinyl alcohol (PVA) or cupric PVA.

71 e methodology (RSM), was evaluated using the Cupric Reducing Antioxidant Capacity (CUPRAC) method.

72 Antioxidant activity was assessed using Cupric Reducing Antioxidant Capacity and DPPH radical sc

73 berries of Rubus and Ribes genera, had high cupric reducing antioxidant capacity, comparable with th

74 say, beta-carotene/linoleic acid, ferric and cupric reducing power.

75 A cupric reductase activity is also increased in copper-de

76 +) abundance, through attenuation of histone cupric reductase activity or depletion of total cellular

77 Thus, CcoG is a bacterial cupric reductase and a founding member of a widespread c

78 al characteristics of the regulation of this cupric reductase are compatible with its involvement in

79 dase (cbb (3)-Cox) assembly factor CcoG as a cupric reductase that binds Cu via conserved cysteine mo

80 e was regulated by copper and could act as a cupric reductase.

81 ns suggest they are physiologically relevant cupric reductases and ferrireductases in vivo.

82 face exposed metalloreductases, but specific cupric reductases have not been identified in bacteria.

83 teap4, are not only ferrireductases but also cupric reductases that stimulate cellular uptake of both

84 days from weaning were evaluated with amino cupric silver neurodegeneration stain.

85 Using the amino-cupric silver stain on brain sections from canine narcol

86 generating neurons was found using de Olmos' cupric silver stain.

87 limited seizure activity (stage 1); however, cupric-silver and Fluoro-Jade B stains revealed signific

88 ed for histological evaluation utilizing the cupric-silver neurodegeneration stain, immunohistochemis

89 Amino-cupric-silver staining demonstrated degenerative changes

90 taneous mutant model of human NP-C, by amino-cupric-silver staining, showed that the terminal fields

91 milar, indicating that the structures of the cupric sites, and the spin density distributions onto th

92 n was also observed when cuprous SOD but not cupric SOD was added to a H(2)O(2) solution.

93 consistently undersaturated with respect to cupric solid phases.

94 n reduction of an initially added air-stable cupric species (Cu(II)/Ligand).

95 uttles via a metastable but activated ferric/cupric state (O(H)), which may decay into a more stable

96 ents have shown that reduction of the ferric/cupric state of the enzyme's binuclear heme a3/CuB cente

97 a fibers formed by hydrodynamic injection of cupric sulfate into silicate solution.

98 a highly bioavailable form of copper such as cupric sulfate or cupric chloride.

99 Previous efforts to synthesize a cupric superoxide complex possessing a thioether donor h

100 The central role of cupric superoxide intermediates proposed in hormone and

101 nded [( (X1)(X2) TMPA)Cu(II) (O(2) (.) )](+) cupric superoxide species was achieved, and they were ch

102 drastic enhancement in the reactivity of the cupric superoxide towards phenolic substrates as well as

103 The latter is the first reported cupric superoxide with an experimentally proven Cu-S bon

104 f a dioxygen adduct with [LCu(I)][B(C6F5)4], cupric superoxo complex [LCu(II)(O2(*-))]+ (1) (L = TMG3

105 ity patterns for copper(I)-O2 adducts, a new cupric-superoxo complex [(DMM-tmpa)Cu(II)(O2(*-))](+) (2

106 2,5-benzimidazole)-modified vanadium dioxide-cupric tungstate (VO(2)-CuWO(4)) as an efficient photose