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1 entions were required in 18 patients (16 for cyanosis).
2 nasal congestion, fever, chest indrawing, or cyanosis.
3 , death or unplanned reintervention to treat cyanosis.
4 , was homebound due to dyspnea and worsening cyanosis.
5 ms of paroxysmal cough, whooping, apnea, and cyanosis.
6 ve chronotropic agents, age, and presence of cyanosis.
7 mutations are a rare but important cause of cyanosis.
8 ysfunction, failed Fontan physiology, and/or cyanosis.
9 rt surgery is dependent on age and degree of cyanosis.
10 PAVM in patients with CPA may cause profound cyanosis.
11 signs are tachypnea, tachycardia, rales, and cyanosis.
12 nt pulmonary arteriovenous malformations and cyanosis.
13 sleep abnormalities, plastic bronchitis, and cyanosis.
14 2.0 [95% CI, 1.0-4.1]; P=0.05) and clinical cyanosis (adjusted odds ratio, 5.0 [95% CI, 1.8-13.4]; P
17 hose with worse physiological stage, such as cyanosis and pulmonary hypertension, whereas anatomic co
18 ation are still unclear, extended periods of cyanosis and pulmonary overcirculation may adversely imp
20 /or Fontan physiology, 87 (8%) patients with cyanosis, and 73 (7%) patients with pulmonary hypertensi
24 They can alleviate obstructions and improve cyanosis by enhancing pulmonary blood flow and oxygen sa
26 Association class, ventricular function and cyanosis: complement system (C1QA, r=0.91), mitochondria
30 indrawing) or very severe pneumonia (central cyanosis, difficulty breastfeeding/drinking, vomiting ev
31 g from supplemental oxygen or flow, apnea or cyanosis during the present illness, neurologic impairme
32 ular function, exercise ability, progressive cyanosis from pulmonary arteriovenous malformations, sys
33 inished ventricular function, arrhythmias or cyanosis, have extracardiac comorbidities, and face addi
34 f death or unplanned reintervention to treat cyanosis (hazard ratio, 0.8; 95% confidence interval [CI
35 ession correlated inversely with severity of cyanosis ie, preoperative hematocrit (P<0.01) and positi
39 splant, or unplanned reintervention to treat cyanosis occurred in 25.7% of DAS and 35.8% of BTS, P=0.
40 daptation, including respiratory difficulty, cyanosis on feeding, and jitteriness (relative risk, 8.7
42 f Fallot (TOF) typically results in clinical cyanosis or volume overload of the left ventricle (LV),
43 r an infant with severe PS will present with cyanosis or without symptoms are not well understood.
44 length of intubation (P=.0009), preoperative cyanosis (P=.002), and younger age at surgery (P=.01).
46 ure, which is accompanied by hypotension and cyanosis, pericardial effusion, low voltage on the elect
47 enrichment for embryonic development and the cyanosis phenotype, a clinical manifestation of BPD.
49 an procedure, including ventricular failure, cyanosis, protein-losing enteropathy, thromboembolism, a
50 ated with death included male sex, diabetes, cyanosis, pulmonary hypertension, renal insufficiency, a
51 ation, anatomic Fontan obstruction, clinical cyanosis (pulse oximetry <90%), polycythemia, portal var
52 f interstitial fibrosis (rales, clubbing, or cyanosis) raised the risk of subsequent death from asbes
55 associated with worsening heart failure and cyanosis, suggesting their potential utility as biomarke
57 is possibly due to the difficulty detecting cyanosis that is masked by naturally occurring dark skin
58 27 infants) with or without hypoxic stress (cyanosis) undergoing open heart surgery with cold-crysta
59 ed differently, depending on the presence of cyanosis, use of rate-lowering medications, and achieved
60 features (breathlessness, sleep disturbance, cyanosis, venous dilatation, paresthesia, headache, and
62 lure was present in 22 patients, significant cyanosis was present in 7, and protein-losing enteropath
63 g, wheezing, chest indrawing, and/or central cyanosis]) were tested for RSV infections using real-tim