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1              Twelve patients (75%) displayed cyanotic acral edema of the extremities.
2                                              Cyanotic acral edema was present in 75% of our patients,
3 proatrial natriuretic peptide (proANP) in 26 cyanotic adults and 28 noncyanotic control subjects.
4 iac surgery and the multisystem disorders of cyanotic adults.
5  surgical repair of TOF from 23 patients: 13 cyanotic and 10 acyanotic.
6 enic factor CCBE1 or VEGFR3 function, appear cyanotic and die shortly after birth due to failure of l
7                     Both had erythematous to cyanotic and hyperplastic gingiva.
8 become immobile, breathe irregularly, appear cyanotic, and die within an hour.
9 cally stable and without a hemoglobinopathy, cyanotic cardiac condition, or severe hypoxemia, the int
10 justed rate ratio, 95% confidence interval): cyanotic CHD (6.44, 3.95-10.50), endocardial cushion def
11 ailed prenatal or early newborn detection of cyanotic CHD (CCHD) can have disastrous consequences.
12 and tetralogy of Fallot, two common forms of cyanotic CHD lesions, as well as dilated and hypertrophi
13 ngitudinal observational study, fetuses with cyanotic CHD underwent late gestational fetal cardiovasc
14 ime, the accurate detection of non-syndromic cyanotic CHD using maternal salivary metabolomics.
15                  A total of 144 fetuses with cyanotic CHD were assessed.
16                                 Infants with cyanotic CHD were identified.
17                           Among fetuses with cyanotic CHD, diminished fetal combined ventricular outp
18  indicating significant lipid dysfunction in cyanotic CHD.
19 developmental (ND) outcomes in patients with cyanotic CHD.
20 HW, 1393 NHB, and 1920 Hispanic infants with cyanotic CHD.
21 renatal detection of isolated, non-syndromic cyanotic CHD.
22                                              Cyanotic children showed a higher peak troponin-T level
23 is did not influence outcome in infants, but cyanotic children showed worse reperfusion injury and cl
24 Tetralogy of Fallot (TOF) is the most common cyanotic congenital heart defect.
25                            Ten patients with cyanotic congenital heart defects and 10 control subject
26 on were upper airway obstruction (13.5%) and cyanotic congenital heart disease (11.5%).
27                                 Fetuses with cyanotic congenital heart disease (CHD) exhibit profound
28     Between 1988 and 1995, 162 patients with cyanotic congenital heart disease (mean age 37 years, ra
29 g nonlinear mixed effects model, presence of cyanotic congenital heart disease (odds ratio, 7.35; p <
30 igned infants 92 days of age or younger with cyanotic congenital heart disease and a systemic-to-pulm
31 erebrovascular events in adult patients with cyanotic congenital heart disease and to evaluate any co
32                                  Adults with cyanotic congenital heart disease are at risk of having
33                                  Adults with cyanotic congenital heart disease are characterized by i
34                             Many adults with cyanotic congenital heart disease are characterized by r
35 he recommendation that routine screening for cyanotic congenital heart disease be added to the panel
36 and BNP are markedly elevated in adults with cyanotic congenital heart disease despite reduced body w
37 ascular events are a serious complication of cyanotic congenital heart disease in infants and childre
38 Tetralogy of Fallot (TOF) is the most common cyanotic congenital heart disease in Taiwan.
39                 Pulse oximetry screening for cyanotic congenital heart disease is specific, sensitive
40 Chronic hypoxia (CH) present in infants with cyanotic congenital heart disease may be responsible for
41          Clopidogrel therapy in infants with cyanotic congenital heart disease palliated with a syste
42                                 Infants with cyanotic congenital heart disease palliated with placeme
43 Tetralogy of Fallot (TOF) is the most common cyanotic congenital heart disease, and sudden cardiac de
44 ble increase in basal coronary blood flow in cyanotic congenital heart disease, flow reserve remains
45 iteria were trainees intubating infants with cyanotic congenital heart disease, known or suspected di
46           Patients with extreme prematurity, cyanotic congenital heart disease, pulmonary hypertensio
47 echanism for preservation of flow reserve in cyanotic congenital heart disease.
48 e success story in the surgical treatment of cyanotic congenital heart disease.
49  toward treating microcytosis in adults with cyanotic congenital heart disease.
50 utflow tract (OFT) are an important cause of cyanotic congenital heart disease.
51 Nkx2-5(Deltaenh/Deltaenh) mice recapitulated cyanotic conotruncal defects seen in patients with NKX2-
52  who undergo heart surgery have a congenital cyanotic defect in which the heart is chronically perfus
53 ho undergo cardiac surgery have a congenital cyanotic defect where the heart is chronically perfused
54                              Noncyanotic and cyanotic defects were classified as simple and severe, r
55 CHD categories (with septal, obstructive and cyanotic defects).
56 rgery do not take into consideration age and cyanotic differences.
57   Tetralogy of Fallot (TOF) is the commonest cyanotic form of congenital heart disease.
58    Tetralogy of Fallot (TOF)-the most common cyanotic heart defect in newborns-has evidence of multip
59                        In human infants with cyanotic heart defects, PKCepsilon, p38 MAP kinase, and
60     Lack of past cardiac surgery (p = 0.04), cyanotic heart disease (p = 0.03), and early postoperati
61 ffered to patients with a variety of complex cyanotic heart disease at younger ages, and has resulted
62 ry syncytial virus infection, or presence of cyanotic heart disease or residual right-to-left intraca
63                       Female sex and complex cyanotic heart disease were significant risk factors for
64 DS criteria more than 24 h before screening, cyanotic heart disease, active perinatal lung disease, a
65 Fallot, transposition of the great arteries, cyanotic heart disease, Ebstein anomaly, and Fontan circ
66 diopathic pulmonary hypertension, congenital cyanotic heart disease, morbid obesity associated with s
67                          In patients without cyanotic heart disease, worse daily fluid overload % pre
68 , 1.02-1.08) after adjusting for presence of cyanotic heart disease.
69 suscitation events occurred in patients with cyanotic heart disease.
70 T angiography in the diagnosis of congenital cyanotic heart diseases.
71  decision-making in patients with congenital cyanotic heart diseases.
72 phometric or hemodynamic differences between cyanotic infants with critical PS and asymptomatic infan
73                        In such severely ill, cyanotic infants, irregularities, however slight, may ha
74                 Women and those with complex cyanotic lesions are at particular risk, as patients may
75               Two children (2/558, 0.4%) had cyanotic lesions.
76 bilaterally localized distal erythematous or cyanotic lesions.
77 sk is primarily represented by patients with cyanotic or left heart obstructive lesions.
78        Plasma proANP levels were elevated in cyanotic patients compared with control subjects (1828+/
79 here was no difference in oxytocin levels in cyanotic patients compared with control subjects (P=0.49
80                                              Cyanotic patients had a significantly lower expression o
81                                         Four cyanotic patients had evidence of iron deficiency.
82                                              Cyanotic patients had lower mean right atrial pressures
83                                              Cyanotic patients had reduced body water compared with c
84 tion and hematocrit significantly greater in cyanotic patients than in control subjects (82+/-6 versu
85 old (P=0.02) and 12-fold (P=0.03) greater in cyanotic patients than in control subjects.
86                                              Cyanotic patients were excluded to avoid confounding fro
87                                          The cyanotic patients were younger at surgery compared with
88 hest potential for vascular injury: younger, cyanotic patients with longer pump times and longer post
89 2)) slope was not predictive of mortality in cyanotic patients.
90 ein kinases (MAP kinases) in 15 infants with cyanotic (SaO2<85%) or acyanotic (SaO2>95%) heart defect
91 ent animals and found that these mice become cyanotic shortly before death because of lung maturation
92 ed generalized swelling of the third finger, cyanotic skin, and fingernail splitting on the second fi
93 Caesarean section at embryonic day 18.5 were cyanotic, suffered from respiratory distress, and failed
94       Clinical presentation can range from a cyanotic toe to a diffuse multiorgan systemic disease th