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1 ination by the deubiquitinases A20 and CYLD (cylindromatosis).
2 complex (LUBAC) and the deubiquitinase CYLD (cylindromatosis).
3 und to TRAF6 and the deubiquitinating enzyme cylindromatosis.
4 tumor suppressor that is mutated in familial cylindromatosis.
5 susceptibility, incontinentia pigmenti, and cylindromatosis.
6 25 tumours from 4 individuals with familial cylindromatosis.
7 that should be considered in the context of cylindromatosis.
8 me that is altered in patients with familial cylindromatosis, a condition characterized by numerous b
9 a tumour suppressor gene mutated in familial cylindromatosis, a genetic disorder leading to the devel
10 a tumor suppressor gene mutated in familial cylindromatosis, an autosomal dominant predisposition to
11 tumor suppressor that is mutated in familial cylindromatosis, an autosomal dominant predisposition to
13 nd validated the function two DUBs including cylindromatosis and ubiquitin-specific protease-34 that
14 which causes a benign human syndrome called cylindromatosis, as a key negative regulator for NF-kapp
17 und for the first time that tumor suppressor cylindromatosis (CYLD) acts as a negative regulator for
18 tudies that identify deubiquitinating enzyme cylindromatosis (CYLD) as a key regulator for T cell dev
21 chymal cell (LPC)-specific disruption of the cylindromatosis (CYLD) lysine 63 deubiquitinase gene (Cy
22 In addition, the deubiquitination enzyme Cylindromatosis (CYLD) negatively regulates the activity
23 s end, we have identified the deubiquitinase cylindromatosis (CYLD) to be a regulator of PARIS protei
25 in a backcross panel of mice, we identified cylindromatosis (CYLD), a deubiquitinase known to act di
27 NF receptor associated factor 3 (TRAF3), and cylindromatosis (CYLD), as well as retinoblastoma protei
28 echanism in which the tumor-suppressor gene, cylindromatosis (CYLD), confers protection from hepatoce
29 enes including the tumor suppressor familial cylindromatosis (CYLD), which was found to be a direct t
33 associated with the hypertrophic skin tumor cylindromatosis, disrupt the USP domain, accounting for
35 d CYLD by detecting germline mutations in 21 cylindromatosis families and somatic mutations in 1 spor
36 Brooke-Spiegler syndrome (BSS), familial cylindromatosis (FC), and multiple familial trichoepithe
40 ed to mimic the smallest truncation found in cylindromatosis patients, CYLD interaction with CAP350 i
41 rt here that silencing of the deubiquitinase cylindromatosis protein (CYLD), increases HIV infection
45 These researchers found that the familial cylindromatosis tumour suppressor gene (CYLD), previousl
46 denced a new locus in the 16q12 region (near cylindromatosis turban tumor syndrome gene [CYLD]) and c