ライフサイエンスコーパス: cytochrome c

1 f hemes through deprotonated histidine (e.g. Cytochrome c).

2 RS) based biosensor for the determination of cytochrome c.

3 led collision-induced unfolding pathways for Cytochrome C.

4 induce cell death through direct release of cytochrome c.

5 es reducing equivalents for the reduction of cytochrome c.

6 ied by the release of toxic proteins such as cytochrome c.

7 ochrome c maturation (Ccm) systems with MR-1 cytochrome c.

8 we probe the dynamics of certain regions in cytochrome c.

9 slightly changing the abundance of the other cytochromes c.

10 state abundances of both c-type cytochromes, cytochrome c (1) and cytochrome c The observed reduction

11 ndance of complex III could be attributed to cytochrome c (1) being one of its subunits.

12 ic bacterium Rhodobacter sphaeroides lacking cytochrome c(2) as natural electron donor to P(+) (mutan

13 des and Desulfovibrio vulgaris Hildenborough cytochrome c(3)).

14 SI centers that are prebound by plastocyanin/cytochrome c (6) in darkness (about 60% in both cyanobac

15 isotopes at the fundamental distribution of cytochrome c(+8) (m/z approximately 1549) were nearly ba

16 the presence of reactive oxygen species and cytochrome c, a proapoptotic peroxidase that is released

17 gly, D62-DPPC acyl chains were unaffected by cytochrome c accumulation, while cardiolipin showed majo

18 In addition, we observed for cytochrome C acidic and basic conformations.

19 tions increased SMC death; delocalization of cytochrome C activated caspases 9 and 3 to induce apopto

20 nificantly lower ROS, cleaved caspase-3, and cytochrome c activities, leading to reduced spinal cord

21 minimal structural changes were observed for Cytochrome C after hundreds of milliseconds, while no ch

22 3 and 9) and in the mitochondrial (cytosolic cytochrome C, AIF, Mcl-1, Bcl-2, Bcl-xl, Bax, Bad, and p

23 to apoptosis, indicating that restoration of cytochrome c alone may be insufficient to induce effecti

24 ilizes the mitochondrial membrane, releasing cytochrome c and activating the apoptosome.

25 Furthermore, translocation of mitochondrial cytochrome C and AIF was significantly alleviated in the

26 tive neurons, translocation of mitochondrial cytochrome C and apoptosis inducing factor (AIF), LC3B-p

27 mbrane potential loss, increase of cytosolic cytochrome c and Bax levels, decrease of Bcl-2 levels an

28 er resolution images of individual proteins (cytochrome C and BSA) as well as of protein complexes (h

29 yze electron transfer (ET) from ubiquinol to cytochrome c and concomitantly translocate protons acros

30 ely related protein variants, such as bovine cytochrome c and equine cytochrome c, which differ by on

31 ed minority MOMP noted by minimal release of cytochrome C and limited caspase 3 activation, which res

32 Broadening our scope to cytochrome c and lysozyme, we showed that although compa

33 n applied to a simple two-protein mixture of cytochrome c and myoglobin.

34 is and the binding of amino acid residues in cytochrome c and neuroglobin.

35 Cytochrome c and p53-related apoptosis mechanisms were i

36 of mitochondrial activity by phosphorylated cytochrome c and to develop novel therapeutic approaches

37 enes changed (increased transcriptional Bax, cytochrome-c and Apaf-1 and downregulated Bcl-2), wherea

38 of mitochondrial transition pore, release of cytochrome c, and activation of caspase-9 and -3.

39 ell death with immunostaining for annexin V, cytochrome C, and caspases 3 and 9 pointing to induction

40 Bax expression, a disturbed distribution of cytochrome c, and cleaved caspase-3 positive staining in

41 CRALBP, cytochrome C, and GNB3 showed that the RPE interdigitati

42 l associated molecules, including clusterin, cytochrome C, and HTRA2/OMI.

43 apped, translated into a solution containing cytochrome c, and monitored for 3-NBS leakage.

44 me using "protein charge ladders" of azurin, cytochrome c, and myoglobin.

45 apparent K(d),of ~15.8 muM and competes with cytochrome c (apparent K(d) of ~1.31 muM) for binding to

46 of free reactive oxygen species, rather than cytochrome c, are rate limiting.

47 probe for detection Fe(3+), Cu(2+) ions and Cytochrome c based on aggregation induced fluorescence o

48 Cytochrome c binds cardiolipin on the concave surface of

49 erobic electron acceptors include oxygen and cytochrome c, but an acceptor that can function under an

50 electrons coming from NADH and ubiquinol to cytochrome c, but it is also capable of producing signif

51 led device (CCD) detector after digesting of cytochrome c by immobilized trypsin enzymes on NAA-NH(2)

52 nal changes in human serum albumin (HSA) and cytochrome C by monitoring crosslink abundances as a fun

53 Tissue-specific regulation of cytochrome c by post-translational modifications: respir

54 Here we mimic phosphorylation of cytochrome c by replacing tyrosine 48 with p-carboxy-met

55 n mitochondrial cytochromes by knocking down cytochrome c/c (1) heme lyase 1 (TgCCHL1), a mitochondri

56 yl arachidonoyl-phospholipids or through the cytochrome c-catalyzed oxidative hydrolysis of the vinyl

57 (CcP)], suggesting both ascorbate (Asc) and cytochrome c (Cc) peroxidase activity.

58 loys a Trp191(*+) radical to oxidize reduced cytochrome c (Cc).

59 ved C-terminal domain comprising a ubiquinol-cytochrome c chaperone region.

60 ESI-QTOF-MS technique, formation of glycated cytochrome C containing up to 12 glucose moieties were o

61 spho-eNOSSer633, and decreased mitochondrial cytochrome c content with increased lactate dehydrogenas

62 ria differed primarily at a Raman biomarker, cytochrome c, corresponding to a bacteroid-specific term

63 ns (i.e., myoglobin, carbonic anhydrase, and cytochrome c) could be analyzed by SEC-ESI-MS using diff

64 that miR-34a regulates the BBB by targeting cytochrome c (CYC) in vitro.

65 rving as respiratory electron shuttle, ferri-cytochrome c (cyt c) acts as a peroxidase; i.e., it cata

66 d, catalytic circuit based on photosystem I, cytochrome c (cyt c) and human sulfite oxidase (hSOX).

67 proach with three variants of a heme protein cytochrome c (cyt c) and show that the method yields a w

68 cribe advancements in sensing technology for cytochrome c (cyt c) detection, at point-of-care (POC) a

69 Cytochrome c (Cyt c) has evolved to become an important

70 CRALBP and cytochrome C (Cyt C) immunolabeling revealed that hyperr

71 It is well known that cytochrome c (Cyt c) is a crucial death regulator that t

72 Cytochrome c (cyt c) is a small hemoprotein involved in

73 Cytochrome c (cyt c) is a small soluble heme protein cha

74 Cytochrome c (Cyt c) is commonly used as intrinsic bioma

75 Cytochrome c (cyt c) is known for its role in the electr

76 Cytochrome c (Cyt c) plays a vital role in the mitochond

77 The multifunctional protein cytochrome c (cyt c) plays key roles in electron transpo

78 Herein, the structural change of cytochrome c (Cyt c) upon encapsulation within a hierarc

79 Globular proteins, such as cytochrome c (cyt c), display an organized native confor

80 Cytochrome c (cyt c), required for electron transport in

81 assembly and prevent apoptosis triggered by cytochrome c (Cyt c).

82 the adsorption behavior of the redox protein cytochrome c (Cyt-C) onto different interfaces, namely,

83 The mesh is functionalized with cytochrome-c (cyt-c) and incorporated as a working elect

84 of mitochondrial permeability and release of cytochrome c (CytC) and apoptosis-inducing factor (AIF)

85 Cytochrome c (Cytc) is a multifunctional protein that op

86 Cytochrome c (Cytc) is a multifunctional protein, acting

87 rovide the first comprehensive evidence that cytochrome c deficiency in AA primary tumors and cancer

88 Cytochrome c deficiency promoted the acquisition of glyc

89 inding to the cytochrome c promoter mediated cytochrome c deficiency.

90 A deviant subgroup is the cytochrome c dependent NO reductases (cNOR), which reduc

91 after three initial reports of NECD from the cytochrome c dimer complex, no further evidence of the e

92 y 20-fold larger than the previously studied cytochrome c dimer.

93 QBET and the dynamics of ET in mitochondrial cytochrome c during cell life and death process.

94 rt via an Hmc complex (high-molecular-weight cytochrome c encoded by dvu0531-dvu0536) and the Fe-only

95 The hybrid CcmH increased cytochrome c expression by increasing the abundance of C

96 moted Nrf1 binding to cytochrome c promoter, cytochrome c expression, caspase activation, and cell de

97 e cofactors (hemes b and copper for CcoN and cytochromes c for CcoO and CcoP) were present within the

98 e detection of apoptosis based on release of cytochrome c from mitochondria in lysates human embryoni

99 ns that contain multiple heme groups (diheme cytochrome c from Rhodobacter sphaeroides and Desulfovib

100 y interact with electrodes by expressing the cytochrome c from Shewanella oneidensis MR-1.

101 which maspin binds to cardiolipin, displaces cytochrome c from the membrane, and facilitates its rele

102 Cytochrome C had multiple charges in non-glycated state,

103 e basal portion of the RPE, as identified by cytochrome C immunoreactivity, and that the hyporeflecti

104 The distribution pattern of cytochrome c in individual cells was used as a measure o

105 The determination of cytochrome c in the human serum sample is a regular medi

106 Serine-47 phosphorylation of cytochrome c in the mammalian brain regulates cytochrome

107 c relationship with the concentration of the cytochrome c in the range of 1-100 nM.

108 cient in 1-electron transfers to a surrogate cytochrome c in vitro.

109 models of the OMM, is investigated to probe cytochrome c-induced permeability.

110 depicts a low barrier for the permeation of cytochrome C into the Bax C-terminal mouth, with the pat

111 ted to the mitochondria's ability to release cytochrome c into the cytosol, which triggers the apopto

112 can trigger intrinsic apoptosis by releasing cytochrome c into the cytosol.

113 collision cross sections of native-like, 7+ cytochrome c ions increase monotonically from 15.1 to 17

114 sensing principle of proposed IRS sensor to cytochrome c is based on a change in the intensity of th

115 istae, we estimate the LPR of cardiolipin to cytochrome c is between 50 and 100.

116 How cytochrome C is released from the mitochondria to the cy

117 We have reported that once in the cytosol, cytochrome c is targeted for degradation by the E3 ligas

118 The example of cytochrome c is used to demonstrate how StructureDistill

119 The glycated species of cytochrome C, lysozyme, and beta-casein formed during gl

120 s circumvented by deleting the gene encoding cytochrome c (M) (CytM), a cryptic c-type heme protein w

121 ologous, nucleus-, and mitochondrion-encoded cytochrome c maturase systems.

122 bacteria, including Rhodobacter capsulatus, cytochrome c maturation (Ccm) is carried out by a membra

123 to the limited compatibility of the E. coli cytochrome c maturation (Ccm) systems with MR-1 cytochro

124 lies that OXA2a is required for the system I cytochrome c maturation of Arabidopsis.

125 umed to form the heme lyase component of the cytochrome c maturation pathway.

126 Due to the interaction of OXA2a with Cytochrome c maturation protein CcmF C-terminal-like pro

127 f a soluble heme lyase from an organism with cytochrome c maturation system III could functionally co

128 Thus, cytochrome c may have adapted to its native environment

129 Therefore, restoring cytochrome c may overcome therapeutic resistance and pro

130 The spectra for smaller proteins (ubiquitin, cytochrome c, myoglobin) again resemble those at AP, pro

131 Tested on cytochrome c, myoglobin, and beta-lactoglobulin cross-li

132 The diffusion rates of lysozyme, cytochrome c, myoglobin, ovalbumin, bovine serum albumin

133 Cytochrome c nitrite reductase (ccNiR) is a periplasmic,

134 Cytochrome c nitrite reductase (NrfA) catalyzes the redu

135 from G. lovleyi represents a new subclass of cytochrome c nitrite reductase.

136 usion rates of 6 Hz are used for analysis of cytochrome c, on a DTIMS Q-TOF similar rates were obtain

137 Here, we identify the proposed cbb (3)-type cytochrome c oxidase (cbb (3)-Cox) assembly factor CcoG

138 latus, Cu-detoxification and Cu delivery for cytochrome c oxidase (cbb(3) -Cox) assembly depend on tw

139 pper importer CcoA, required for cbb(3)-type cytochrome c oxidase (cbb(3)-Cox) biogenesis, revealed a

140 Cytochrome c oxidase (CcO) catalyzes the reduction of di

141 Cytochrome c oxidase (CcO) catalyzes the reduction of ox

142 Cytochrome c oxidase (CcO) is a transmembrane protein th

143 Cytochrome c oxidase (CcO) reduces dioxygen to water and

144 Proton pumping A-type cytochrome c oxidase (CcO) terminates the respiratory ch

145 Cytochrome c oxidase (CcO), the terminal enzyme in the e

146 o hemoglobin and by inhibiting mitochondrial cytochrome c oxidase (CcO), thereby decreasing oxygen de

147 by knockdown (KD) of subunits IVi1 or Vb of cytochrome c oxidase (CcO).

148 ivities of succinate dehydrogenase (SDH) and cytochrome c oxidase (CCO).

149 ctor of the mitochondrial respiratory enzyme cytochrome c oxidase (CcO).

150 gen that we consume, reducing it to water by cytochrome c oxidase (CcO).

151 nalysis revealed R25 to be the subunit II of cytochrome c oxidase (CCOX), an essential constituent of

152 In Saccharomyces cerevisiae, cytochrome c oxidase (CIV) forms SCs of varying stoichio

153 dendritic mitochondria and histochemistry of cytochrome c oxidase (CO) activity were examined in pre-

154 We describe human COX6B2, a component of cytochrome c oxidase (complex IV).

155 Cytochrome c oxidase (complex IV, CIV) is known in mamma

156 bly and/or modulation of the activity of the cytochrome c oxidase (complex IV, CIV).

157 itochondrial protein with essential roles in cytochrome c oxidase (COX) assembly and the regulation o

158 dentify an interaction between EGFL9 and the cytochrome c oxidase (COX) assembly factor COA3.

159 llar cristae was associated with the gain of cytochrome c oxidase (COX) function, and the COX subunit

160 rone COX19 participates in the biogenesis of cytochrome c oxidase (COX) in yeast and humans.

161 ng cox10 gene encoding an assembly factor of cytochrome c oxidase (COX) specifically in mouse ECs, pr

162 Cytochrome c oxidase (COX) was initially purified more t

163 it shuttles electrons from bc(1) complex to cytochrome c oxidase (COX), and as a trigger of type II

164 Copper is required for the activity of cytochrome c oxidase (COX), the terminal electron-accept

165 tes to fail in the assembly of mitochondrial cytochrome c oxidase (COX).

166 We focused on subunit II of cytochrome c oxidase (Cox2) to study which modifications

167 In cytochrome c oxidase (CytcO) reduction of O(2) to water

168 his prediction measuring oxidation states of cytochrome c oxidase (oxCCO), an intracellular marker of

169 enase, pyruvate kinase, creatine kinase, and cytochrome c oxidase activities, and increased myoglobin

170 ction by measuring H2O2, lipid peroxidation, cytochrome c oxidase activity and mitochondrial ATP.

171 synthase activity was lower (P < 0.0001) and cytochrome c oxidase activity per Mt unit was higher (P

172 ator of mitochondrial bioenergetics enhanced cytochrome c oxidase activity, reversed oxidative stress

173 tected the activity of mitochondrial enzymes cytochrome c oxidase and aconitase in differentiating NS

174 tected the activity of mitochondrial enzymes cytochrome c oxidase and aconitase.

175 ytochrome c in the mammalian brain regulates cytochrome c oxidase and caspase-3 activity.

176 iron-sulfur assembly proteins)1/2 and COX10 (cytochrome c oxidase assembly protein-10), indicating en

177 he matrix side into the inner membrane while Cytochrome c oxidase assembly protein18 (Cox18/Oxa2) is

178 otein synthesis but rather have a problem in cytochrome c oxidase complex (COX) assembly.

179 ression of key mitochondrial genes including cytochrome C oxidase copper chaperone (COX17) and ATP Sy

180 der characterized with neurodegeneration and cytochrome c oxidase deficiency.

181 Cytochrome c oxidase dysfunction enhances phagocytic fun

182 cluding impaired energy generation caused by cytochrome c oxidase dysfunction in the mitochondria.

183 osensor based on the bacterial expression of cytochrome c oxidase for the selective detection of clin

184 Proton transfer in cytochrome c oxidase from the cellular inside to the bin

185 ulated with the Anr-controlled high-affinity cytochrome c oxidase genes.

186 Here, metabarcoding of cytochrome c oxidase I (COI) region of mitochondrial DNA

187 , extracted DNA and sequenced mitochondrial (cytochrome c oxidase I [mtCOI]) and nuclear (small subun

188 e, including genes encoding cytochrome b and cytochrome c oxidase I.

189 nd decreased levels of mitochondrial complex cytochrome c oxidase I/IV and lower ATP levels.

190 tochondrial function that directly activates cytochrome c oxidase in the mitochondria and functions i

191 on function (Q) of the redox center CuA from cytochrome c oxidase is attained by tuning the accessibi

192 ted copper to the mitochondria and increased cytochrome c oxidase levels in the brain.

193 ong with a correlation between the number of cytochrome c oxidase operons and heterotrophic or diazot

194 Defects in mitochondrial cytochrome c oxidase or respiratory chain complex IV (CI

195 We assembled individual mitochondrial cytochrome c oxidase subunit 1 (COI) sequences (n = 866)

196 in a stretch of 22 identical amino acids in cytochrome c oxidase subunit 1 and NADH dehydrogenase su

197 TPR domain of OXA2b directly interacts with cytochrome c oxidase subunit 2, aiding in efficient memb

198 voltage-dependent anion channel (VDAC), and cytochrome c oxidase subunit 4 (COX IV).

199 Complex III's Rieske Fe-S center and COX4i2 [cytochrome c oxidase subunit 4 isoform 2] in Complex IV)

200 in fragments of the mitochondrial (mt) genes cytochrome c oxidase subunit I (COI) and Cytochrome b as

201 A 450 base pairs (bp) segment of cytochrome c oxidase subunit I (COI) was amplified and s

202 Here we used eDNA metabarcoding of COI (cytochrome c oxidase subunit I) and 18S (nuclear small s

203 Pyrosequencing of genetic marker, COI (cytochrome c oxidase subunit I) and subsequent sequence

204 EPAS1, and the nuclear-encoded mitochondrial cytochrome c oxidase subunit, COX4I2, in glomus cell sen

205 ons: Cox6a2, which codes for an isoform of a cytochrome c oxidase subunit.

206 and genes coding for NADH dehydrogenase and cytochrome c oxidase subunits.

207 ith a subpopulation of the cytochrome bc (1)-cytochrome c oxidase supercomplex and have been proposed

208 gly, yeast, mouse liver, and isolated bovine cytochrome c oxidase were directly inhibited by the drug

209 biopsies, cocoa improved mitochondrial COX (cytochrome c oxidase) activity (P=0.013), increased capi

210 ansport chain complexes) markers and COX IV (cytochrome C oxidase) activity in myocardium from patien

211 tion (oxygenated hemoglobin) and metabolism (cytochrome c oxidase) by 15% +/- 6.7% and 49% +/- 18%, r

212 enzymes (P-450s, peroxidases, catalases, and cytochrome c oxidase) involved in biological respiration

213 We measured the oxidation state of cytochrome c oxidase, an intracellular measure of metabo

214 Conditional deletion of cytochrome c oxidase, the terminal enzyme in the respira

215 In the hypoxic mitochondrion, cytochrome c oxidase, which is a major source of NO, als

216 Oxidative histochemistry showed cytochrome c oxidase-deficient fibres in skeletal and ca

217 mtDNA depletion and cytochrome c oxidase-negative cells were found ex vivo i

218 nce of ragged-red fibers, however, scattered cytochrome c oxidase-negative staining and electron dens

219 rget, COX2 mRNA, which encodes subunit II of cytochrome c oxidase.

220 enosine triphosphate (ATP) and a decrease in cytochrome c oxidase.

221 is also utilized in other enzymes, including cytochrome c oxidase.

222 etics based on the activity of mitochondrial Cytochrome-C Oxidase (CCO).

223 Muscle studies showed global cytochrome-c oxidase deficiency in all patients tested a

224 vity of the mitochondrial respiratory chain (cytochrome c-oxidase/succinate dehydrogenase [COX/SDH]-r

225 I (NADH dehydrogenase, NDU) and complex IV (cytochrome-c-oxidase, COX) of the mitochondrial electron

226 The largest subgroup contains the cytochrome c oxidases (CcO), which reduce molecular oxyg

227 Cytochrome c oxidases (CcOs) are the terminal enzymes in

228 are structurally unrelated to mitochondrial cytochrome c oxidases and are therefore a prime target f

229 Cytochrome c oxidases of facultative members of the comm

230 In the absence of high-affinity cytochrome c oxidases, deletion of mhr no longer caused

231 cbb3-type cytochrome c oxidases, which catalyze the terminal step

232 Ubiquinol cytochrome c oxidoreductase (bc1 complex) serves as an i

233 e, the flexible pore may selectively aid the cytochrome C passage.

234 ndrial respiration via the energy-conserving cytochrome c pathway in both strains, the mutant was una

235 idopsis (especially after restriction of the cytochrome c pathway) but cannot compensate for the lack

236 MbnH belongs to the bacterial diheme cytochrome c peroxidase (bCcP)/MauG protein family, and

237 Cytochrome c peroxidase (CcP) employs a Trp191(*+) radic

238 ned in a structurally nonhomologous protein, cytochrome c peroxidase (CcP), by only two mutations (Cu

239 drogen peroxide (H(2)O(2)) respiration using cytochrome c peroxidase (Ccp).

240 A member of class I heme peroxidases [TcAPx-cytochrome c peroxidase (CcP)], suggesting both ascorbat

241 eme enzyme that is a member of the bacterial cytochrome c peroxidase superfamily, capable of generati

242 ape routes in cytochrome P450 monooxygenase, cytochrome c peroxidase, and benzylsuccinate synthase (B

243 Microbial cytochrome c peroxidases (Ccp) have been studied for 75

244 Although cytochrome c phosphorylation-in particular, at tyrosine

245 1) and its subsequent loss of binding to the cytochrome c promoter mediated cytochrome c deficiency.

246 r activation of AKT promoted Nrf1 binding to cytochrome c promoter, cytochrome c expression, caspase

247 the macromolecular level by conjugating the cytochrome c protein to a complementary polyethylene gly

248 mediate hydrazine is produced by a multiheme cytochrome c protein, hydrazine synthase, through the co

249 ultiheme enzyme complexes that rely on small cytochrome c proteins for electron shuttling.

250 When the vesicle-associated cytochrome c Raman spectrum is compared with a spectrum

251 oscopy, we optically capture real-time ET in cytochrome c redox dynamics during cellular apoptosis an

252 ytochrome bc1 :aa3 consists of a menaquinone:cytochrome c reductase (bc1 ) and a cytochrome aa3 -type

253 In this work, cytochrome c reductase (CcR) biofunctionalized self asse

254 fide bond cross-link caused a >/=95% loss of cytochrome c reductase activity that was reversible with

255 (p.Tyr314Ser) in the mitochondrial ubiquinol-cytochrome c reductase core protein 1 (UQCRC1) gene, whi

256 Ubiquinol-cytochrome c reductase hinge protein (UQCRH) is the hing

257 ter assembly and newly synthesized ubiquinol-cytochrome c reductase Rieske iron-sulfur polypeptide 1

258 Superoxide generation was measured by cytochrome C reduction in the presence and absence of N-

259 In wild-type cells, H(2)O(2) caused cytochrome c release and apoptosis, both of which were p

260 e inner mitochondrial membrane and decreases cytochrome c release and apoptosis.

261 chondrial outer membrane (MOM), which causes cytochrome c release and apoptosis.

262 K)-mediated phosphorylation and elevation of cytochrome c release and caspase 9 activity.

263 Cytochrome c release and caspase-3 activation in respons

264 ostate cancer cells contributed to defective cytochrome c release and increased resistance to apoptos

265 totic signalling downstream of MOMP involves cytochrome c release from mitochondria and subsequent ca

266 ins in targeting the mitochondria to promote cytochrome c release to augment the mitochondrial apopto

267 fingerprint to confirm the cellular uptake, cytochrome c release, and DNA fragmentation in a label-f

268 o the MOM bypasses the need for Mff to evoke cytochrome c release, and occludes the effect of SENP3 o

269 Active caspase-6 cleaved Bid to induce cytochrome c release, generating a feedforward loop that

270 d functionally uncouples BAX clustering from cytochrome c release, while knockdown of the Rab5 exchan

271 spases activation, Bcl-xL sequestration, and cytochrome c release.

272 both Drp1 binding to Mff and stress-induced cytochrome c release.

273 ncides with mitochondrial BAX clustering and cytochrome c release.

274 ctor Rabex-5 impairs both BAX clustering and cytochrome c release.

275 s hemoglobin, myoglobin, cytochrome P450 and cytochrome c, respectively.

276 quantifiable across triplicates for HSA and cytochrome C, respectively.

277 types and mitochondrial dysfunction, whereas cytochrome c restoration via inhibition of c-Myc and NF-

278 hat oxidative alphaSyn aggregation scavenges cytochrome c's activity into the formation of amorphous,

279 pathological changes and increased levels of cytochrome c, Smac/DIABLO and AIF in the cytosol while t

280 d via comet assay, CyQuant, annexin V, JC-1, cytochrome C subcellular localization, caspase 3 activat

281 ubunits: the catalytic CcoN subunit, the two cytochrome c subunits (CcoO and CcoP) involved in electr

282 S. oneidensis MR-1 encodes two cytochrome c synthetases (CcmF and SirE) and two apocyto

283 oprotein biocatalyst based on a thermostable cytochrome c that directly transforms alkenes to amino a

284 oth c-type cytochromes, cytochrome c (1) and cytochrome c The observed reduction in the steady-state

285 chrome bc (1) complex and its redox partner, cytochrome c These results suggest that a chemerin p4-ba

286 imilar to the previously reported results on cytochrome c, these fragment ions form near residues kno

287 Rather than inert passing of the cytochrome C through a rigid pore, the flexible pore may

288 es reducing equivalents for the reduction of cytochrome c to facilitate heme attachment.

289 nge of mitochondrial function and release of cytochrome c to initiate apoptosis.

290 allenge correlated with increased release of cytochrome c to the cytosol, activation of caspase-3, an

291 mplex 1 and NADH synergistically photoreduce cytochrome c under hypoxia.

292 reactivity of the reduced FMN domain toward cytochrome c; (v) response to calmodulin binding; and (v

293 The limit of detections (LOD) for cytochrome c was 0.5 nM.

294 the SH3 domain, dihydrofolate reductase, and cytochrome c, where the transparent window vibrational p

295 ants, such as bovine cytochrome c and equine cytochrome c, which differ by only three amino acids.

296 anges to key apoptotic regulators, including cytochrome c, which primes cells for death.

297 he dissociated heme to the protein moiety in cytochrome c, which we assign to the presence of covalen

298 on proceeds through selective interaction of cytochrome c with cardiolipin, resulting in protein unfo

299 To verify selective interactions of cytochrome c with cardiolipin, these experiments were re

300 In this work, the interaction of cytochrome c with cardiolipin-containing phospholipid ve