ライフサイエンスコーパス: cytokine

1 to sustained production of pro-inflammatory cytokines.

2 exposed to allergic inflammation-associated cytokines.

3 D8 T-cell subset with the ability to secrete cytokines.

4 motif in the promoter region of inflammatory cytokines.

5 rks in Matrigel, but did generate angiogenic cytokines.

6 ocytosis and decrease production of iNOS and cytokines.

7 d upregulated expression of immunomodulatory cytokines.

8 the transcription of several proinflammatory cytokines.

9 s well as other TLR ligands and inflammatory cytokines.

10 ction in hepatotoxicity from proinflammatory cytokines.

11 acellular IL-17A by inducing proinflammatory cytokines.

12 reduced systemic levels of pro-inflammatory cytokines.

13 2 inflammation, immune cell populations, and cytokines.

14 ia, elevated serum Igs, and induction of Th2 cytokines.

15 Tumor necrosis factor-like cytokine 1A (TL1A, TNFSF15) is implicated in inflammator

16 cluding increased levels of pro-inflammatory cytokines(3,4) that may be produced by a subset of infla

17 Elafin, via immune-derived miRNAs and cytokine, activates leptin sensitivity and expression th

18 cting how different myeloid cells respond to cytokine activation, we can delineate biological roles f

19 Increases in airway type 2 cytokine activity, including interleukin-4 (IL-4), IL-5,

20 of these diseases is related to dysregulated cytokine activity.

21 appaB-dependent induction of proinflammatory cytokines after FcepsilonR1 stimulation in mast cells, i

22 t but complementary molecular pathways, both cytokines also induced epithelial-mesenchymal transition

23 that overexpression of MEGF11 induces both a cytokine and a chemokine cascade, which will favour the

24 r, type I IFN alone was sufficient to induce cytokine and chemokine production by macrophages and B16

25 lly potentiate immune responses by elevating cytokine and chemokine production via triggering multipl

26 thesis pathways were activated, and specific cytokine and chemokine receptors were up-regulated in CS

27 hages with upregulation of immunity relevant cytokine and co-stimulatory markers.

28 otype but also tempers their proinflammatory cytokine and ferritin secretion by negatively regulating

29 scovery of JAKs and STATs and their roles in cytokine and IFN action represented a significant basic

30 min of 40 Hz or control flicker and assessed cytokine and phosphoprotein networks known to play a rol

31 the importance of the link between perinatal cytokines and abnormal behaviors in offspring, human epi

32 cular, analysis of secreted proteins such as cytokines and antibodies.

33 nied by the reduction of key proinflammatory cytokines and changes in plaque morphology.

34 LPA-stimulated expression of pro-tumorigenic cytokines and chemokines overexpressed in ovarian cancer

35 lial cells secreted large amounts of several cytokines and chemokines, especially tumor necrosis fact

36 ly reduced the concentration of inflammatory cytokines and chemokines, including IL-1, TNF-alpha, IL-

37 sclerosis (MS): secretors of proinflammatory cytokines and chemokines, presenters of autoantigens to

38 f TNF-alpha- and STS-induced proinflammatory cytokines and chemokines.

39 enhancing the production of proinflammatory cytokines and chemokines.

40 c molecules, e.g. lipopolysaccharides (LPS), cytokines and damage- or pathogen-associated molecular p

41 oting passage of circulating proinflammatory cytokines and depression-like behaviors.

42 antiviral function: memory T cells secreted cytokines and expanded upon antigen re-encounter, wherea

43 mediated production of both proinflammatory cytokines and interferons.

44 thresholds were also observed for different cytokines and killing.

45 Elevated local production of inflammatory cytokines and MMPs, together with apparent mononuclear i

46 eased lung concentrations of proinflammatory cytokines and neutrophil-attracting chemokines, and enha

47 S in RA produce pathogenic mediators such as cytokines and proteases that contribute to disease patho

48 evation of IL-6, IL-10, TNF-alpha, and other cytokines and severe CD4(+) and CD8(+) T-cell lymphopeni

49 ppressive, unable to produce proinflammatory cytokines, and exhibit the epigenetic modifications of t

50 , dampen their capacity to make inflammatory cytokines, and increase their responsiveness to adenosin

51 Systemically elevated cytokines are also known to be cardiotoxic and have the

52 Cytokines are small proteins secreted by cells in innate

53 ur knowledge, identify a novel role for this cytokine as a central regulator of immunity in lymphatic

54 ix components; and reductions in markers and cytokines associated with immunosuppression.

55 modulates the release of key proinflammatory cytokines associated with periodontal disease pathogenes

56 ioral and psychological surveys and analyzed cytokines at three time points.

57 human IgG1 monoclonal antibody targeting the cytokine B cell-activating factor (BAFF).

58 ic inflammatory diseases in combination with cytokine-blocking drugs.

59 Interleukin-33 (IL-33) acts as an alarmin cytokine by alerting the system of potential environment

60 kines by two or more unique peptides and two cytokines by a single unique peptide.

61 Our MRM method was able to identify 21 cytokines by two or more unique peptides and two cytokin

62 Age-associated cytokine changes differed among CRS subtypes and control

63 ation expressing high levels of Th2 and Th17 cytokines, chemokine receptors CCR4 and CCR6, and the tr

64 Inflammatory cytokine/chemokine concentrations were determined using

65 The role of Mincle for MERS-CoV-triggered cytokine/chemokine induction was established based on th

66 biased clustering approach revealed distinct cytokine/chemokine patterns, and these aligned with path

67 lls reactivates their antitumor inflammatory cytokine/chemokine production.

68 n effectively kill target cells by producing cytokines, chemokines, and granzymes.

69 hese transplants induced mRNAs for >40 to 50 cytokines, chemokines, and receptors.

70 t notable fever-mediated modulation of their cytokine commitment.

71 Cytokine concentrations in supernatants of culture and i

72 No association between HIV status and cytokine concentrations was found.

73 7 cells were more activated and Th17-related cytokine concentrations were elevated.

74 activation, vaginal microbial diversity and cytokine concentrations.

75 This was associated with lower levels of Th1 cytokines, decreased T cell infiltration, increased B ce

76 resentation; more lymphocytes and associated cytokines; decreased extracellular matrix components; an

77 gnal transduction, indicated by induction of cytokine-dependent cellular proliferation, signal transd

78 Proinflammatory cytokines directly modulate RANKL/OPG expression and con

79 utically beneficial if cocultured with IL-12 cytokine during in vitro expansion and highly effective

80 In response to secretion of inflammatory cytokines (e.g., IL1B) from immune infiltrates, ELF3 in

81 alloantigen to donor T cells while releasing cytokines (eg, interleukin-12 [IL-12], IL-23, IL-6, IL-2

82 logical roles for myeloid cells in different cytokine environments during disease processes, especial

83 pid elimination, which could be coupled with cytokine expansion and contraction.

84 es correlated with increased proinflammatory cytokine expression and with lung inflammatory pathology

85 e of bacterial and viral pathogens; however, cytokine expression patterns manifested independently of

86 gene expression indicates alterations in the cytokine expression that could be related to reduced par

87 Cytokine expression was examined by reverse-transcriptas

88 e content, plaque size, and pro-inflammatory cytokine expression.

89 ured brain areas by suppressing inflammatory cytokines expression whereas free Sino treatment did not

90 in-10 (IL-10), a classical anti-inflammatory cytokine, extends lifespan in the SOD1-G93A mouse model

91 and the common IL-10R2, belongs to the IL-10 cytokine family, and is critically involved in tissue re

92 ized by the rapid production of inflammatory cytokines following delivery of therapy, with symptoms r

93 Interleukin 15 (IL-15) is an essential cytokine for the survival and proliferation of natural k

94 play a key role in providing chemokines and cytokines for the localization, differentiation, and sur

95 he levels of immune stimulatory inflammatory cytokines, for example, IFN-gamma and IL-12, in CCR2i- v

96 the activation of STAT3 and STAT6 as well as cytokine gene expression in ECs challenged with IL-4/IL-

97 ines, resulting in an incomplete portrait of cytokine gene regulation.

98 Studies on the transcriptional control of cytokine genes have mostly focused on highly researched

99 Immunotherapies with cytokines have also been extensively explored, but they

100 g alone, we show that different inflammatory cytokines have comparable Ag dose thresholds across a 25

101 gs suggest mechanisms by which the enigmatic cytokine IL-17F contributes to host defense against fung

102 Herein we identify the dual function cytokine IL-33 as an orchestrator of the glioblastoma mi

103 This process is inhibited by the cytokine IL-37.

104 epatic MPhis to produce the hepatoprotective cytokine IL-6, thereby ameliorating AILI.

105 ronic overexpression of the pro-inflammatory cytokine IL-6.

106 maintained by the stroma-derived homeostatic cytokine IL-7, and priming diminishes if Il7r is subsequ

107 panel of 10 cytokines, the pro-inflammatory cytokine IL-8 exhibited a strong correlation with deliri

108 via its involvement in the production of the cytokines IL-1 and/or IL-18.

109 sulted in the cellular release of the mature cytokines IL-1beta and IL-18 and induction of pyroptosis

110 and drive the maturation of proinflammatory cytokines IL-1beta and IL-18.

111 ortic diameter, decrease in pro-inflammatory cytokines (IL-1beta, IL-6, IL-17, MCP-1, MIP-1alpha, MIP

112 correlated with suppression of two major Th2 cytokines, IL-10 and IL-13.

113 hemokines (CXCL5, CCL20, CXCL13, and CCL18), cytokines (IL23A, IL19, and IL1B), matrix modifiers (MMP

114 rises the high affinity receptor for IL-2, a cytokine important in immune proliferation, activation,

115 application of the utility to a panel of 62 cytokines in a sample of human patients diagnosed with s

116 on markers and production of proinflammatory cytokines in a similar manner to B. burgdorferi.

117 rative intravenous n-3 PUFAs on inflammatory cytokines in colon cancer surgery.

118 nd underscore the importance of inflammatory cytokines in mucosal HIV infection, demonstrating the li

119 ors (FGF) act as proangiogenic and mitogenic cytokines in multiple myeloma.

120 n pattern-recognition receptor (PRR)-induced cytokines in myeloid cells.

121 olated from intestinal biopsies, with gammac cytokines in presence or absence of BNZ-2.

122 tion and release; and the functions of these cytokines in protective and pathological inflammation.

123 DNA sensing, PYHIN1 induced proinflammatory cytokines in response to interleukin-1 (IL-1) or tumor n

124 ment, mucin production and asthma-associated cytokines in the bronchoalveolar lavage fluid.

125 he percentage of cells expressing all tested cytokines in the lamina propria and the epithelium was h

126 elease of myokines, skeletal muscle-specific cytokines, in response to exercise.

127 cells engrafted, induced release of multiple cytokines including IL6, IL17, MCP-1, and GM-CSF in the

128 rther enhances expression of proinflammatory cytokines (including monocyte chemoattractant protein 1,

129 classic roles attributed to this pleiotropic cytokine, including how IL-10 regulates basic processes

130 ta is the most studied of the IL-1 family of cytokines, including 11 members, among which are IL-1alp

131 ta-cell apoptosis induced by proinflammatory cytokines, increasing the possibility that they can be b

132 NTRK3E176D, and NTRK3L449F), determined via cytokine-independent cellular assays.

133 Inflammatory cytokines induce the expression of IFITM3 in neurons and

134 The pattern of pro-inflammatory cytokines induced in COVID-19 has similarities to those

135 ss effector T cell-mediated and inflammatory cytokine-induced CEnC death, and to elucidate the mechan

136 tumor necrosis factor-alpha (TNF-alpha) and cytokine-induced neutrophil chemoattractant-1 (CINC-1).

137 ell RNA sequencing revealed that a number of cytokine-inducible genes shared this heterogeneous respo

138 on with high levels of serum proinflammatory cytokines, inflammatory infiltrates in various organs, a

139 We also demonstrate a role for the cytokine interferon-gamma (IFNgamma) and the enzyme tran

140 The cytokine interleukin (IL)-1beta is a key mediator of ant

141 report a role for the type 2 cytokine interleukin-13 in orchestrating metabolic repro

142 e maturation and release of the inflammatory cytokine interleukin-1beta (IL-1beta).

143 increased the sensitivity of T cells to the cytokine interleukin-2 (IL-2) through a positive feed-fo

144 The cytokine interleukin-22 (IL-22) is a critical regulator

145 We established that the immune-suppressive cytokine interleukin-27 (IL-27) is elevated in neonatal

146 The anti-inflammatory cytokines (interleukin-4 [IL-4], IL-6, and IL-10) and MC

147 hrough the production of two proinflammatory cytokines: interleukin-17 (IL-17) and GM-CSF.

148 tribute to the development of EoE, but other cytokines involved in pathogenesis are unknown.

149 sforming growth factor beta 1 (TGF-beta1), a cytokine known to inhibit CD56(+) cell development.

150 oduced significantly more IL-1beta and IL-6, cytokines known to drive Th17 cell differentiation.

151 tion, release granzyme B and proinflammatory cytokines, leading to target cell killing.

152 l cells showed that receptors of the hormone/cytokine leptin were highly expressed, and we found a de

153 Bacteria, cytokines, leukocytes, and hematopoietic precursors were

154 An examination of 40 cytokine levels across the study groups revealed that an

155 mmune activation (MIA) elevates inflammatory cytokine levels in the maternal and fetal compartments a

156 Galactomannan and relevant cytokine levels were assessed in serum, plasma and BAL.

157 s analyzed by next-generation sequencing and cytokine levels were determined in SAT.

158 e hybrid system locally reduces inflammatory cytokine levels.

159 , leading to a reduction in pro-inflammatory cytokine levels.

160 ng, but those cells release decreased T(H) 2 cytokine levels.

161 ization of the microbiota and measurement of cytokine levels; primary endpoints were cervical T cell

162 s activation as well as suppressing cerebral cytokines levels.

163 beta (TGFbeta) and LIF interleukin-6 family cytokine (LIF) signaling pathways.

164 racterized the pivotal role of the versatile cytokine macrophage migration inhibitory factor (MIF) in

165 suggests the possibility that targeting this cytokine may restore antiviral mechanisms.FUNDINGThis st

166 a link between contact system activation and cytokine-mediated inflammation.

167 vely, our findings suggest that blocking the cytokine-mediated inflammatory cell death signaling path

168 We found that type-2 cytokine-mediated inhibition of the expression of genes

169 ndicate a pivotal role for LDH in modulating cytokine-mediated T cell differentiation and underscore

170 ntral inhibition of RAS and pro-inflammatory cytokines normalizes sympathetic drive and improves card

171 hese responses differ from the activities of cytokines on beta cells, which are mediated by inducible

172 can suppress production of T-cell polarizing cytokines or induce inhibitory antigen-presenting cells

173 nt that examined multiple lots of a human 51-cytokine panel.

174 ncer, our in vivo screen of inflammatory and cytokine pathway genes revealed IL26 to be one of the mo

175 phagocytosis, Ag presentation, and chemokine/cytokine pathways.

176 investigating temporal IFN and inflammatory cytokine patterns in 32 moderate-to-severe patients with

177 Cytokines play critical roles in regulating the developm

178 lopoietic growth factor and pro-inflammatory cytokine, plays a critical role in alveolar macrophage h

179 ion in vivo but was remarkably distinct from cytokine-polarized Th17 cells.

180 that SKAP2 controls beta-cell sensitivity to cytokines possibly by affecting the NF-kappaB-inducible

181 Cytokine-primed neutrophils can undergo a nonapoptotic t

182 IL-21 is a pleiotropic cytokine produced predominantly by CD4+ T cells.

183 ts with MS and animal models of how specific cytokines produced by autoreactive CD4 T cells contribut

184 Cytokines produced by T-helper type 2 cells and transfor

185 erved in regard to lymphoblast expansion and cytokine production (IFN-gamma, IL-2, and TNF), with the

186 ia, however, glycolysis becomes critical for cytokine production and cell survival.

187 emphasis on recent work on the regulation of cytokine production and proliferation.

188 IFN-beta did not induce inflammatory cytokine production by MDMs or PBECs but reduced influen

189 Cytokine production by vaginal epithelial cells in respo

190 y morning vaccination resulted in a superior cytokine production capacity compared with later morning

191 A cohort of septic patients had increased cytokine production compared with controls consistent wi

192 te and adaptive immune responses, and type 2 cytokine production in a model of airway sensitization a

193 ogy, leukocyte recruitment, and inflammatory cytokine production in lungs including TNF, IL-6, IL-10,

194 gly, GM-CSF signaling amplifies inflammatory cytokine production in recruited monocytes by enhancing

195 ession in DC enhances proinflammatory innate cytokine production to promote an altered Th2 immune res

196 Cytokine production was measured to assess the induction

197 Cytokine production was measured using flow cytometry, E

198 No significant differences in cytokine production were found in Th1, Th2, and Th17 imm

199 e-mediated inflammation and pro-inflammatory cytokine production, a shift towards ketone bodies as th

200 creases allergen-induced IL-33 release, ILC2 cytokine production, and airway inflammation.

201 Necrosome signaling, cytokine production, and cell death were evaluated by im

202 pram inhibits organ damage, pro-inflammatory cytokine production, and intracellular migration of earl

203 ecule retinoic acid, inhibiting inflammatory cytokine production, and making macrophages more suscept

204 cific T cell receptors (TCRs) show increased cytokine production, migration toward tumor cells, and t

205 y inflammation and significantly reduced Th2 cytokine production, serum IgE levels, and airway hyperr

206 diversity, is associated with increased FGT cytokine production.

207 ing sustained immune activation and effector cytokine production.

208 cules, and they were polyfunctional based on cytokine production.

209 (bright) NK cell subset characterized by low cytokine production.

210 amma on AML blasts generated an inflammatory cytokine profile and activated NK cells.

211 tive signs on the day of blood drawing had a cytokine profile that was similar to that of non-AMD ind

212 The aim of this study was to compare cytokine profiles in stimulated primary mononuclear cell

213 h is able to regulate CD8(+) T cell effector cytokine R production independent of TCR Ag affinity.

214 it a STAT3-activating complex, circumventing cytokine receptor activation.

215 is insufficient to eradicate the most common cytokine receptor-like factor 2-rearranged (CRLF2-rearra

216 Knowledge of cytokine-receptor interaction (CRI) is very important fo

217 the T-cell receptor (TCR), co-receptors, and cytokine receptors.

218 However, cytokine release and microgliosis are consistently obser

219 LKB1 deficiency was associated with reduced cytokine release into the airways upon local LPS instill

220 A proinflammatory dysregulation of cytokine release is associated with various diseases, in

221 Rab27 contributes to airway inflammation and cytokine release remain ambiguous.

222 Cytokine release syndrome (CRS) is a life-threatening tr

223 tential for severe adverse events, including cytokine release syndrome (CRS).

224 ong the axi-cel-treated patients, grade >= 3 cytokine release syndrome and neurotoxicity occurred in

225 Cytokine release syndrome was the most common adverse ev

226 of its protumor effects and its role in the cytokine release syndrome.

227 19 patients (91%); three (14%) had grade 3-4 cytokine release syndrome.

228 normal cell-cycle transition and profiles of cytokine release that resembled those of normal memory T

229 interactions with each cell type, alongside cytokine release.

230 interleukin-1 (IL-1)-dependent inflammatory cytokine response by recruited monocytes and other cells

231 characteristics, outcomes, and chemokine and cytokine response in transplant recipients to immunocomp

232 PUFAs and specifically AA trigger a cytokine response of IECs which is restricted by GPX4.

233 ellular antioxidants, to facilitate enhanced cytokine responses in HIEs.

234 Type 2 cytokine responses promote parasitic immunity and initia

235 al sepsis, and contribute to tissue-specific cytokine responses that are protective against mortality

236 n resulted in increased schistosome-specific cytokine responses that were negatively associated with

237 and long-term repression of monocyte-derived cytokine responses, and short-term as well as long-term

238 phages impairs their ability to mount robust cytokine responses.

239 select patients helps elicit favorable host cytokine responses.

240 cells (PBMCs), that is highly predictive of cytokine responses.

241 Altered cytokine responsiveness to endotoxin is also observed in

242 y researched transcription factors (TFs) and cytokines, resulting in an incomplete portrait of cytoki

243 -T-cells in the spleen, and enhanced overall cytokine-secreting T cell percentages upon antigen resti

244 (CD16) and by high levels of proinflammatory cytokine secretion and cytotoxic activity.

245 C reparative activities, modulating in vitro cytokine secretion and in vivo gene expression for effec

246 teen abacavir analogues were synthesized and cytokine secretion from abacavir/abacavir analogue-respo

247 in from S Typhimurium induced cell death and cytokine secretion in THP-1 cells and primary human mono

248 3(+) BMRMs show a specific transcriptome and cytokine secretion pattern demonstrating a specific immu

249 cultured immune cells induced a differential cytokine secretion that may contribute to CRC metastasis

250 atory lung disease associated with increased cytokine secretion.

251 sis, hemophagocytic lymphohistiocytosis, and cytokine shock.

252 lular protein SOCS1 is known to downregulate cytokine signaling by inhibiting the JAK-STAT pathway.

253 nrichment analysis revealed pro-inflammatory cytokine signaling pathways as dysregulated, and this wa

254 can regulate both pro- and anti-inflammatory cytokine signaling.

255 naling, including induction of suppressor of cytokine signaling.

256 sseminated intravascular coagulation, with a cytokine signature similar to that of macrophage activat

257 HMT2 causes strong increases in inflammatory cytokine signatures associated with redox dependent indu

258 IL33 was the only cytokine significantly correlated with miRNAs (rho > 0.9

259 tic activation of phenoloxidase (PO) and the cytokine Spatzle during immune responses of insects is m

260 iatric kidney recipients using intracellular cytokine staining and flow cytometry.

261 e inflammation and respiratory disease, with cytokine storm and acute respiratory distress syndrome i

262 ctivity, chloroquine might also mitigate the cytokine storm associated with severe pneumonia caused b

263 A hyperinflammatory state referred to as cytokine storm in its severest form has been marked by e

264 RS-CoV-2) pneumonia patients indicate that a cytokine storm may increase morbidity and mortality.

265 lays a previously unappreciated role in sHLH/cytokine storm syndrome pathogenesis by preventing macro

266 into the bloodstream, causing the so-called "cytokine storm".

267 illness characterized by hyperinflammation, cytokine storm, and elevations of cardiac injury biomark

268 evented the debris-stimulated eicosanoid and cytokine storm, down-regulated ER stress genes, and prom

269 ma (HCC) tumor growth via an "eicosanoid and cytokine storm." AFB(1)-generated debris up-regulates cy

270 Elevated inflammatory cytokines such as IL-1beta and IL-18 and concurrent late

271 d expression of pro-inflammatory tumorigenic cytokines, such as IL-17A and IL-6, and increased STAT3

272 flammatory transcription factor required for cytokine synthesis in psoriasis.

273 erized by RV species, microbiome, and type 2 cytokine (T2) response: endotype A, virus(RV-C)microbiom

274 ressing Hu19-CD828Z released lower levels of cytokines than T cells expressing FMC63-28Z.

275 Midkine-a, an injury-induced growth factor/cytokine that is expressed by Muller glia following neur

276 hogenetic protein-9 (BMP-9) is a circulating cytokine that is known to play an essential role in the

277 stimulating factor (GM-CSF) is a multipotent cytokine that prompts the proliferation of bone marrow-d

278 nterleukin-2 (IL-2) is a small alpha-helical cytokine that regulates immune cell homeostasis through

279 cells, in cooperation with a combination of cytokines that are abundant at the tumor site.

280 resolution as macrophages are polarized with cytokines that drive them into "M1" and "M2" molecular s

281 These macrophages generate inflammatory cytokines that induce cancer cell LCN2 expression but do

282 C2s) are a potent source of T-helper 2 (Th2) cytokines that promote AHR and lung inflammation.

283 Cytokines that stimulate T cell proliferation, such as i

284 From a panel of 10 cytokines, the pro-inflammatory cytokine IL-8 exhibited

285 revealed the upregulation of proinflammatory cytokines (TNF-alpha, IL-6, and IL-1beta) that are assoc

286 the chemokine Ccl2 and the pro-inflammatory cytokine Tnfalpha were observed at the protein level in

287 py-induced secretion of the pro-inflammatory cytokines TNFalpha, IL-1beta, and IL-8.

288 d does not induce expression of inflammatory cytokines TNFalpha, IL-6, IL-8 or IFNbeta.

289 to express isoforms of the antiinflammatory cytokine transforming growth factor beta (TGF-beta).

290 Like many mammalian cytokines, unpaireds can be activated by infection and o

291 HCV-specific cytokines were also improved post DAA.

292 Lower levels of cytokines were detected in blood from patients who recei

293 Cytokines were quantified from culture supernatants by E

294 ere higher levels of IL-1beta, IL-6 and IL-8 cytokines were quantified in keratitis caused by Gram-ne

295 Interleukin-10 (IL-10) is a dimeric cytokine with both immunosuppressive and immunostimulato

296 Interleukin-6 (IL-6) is a cytokine with critical innate and adaptive immunity func

297 Interleukin (IL)-13 is a type 2 cytokine with important roles in allergic diseases, asth

298 Treatment with interleukin (IL)-22, a cytokine with multiple targets, ameliorated CXCL1/HFD-in

299 terleukin-17A (IL-17A) is a pro-inflammatory cytokine with well-characterized biological effects on s

300 the proapoptotic molecule Hrk in response to cytokine withdrawal.