ライフサイエンスコーパス: cytokine secretion

1 increased induction of myeloid cell-derived cytokine secretion.

2 eceptors (TLRs), stimulating proinflammatory cytokine secretion.

3 old for new cardiomyocytes, angiogenesis and cytokine secretion.

4 face responsible for the galectin-3-mediated cytokine secretion.

5 resulted in increased Th17 polarization and cytokine secretion.

6 ulator of interferon genes (STING)-dependent cytokine secretion.

7 onal T-cell receptor repertoire and enhanced cytokine secretion.

8 We analyzed caspase 1 activity and cytokine secretion.

9 lity, growth, gene expression signature, and cytokine secretion.

10 ability to induce T-cell proliferation, and cytokine secretion.

11 ly kinases (SFKs) and SFK inhibition blocked cytokine secretion.

12 signaling events that drive ISG15-dependent cytokine secretion.

13 ation also inhibits innate gammadelta17 cell cytokine secretion.

14 ctivation, secondary granule exocytosis, and cytokine secretion.

15 ells to degranulate and induce chemokine and cytokine secretion.

16 potent than regulatory T cells at inhibiting cytokine secretion.

17 n factor NF-kappaB, leading to chemokine and cytokine secretion.

18 rway hyperreactivity (AHR) and driven by Th2 cytokine secretion.

19 atory lung disease associated with increased cytokine secretion.

20 ith CHS had normal cytokine compartments and cytokine secretion.

21 igration, proliferation, differentiation and cytokine secretion.

22 igen expression, angiogenic and inflammatory cytokine secretion.

23 ligase gene (waaL) enhanced proinflammatory cytokine secretion.

24 effect is mediated by changes in tumor cell cytokine secretion.

25 e immune signaling, culminating in antiviral cytokine secretion.

26 nctions such as proliferation, survival, and cytokine secretion.

27 ry arthritis and colitis, and its effects on cytokine secretion.

28 l activation, metabolism, proliferation, and cytokine secretion.

29 induced caspase-1 activation, signaling, and cytokine secretion.

30 recruitment of Th cells and increased type 1 cytokine secretion.

31 the upregulation of costimulatory ligands or cytokine secretion.

32 plays a role in regulating pro-inflammatory cytokine secretion.

33 responses to mitogens as well as diminished cytokine secretion.

34 iate protective and repair functions through cytokine secretion.

35 nflammatory cell recruitment, and airway-Th2 cytokine secretion.

36 lA nuclear translocation, NO production, and cytokine secretion.

37 NFkappaB signaling, and, ultimately, overall cytokine secretion.

38 ired cell autonomously for optimal iNKT cell cytokine secretion.

39 vascular cell proliferation, migration, and cytokine secretion.

40 tivity was measured through proliferation or cytokine secretion.

41 ells, suppressing Th1 cell proliferation and cytokine secretion.

42 nces, as reported by T-cell proliferation or cytokine secretion.

43 oliferation and expression of Bcl-xL but not cytokine secretion.

44 y with monocytes and induced proinflammatory cytokine secretion.

45 n, surfactant clearance, and proinflammatory cytokine secretion.

46 ng oligomerization domain 2 (NOD2)-initiated cytokine secretion.

47 ilia beat frequency, and increased fluid and cytokine secretion.

48 nel in the regulation of alveolar epithelial cytokine secretion.

49 PS28, as negative regulators of NOD2-induced cytokine secretion.

50 ent of T cells did not directly alter T cell cytokine secretion.

51 l clearance and downregulate proinflammatory cytokine secretion.

52 ignalling increases Mtb growth, and augments cytokine secretion.

53 er type 1 (T(H)1) cells with proinflammatory cytokine secretion.

54 al M2 phenotype leading to suppressed T-cell cytokine secretion.

55 ects on M/M polarization, proliferation, and cytokine secretion.

56 ens, and they regulate other immune cells by cytokine secretion.

57 hannels (Ca(V)) as a downstream mechanism of cytokine secretion.

58 changing both cellular interaction sites and cytokine secretion.

59 e lung and maintain lung homeostasis through cytokine secretion.

60 TCR itself encodes information for specific cytokine secretion.

61 lular bacterial survival and proinflammatory cytokine secretion.

62 ating immune cell composition and peripheral cytokine secretion.

63 lated macrophages at the gene expression and cytokine secretion.

64 barrier function and reduced gliadin-induced cytokine secretion.

65 specific T-cell proliferation and pathogenic cytokine secretion.

66 ation to phagosomes, reduces proinflammatory cytokine secretion, abolishes phagosomal tubule formatio

67 downregulating signaling and proinflammatory cytokine secretion after chronic NOD2 and TLR4 stimulati

68 or (TLR) expression by quantitative PCR, and cytokine secretion after stimulation with mitogenic, TLR

69 iated with increased GM-CSF, IL-4, and IL-13 cytokine secretion among Ag-stimulated low-affinity iNKT

70 six statins tested, suppressing IgE-induced cytokine secretion among mouse MCs and basophils.

71 ted induction of endothelial proinflammatory cytokine secretion and adhesion molecule expression.

72 gainst TLR2 and TLR4 significantly inhibited cytokine secretion and attenuated Lsa21 induced phosphor

73 ate decreased PRR-induced mtROS, signalling, cytokine secretion and bacterial clearance.

74 lling, mitochondrial ROS (mtROS) production, cytokine secretion and bacterial clearance.

75 CRS arises from massive cytokine secretion and can be life-threatening, but it i

76 possess a range of suppressive function and cytokine secretion and can exert a genomic footprint on

77 ases caspase-8 and caspase-1 in coordinating cytokine secretion and cell death in response to immunos

78 pathogens by activating caspase-1-dependent cytokine secretion and cell death.

79 altered phagocytosis and synaptic pruning to cytokine secretion and changes in trophic support.

80 d T-cell cytokine interleukin-2, we show how cytokine secretion and competitive uptake determine this

81 (CD16) and by high levels of proinflammatory cytokine secretion and cytotoxic activity.

82 proliferation, differentiation, antibody and cytokine secretion and cytotoxicity.

83 or attachment protein receptor in AD-related cytokine secretion and epidermis-nerve communication.

84 d-type counterparts, but they exhibit normal cytokine secretion and expression of cytotoxic mediators

85 ion of the B-cell compartment and influences cytokine secretion and immunoglobulin production.

86 with sham-operated mice, including elevated cytokine secretion and impaired bacterial clearance.

87 ose from WT mice had reduced proinflammatory cytokine secretion and impaired macrophage activation.

88 aB expression in KCs in obese mice decreased cytokine secretion and improved insulin sensitivity and

89 C reparative activities, modulating in vitro cytokine secretion and in vivo gene expression for effec

90 phagy-related 5 (Atg5) resulted in decreased cytokine secretion and increased apoptosis.

91 is of bacteria while decreasing inflammatory cytokine secretion and increased intracellular ATP level

92 -induced STAT1 and STAT4 phosphorylation and cytokine secretion and increased TLR4-enhanced antimicro

93 dotoxin sensitivity, as measured by enhanced cytokine secretion and lethality following LPS challenge

94 in vitro priming of CD8(+) T cells improved cytokine secretion and lytic capacity of high-avidity T

95 C growth advantage through NF-kappaB-related cytokine secretion and metastatic TNBC cells exhibit gai

96 ergistically with the TLR pathway to promote cytokine secretion and neutrophil migration, whereas the

97 e-1 or caspase-11 activation, culminating in cytokine secretion and obliteration of the replicative n

98 cell migration to inflamed skin, but not for cytokine secretion and proliferation in regional lymph n

99 The link between cytokine secretion and proliferation rate holds both in

100 ivating both caspase-1-mediated inflammatory cytokine secretion and pyroptosis.

101 Inhibiting TIFA perturbed leukemic cytokine secretion and reduced the IC50 of chemotherapeu

102 The helminth-induced anti-inflammatory cytokine secretion and regulatory T cell suppressor acti

103 phages that coincides with the modulation of cytokine secretion and specific cellular processes.

104 of Pol III activity in macrophages restrains cytokine secretion and suppresses phagocytosis, two key

105 Early postvaccination cytokine secretion and T lymphocyte and plasmablast acti

106 egulated genes and reduced anti-inflammatory cytokine secretion and the number of T(reg) cells in vit

107 ed in the inhibition of both proinflammatory cytokine secretion and the upregulation of costimulatory

108 nction, likely further disrupting regulatory cytokine secretion and ultimately exacerbating inflammat

109 c infections induce T cells showing impaired cytokine secretion and up-regulated expression of inhibi

110 Therefore, we studied B cell cytokine secretion and/or Ab production across obesity m

111 over time, and functional (proliferation and cytokine secretion) and phenotypic (cell surface and int

112 could include roles in lymphoid development, cytokine secretion, and Ag presentation; however, these

113 nd were analyzed for survival, inflammation, cytokine secretion, and amyloidosis development.

114 T-cell proliferation, cytokine secretion, and antibody titers were measured by

115 o exhibit varying levels of phagocytosis and cytokine secretion, and are differentially expanded in c

116 f DC, through cyclin-dependent pathways, Th1 cytokine secretion, and by adding a nonviral Ag highly o

117 hallenge with decreases in mucus production, cytokine secretion, and collagen deposition.

118 3(bright) surface phenotype, proinflammatory cytokine secretion, and cytotoxicity.

119 of senescence associated secretory factors, cytokine secretion, and deposition of laminin 332 for se

120 Mast cell degranulation, cytokine secretion, and early signaling events in the Fc

121 in upregulation of antiapoptotic molecules, cytokine secretion, and enhanced effector function.

122 t v 1, epitope specificity, allergen-induced cytokine secretion, and expression of integrins alpha4be

123 mediators, rapidly communicating danger via cytokine secretion, and functioning as guardians of tiss

124 ll myeloperoxidase activity, proinflammatory cytokine secretion, and inflammation, were significantly

125 d for optimal MAPK and NF-kappaB activation, cytokine secretion, and intracellular bacterial clearanc

126 conjugate and analyzed for cell activation, cytokine secretion, and metabolic state.

127 e oxygen species formation, phagocytosis, or cytokine secretion, and neutrophils treated with azithro

128 urface strongly inhibited B cell activation, cytokine secretion, and proliferation.

129 ages were used to evaluate barrier function, cytokine secretion, and protein expression under basal c

130 ted cytokine secretion, elevated TH1-related cytokine secretion, and reduced human monocyte recruitme

131 s of immunity including antibody production, cytokine secretion, and T-cell activation; moreover, B c

132 The maturation, cytokine secretion, and T-cell stimulatory capacity of r

133 overexpressing PC cells, inducing apoptosis, cytokine secretion, and the recruitment of human periphe

134 ) through the alteration of surface markers, cytokine secretion, and their ability to activate T cell

135 accumulation, phosphorylation of tau and/or cytokine secretion, and, as illustrated with FERMT2, it

136 raneoplastic syndrome mechanisms via ectopic cytokine secretion, and/or a non-HHV-8 virus.

137 n predominantly on dying cells and stimulate cytokine secretion as well as leukocyte recruitment in v

138 The newly developed cytokine secretion assay consists of anti-IL-22 and anti

139 ory activity in a lipopolysaccharide-induced cytokine secretion assay.

140 The cytokine-secretion assay (CSA) can overcome this limitat

141 d cells infected with HBV was assessed using cytokine secretion assays and imaging-based killing assa

142 et, and the combination of several different cytokine secretion assays can be used to purify and char

143 ls was performed with the combination of two cytokine secretion assays detecting IL-17A- and IL-22-pr

144 ode cells were analyzed in proliferation and cytokine secretion assays or by flow cytometry, RNA sequ

145 (controls) and analyzed in proliferation and cytokine secretion assays or by flow cytometry.

146 aded with tumor peptide and antigen-specific cytokine secretion assays, we determined that TNF-alpha

147 de data to clinicians and doctors concerning cytokines secretion at minute concentrations and the pre

148 STK4 dampened TLR4/9-induced proinflammatory cytokine secretion but enhanced TLR3/4-triggered IFN-bet

149 rferon-gamma and tumor necrosis factor-alpha cytokine secretion by CD4(+) T cells and antibody produc

150 n macrophages, and increased proinflammatory cytokine secretion by colon tissue and macrophages.

151 iacetate succinimidyl ester dye dilution and cytokine secretion by ELISpot.

152 nts demonstrate that nGO-PEG indeed provokes cytokine secretion by enhancing integrin beta8-related s

153 I interferon and stimulated proinflammatory cytokine secretion by human peripheral blood cells.

154 as IL-10 and TGF-beta1 significantly reduce cytokine secretion by ILC2s.

155 In this study, we assessed rhythms of cytokine secretion by immune cells and tested their resp

156 yanin resulted in enhanced proliferation and cytokine secretion by keyhole limpet hemocyanin-experien

157 to A1 and A3 adenosine receptors, increases cytokine secretion by LPS activated monocytes.

158 xin-43 blockade were associated with reduced cytokine secretion by macrophages in response to LPS and

159 extracellular DNA to trigger proinflammatory cytokine secretion by monocytes, in a STING- and inflamm

160 t TH17 subsets on modulating proinflammatory cytokine secretion by monocytes.

161 esion area, immune cell composition, ex vivo cytokine secretion by peritoneal cells or bone marrow de

162 leukin-15 (rhIL-15) synergistically enhanced cytokine secretion by proliferating HIVGag-specific CD8

163 integrity, and induction of proinflammatory cytokine secretion by S. Paratyphi A but not by S. Typhi

164 s by dying radiosensitive cells, and altered cytokine secretion by surviving radioresistant cells.

165 differentiation by altering proinflammatory cytokine secretion by T and B cells.

166 gnition receptor (PRR)-induced signaling and cytokine secretion can lead to inflammatory bowel diseas

167 latory molecule expression and chemokine and cytokine secretion compared with monocyte-derived dendri

168 ent the importance of PKR, TRIF, and TBK1 in cytokine secretion during L. pneumophila infection of ma

169 ation, inflammatory cell death pathways, and cytokine secretion during SARS-CoV, MERS-CoV, and SARS-C

170 In contrast, a lack of Kif5b did not affect cytokine secretion, early FcepsilonRI-initiated signalin

171 LO/LTB4 inhibition downregulated TH2-related cytokine secretion, elevated TH1-related cytokine secret

172 can modulate the immune response by inducing cytokine secretion, especially IL-10 and MIP-1beta.

173 locks both TCR-dependent and TCR-independent cytokine secretion from a Sezary syndrome-derived cell l

174 teen abacavir analogues were synthesized and cytokine secretion from abacavir/abacavir analogue-respo

175 gh fibrillar amyloid beta (Abeta)-stimulated cytokine secretion from both wild-type and APP knock-out

176 specific innate immune cells, we quantified cytokine secretion from H. pylori-infected primary gastr

177 Cytokine secretion from HCT116 cells and cellular migrat

178 for functional immunophenotyping to examine cytokine secretion from human immune cells stimulated ex

179 tionally similar to IL-25 and elicits type 2 cytokine secretion from innate type 2 lymphocytes, NKT,

180 philia and suppressed ex vivo OVA-stimulated cytokine secretion from lung cells in the OVA respirator

181 AMA0825 did not affect pro-inflammatory cytokine secretion from other ROCK-positive cell types,

182 study immune responses by measuring rates of cytokine secretion from single T cells, and to measure a

183 trated by potent and sustained inhibition of cytokine secretion from T cells, a therapeutic target fo

184 Here we show that TRAIL-triggered cytokine secretion from TRAIL-resistant cancer cells is

185 Tregs have more potent suppressive effect on cytokines secretion from CD4(+) CD25(-) responder T cell

186 development, sublineage differentiation, and cytokine secretion function.

187 1BB CAR T cells retained their cytotoxic and cytokine secretion functions longer than CD28 CAR T cell

188 olated to evaluate proliferation, apoptosis, cytokine secretion, gene expression and cell metabolism.

189 of IL-33 on goblet cell differentiation and cytokine secretion has not been described.

190 ffects of lung endothelium and epithelium on cytokine secretion, identify new biomarkers of disease e

191 a higher threshold to induce TRIF-dependent cytokine secretion (IL-1beta, IL-6, IL-10, and TNF-alpha

192 al key parameters such as immunoglobulin and cytokine secretion immediately after AIT.

193 injury, bacterial clearance from the lungs, cytokine secretion in bronchoalveolar lavage, lung antim

194 e the link between cell stress and increased cytokine secretion in CAPS.

195 However, EVT did not enhance cytokine secretion in dNK, whereas stimulation of dNK wi

196 protein 3 (MTMR3) in amplifying PRR-induced cytokine secretion in human macrophages and defined MTMR

197 tors of proinflammatory signaling leading to cytokine secretion in Listeria monocytogenes-infected ma

198 arasite replication in mouse infections, and cytokine secretion in macrophage infections.

199 quired for upregulation of these CLR and for cytokine secretion in macrophages stimulated with the MI

200 urium-induced pyroptosis and proinflammatory cytokine secretion in macrophages.

201 -initiated MAPK/NF-kappaB/PI3K signaling and cytokine secretion in macrophages.

202 IL-1 signaling, thereby leading to decreased cytokine secretion in MDMs upon stimulation of a broad r

203 cantly reduced crypt damage and inflammatory cytokine secretion in NOD2(-/-) mice 3 d after anti-CD3

204 pecific CLA(+) and CCR4(+) proliferation and cytokine secretion in patients with and without APT reac

205 The nitric oxide production and cytokine secretion in rat peritoneal cells indicate immu

206 gradation, preventing gene transcription and cytokine secretion in response to cytokine stimulation.

207 8.0 trauma, p < 0.05) and reduced leukocyte cytokine secretion in response to lipopolysaccharide sti

208 e diseases are characterized by dysregulated cytokine secretion in response to pattern-recognition re

209 ired for B cell survival, proliferation, and cytokine secretion in response to signaling through seve

210 Cytokine secretion in response to VLY and G. vaginalis a

211 induction of pyroptosis and proinflammatory cytokine secretion in the control of growth and eliminat

212 associated with increased proliferation and cytokine secretion in the spleen, intraepithelial lympho

213 in from S Typhimurium induced cell death and cytokine secretion in THP-1 cells and primary human mono

214 ished their proliferation, cytotoxicity, and cytokine secretion in vitro in response to CD19 recognit

215 MDA-induced cytokine secretion in vitro was dependent on the presenc

216 ge associated with decreased proinflammatory cytokine secretion in vivo.

217 liferation (in models 1 and 2), inflammatory cytokine secretion (in models 1, 2, and 3), circulating

218 tion, its anti-inflammatory activity against cytokines secretion induced by these pathogens was deter

219 erized by enhanced glycolysis and an altered cytokine secretion (interleukin-6 P<0.0001, interleukin-

220 (mtDNA) integrity, morphology, phenotype and cytokine secretion into storage buffer.

221 Paracrine signalling mediated by cytokine secretion is essential for liver regeneration a

222 Abnormal cytokine secretion is often regarded as an early cue of

223 e underlying mechanisms are unknown, massive cytokine secretion is thought to be involved.

224 li modulating amino acid catabolism, as were cytokine secretion levels and regulatory T cell numbers.

225 e cell lines changed the gene expression and cytokine secretion levels of CD8(+) T cells.

226 agreement, CD28 blockade suppressed NKT cell cytokine secretion, lowering the ratio of IFN-gamma:IL-4

227 udy, we show that M. tuberculosis impairs DC cytokine secretion, maturation, and Ag presentation thro

228 Despite the absence of inflammatory cytokine secretion, mDCs incubated with P. falciparum-in

229 ged with lipopolysaccharide (LPS) to measure cytokine secretion; monocytes were also challenged with

230 Furthermore, reduced cytokine secretion observed at high Ag density for both

231 ine promoters, consistent with the increased cytokine secretion observed.

232 The H4R modulated the cytokine secretion of CD4+ T cells and splenocytes and a

233 n vitro MVC treatment reduced activation and cytokine secretion of CD8(+) T cells via a CCR5-independ

234 effect of synthesized iminosugars 1-3 on the cytokine secretion of IL-4, IL-6, and IFN-gamma was eval

235 dditionally, IL4M markedly suppressed type-2 cytokine secretion of splenocytes beyond the effect of A

236 ne diseases through autoantibody production, cytokine secretion, or antigen presentation to T cells.

237 activation test (MAT), MPLA induced the same cytokine secretion pattern as LPS (ESM: IL-6, IL-12, TNF

238 3(+) BMRMs show a specific transcriptome and cytokine secretion pattern demonstrating a specific immu

239 ), such as Sezary syndrome, display aberrant cytokine secretion patterns that contribute to pathology

240 8) changed CD8(+) T cell gene expression and cytokine secretion patterns to create an immunosuppressi

241 T2, and NKT17 subsets, defined through their cytokine-secretion patterns and the expression of key tr

242 ntly infected cells to limit proinflammatory cytokine secretion, perhaps as an immune evasion strateg

243 pon FcgammaR cross-linking without affecting cytokine secretion, phagocytosis, or nitrate/nitrite pro

244 maturation and their acquisition of an NKT17 cytokine secretion phenotype in the thymus.

245 markers, T lymphocyte proliferation/ex-vivo cytokine secretion, plasma cardiometabolic risk factors,

246 show here that IFN-alpha treatment enhanced cytokine secretion, polyfunctionality, degranulation, an

247 n accessory cells and TLR-4-dependent innate cytokine secretion (predominantly from CD14+ monocytes)

248 e detected in most subjects with CSU, with a cytokine secretion profile more typical of a TH1-cell re

249 We observed that the magnitude, breadth, and cytokine secretion profile of HIV-1-specific CD8 T cell

250 tro GALNS stimulation, (b) modulation of the cytokine secretion profile, (c) decrease in GALNS-specif

251 HP-NAP-activated DCs had a Th1 cytokine secretion profile, with high IL-12 and relative

252 patients with ABPA is skewed to a T helper 2 cytokine secretion profile.

253 Cultured lymphocytes were analyzed for cytokine secretion profiles and cellular polarization by

254 atural killer targets confirmed the distinct cytokine secretion profiles of dNK and peripheral blood

255 n primary T cells: they can drive a la carte cytokine secretion profiles, biased T cell differentiati

256 h together with specific gene expression and cytokine secretion profiles, FL-TFH constitute a heterog

257 AMPK and reductions in NK cell cytotoxicity, cytokine secretion, proliferation, and telomerase expres

258 trophoblast cells in vitro are by modulating cytokine secretion, reducing apoptosis to levels seen in

259 or attachment protein receptors in mediating cytokine secretion related to AD.

260 However, the gene expression and cytokine secretion response to combinatorial stimuli wer

261 rsed the proinflammatory gene expression and cytokine secretion seen in BMDM from high fat-fed mice.

262 nged their signalling pathways and adipokine/cytokine secretions, such as adiponectin and leptin, res

263 at could be targeted to inhibit the aberrant cytokine secretion that drives the immunopathogenesis of

264 a combination of cell-to-cell signalling and cytokine secretion that elicit unique biological effects

265 cultured immune cells induced a differential cytokine secretion that may contribute to CRC metastasis

266 that CRC cell-resident F. nucleatum promotes cytokine secretion that may potentiate tumor growth and

267 ed with this, and based on their patterns of cytokine secretion, there was a difference in their capa

268 osomal proteins and inducing proinflammatory cytokine secretion through protease-activated receptor 2

269 dose-dependently promoted DC maturation and cytokine secretion through TLR4 signaling pathway.

270 alters how DCs metabolize glutamate, skewing cytokine secretion to bias T cell function.

271 imulated ILC2(10)s were isolated to evaluate cytokine secretion, transcription factor signaling, meta

272 sess the effects of the miRNAs identified on cytokine secretion (tumor necrosis factor alpha [TNF-alp

273 overall magnitude of TNF-alpha and IFN-gamma cytokine secretion upon Ab-dependent and -independent ac

274 (CTLR) triggering cellular cytotoxicity and cytokine secretion upon high-affinity interaction with t

275 -) mice displayed enhanced proliferation and cytokine secretion upon receiving OX40 cosignaling.

276 c cells, were used to compare differences in cytokine secretions upon stimulation.

277 -alpha production, IL-8 gene expression, and cytokine secretion, upregulated IL-10 secretion, and inh

278 Type 1 cytokine secretion was also enhanced by DC and was most

279 based on HbA(1c) levels revealed that lower cytokine secretion was coupled to higher glycolytic rate

280 Both pro- and anti-inflammatory cytokine secretion was decreased in MDMs from GG carrier

281 ng TLRs were exposed to cSSSI pathogens, and cytokine secretion was determined by enzyme-linked immun

282 Plasma cytokine secretion was higher in both female and male ra

283 Cytokine secretion was measured 24 hours postinfection b

284 Myeloid and lymphoid cell cytokine secretion was measured after in vitro PBMC rest

285 Cytokine secretion was measured by multiplex analysis an

286 Cytokine secretion was measured using multiplex and ELIS

287 that were sufficient in LAIR-1, CD3-induced cytokine secretion was significantly suppressed in the p

288 ng night shifts, with the exception of IL-2, cytokine secretion was still rhythmic but with peak leve

289 ted IL-10 secretion, but not proinflammatory cytokine secretion, was inhibited by rapamycin in mDCs.

290 ell tropism, effect on tissue integrity, and cytokine secretion were compared.

291 infection, studies of inflammasome-dependent cytokine secretion were conducted with the human THP-1 m

292 d NOD2 stimulation, both the acetylation and cytokine secretion were dramatically decreased.

293 Drug-specific proliferative responses and cytokine secretion were measured.

294 lav was the culprit, although differences in cytokine secretion were observed.

295 udies, including migration, Ca(2+) flux, and cytokine secretion, were conducted with primary human mo

296 e-like cells (THP-1) significantly increased cytokine secretion, whereas A20 overexpression using len

297 Pam-(Lys-betaNSpe)6-NH2, blocks LPS-induced cytokine secretion with a potency comparable to that of

298 tion on cytokine promoters and the decreased cytokine secretion with chronic NOD2 stimulation.

299 to 37 degrees C does not normalize monocyte cytokine secretion within 36 hours, whereas warming to 3

300 diated indirect alloantigen presentation and cytokine secretion within the GI tract.