ライフサイエンスコーパス: cytokine signal

1 an important transducer of growth factor and cytokine signals.

2 , including those derived from extracellular cytokine signals.

3 tagonists, represent new ways of fine-tuning cytokine signals.

4 or memory CD4(+) T cells sense and interpret cytokine signals.

5 L-10, indicating a broader role for ISG15 in cytokine signaling.

6 etic diseases of immune deficiency affecting cytokine signaling.

7 mature lymphocytes or of Stat6-dependent Th2 cytokine signaling.

8 ein hypothetically involved in regulation of cytokine signaling.

9 bers, including the end-to-end regulation of cytokine signaling.

10 newly minted ASCs, by enhancing pro-survival cytokine signaling.

11 -type placenta or by genetically restricting cytokine signaling.

12 gamma-secretase protease to pro-inflammatory cytokine signaling.

13 ced by activated T cells and inhibits gammac cytokine signaling.

14 innate lymphoid progenitor (ChILP), but not cytokine signaling.

15 can regulate both pro- and anti-inflammatory cytokine signaling.

16 zing the need for spatially resolved data on cytokine signaling.

17 damage potentiates senescence and activates cytokine signaling.

18 proliferation, immune synapse formation, and cytokine signaling.

19 s a cytoplasmic tyrosine kinase critical for cytokine signaling.

20 me activation, and autocrine proinflammatory cytokine signaling.

21 that suppress COX-2 as well as inflammatory cytokine signaling.

22 nce by regulating cross talk between TCR and cytokine signaling.

23 naling, including induction of suppressor of cytokine signaling.

24 lete immune cells, and IL4 or IL7RA to block cytokine signaling.

25 ell differentiation, thymic development, and cytokine signaling.

26 target genes involved in innate immunity and cytokine signaling.

27 r profile represents protein translation and cytokine signaling.

28 nctions, including spatial learning, through cytokine signaling.

29 en endocytosis in the positive regulation of cytokine signalling.

30 e effects on adipocyte-derived NF-kappaB and cytokine signalling.

31 t these pathways converge on pro-tumorigenic cytokine signalling.

32 155 (miR-155) and its target, suppressor of cytokine signaling 1 (SOCS-1).

33 the methylation pattern in the suppressor of cytokine signaling 1 (SOCS1) gene in smokers and non-smo

34 p120-induced IL-23 upregulated suppressor of cytokine signaling 1 (SOCS1) protein in T cells, which i

35 phages leads to proteolysis of suppressor of cytokine signaling 1 (SOCS1), alleviating its repression

36 inducible negative regulators suppressor of cytokine signaling 1 (SOCS1), SOCS3, and ubiquitin-speci

37 rozygous truncation variant in suppressor of cytokine signaling 1 (SOCS1).

38 Furthermore, suppressor of cytokine signaling 1, an immune suppressive molecule in

39 Basal expression levels of Suppressor of Cytokine Signalling 1 (chSOCS1), a negative regulator of

40 MyD88 expression by decreasing suppressor of cytokine signaling-1 (SOCS-1) mRNA stability.

41 -gamma production by targeting suppressor of cytokine signaling-1 (SOCS1) and Src homology-2 domain-c

42 Expression of the suppressor of cytokine signaling-1 (SOCS1) is inactivated in hematopoi

43 cient mice, likely via reduced suppressor of cytokine signaling-1 (Socs1) regulation by miR-142-5p.

44 he kinase-inhibitory region of suppressor of cytokine signaling-1 (SOCS1) regulatory protein protects

45 ented by overexpression of the suppressor of cytokine signaling-1 or inactivation of the BH3-only pro

46 iated with increased levels of suppressor of cytokine signaling-1, implicating increased degradation

47 sion, with a resulting loss of suppressor of cytokine signaling-1.

48 The suppressor of cytokine signaling 2 (SOCS2) acts as substrate recogniti

49 dentified the ubiquitin ligase suppressor of cytokine signaling 2 (SOCS2) as a direct, biologically r

50 interleukin (IL)-8, IL-11, and suppressor of cytokine signaling 2 (SOCS2) messenger RNAs confirmed st

51 screen for candidates revealed suppressor of cytokine signaling 2 (SOCS2), an inhibitor of growth hor

52 ocarbon receptor (AHR) and the suppressor of cytokine signaling 2 (SOCS2).

53 STAT3 target genes, including suppressor of cytokine signaling 2/3 (Socs2/3); Pim-1 proto-oncogene,

54 counter of inflammatory signals alters their cytokine (signal 3) secretion pattern.

55 is factor alpha (TNFalpha) and suppressor of cytokine signaling 3 (SOCS3) activities in whole retina

56 Here, we show that host suppressor of cytokine signaling 3 (SOCS3) can also bind to EBOV VP40,

57 Suppressor of cytokine signaling 3 (SOCS3) is involved in regulating t

58 Suppressor of cytokine signaling 3 (SOCS3) regulates STAT3 activation

59 on proteolytic degradation of suppressor of cytokine signaling 3 (SOCS3) resulting in low levels of

60 Epigenetic changes in suppressor of cytokine signaling 3 (SOCS3) were determined by using me

61 ) by transcellular delivery of suppressor of cytokine signaling 3 (SOCS3) within extracellular vesicl

62 sed the gene transcription of suppressors of cytokine signaling 3 (SOCS3), a critical negative regula

63 find that deletion of cortical suppressor of cytokine signaling 3 (SOCS3), a negative regulator of cy

64 ases in dipeptidylpeptidase 4, suppressor of cytokine signaling 3 (SOCS3), and insulin receptor subst

65 activated STAT3 (PIAS3) and/or suppressor of cytokine signaling 3 (SOCS3), factors that inhibit STAT3

66 ile comprising upregulation of suppressor of cytokine signaling 3 (SOCS3), glycoprotein A repetitions

67 to decrease the expression of Suppressor of Cytokine Signaling 3 (SOCS3), leading to increases in ST

68 Expression of one such gene, Suppressor of Cytokine Signaling 3 (Socs3), rescued the self-renewal d

69 educed amount of expression of suppressor of cytokine signaling 3 (SOCS3)-a key negative regulator of

70 vered that CD37 interacts with suppressor of cytokine signaling 3 (SOCS3); therefore, absence of CD37

71 itor of the JAK/STAT3 pathway [suppressor of cytokine signaling 3 (SOCS3)].

72 constitutive, independent from suppressor of cytokine signaling 3 activity.

73 Th17 cells due to induction of suppressor of cytokine signaling 3 and 5.

74 Suppressor of cytokine signaling 3 elicited the immunosuppressive acti

75 ges was accompanied by reduced suppressor of cytokine signaling 3 expression and an inadequacy of IL-

76 STAT3 inhibition, by restoring suppressor of cytokine signaling 3 expression and thus inhibiting exce

77 Restoring suppressor of cytokine signaling 3 expression under TAM knockdown cond

78 penditure, and reduced arcuate suppressor of cytokine signaling 3 expression, indicative for enhanced

79 s, which, in turn, upregulated suppressor of cytokine signaling 3 expression.

80 Rosi-mediated upregulation of suppressor of cytokine signaling 3 leads to an altered ratio of nuclea

81 rmore, exogenous expression of suppressor of cytokine signaling 3 or either T-bet or STAT1 RNA interf

82 STAT3 and suppressor of cytokine signaling 3 protein levels were measured by imm

83 n, which, in turn, may inhibit suppressor of cytokine signaling 3 transcription.

84 ated TNFAIP3 suppressed SOCS3 (suppressor of cytokine signaling 3)-activated STAT3/VEGFA indirectly.

85 ne-mediated signaling pathway (suppressor of cytokine signaling 3, C-X-C ligand 8 [CXCL8]), and respo

86 nal deletion in basal cells of suppressor of cytokine signaling 3, encoding a negative regulator of t

87 nsitive to the STAT3 inhibitor suppressor of cytokine signaling 3.

88 y BA-FXR through activation of suppressor of cytokine signaling-3 (SOC3).

89 was mediated through transient suppressor of cytokine signaling-3 (SOCS3) inhibition of the STAT5b tr

90 , rs4251961; IL-10, rs1800871; suppressor of cytokine signaling-3, rs4969170; nucleotide-binding olig

91 tes that DRD3 signaling evokes suppressor of cytokine signaling 5 expression, a negative regulator of

92 ppel-like factor 4 (KLF4), and suppressor of cytokine signaling 5.

93 Here we show that suppressor of cytokine signaling 6 (SOCS6) and Cullin 5, two component

94 re we developed antibody-based activators of cytokine signaling (AcCS), which recognize cytokines onl

95 IL-6-type cytokine signalling additionally involves the recruitmen

96 To explore how anti-inflammatory cytokine signaling affects Abeta pathology, we investiga

97 Besides physiological fine-tuning of cytokine signals, altering the nature or potency of the

98 It can both enhance and suppress cytokine signalling, although the role of endocytosis fo

99 , was critical for generation of both innate cytokine signaling and Ag-specific B and T cell response

100 We, therefore, examined cytokine signaling and CD4(+) T cell differentiation in

101 here it functions as a negative regulator of cytokine signaling and cell proliferation.

102 pport, neurotransmitter and ion homeostasis, cytokine signaling and immune function, and debris engul

103 roles of FAK/Pyk2 in mediating inflammatory cytokine signaling and implicate FAK/Pyk2 inhibitors as

104 This gain-of-function mutation dysregulates cytokine signaling and is associated with increased accu

105 roach for identifying new genes that promote cytokine signaling and leukemogenesis.

106 by eosinophil infiltration, enhanced type 2 cytokine signaling and M2 macrophage polarization in the

107 ew evidence supporting the interplay between cytokine signaling and mitochondrial physiology in skele

108 e SH2B3 gene encodes a negative regulator of cytokine signaling and naturally occurring loss-of-funct

109 nvolved in lipid metabolism, immune response/cytokine signaling and other diverse pathways, including

110 and its receptor c-Mpl regulates downstream cytokine signaling and platelet homeostasis.

111 1 is a critical effector of pro-inflammatory cytokine signaling and plays important roles in immune f

112 xpansion and survival, and more globally ILC cytokine signaling and proliferation.

113 nd IL-7Ralpha proteins on T cells to inhibit cytokine signaling and promote inflammation.

114 of inflammasomes results in proinflammatory cytokine signaling and pyroptosis.

115 e-BII cell stage was exacerbated by abnormal cytokine signaling and repetitive inflammatory stimuli.

116 ed from reduced survival following deficient cytokine signaling and STAT5 activation.

117 c instruments to clarify the causal roles of cytokine signaling and upstream inflammation in immune-r

118 p OA exhibited differential pro-inflammatory cytokine signalling and peripheral and local lymphocyte

119 thin the innate immune system as chemotactic cytokines, signaling and recruiting host immune cells to

120 to regulate differentiation, lineage choice, cytokine signaling, and cell cycle.

121 lved in interferon-mediated innate immunity, cytokine signaling, and cellular defenses were downregul

122 ncluding matrix remodeling, chemokine and/or cytokine signaling, and immunological functions such as

123 Pases in early erythropoiesis, downstream of cytokine signaling, and in terminal erythroblast maturat

124 face receptors to regulate STAT3 activation, cytokine signaling, and the induction of both vascular e

125 at are impaired by the suppression of type 2 cytokine signaling, and they are reversed by recolonizat

126 were seen in pathways involved in apoptosis, cytokine signaling, and TLR pathways.

127 We have identified cytokine, signaling, and transcription factors that are

128 which offers novel approaches for targeting cytokine signaling as well as potential therapeutic appl

129 progenitor cells exhibited severely impaired cytokine signaling as well as upregulation of p53 and ex

130 Cytokine array analysis suggested changes to cytokine signaling associated with iAs exposure.

131 eaction-diffusion models for the dynamics of cytokine signaling at two successive scales: in immunolo

132 SPRY domain-containing suppressor of cytokine signaling box protein (SPSB) 2-deficient macrop

133 S703I JAK1 is not only hypermorphic for cytokine signaling but also neomorphic, as it enables si

134 lular protein SOCS1 is known to downregulate cytokine signaling by inhibiting the JAK-STAT pathway.

135 uncover the targeting of a lineage-specific cytokine signaling by miRNAs as a mechanism regulating i

136 entified several relevant targets, including cytokine signaling by the IL-1 receptor (IL-1R) pathway

137 SHMT2 also promotes inflammatory cytokine signalling by interacting with the deubiquityla

138 In turn, miR-193b restricts cytokine signalling, by targeting the receptor tyrosine

139 In contrast, aberrant cytokine signaling can also result in conditions of immu

140 teraction whereby IL4Ralpha-dependent type 2 cytokine signaling can directly inhibit hypoxic neutroph

141 IL-6 cytokine signaling can enhance ASC production and has be

142 lly important, and augmenting or attenuating cytokine signals can have deleterious or therapeutic eff

143 lk between sphingosine-1-phosphate (S1P) and cytokine signaling cascades in astrocyte activation and

144 pathogen binding, TLRs initiate specialized cytokine signaling catered to the class of invading path

145 resistant cells through an IL1beta/IL8/CXCR1 cytokine signaling circuit.

146 deficient in Toll-like receptors (TLRs) and cytokine signaling components (TLR-2(-/-), TLR-4(-/-), T

147 However, whether cytokine-signaling contributes to negative selection rem

148 ddition of IL-12 to cultures, revealing that cytokine signaling could restore the acquisition of effe

149 Cytokine signaling dependent on JAK3 and JAK1 is critica

150 iseases provides new opportunities to target cytokine signaling directly as a novel treatment strateg

151 line of transgenic mice, we show that these cytokines signal directly into cardiomyocytes, limiting

152 CA/B) and not to be associated with specific cytokine signaling events.

153 -31 induces expression of four suppressor of cytokine signaling family members and provide evidence t

154 rotein (CISH), a member of the suppressor of cytokine signaling family of negative feedback regulator

155 encoding members of the SOCS (suppressor of cytokine signaling) family are critical targets of ThPOK

156 slationally, such as the SOCS (suppressor of cytokine signaling) family.

157 This suggests AcCS can be used to manipulate cytokine signaling for basic science and possibly for th

158 hem to convert danger signals into versatile cytokine signals for the regulation of stress hematopoie

159 lls depend on host-derived costimulation and cytokine signals for their full and sustained activation

160 Excessive cytokine signaling frequently exacerbates lung tissue da

161 s substantial increases in the expression of cytokine signaling genes relative to wild-type HCMV.

162 ifferentiation and lineage commitment, while cytokine signaling has been shown to instruct the fate o

163 he role of RAGE in directly mediating type 2 cytokine signaling has never been investigated.

164 wever, the identity of responsible PTK(s) in cytokine signaling have not been elucidated.

165 that regulates inflammation (suppressors of cytokine signaling, high-mobility group protein B1, oxid

166 Therefore, blocking IL-6 cytokine signaling in (mesenteric) adipocytes may be a n

167 recently showed that interleukin (IL)-6-type cytokine signaling in adipocytes induces free fatty acid

168 We thus hypothesized that IL-6-type cytokine signaling in adipocytes may regulate insulin se

169 tly, targeting PERK neither disrupted normal cytokine signaling in astrocytes or microglia nor impair

170 These findings provide novel evidence that cytokine signaling in beta-cells functions to limit vira

171 signaling and pathways related to chemokine/cytokine signaling in both compartments.

172 role of genetic background, lymphocytes, and cytokine signaling in diet-induced obesity and insulin r

173 In summary, our dissection of cytokine signaling in embryonic HSCs has uncovered uniqu

174 Inhibition of cytokine signaling in mice with X-CGD reduced HPC number

175 ficial effects of blocking anti-inflammatory cytokine signaling in preclinical mouse models provide s

176 between NQO1 expression and proinflammatory cytokine signaling in prostate cancer.

177 peroxide (H(2)O(2)) permeability can enhance cytokine signaling in several cell types.

178 However, constitutive cytokine signaling in T cells and activation of bystande

179 phatase PTPN2 attenuates T-cell receptor and cytokine signaling in T cells to maintain peripheral tol

180 However, little is known about how cytokine signaling in the brain may influence higher-ord

181 of TLR4, TLR5, and TLR9 and their downstream cytokine signaling in the gingival epithelial cells in p

182 ier function, the intestinal microbiota, and cytokine signaling in the pathogenesis of inflammatory b

183 nalling 1 (chSOCS1), a negative regulator of cytokine signalling in mammals, are 16-fold higher in DF

184 arisons support a role for IL-6 inflammatory cytokine signalling in OCCC pathogenesis.

185 Many aspects of cytokine signalling in the immune system have been explo

186 JAK1 to JAK3 and tyrosine kinase 2) mediate cytokine signals in the regulation of hematopoiesis and

187 luding epigenetic, cell-fate regulation, and cytokine signaling, in MF tumors and CTCL cell lines.

188 This reduces cytokine signaling including IL6 that is implicated in m

189 of gamma-secretase substrates have a role in cytokine signaling, including the IL-6 receptor, IL-1 re

190 While it is known that cytokine signals induce thymocytes to express Runx3, it

191 domains of cytokine receptors that phenocopy cytokine signaling induced by nonphysiological homo- and

192 eptor ligation-along with co-stimulatory and cytokine signals-induces a glycolytic anabolic program t

193 Unexpectedly, IL6-type cytokine signaling inducing STAT3 activation rendered ce

194 nd whether such T cells are sensitive to the cytokine-signaling inhibitor tofacitinib, a Janus kinase

195 Aberrant cytokine signaling initiated from mutant receptor tyrosi

196 We find that incorporating dynamic cytokine signaling into a simple model of Th differentia

197 lambdaR expression was independent of direct cytokine signaling into T cells.

198 se 1 and Janus kinase 2, potently suppresses cytokine signaling involved in AD pathogenesis.

199 We investigated additional cytokine signals involved in residual eosinophilia in th

200 findings provide evidence that inflammatory cytokine signaling is a key process underlying epilepsy

201 n of a functional pre-B-cell receptor (BCR), cytokine signaling is attenuated and the tonic/autonomou

202 s of P. aeruginosa We demonstrate that IL-17 cytokine signaling is essential for mouse survival and p

203 that targeting both JAK1- and TYK2-mediated cytokine signaling is more effective than TYK2 inhibitio

204 Cytokine signaling is transmitted by cell-surface recept

205 How structural dynamics affects cytokine signaling is under debate.

206 d lymphocyte activation, but its function in cytokine signaling is unknown.

207 h codes for a receptor implicated in myeloid cytokine signaling, is a direct target for both Rcor1 an

208 gnaling; (2) removal of keratinocyte-induced cytokine signaling, leading to reductions in pathologic

209 GF-beta, although they clearly require other cytokine signals, leading to the activation of transcrip

210 Aside from downstream cytokine signaling, little is known about the regulation

211 ty was associated with up-regulation of IL-6 cytokine signaling machinery, which might be attributabl

212 aling pathway, suggesting alternative gammac cytokine signaling may support TEM homeostasis in the ab

213 Thus, intracellular cytokine signalling may serve as 'fate determinants' use

214 Therefore, the local interference of cytokine signaling mediated by siRNA-loaded nanoparticle

215 DNA regulatory protein and an extracellular cytokine signaling molecule that promotes airway inflamm

216 nes clustered in multiple pathways including cytokine signaling, mRNA processing, endosomal trafficki

217 contribute to treatment tolerance through a cytokine-signaling network that involves macrophage-deri

218 rter of TCR signaling that is insensitive to cytokine signaling, Nur77-eGFP, we identify a sharp, min

219 es and offer new insights into the impact of cytokine signaling on leukemia development.

220 of IL-2R gamma-chain (Il-2rgamma)-dependent cytokine signaling only to donor cells in NSG recipients

221 ns affecting the common gamma-chain (gammac) cytokine signaling pathway and mice with similar mutatio

222 anism dependent on NF-kappaB proinflammatory cytokine signaling pathway in both normal and steatotic

223 Focusing on a key cytokine signaling pathway previously implicated in cuta

224 The cytokine-cytokine signaling pathway was enriched in both sexes.

225 l development critically depends on the IL-7 cytokine signaling pathway.

226 nitial interaction with APCs, altering early cytokine signal pathways and leading to T cell unrespons

227 ), which is a negative regulator of multiple cytokine signaling pathways and is associated with incre

228 signaling (SOCS) proteins are inhibitors of cytokine signaling pathways and may play a role in restr

229 nrichment analysis revealed pro-inflammatory cytokine signaling pathways as dysregulated, and this wa

230 afenib would also have inhibitory effects on cytokine signaling pathways in immune cells.

231 We set out to investigate the role that cytokine signaling pathways play in these early processe

232 ssociated with AEC2-derived up-regulation of cytokine signaling pathways that are known to provoke in

233 anus kinases (JAKs) is required for multiple cytokine signaling pathways, and as such, JAK inhibitors

234 iption factor network, chromatin remodeling, cytokine signaling pathways, cell adhesion, and cell pro

235 naling, T cell activation, and chemokine and cytokine signaling pathways.

236 gy to identify known oncogenic components of cytokine signaling pathways.

237 performance of -lactams may be linked to key cytokine signaling pathways.

238 of toll-like receptors and pro-inflammatory cytokine signaling pathways.

239 e cross talk between the T-cell receptor and cytokine signalling pathways to limit inappropriate T-ce

240 -related genes that are involved in multiple cytokine signalling pathways.

241 Cytokine-signaling pathways are very tightly regulated b

242 ndicate that targeting specific cytokines or cytokine-signaling pathways to reduce or ameliorate lung

243 cally focus on the roles of pro-inflammatory cytokine signaling, peripheral monocyte infiltration, mi

244 of gp130 suggests GSK-3 can regulate normal cytokine signaling, potentially enabling metabolic and i

245 mined by whether persistent TCR signaling or cytokine signaling predominates, respectively.

246 these findings reveal that IKBKE-associated cytokine signaling promotes tumorigenicity of immune-dri

247 nscription factor NF-kappaB, ubiquitination, cytokine signaling, protein folding, type I interferon p

248 at this mechanism integrating BCR, TLR9, and cytokine signals provides a peripheral checkpoint for DN

249 iers, transcription factors and mediators of cytokine signaling, recapitulating the combinations of m

250 By contrast, long-range cytokine signaling requires a high density of cytokine p

251 and intratumoral T(reg) cells, and highlight cytokine signaling responsiveness as a key determinant o

252 Normal cytokine signaling shows distinct differences in activat

253 cell receptor and B cell receptor signaling, cytokine signaling, skin barrier function, and mast cell

254 s, expression of AhR/cytokines/suppressor of cytokine signaling (SOCS) (spleen/heart), and production

255 Suppressors of cytokine signaling (SOCS) 1 and 3 proteins and reactive

256 investigate the expression of suppressor of cytokine signaling (SOCS) 1 in tissues from asthmatic pa

257 anistically distinct from the suppressors of cytokine signaling (SOCS) family of genes.

258 und that Cish, a member of the suppressor of cytokine signaling (SOCS) family, is induced by TCR stim

259 Suppressor of cytokine signaling (SOCS) proteins are inhibitors of cyt

260 The suppressor of cytokine signaling (SOCS) proteins are negative regulato

261 Suppressors of cytokine signaling (SOCS) proteins inhibit signaling by

262 The suppressors of cytokine signaling (SOCS) proteins play important roles

263 ni involve the exploitation of suppressor of cytokine signaling (SOCS) proteins that are well-known n

264 etermined that AMs can secrete suppressor of cytokine signaling (SOCS) proteins within microparticles

265 endogenous inhibitors, namely, suppressor of cytokine signaling (SOCS) proteins, phosphatases, and pr

266 Interestingly, suppressor of cytokine signaling (SOCS)-1 directly associated with TLR

267 s like cyclooxygenase (COX)-2, suppressor of cytokine signaling (SOCS)-3, and matrix metalloproteinas

268 Suppressor of cytokine signaling (SOCS)1 is a cross-functional negativ

269 Following IL-4 exposure, suppressor of cytokine signaling (SOCS)1 is highly induced in human mo

270 ut may involve upregulation of suppressor of cytokine signaling (SOCS-3) proteins that are associated

271 Members of the suppressor of cytokine signalling (SOCS) family have been implicated i

272 b, a negative regulator of the suppressor of cytokine signalling (SOCS) genes, which inhibit HSC migr

273 Suppressor of cytokine signalling (SOCS) proteins are a family of indu

274 transcription (e.g., IRF1 and suppressor of cytokine signaling [SOCS] 1) was strongly impaired.

275 tutive activation of STAT1 through autocrine cytokine signaling, suggesting that subclinical endotoxe

276 ted with symptom scores, and these mapped to cytokine signalling/T-cell activation-associated pathway

277 Thus, we identify a cytokine signal that must be received in muscle to contr

278 Thus, Otop1 defines a unique target of cytokine signaling that attenuates obesity-induced adipo

279 esearch aims to define immune parameters and cytokine signaling that distinguish cohorts of IL-2Ralph

280 enotypic switch is associated with increased cytokine signaling that promotes nonautonomous resident

281 p among the B cell receptor (BCR), TLR9, and cytokine signals that regulate B cell responses to DNA-c

282 hrough a temporally orchestrated sequence of cytokine signals that sustain division rather than survi

283 that, in response to antigen receptor and/or cytokine signaling, the E-Id protein axis modulates the

284 controlling TLR4 signaling and inflammatory cytokine signaling through a negative feedback regulatio

285 rotein that functions to negatively regulate cytokine signaling through GP130 and pSTAT3Y705 and is m

286 as advanced, mechanism-based manipulation of cytokine signaling through protein engineering has becom

287 Moreover, these three cytokines signal through a common beta-chain receptor bu

288 Although members of this family of cytokines signal through a single shared receptor, bioch

289 IL-36 cytokines signal through the IL-36 receptor (IL-36R) and

290 e rules reveal that Th cells harness dynamic cytokine signaling to implement a system of quorum sensi

291 inase is essential for coupling inflammatory cytokine signals to the cell death machinery in the diff

292 that RAGE is a critical component of type 2 cytokine signal transduction mechanisms, which is a driv

293 ization ratio" as a metric to understand how cytokine signaling translates into polarization of singl

294 gival epithelial cells expressing diminished cytokine signaling upon P. gingivalis stimulation.

295 nhibiting IL-6, IL-1beta or TNF or targeting cytokine signalling via Janus kinase inhibition in the t

296 Transforming growth factor (TGF)-beta cytokines signal via a complex network of pathways to re

297 CDK4 regulated prometastatic inflammatory cytokine signaling, whereas CDK6 mainly controlled DNA r

298 F signaling system indicates perceiving more cytokine signals which in fact do not exist at the syste

299 affected extracellular matrix remodeling and cytokine signaling, with expression profiles indicating

300 associations between vaccine responsiveness, cytokine signaling within peripheral immune cells, and b