ライフサイエンスコーパス: cytotoxicity assay

1 etion system dependent Y. pseudotuberculosis cytotoxicity assay).

2 ing assay to breast cancer cell lysate and a cytotoxicity assay.

3 icrog/ml, as determined by a Vero cell-based cytotoxicity assay.

4 ssay results were confirmed by the Vero cell cytotoxicity assay.

5 NAs to induce cell death in an in vitro cell cytotoxicity assay.

6 assay, and antibody-dependent cell-mediated cytotoxicity assay.

7 sensitivities and specificities compared to cytotoxicity assay.

8 s conferring unusual potency in a tumor cell cytotoxicity assay.

9 oth mAbs in antibody-dependent cell-mediated cytotoxicity assay.

10 hat these variants are active in the cognate cytotoxicity assay.

11 44 lacks inhibitory capacity in a redirected cytotoxicity assay.

12 a simple anti-ganglioside antibody-mediated cytotoxicity assay.

13 le were identified by using a (51)Cr release cytotoxicity assay.

14 spot assay and the standard chromium release cytotoxicity assay.

15 n production was demonstrated by a HeLa cell cytotoxicity assay.

16 roduction as assessed by PCR and a HeLa cell cytotoxicity assay.

17 tolytic activity against leukemia cells in a cytotoxicity assay.

18 eir effects against rat P. carinii by an ATP cytotoxicity assay.

19 red the presence of high dose SEB during the cytotoxicity assay.

20 ermined serially with a complement-dependent cytotoxicity assay.

21 ically active TNF-alpha, using the L929 cell cytotoxicity assay.

22 gically active TNF-alpha using the L929 cell cytotoxicity assay.

23 ct receptor on cells as well as an apoptosis/cytotoxicity assay.

24 he activity of the preparation in a WEHI 164 cytotoxicity assay.

25 diagnosing FHL than hemophagocytosis and the cytotoxicity assay.

26 in B digital ELISA was 100% sensitive versus cytotoxicity assay.

27 the National Cancer Institute's 60 cell line cytotoxicity assay.

28 ant was cytotoxic to BHS PMNs in an in vitro cytotoxicity assay.

29 toxin and similar IC50 values in a Vero cell cytotoxicity assay.

30 as neutralize purified toxins in an in vitro cytotoxicity assay.

31 analyzed by focal expansion assay and CD107 cytotoxicity assay.

32 eliable, quantitative, and robust cell-based cytotoxicity assay.

33 lene glycol), is demonstrated using standard cytotoxicity assays.

34 cellular injury was evaluated using the same cytotoxicity assays.

35 ility was assessed using TUNEL and viability/cytotoxicity assays.

36 were examined by flow cytometry and in vitro cytotoxicity assays.

37 nel formation, N-terminus translocation, and cytotoxicity assays.

38 analyses and to neutralize them in Vero cell cytotoxicity assays.

39 7-aminoactinomycin D staining in flow-based cytotoxicity assays.

40 nstrated through fluorescence microscopy and cytotoxicity assays.

41 efficacy was analyzed in in vitro complement cytotoxicity assays.

42 llular cytotoxicity and complement-dependent cytotoxicity assays.

43 found to be nontoxic in preliminary in vitro cytotoxicity assays.

44 acetate succinimidyl ester dye dilution) and cytotoxicity assays.

45 by tetramer binding, IFN-gamma ELISPOT, and cytotoxicity assays.

46 ular phagocytosis, and Ab-dependent cellular cytotoxicity assays.

47 the normal hematopoietic cells in short-term cytotoxicity assays.

48 tectable by standard lymphoproliferation and cytotoxicity assays.

49 esponses, as detected by in vivo or in vitro cytotoxicity assays.

50 L39) and transfected into HCC cells for G207 cytotoxicity assays.

51 ormoxia (21% O2), and infected with G207 for cytotoxicity assays.

52 active as paclitaxel in tubulin assembly and cytotoxicity assays.

53 lytic function in both direct and redirected cytotoxicity assays.

54 fluorescein succinimidyl ester staining, and cytotoxicity assays.

55 ro, as determined by IFN-gamma secretion and cytotoxicity assays.

56 ts of E1A are easily measured in vitro using cytotoxicity assays.

57 EGF receptor, Flk-1, as measured by in vitro cytotoxicity assays.

58 ved as a model for xenograft target cells in cytotoxicity assays.

59 cells with different VHL status in TNFalpha cytotoxicity assays.

60 d HLA-class I- restricted manner in standard cytotoxicity assays.

61 ating alloantibody levels were determined by cytotoxicity assays.

62 e-linked immunospot (ELISPOT), tetramer, and cytotoxicity assays.

63 ions (XMLRs) and in natural killer (NK) cell cytotoxicity assays.

64 ften do not lyse HIV-infected targets in 4-h cytotoxicity assays.

65 , and potentiated the activity of CT-2584 in cytotoxicity assays.

66 ed in gamma-IFN enzyme-linked immunospot and cytotoxicity assays.

67 lpha RI as measured in Ab-dependent cellular cytotoxicity assays.

68 or frequency (limiting dilution assay [LDA]) cytotoxicity assays.

69 TAT ELISA system and by complement-dependent cytotoxicity assays.

70 s systems of infected mice in direct ex vivo cytotoxicity assays.

71 the MART-1(27-35) peptide, AAGIGILTV, in 4-h cytotoxicity assays.

72 and perforin-dependent killing in redirected cytotoxicity assays.

73 he presence or absence of AH in conventional cytotoxicity assays.

74 of chickenpox, by T lymphoproliferation and cytotoxicity assays.

75 c cytotoxic T cells (CTLs) in direct ex vivo cytotoxicity assays.

76 ive to CMV peptides in IFN-gamma release and cytotoxicity assays.

77 bolished the inhibitory function of Ly49A in cytotoxicity assays.

78 e variant epitopes were poorly recognized in cytotoxicity assays.

79 cells was evaluated in in vitro and in vivo cytotoxicity assays.

80 prescription drugs in p-tau aggregation and cytotoxicity assays.

81 in two different P. aeruginosa T3SS-mediated cytotoxicity assays.

82 ed effector functions as defined by in vitro cytotoxicity assays.

83 ry, and enzyme-linked immunosorbent spot and cytotoxicity assays.

84 In a Vero cell cytotoxicity assay, 24B11 was approximately two times mo

85 In 4-day cytotoxicity assays, ABCG2-mediated resistance to SN-38

86 ein endothelial cells (HUVECs) in redirected cytotoxicity assays after T-cell receptor triggering and

87 KO/gld LAK cells were cytotoxic in long term cytotoxicity assays against TNF-sensitive tumor lines, a

88 nzyme-linked immunospot (ELISPOT) assay, and cytotoxicity assay all showed that the Ad-sig-TAA/ecdCD4

89 biological context is also demonstrated in a cytotoxicity assay and a cell internalization study with

90 e cell and to the lungs, as evidenced by the cytotoxicity assay and analyses of the injury markers in

91 ines and the results were validated by WST-1 cytotoxicity assay and annexin V-FITC/propidium iodide (

92 ositive B16 and CT26 tumor cells in vitro by cytotoxicity assay and antitumor activity in vivo using

93 MTT cytotoxicity assay and confocal microscopy images were c

94 s was predictive for rejection in an in vivo cytotoxicity assay and correlated with skin allograft re

95 noplexes in fibroblasts were evaluated using cytotoxicity assay and florescence microscopy, respectiv

96 ally active, as demonstrated by a cell-based cytotoxicity assay and inhibition of microglia activatio

97 MTS assay cytotoxicity assay and live-dead cell viability test wer

98 esence of Shiga toxin using both a Vero cell cytotoxicity assay and the Shiga Toxin Quik Chek test (S

99 In vitro cytotoxicity assay and tumor S-phase fraction can be use

100 cells were quantified by the standard YAC-1 cytotoxicity assay and were found to appear in the genit

101 nontoxigenic in the Vero cell tissue culture cytotoxicity assay and were therefore nontoxigenic for d

102 of higher avidity measured both by in vitro cytotoxicity assays and a new methodology that measures

103 We have used cytotoxicity assays and a variety of biophysical approac

104 ble alloreactivity in both proliferation and cytotoxicity assays and are able to proliferate and secr

105 e recognized AFP-transfected targets in both cytotoxicity assays and cytokine release assays.

106 th Flu-M1 and G9(280)-9V-specific T cells in cytotoxicity assays and IFN-gamma ELISPOT assays.

107 ing HLA-A*0201(+)/AFP(+) tumor cells in both cytotoxicity assays and IFN-gamma enzyme-linked immunosp

108 n, cytotoxicity, and antibody-dependent cell cytotoxicity assays and in vivo by human lymphoma xenogr

109 ndemic H1N1 strain, and influenza B virus in cytotoxicity assays and intracellular-cytokine-staining

110 icide gene therapy for neuroblastomas by GCV cytotoxicity assays and noninvasive bioluminescent imagi

111 e peptide KIF and HER-2-expressing tumors in cytotoxicity assays and secreted gamma interferon (IFN-g

112 ated by both inhibition studies and in vitro cytotoxicity assays and shows a correlated structure-act

113 In vivo cytotoxicity assays and various gene-deficient recipient

114 ntihuman-globulin-enhanced T cell and B cell cytotoxicity assays and/or flow cytometry.

115 radioimmunoassay (RIA), neuroblastoma (N2A) cytotoxicity assay, and liquid chromatography/mass spect

116 heir viability was assessed by a BCECF-based cytotoxicity assay, and their function was assessed by a

117 ng flow cytometry, [(125)I]IAAP binding, and cytotoxicity assays, and interaction was documented in 2

118 Toxin-specific enzyme immunoassays, cytotoxicity assays, and PCR were used to analyze 48 tox

119 etermined in both tubulin polymerization and cytotoxicity assays, and several analogues with enhanced

120 e antigen proteins were analyzed by cellular cytotoxicity assays, and their interactions with antibod

121 For this reason, ecotoxicity and cytotoxicity assays are of special interest in order to

122 Enzyme-linked immunospots, cytotoxicity assays as well as adoptive transfer in NOD/

123 (1) Proliferation and cytotoxicity assays, as well as skin graft survival, dem

124 a more traditional methyl tetrazolium (MTT) cytotoxicity assay at selected time points following app

125 ific CD8(+) T cells, using a flow-cytometric cytotoxicity assay based on caspase-3 activation in dyin

126 Cytotoxicity assays based on colony formation revealed t

127 ur IgA MAbs neutralized ricin in a Vero cell cytotoxicity assay, blocked toxin-induced interleukin-8

128 ytolytic properties, as measured in vitro by cytotoxicity assays, but differed markedly in their capa

129 darabine to bendamustine resulted in maximum cytotoxicity assayed by 3,3'-dihexyloxacarbocyanine iodi

130 ciency of peptide generation was measured in cytotoxicity assays by determining 1) the kinetics of pr

131 This simple cytotoxicity assay can potentially be used for screening

132 ibrils, and that the PC 12-based comparative cytotoxicity assay can provide insights into toxicity di

133 Natural killer (NK)-cell cytotoxicity assays can quickly screen for all of these

134 ate drug safety in a regime that the current cytotoxicity assays cannot cover and be generally applie

135 during a 3-month period using a cell culture cytotoxicity assay (CCCA).

136 As assessed by MTT cytotoxicity assay, CEM cells were also significantly mo

137 cence microscopy using a live-dead viability-cytotoxicity assay conducted by an observer uninformed o

138 Standard degranulation and chromium release cytotoxicity assays confirm the ability of cryopreserved

139 Finally, in vitro dose-escalation cytotoxicity assays confirm the biocompatibility of the

140 In vitro cytotoxicity assays confirmed that mouse neutrophils lys

141 Cytotoxicity assays confirmed that the LF(N) mutations i

142 Data derived from cytotoxicity assays, confocal laser scanning microscopy,

143 Cytotoxicity assays, confocal laser scanning microscopy,

144 sensitization of target cells in an in vitro cytotoxicity assay, consistent with enhanced antigen pre

145 T cell proliferation assays and NK cytotoxicity assays demonstrate that this fusion protein

146 A Vero cell cytotoxicity assay demonstrated that 90% of the represen

147 sistent with chemistry observation, in vitro cytotoxicity assay demonstrated that these agents induce

148 Cytotoxicity assays demonstrated at least additivity in

149 Cytotoxicity assays demonstrated higher responses to don

150 Cr51 release cytotoxicity assays demonstrated less susceptibility to

151 In vitro cytotoxicity assays demonstrated that activity of an unt

152 Cytotoxicity assays demonstrated the crucial role of the

153 Moreover, NK cell receptor blockade in cytotoxicity assays demonstrates that NCRs play a major

154 s determined by LC/MS, the RBA, RIA, and N2A cytotoxicity assay detected 73, 83, and 51% of the total

155 e a mechanistic understanding of an in vitro cytotoxicity assay developed while he was a fellow with

156 Although cytotoxicity assays did not support differences between

157 and B by enzyme immunoassay and cell culture cytotoxicity assay (EIA/CCA).

158 In vitro cytotoxicity assays established that the mAb-MGB drug co

159 also were inactive in antiproliferative and cytotoxicity assays, except for 3-methyl-6-beta-D-ribofu

160 hiazol-2-yl)-2,5-diphenyltetrazolium bromide cytotoxicity assay, flow cytometric apoptosis quantifica

161 this work, we have developed a microfluidic cytotoxicity assay for a cell culture and detection plat

162 The CC50 value obtained in a cytotoxicity assay for compound 9 was >100 muM, correspo

163 ed bioactivity assays along with traditional cytotoxicity assays for in vitro screening.

164 om proteasomal breakdown and, as measured in cytotoxicity assays, from major histocompatibility compl

165 Cytotoxicity assays further confirmed that these melanom

166 ficile by anaerobic bacterial culture and/or cytotoxicity assays has been largely replaced by rapid e

167 In a 3-day cytotoxicity assay, human SPF45 overexpression conferred

168 HPLC, mass spectrometry, cytotoxicity assays, IFN-gamma ELISPOT, and human breast

169 pathway, we developed an indirect glycolate cytotoxicity assay in a 1,536-well plate format for high

170 e complexes were determined through in vitro cytotoxicity assay in human fibroblasts (MRC5) and two c

171 ted against the standard LIVE/DEAD Viability/Cytotoxicity Assay in small tumor spheroid cultures, sho

172 ctivity was established in an A2780 cellular cytotoxicity assay in which 21 showed an IC(50) = 95 nM.

173 logical activity in the complement-dependent cytotoxicity assay in which the wild-type IgG2 is inacti

174 say, along with in vitro phosphorylation and cytotoxicity assays in leukemia cells, we compared PKC41

175 malaria parasite was validated by performing cytotoxicity assays in red blood cells infected with Pla

176 Estimates have been obtained using in vivo cytotoxicity assays in which peptide-pulsed splenocytes

177 In cytotoxicity assays, incubation of 1 microM CPT-11 with

178 Viability/cytotoxicity assays indicate dose-dependent cytotoxic ef

179 In vitro cytotoxicity assays indicate that Ly49I recognizes H2(q)

180 In vitro cytotoxicity assays indicated that 27 was more potent th

181 In vitro cytotoxicity assays indicated that donor immunization in

182 Results of cytotoxicity assays indicated that the nonstructural pro

183 CD8 T cells in the spleen, as determined by cytotoxicity assays, intracellular staining for gamma in

184 Mouse or human ESCs were subjected to cytotoxicity assays involving their respective species-m

185 The commonly performed complement-dependent cytotoxicity assay is insensitive compared with newer me

186 In cytotoxicity assays, ISG15 was a potent inducer of cytol

187 ified stool toxin levels via a modified cell cytotoxicity assay, isolated C. difficile by anaerobic c

188 he present study, using liposome-leakage and cytotoxicity assays, LC-MS/MS-based proteomics, and CCF-

189 Furthermore, lactate dehydrogenase cytotoxicity assay, live-dead cell staining and transmis

190 primate and human subjects using traditional cytotoxicity assay methods.

191 cal, superoxide radical, hypochlorous acid), cytotoxicity assay (MTT) and quantification of TNF-alpha

192 From these preliminary cytotoxicity assays (MTT) we found that C8-propyl-catech

193 The cytotoxicity assay of nanoMgO particles, examined using

194 al blood mononuclear cells, and 51Cr release cytotoxicity assay of rNcSRS2-stimulated effector cells.

195 Competitive uptake and cytotoxicity assays of 2 were investigated and interacti

196 Comparative cytotoxicity assays of 41 MTM(ox) analogues using E-twen

197 fluorescence microscope images and live/dead cytotoxicity assays of HeLa cancer cells suggested that

198 sitive or negative results from conventional cytotoxicity assays of nanomaterials (NMs) suggests that

199 ldin A are likely to be generated during the cytotoxicity assays of the sulfoxide derivatives.

200 etermined by use of the complement-dependent cytotoxicity assay, of 77+/-19% or with donor-specific a

201 tomy, sera were tested by flow cytometry and cytotoxicity assay on porcine peripheral blood mononucle

202 The cytotoxicity assay on RAW264.7 cells suggested that the

203 on linoleate uptake in vitro was measured by cytotoxicity assays on Caco-2 cells.

204 Cytotoxicity assays on human epithelial ovarian carcinom

205 Cytotoxicity assays on primary clinical DS-ALL samples d

206 sis, with biological activity as assessed by cytotoxicity assays performed in adenocarcinoma human al

207 In vitro cytotoxicity assays performed on H2987 lung adenocarcino

208 ile isolates selected from 50 tissue culture cytotoxicity assay-positive clinical stool samples.

209 This rapid and efficient in vivo cytotoxicity assay recapitulated the specificity of NK c

210 and 3 h in sensitized recipients in in vivo cytotoxicity assay, reflecting Ab-mediated cytotoxicity.

211 125% as determined by the RBA, RIA, and N2A cytotoxicity assay, respectively.

212 osine triphosphatase (ATPase) and cell-based cytotoxicity assays, respectively.

213 iazol-2-yl]-2,5-diphenyltetrazolium bromide) cytotoxicity assay revealed that Sf9 cells expressing CY

214 Cytotoxicity assays revealed that LF was cytotoxic to WE

215 Cytotoxicity assays revealed that PZS was highly toxic.

216 Cytotoxicity assays revealed that the compounds inhibite

217 Cytotoxicity assays revealed that the TDN p53 cell lines

218 Cytotoxicity assays revealed that the two trophoblast ce

219 Cytotoxicity assays show good correlation at the outer l

220 In addition, cytotoxicity assays show that the LLC-MDR1-3H cells are

221 A dye reduction cytotoxicity assay showed that IL-4 induced resistance t

222 Cytotoxicity assay showed that NK cells from HLA-C1 posi

223 In vitro cytotoxicity assays showed CD8+ T cell-mediated eliminat

224 is, reverse antibody-dependent cell-mediated cytotoxicity assays showed potent degranulation and cyto

225 Cytotoxicity assays showed that both nonfunctionalized a

226 onding to variant epitopes in direct ex vivo cytotoxicity assays showed that each mutation caused a l

227 In vitro cytotoxicity assays showed that T lymphocytes or CD8(+)

228 Cytotoxicity assays showed that the date extracts were a

229 T-cell cytotoxicity assays showed that TIV is processed and the

230 tumors did not lyse 9L cells in 51Cr-release cytotoxicity assays, specific cytotoxicity was demonstra

231 Cytotoxicity assays suggest that toxicity is a property

232 The cytotoxicity assay suggested that the inhibition of PAM

233 -deficient and wild-type controls in in vivo cytotoxicity assays, suggesting donor-specific tolerance

234 Using specimens that were negative by cytotoxicity assay/TC/NAAT, clinical cutoffs were set at

235 of UV-irradiated AdLacZ adenovirus and in a cytotoxicity assay that appears to be the result of aber

236 virus-infected mice in vivo using an in vivo cytotoxicity assay that features class II-expressing B c

237 g a multilayered multiple myeloma cell-based cytotoxicity assay that modeled disease niche, normal li

238 e compounds were evaluated using an in vitro cytotoxicity assay that utilizes a hippocampal-derived c

239 Safety of drugs is regularly evaluated using cytotoxicity assays that measure cell death.

240 In the plaque reduction and cytotoxicity assays, the 5-bromo analogue (3) showed goo

241 ed in interferon-gamma Elispot assays and in cytotoxicity assays, these responses were of low frequen

242 to standard Hsp90alpha binding and cell line cytotoxicity assays, this study also highlights the firs

243 ected DCs also serve as effective targets in cytotoxicity assays, thus providing a general method for

244 In the cytotoxicity assay to correlate intracellular anthracycl

245 them in a murine fibroblast cell line (L929) cytotoxicity assay to investigate whether these mycobact

246 Compound 28 at 3 muM reduced resistance in cytotoxicity assay to paclitaxel in P-gp-expressing SW62

247 One of the transconjugants is shown by cytotoxicity assay to produce toxin B at a similar level

248 virus 68 (gammaHV68) in in vitro and in vivo cytotoxicity assays to show CD4-dependent killing of gam

249 ts" was further refined using cell viability-cytotoxicity assays to two 1,3,5-triaryl pyrazoline comp

250 mmunoassays are insufficiently sensitive and cytotoxicity assays too complex; assays that detect toxi

251 blot and immunoprecipitation, as well as by cytotoxicity assay using Fas-expressing target cells.

252 sitive and negative signaling in an in vitro cytotoxicity assay using sorted NK cell subsets as effec

253 analogues proved to be highly active in the cytotoxicity assay using the human cervix carcinoma cell

254 Cytotoxicity assays using A549 and H596 lung cancer cell

255 In vitro cytotoxicity assays using hematopoietic cells and fibrob

256 In vitro cytotoxicity assays using multiple different cell lines

257 LA-DR molecules and in IFN-gamma ELISPOT and cytotoxicity assays using peripheral blood lymphocytes f

258 Antibody-dependent cellular cytotoxicity assays using purified peripheral blood mono

259 s by RT-PCR, Western blot analysis, IHC, and cytotoxicity assays using siRNA or transfection-modified

260 minor histocompatibility Ag, as detected by cytotoxicity assays using SMCY(311-319)-specific CTL.

261 ing results were confirmed functionally with cytotoxicity assays using sorted 129/J NK cells.

262 Biocompatibility was evaluated using cytotoxicity assays utilising L-929 cell line.

263 NK cell function was tested using a cytotoxicity assay versus YAC-1 target cells on day 14 o

264 Metformin cytotoxicity assay was performed using the MTS assay.

265 NK cells in a redirected, antibody-dependent cytotoxicity assay was triggered by a number of receptor

266 f lethal concentration derived from standard cytotoxicity assays was not.

267 Using in vivo cytotoxicity assays we demonstrated that early up-regula

268 analysis, mutagenesis, NMO-IgG binding, and cytotoxicity assay, we have disclosed the key role of as

269 Using an ultrasensitive cytotoxicity assay, we measured C. difficile toxin A (Tc

270 unoassays compared to the accepted standard, cytotoxicity assay, we studied three enzyme immunoassays

271 Using NK cell activation and NK cytotoxicity assays, we compared ADCC-mediating antibodi

272 is of intracellular IFN-gamma production and cytotoxicity assays, we determined that CD16(+) liver NK

273 Using in vivo cytotoxicity assays, we evaluated the effector systems m

274 ate dehydrogenase release and live/dead cell cytotoxicity assays, we found in different cell lines, a

275 Using protein aggregation and cytotoxicity assays, we found that the dissociation of t

276 ng with resistance/enzymatic, hemolytic, and cytotoxicity assays were also studied.

277 Complement-dependent cytotoxicity assays were performed to determine the cyto

278 cally, Affymetrix U133 Plus 2.0 GeneChip and cytotoxicity assays were performed to obtain basal mRNA

279 Cytotoxicity assays were performed using osteoblast and

280 ctin expression, induction of apoptosis, and cytotoxicity assays were performed.

281 ELISPOT and cytotoxicity assays were used to evaluate tumor-reactive

282 sponded to VP16 393-405 in proliferation and cytotoxicity assays when presented by DRB1*0402, DRB1*11

283 NM interactions compared to the conventional cytotoxicity assays where only one aspect of toxicity ca

284 SPOT), interferon (IFN)-gamma secretion, and cytotoxicity assays, whereas HCV-specific antibodies wer

285 toma cells and medulloblastoma cell lines in cytotoxicity assays, whereas HER2-negative tumor cells w

286 e standard Chinese hamster ovary (CHO)-based cytotoxicity assay, which took about 72 h to complete.

287 s of memory T cells was reflected by in vivo cytotoxicity assays, which showed decreased clearance of

288 In cytotoxicity assays, wild-type Chinese hamster ovary (CH

289 body-positive sera in a complement-dependent cytotoxicity assay with cultured melanoma cell lines.

290 A mammalian cytotoxicity assay with human keratinocytes (HaCaTs) yie

291 A cytotoxicity assay with NCI/ADR-RES, a drug resistant ce

292 ue extracts, is even more active than QL9 in cytotoxicity assays with 2C CTL.

293 utralization of diphtheria toxin in in vitro cytotoxicity assays with a 50% effective concentration o

294 Candidates were characterized in cellular cytotoxicity assays with Chinese hamster ovary (CHO) cel

295 efficacy as hapalosin in antagonizing MRP in cytotoxicity assays with HL-60/ADR cells.

296 Cytotoxicity assays with human breast cancer cells showe

297 In vitro tumor cytotoxicity assays with lymph node effector cells revea

298 In vitro cytotoxicity assays with peripheral blood mononuclear ce

299 In vitro cancer cell cytotoxicity assays with the human colon carcinoma cell

300 particularly in localities where performing cytotoxicity assays would be problematic.