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1 ed by nail psoriasis and current or previous dactylitis.
2 h groups, however, share a high frequency of dactylitis.
3 d clinical signs of psoriatic enthesitis and dactylitis.
4  most common severe disease thought to cause dactylitis.
5 rs of life for (1) painful events other than dactylitis, (2) dactylitis, and (3) acute chest syndrome
6  patients in the placebo group, p=0.002) and dactylitis (24 events in 14 patients vs 123 events in 42
7 s of initial and recurrent episodes of pain, dactylitis, acute chest syndrome, and hospitalization; e
8 ical outcome (vaso-occlusive painful crisis, dactylitis, acute chest syndrome, splenic sequestration,
9  malaria, but DP was associated with reduced dactylitis and P. falciparum parasitization.
10 tients are more likely to be female, exhibit dactylitis and small joint involvement, and express anti
11 ation (including joint count, evaluation for dactylitis and/or enthesitis, and skin examination) and
12 1) painful events other than dactylitis, (2) dactylitis, and (3) acute chest syndrome (ACS).
13 ed development of PsA, including enthesitis, dactylitis, and bone destruction.
14 pheral arthritis, axial disease, enthesitis, dactylitis, and skin and nail psoriasis; additional sear
15 rd digit of the right hand, corresponding to dactylitis (arrow).
16 the current knowledge comes from analysis of dactylitis as a secondary outcome measure.
17 cal trials in PsA have increasingly included dactylitis as an important secondary outcome measure.
18 ylitis, no studies have been conducted using dactylitis as the primary outcome measure, and the curre
19 who were asymptomatic at enrollment had less dactylitis as well as fewer hospitalizations and transfu
20 dictors of an adverse outcome: an episode of dactylitis before the age of one year (relative risk of
21 rstanding of the pathogenesis of PsA-related dactylitis comes from experimental animal models of PsA-
22                                              Dactylitis had limited utility as a predictor.
23 nt adaptive immune mechanisms amplifying the dactylitis inflammatory response.
24 hly DP was associated with a reduced rate of dactylitis (IRR: 0.47; 95% CI: 0.23 to 0.96; p = 0.038).
25 -inflammatory cytokines, the pathogenesis of dactylitis is best understood as an initial aberrant inn
26                                              Dactylitis is diffuse swelling of the digits that is usu
27                   Regarding the treatment of dactylitis, no studies have been conducted using dactyli
28                           The combination of dactylitis of a toe, heel pain, and oligoarthritis appea
29 subgroups but suggested that the presence of dactylitis, rather than age, has the greatest capacity t
30 e Study of Sickle Cell Disease reported that dactylitis, severe anemia, and leukocytosis in very youn
31 t may appear in the first two years of life (dactylitis, severe anemia, and leukocytosis) can help to
32  of key clinical domains (joints, enthesium, dactylitis, spine, and skin and nails), and coming to co
33 ng the gene dose (Ikbkb(GoF/GoF)) results in dactylitis, spondylitis, and characteristic nail changes
34            The development of enthesitis and dactylitis was not due to high circulating levels of IL-