ライフサイエンスコーパス: damage in

1 7.5% of nuclei analyzed) and plasma membrane damage (in 100% of treated hyphae).

2 red: the prokaryotic type, which removes the damage in 11-13-nucleotide-long oligomers, and the eukar

3 , and the eukaryotic type, which removes the damage in 24-32-nucleotide-long oligomers.

4 the association of sciatic nerve structural damage in 3 Tesla (3T) magnetic resonance neurography (M

5 ilibrium, however, is displaced by extrinsic damage in a dosage-dependent response.

6 organisms protect their future progeny from damage in a fluctuating environment is a fundamental que

7 tive, human-imposed management suffered less damage in a hurricane's path than unmanaged systems.

8 impact of these different stages of cardiac damage in a large, real-world, multicenter cohort of sym

9 rotein of Mycobacterium tuberculosis removes damage in a manner analogous to yeast and humans in a 25

10 rt-hypothermic treatment can ameliorate this damage in a model of PA.

11 joints and is a negative regulator of tissue damage in a mouse model of inflammatory arthritis.

12 T cell axis triggered corneal sensory nerve damage in a mouse model of ocular graft-versus-host dise

13 Here, we show that upon damage in a mouse model, epidermal cells at the wound ed

14 ncreased carbon tetrachloride-mediated liver damage in a mouse model.

15 CGS21680, significantly reduced OA cartilage damage in a murine model of obesity-induced OA.

16 and increase the risk of TCDD-elicited liver damage in a sex-specific manner.

17 blood proportionally to the degree of axonal damage in a variety of neurological disorders, including

18 pV1 sensed heat normally but suffered scald damages in a hot environment.

19 nd adduct that help to accommodate the bulky damages in a hydrophobic pocket to facilitate TLS.

20 esized that S epidermidis could promote skin damage in AD by the production of a protease that damage

21 d plays an important role in cerebrovascular damage in AD, we investigated the role of the Abeta-fibr

22 treat hyperexcitability and related synaptic damage in AD.

23 tended to correlate positively with protein damage in adult birds while in nestlings they positively

24 llum is a target of alcoholism-related brain damage in adults, yet no study has prospectively tracked

25 cell culture and severe fever-induced brain damage in affected individuals.

26 d response in macrophage phenotype following damage in aged human skeletal muscle: implications for s

27 us end joining (NHEJ) repair pathway and DNA damage in alveolar type II (ATII) cells and emphysema de

28 starch is associated with reduced histology damage in animal studies, and improvements in clinical r

29 itical thresholds at 6% and 64% of cartilage damage in area, and 22% and 56% in depth were predicted

30 observations might reflect structural brain damage in areas that are related to cognition; however,

31 pheral inflammation and contribute to tissue damage in arthritis.

32 for mediating either inflammation or tissue damage in arthritis.

33 sses cause neurodegeneration and this causes damage in associated white matter tracts.

34 r reactive oxygen species generation and DNA damage in ATII cells obtained from individuals with this

35 IgG antibodies cause inflammation and organ damage in autoimmune diseases such as systemic lupus ery

36 response, a cellular system triggered by DNA damage in bacteria, depends on DNA replication for the g

37 tic leakage and SEM analysis showed membrane damage in bacterial cells.

38 reveals the dynamic molecular process of RBC damage in biomedical devices and mechanoporation that, t

39 Acute vascular damage in biopsies that would be classified as mild acut

40 ral induced hemarthrosis causes further bone damage in both FVIII(-/-) and FIX(-/-) mice, but has lit

41 IC1a NT residues, NT(1-20), reduced neuronal damage in both in vitro model of acidotoxicity and in vi

42 lammation in skin and reduce UVB-induced DNA damage in both melanocytes and keratinocytes.

43 opose that UV-DDB is a general sensor of DNA damage in both NER and BER pathways, facilitating damage

44 Herpes simplex virus 1 (HSV-1) can induce damage in brain regions that include the hippocampus and

45 ities, which reflects the degree of neuronal damage in brain tissue in a CJD subtype manner.

46 w that RAD52 NTD forms nuclear foci upon DNA damage in BRCA-deficient human cells and promotes DNA do

47 and inhibits micronucleus formation and DNA damage in breast cancer cells.

48 of estrogen have been shown to stimulate DNA damage in breast epithelial cells through mechanisms med

49 The increased cell death caused by DNA damage in budding yeast cells lacking the Rad53 checkpoi

50 Dampness or water damage in buildings and human exposure to the resultant

51 vel target to dampen the inflammatory tissue damage in C difficile infections.

52 ays, suggesting that Rhizobium may cause DNA damage in C. elegans intestinal cells.

53 overload and ECM degradation-mediated muscle damage in C. elegans.

54 Emerging studies indicate that DNA damage in cancer cells triggers antitumor immunity, but

55 mutagenesis, DNA replication stress and DNA damage in cancer cells.

56 -loops to prevent replication-associated DNA damage in cancer cells.

57 es as a model to study anthracycline-induced damage in cardiac cells.

58 Length-independent telomere damage in cardiomyocytes activates the classical senesce

59 e utility of CAP to model membrane oxidative damage in cells and characterise a previously unreported

60 survival pathway triggered by IR-induced DNA damage in cells of the hematolymphoid lineage and propos

61 ligands offer an opportunity to trigger DNA damage in cells with high levels of transcription and re

62 as was recapitulated by temozolomide-induced damage in cells with MMR deficiency.

63 f estrogen receptor alpha, and increased DNA damage in cells.

64 on across many domains of life and cause DNA damage in certain contexts.

65 ysiological processes but can also cause DNA damage in certain contexts.

66 mage in HL; and spontaneous forelimb use, by damage in CFA.

67 9 gene targeting in PECs prevents glomerular damage in CGN and FSGS mouse models.

68 ts that decrease oxidative stress and axonal damage in chronic and relapsing multiple sclerosis model

69 ction and reduction of PMN-associated tissue damage in chronic inflammatory diseases.

70 cl2 levels are inversely correlated with DNA damage in chronic lymphocytic leukemia (CLL) and lymphom

71 floods cause deaths, injuries, and property damages in communities around the world.

72 to prevent cytotoxic T-cell-mediated tissue damage in complex immune disorders exhibiting upregulati

73 tion of APAP increased the severity of liver damage in control mice.

74 While smoking cessation can slow the damage in COPD, lung immunity remains impaired.

75 eukaemia by releasing factors that cause DNA damage in cord blood and bone marrow cells, including st

76 the magnitude and functional form of climate damages in coupled human-natural systems.

77 chanisms and mediators of inflammatory organ damage in COVID-19.

78 selectively cause DNA re-replication and DNA damage in cSCC cells.

79 LRRK2 mutations can commonly induce neuronal damage in culture models, the mechanisms underlying thes

80 rMSCs prevented the accumulation of DNA damage in cultured HSCs, a hallmark of ageing and replic

81 primary glial cultures exacerbated oxidative damage in cultured neurons.

82 rve microcirculation may contribute to nerve damage in diabetic polyneuropathy (DN).

83 iRNAs that are associated with mitochondrial damage in different MD groups, therefore contributing to

84 tures that are associated with mitochondrial damage in different muscular dystrophies (MDs; Duchenne

85 o and generate dense swarms at sites of cell damage in diverse tissues, often extending the local dis

86 Recurrent muscle damages in DMD patients and DMD mouse model, mdx, lead t

87 yl methanesulfonate (MMS)-induced alkylation damage in DNA involves Mag1 but not Tpa1.

88 have both been reported to repair alkylation damage in DNA.

89 Cells confront DNA damage in every cell cycle.

90 astrocyte reactivity, and attenuate neuronal damage in FBN-ARO-KO mice.

91 isk factors for established clinical cardiac damage in FD.

92 chondrial DNA (mtDNA) and nuclear DNA (nDNA) damage in female mice, indicative of the sex-driven diff

93 induced dry eye produces more severe corneal damage in female mice, yet signs of LGE-induced ocular p

94 ted infections that can produce neurological damage in fetuses and immunocompromised individuals.

95 detrimental by inducing inflammatory tissue damage in, for example, ischemia-reperfusion injury and

96 tween real-life benzene exposure and genetic damage in future risk assessment.

97 s an inflammatory response and oxidative DNA damage in gastric epithelial cells that can lead to gast

98 n photodamage) was used to cause single-cell damage in gastroids, and repair of the damage was monito

99 mediated H(2)O(2) production and induced DNA damage in gastrointestinal malignancies.

100 ral nerve function and can be the target for damage in GBS, we characterized the interactions of ZIKV

101 DNA damage in Gimap5-deficient CD4(+) T cells could be contr

102 able to identify pre-perimetric glaucomatous damage in glaucoma suspects than BMO-MRW.

103 (1) acute elevations in heme lead to kidney damage in hemopexin-deficient states, and (2) a compensa

104 be an attractive new target to prevent joint damage in hemophilia patients.

105 by causing lethal replication stress and DNA damage in HGSOC, warranting further clinical development

106 However, such nano-structures can be damaged in high power applications such as heat resisted

107 encompassing CFA/RFA; hindlimb placement, by damage in HL; and spontaneous forelimb use, by damage in

108 omologue, PARP2, are early responders to DNA damage in human cells(1,2).

109 he addition of ADP-ribose moieties after DNA damage in human cells.

110 orrelate CEACAM1 levels with postreperfusion damage in human liver transplant recipients.

111 r GDR and cell survival after IR-induced DNA-damage in human lymphocytes.

112 reat similar cases of blast-induced auditory damage in human subjects.

113 Modelling neural damage in human systems is crucial to identifying specie

114 of hypertension, vascular disease and kidney damage in humans and animals for many decades.

115 the chronic inflammation elicited by muscle damage in humans.

116 may lead to liver and central nervous system damages in humans and animals, while existing detection

117 g attenuates inflammation and prevents joint damage in inflammatory arthritis.

118 n described as important mediators of tissue damage in inflammatory diseases, we investigated whether

119 zed (e.g. due to inadequate size or physical damage) in infusions and decoctions.

120 tions in complement activation and oxidative damage in IPF patients and provides haptoglobin-related

121 roptosis and ACSL4 mitigated the ferroptotic damage in IR-induced lung injury by reducing lipid perox

122 e before ischemia diminished the ferroptotic damage in IR-injured lung tissue, consistent with the pr

123 tigens favoring autoimmunity, and aggravates damage in ischemia/reperfusion injury.

124 types of dementia (bvFTD and AD) presenting damage in key monitoring areas.

125 Notably, they also present damage in key monitoring areas.

126 tory episodes and main contributors to nerve damage in leprosy.

127 cerebral amyloid angiopathy-related vascular damage) in leptomeningeal vessels (P < 0.0001), but redu

128 lic acid (DCA), induce proliferation and DNA damage in Lgr5(+) cells.

129 nflammatory cytokine response and acute lung damage in linezolid-treated mice.

130 knowledge might help in detecting local DNA damage in live cells, as well as in aiding our biophysic

131 recovery of 53BP1-mCherry, a marker for DNA damage, in live MDA-MB-231 cells.

132 to the real-time evaluation of the oxidative damage in living cells.

133 s to detect UV radiation-induced deep tissue damage in living organisms using bioimpedance analysis (

134 mmunity-acquired pneumonia, led to exclusive damage in lung alveoli, followed by alveolar epithelial

135 assed ABCB1-mediated resistance, induced DNA damage in lung carcinoma cells but exerted DNA protectiv

136 ion, epithelial barrier dysfunction, and DNA damage in lung cells.

137 NO(2) exposure may induce epithelial damage in lungs and alter club cell proliferation and mo

138 k) E-cig smoke (ECS) sustained extensive DNA damage in lungs, heart, and bladder mucosa and diminishe

139 ations of CCSNs induced by peripheral axonal damage in male mice.

140 Ranitidine decreases liver damage in Mdr2(-/-) (ATP binding cassette subfamily B me

141 Fatigue damage in metals manifests itself as irreversible disloc

142 prominently ameliorated acute renal tubular damage in mice exposed to cisplatin insult, associated w

143 own of GPR55 was sufficient to improve liver damage in mice fed a high-fat diet and in mice fed a met

144 tion of Bbeta2 can rescue excessive ischemic damage in mice lacking the mitochondrial PKA scaffold AK

145 issue, we inflicted substantial DG-specific damage in mice of either sex either by diphtheria toxin-

146 ng GHRH treatment also induced pituitary DNA damage in mice.

147 abrogation of the TRe response leads to DNA damage in mitosis, and promotes chromosome instability a

148 , in turn, mediated the reduction in chewing damage in mixed stands.

149 bition of MYCN caused profound mitochondrial damage in MNA neuroblastoma cells through downregulation

150 h VF_MD in early glaucoma but underestimates damage in moderate~advanced stages.

151 duced pain behavior but did not affect joint damage in monosodium iodoacetate-induced OA.

152 iver-specific AMPK knockout aggravated liver damage in mouse NASH models.

153 e of the mechanisms that lead to gray matter damage in MS is limited, because the most widely used an

154 l modification associated with laser-induced damage in multilayer dielectric high reflectors is inves

155 lement in CD4 T cell-dependent corneal nerve damage in multiple disease settings and indicate the pos

156 Whether these connections are equally damaged in multiple sclerosis is unknown, as is their re

157 reveal that excessive placental DNA damage in murine models for Cornelia de Lange syndrome r

158 hus, the AMPK-caspase-6 axis regulates liver damage in NASH, implicating AMPK and caspase-6 as therap

159 hain (sNfL) and its ability to expose axonal damage in neurologic disorders have solicited a consider

160 n AIM2 lead to excessive accumulation of DNA damage in neurons as well as an increase in the number o

161 ochondrial respiration and induces oxidative damage in neurons through mammalian target of rapamycin-

162 ng neuroprotective factors, and reducing DNA damage in neurons while also reducing inflammatory respo

163 racytoplasmic calcium release and subsequent damage in neurons.

164 study demonstrated different patterns of ON damage in NMOSD and MS.

165 de degeneration rather than diffuse thalamic damage in NMOSD.

166 sizing cancer cells but could also cause DNA damage in non-migrating cells and tissues that experienc

167 e-wide repair data to show that repair of UV damage in nucleosomes is asymmetric.

168 ng the sensing, repair and resolution of DNA damage in order to avoid excessive spreading of ubiquiti

169 y an important role in preventing myocardial damage in OSAHS rabbit model.

170 feeding a high-fat diet induced greater DNA damage in osteoblast of Fto (Oc KO) mice compared to con

171 d chromatin accessibility, ameliorated light damage in our mouse model, supporting a causal link betw

172 that excess NEIL3 Zf-GRF repeat reduces DNA damage in oxidative stress in Xenopus egg extracts.

173 RRX1 isoforms may limit the induction of DNA damage in pancreatic cancer cells.

174 ssibly through the accumulation of oxidative damage, in particular in the mitochondrial genome.

175 the kidney and the development of end-organ damage in patients and animal models with sodium-sensiti

176 ion and neutrophil-mediated endothelial cell damage in patients with ANCA-associated vasculitis.

177 e prognostic value of the staging of cardiac damage in patients with asymptomatic moderate to severe

178 al and mechanistic insights into ferroptotic damage in PDAC tumorigenesis in mice.

179 und to facilitate radical-induced side-chain damage in phenylalanine, was found for the reaction of N

180 ing ploidy and the repair of spontaneous DNA damage in placental cells, suggesting that genotoxic str

181 tes a multi-level response to Al-induced DNA damage in plants.

182 undant antioxidant protecting against stress damage in plants.

183 roles of nucleotide excision repair and DNA damage in platinum chemotherapy resistance by profiling

184 nosine (8-OHdg, a byproduct of oxidative DNA damage) in podocytes and tubular epithelial cells.

185 ults show that mitophagy increases after DNA damage in primary fibroblasts, murine neurons and Caenor

186 CA1, NUMB, or HES1 or chemically induced ICL damage in primary murine luminal MECs results in persist

187 al for specific language functions and often damaged in primary progressive aphasia (PPA).

188 a key factor promoting autoinflammatory bone damage in Pstpip2(cmo) mice.

189 in, we assessed REG3alpha as a marker of gut damage in PWH.

190 hout the course of MS, reflecting the axonal damage in pwMS.

191 neutrophils modulate autoimmunity and tissue damage in RA may lead to the development of novel effect

192 nd demonstrate their capacity to trigger DNA damage in rapidly dividing human cancer cells.

193 e isoprenaline (ISO)-induced renal oxidative damage in rats, a model that mimics SNS overstimulation-

194 nst kanic acid-induced hippocampal oxidative damage in rats.

195 C-PAC also reduced levels of DNA damage in reflux-exposed rat esophagi, as observed by re

196 Patients with T2DM show brain damage in regions that are involved in cognition, anxiet

197 o quantify the severity of small nerve fibre damage in relation to the severity of neuropathic pain a

198 ppressed by homologous RV, as was intestinal damage in response to endotoxin.

199 ce displayed greater inflammation and tissue damage in response to influenza A, which may be due to d

200 Epithelial damage in response to RSV infection was associated with

201 us forms of replication perturbation and DNA damage in S phase, suggesting it acts as a post-replicat

202 ollect high quality data and minimize sample damage in SAIM acquisitions.

203 ighly effective in reducing inflammation and damage in SAMP mice, mice that spontaneously develop a C

204 zed as a biomarker of endothelial glycocalyx damage in select pathologies.

205 Studying early immune responses to organ damage in situ requires animal models amenable to intrav

206 sensors for measuring mechanical stress and damage in situ.

207 icate obesity as a larger contributor to DNA damage in skeletal muscle than aging; however, more sens

208 Secondary mitochondrial damage in skeletal muscles is a common feature of differ

209 reduces R-loop accumulation and rescues DNA damage in SMA mice, motor neurons and patient cells.

210 chanistically, nuclear and mitochondrial DNA damage in SMCs and the subsequent leak of DNA to the cyt

211 ve stress and the induction of oxidative DNA damage in spermatozoa.

212 oper ROS homeostasis against isoAsp-mediated damage in stressful environments.

213 eleterious estrogenic responses, such as DNA damage, in susceptible individuals.

214 nificantly curtails chemotherapy-induced DNA damage in T-lymphocytes.

215 s a potential indicator for structural nerve damage in T2D.

216 roangiopathy contributes to structural nerve damage in T2D.

217 s SNP may identify high-risk groups for lung damage in TB.

218 nsor imaging provided evidence for long-term damage in the ablation core and in the thalamus and red

219 major pulmonary finding was diffuse alveolar damage in the acute or organising phases, with five pati

220 Brain and eyes histology presented severe damage in the bilateral group, compared to the unilatera

221 In this study, we demonstrate reduced stroke damage in the brain of mice lacking the Bbeta2 regulator

222 HL as well as neurodegeneration and synaptic damage in the brain.

223 ndent gene expression without any mechanical damage in the brain.

224 e safety indicator able to predict potential damage in the brain.

225 While this damage in the case of plasma treatment was extensive and

226 S) are unable to prevent inflammatory tissue damage in the central nervous system (CNS), and none dir

227 nine pathway mediators, TXNIP, and oxidative damage in the cerebrum and spleen, including inflammator

228 xposure significantly increased histological damage in the colon compared to control.

229 s TcdA-induced fluid accumulation and tissue damage in the colon in a mouse model in which TcdA is in

230 f the mechanics that drive the initiation of damage in the complex microstructures of discontinuous f

231 y glycosylases are inhibited from working on damage in the context of chromatin, we detail how we bel

232 of the mechanisms that contribute to kidney damage in the context of diabetes, include use of drugs

233 pulp cells in the immediate location of the damage in the coronal pulp tissue with no drug action de

234 en valuable in determining the type of acute damage in the developing brain induced by AASD exposures

235 Immunohistochemistry showed significant DNA damage in the duodenum and ileum and apoptosis in the lu

236 n prevented oligodendrocyte death and myelin damage in the EAE model.

237 A contributes to the development of synaptic damage in the early stages of AD.SIGNIFICANCE STATEMENT

238 The DNA damage in the excised oligomers is then reversed by enzy

239 y incorporating knowledge about the level of damage in the fellow eye (P > 0.61).

240 Defective repair of such oxidative damage in the fertilized oocyte results in the creation

241 ple insertions, others showed extreme genome damage in the form of chromosome truncations, large dele

242 d with apparent glia but not neurite density damage in the fornix and the hippocampus.

243 on by CD4+ T cells and mediates pathological damage in the GI tract during GVHD.

244 also show that hills could elevate potential damage in the immediate vicinity of the hills where flow

245 s revealed swollen mitochondria with cristae damage in the kdsr(I105R) mutant hepatocytes, which can

246 y in multiple sclerosis (MS): Lesion-induced damage in the lateral funiculi and gray matter (GM) in r

247 The disease causes damage in the liver where the majority of the AAT protei

248 BACS attenuated microvasculature damage in the lung grafts when compared with conventiona

249 h our previous findings that ECS induces DNA damage in the lungs and bladder and inhibits DNA repair

250 n is not a driving force behind acute muscle damage in the mdx mouse strain.

251 rize the effects of spaceflight on oxidative damage in the mouse brain and its impact on blood-brain

252 auses paralysis as well as muscle structural damage in the nematode Caenorhabditis elegans.

253 background levels of oxidatively induced DNA damage in the nematode or how culturing methods affect D

254 Such waves were initiated by local tissue damage in the outer hair cell region.

255 The importance of tissue damage in the pathophysiology of GVHD rationalizes the d

256 s of gingival epithelium revealed minor cell damage in the plasma groups (score 1).

257 resents a more efficient grader for ischemic damage in the posterior pole.

258 ess may be costly due to increased oxidative damage in the postmenopausal period.

259 e perturbation of vdW forces upon mechanical damage in the presence of controllable amount of confine

260 emalignant changes and susceptibility to DNA damage in the prostate.

261 markedly suppressed tubular epithelial cell damage in the renal interstitium of ICGN mice.

262 ne expression profiles and induces oxidative damage in the retina.

263 purpose of this study was to evaluate focal damage in the retinal pigment epithelium (RPE) layer in

264 protects dopaminergic neurons against toxic damage in the rodent brain and is in clinical trials to

265 s in reported levels of baseline and induced damage in the same experimental systems.

266 ontributes to calcium leak and mitochondrial damage in the senescent myocardium.

267 immune cell activation and associated organ damage in the skin and kidneys without effects on autoan

268 oidal anti-inflammatory drugs (NSAIDs) cause damage in the small intestine in a bacteria-dependent ma

269 eased susceptibility to indomethacin-induced damage in the small intestine; this phenotype was observ

270 utant strains, LA induced more cell membrane damage in the T7SS mutants compared to the WT.

271 therapy, which results in localized chemical damage in the target lesions, and photothermal therapy,

272 ther illuminates the concentration of severe damage in the town of Compton, where accounts suggest ve

273 mal markers indicated the absence of thermal damage in the UHPH-treated musts, since 5-hydroxymethylf

274 e-offs between consumption today and unknown damages in the (distant) future.

275 methane leakage, climate damages, and health damages in the power plant dispatch maximizes the net be

276 shelf weakening triggers the development of damage in their shear zones, which results in further sp

277 K3CB (in trans) in U138MG cells prevents DNA damage in these AEBP1 depleted cells.

278 To investigate the impact of damage in these connections for structural network effic

279 ogic findings emphasize the risk for retinal damage in these highly myopic eyes, indicating that indi

280 ume as an imaging marker of structural brain damage in these patients.

281 and repair of DNA topological-stress-linked damage in these regions.

282 The excess burden of subclinical myocardial damage in this population might not be fully explained b

283 ug action and the proximal cause of cellular damage in tubercle bacilli will make it applicable to ot

284 In vivo, rtPDT induces cellular damage in tumors, shown by strong expression of cleaved

285 novel mechanism of Nrf2-mediated myocardial damage in type 1 diabetes (T1D).

286 , leads to cystine accumulation and cellular damage in various organs, particularly in the kidney.

287 hat has been suggested as mediating neuronal damage in vascular brain injuries.

288 livery with high efficiency and minimal cell damage in vitro and in vivo.

289 mulated neutrophil-mediated endothelial cell damage in vitro, as well as on crescentic GN severity an

290 ediately following chemical induction of DNA damage in vitro, indicating that the canonical immediate

291 To recapitulate hepatocyte (HC) damage in vitro, primary C57BL/6 HC and HC-specific HMGB

292 lf-assembly and related pancreatic beta-cell damage in vitro.

293 significantly alleviated APAP-induced liver damage in vivo and correspondingly reduced serum alanine

294 penetrating property attributes to synaptic damage in vivo, we have generated adeno-associated virus

295 um was performed to quantify R-loops and DNA damage in vivo.

296 ury in unique regions: impaired reaching, by damage in voxels encompassing CFA/RFA; hindlimb placemen

297 parative analysis of predator-induced bodily damage in wild animals.

298 bridging of RGII, we assessed freeze-induced damage in wild-type and sfr8 plants by measuring electro

299 alicylate biosynthesis genes and caused leaf damage in wild-type plants but not in the salicylate bio

300 Here we model chronic intestinal damage in zebrafish larvae using the nonsteroidal antiin