ライフサイエンスコーパス: damage-associated molecular pattern

1 le of a cancer/testis antigen that is a also damage-associated molecular pattern.

2 s that senses PtdSer on apoptotic cells as a damage-associated molecular pattern.

3 ng to reactive oxygen species (ROS), a known damage-associated molecular pattern.

4 ecule that promotes airway inflammation as a damage-associated molecular pattern.

5 flammation and is therefore referred to as a damage-associated molecular pattern.

6 ate immune cells in recognizing pathogen and damage associated molecular patterns.

7 perspective and could possibly be decoded as damage-associated molecular patterns.

8 33 might function as an alarmin triggered by damage-associated molecular patterns.

9 ffectors, such as cytokines, chemokines, and damage-associated molecular patterns.

10 istic biomarkers of mitochondrial damage and damage-associated molecular patterns.

11 early inflammatory responses to pathogen and damage-associated molecular patterns.

12 acrophages stimulated with Wt or Cot/tpl2 KO damage-associated molecular patterns.

13 lammation and innate immunity, in particular damage-associated molecular patterns.

14 platforms that form upon sensing microbe- or damage-associated molecular patterns.

15 nction as sensors of endogenous or exogenous damage-associated molecular patterns.

16 f pathogen-associated molecular patterns and damage-associated molecular patterns.

17 pathogen-associated molecular patterns, and damage-associated molecular patterns.

18 rom dying cells may also engage with TLRs as damage-associated molecular patterns.

19 flammation through the secretion of exosomal damage-associated molecular patterns.

20 (such as lipopolysaccharide), cytokines, and damage-associated molecular patterns.

21 in plasma membrane damage and the release of damage-associated molecular patterns.

22 cells secreted exosomes with proinflammatory damage-associated molecular patterns.

23 -associated immune activation via release of damage-associated molecular patterns.

24 f pathogen-associated molecular patterns and damage-associated molecular patterns.

25 ell responses via the release or exposure of damage-associated molecular patterns.

26 eath trigger inflammation through release of damage-associated molecular patterns.

27 les such as granular enzymes, cytokines, and damage-associated molecular patterns.

28 artner of distinct receptors of microbe- and damage-associated molecular patterns.

29 kers as important signals of sepsis, whereas damage-associated molecular patterns, a class of inflamm

30 urs when a dying cell releases cytokines and damage-associated molecular patterns, acting as adjuvant

31 For example, damage-associated molecular patterns activate an inflamm

32 luding calgranulin A (S100a8), an endogenous damage-associated molecular pattern and TLR4 agonist.

33 avenger receptor of the Ig family that binds damage-associated molecular patterns and advanced glycat

34 t are assembled in response to pathogen- and damage-associated molecular patterns and cellular stress

35 rn recognition receptors sense pathogen- and damage-associated molecular patterns and induce an innat

36 immunogenic cell death due to the release of damage-associated molecular patterns and pro-inflammator

37 such as oxidized low-density lipoprotein and damage-associated molecular patterns and that necroptosi

38 The lipoproteins acquire features of damage-associated molecular patterns and trigger first a

39 -specific immune response via the release of damage-associated molecular patterns and tumor-associate

40 (i.e., presence of circulating pathogen- and damage-associated molecular patterns), and their implica

41 P90), a common stress-response protein and a damage-associated molecular pattern, and showed the high

42 ens, pathogen-associated molecular patterns, damage-associated molecular patterns, and homeostatic di

43 isms by which different modes of cell death, damage-associated molecular patterns, and specific cell

44 ological signals induced by pathogen- and/or damage-associated molecular patterns are essential for a

45 unt for the chronic autoimmune response when damage-associated molecular patterns are released after

46 Cytosolic pathogen- and damage-associated molecular patterns are sensed by patte

47 Pathogen- and damage-associated molecular patterns are sensed by the i

48 The data suggest a paradigm of damage-associated molecular pattern-based signaling wher

49 ed molecular pattern-based signaling whereby damage-associated molecular pattern binding integrins co

50 ognition of microbe, pathogen-associated and damage-associated molecular patterns by cytosolic sensor

51 e release of inflammatory mediators, such as damage-associated molecular patterns, by dying cells was

52 c anticancer treatment, tumor-derived DAMPs (damage-associated molecular patterns) can be sensed by i

53 ptors and, when challenged with pathogen- or damage-associated molecular patterns, can deliver cell-a

54 Breast CSCs treated with 1 exhibit damage-associated molecular patterns characteristic of i

55 issue damage and the release of host-derived damage-associated molecular patterns, contributing to sh

56 in-like lectin-G (Siglec-G) by noninfectious damage-associated molecular patterns controls innate imm

57 at brains following ICAHM included increased damage-associated molecular patterns, cytokines, chemoki

58 factors, such as mitochondrial dysfunction, damage-associated molecular patterns, cytokines, metabol

59 n pneumonia and increased levels of the mito-damage-associated molecular pattern (DAMP) cardiolipin c

60 vely during cell death; it is the prototypic damage-associated molecular pattern (DAMP) molecule and

61 phages that expressed high levels of the key damage-associated molecular pattern (DAMP) molecule high

62 ease in HMGB1 (high mobility group box 1), a damage-associated molecular pattern (DAMP) molecule that

63 ld-inducible RNA-binding protein (CIRP) is a damage-associated molecular pattern (DAMP) molecule whic

64 ages stimulated by injury-induced release of damage-associated molecular pattern (DAMP) molecules.

65 age through the recognition of extracellular damage-associated molecular pattern (DAMP) molecules.

66 Downstream damage-associated molecular pattern (DAMP) pathway activ

67 Activation of damage-associated molecular pattern (DAMP) receptors and

68 trast-enhanced MRI, resulted in an immediate damage-associated molecular pattern (DAMP) response incl

69 pathogen-associated molecular pattern (PAMP)/damage-associated molecular pattern (DAMP) signals deriv

70 phosphorylation system, but is also a major damage-associated molecular pattern (DAMP) that engages

71 d, Brennan et al report that a noninfectious damage-associated molecular pattern (DAMP), heparan sulf

72 Uric acid is a damage-associated molecular pattern (DAMP), released fro

73 During ER stress, calreticulin, a damage-associated molecular pattern (DAMP), translocates

74 xpression in NRVMs treated with LPS to model damage-associated molecular pattern (DAMP)-mediated acti

75 Damage-associated molecular pattern (DAMP)-triggered imm

76 d-inducible RNA-binding protein (eCIRP) is a damage-associated molecular pattern (DAMP).

77 ile "danger signals" that are constituted by damage-associated molecular patterns (DAMP).

78 sociated immunostimulatory endogenous danger/damage associated molecular patterns (DAMPs) and a reduc

79 uently evaluated in musculoskeletal disease, damage associated molecular patterns (DAMPs) respond to

80 role of the inflammasome from one of sensing damage associated molecular patterns (DAMPs) to sensing

81 hare with immune cells the ability to detect damage associated molecular patterns (DAMPs), molecules

82 ell wall degradation, they are classified as Damage Associated Molecular Patterns (DAMPs).

83 rly, cellular injury can release endogenous 'damage'-associated molecular patterns (DAMPs) that activ

84 Mitochondrial DNA damage-associated molecular patterns (DAMPs) accumulate

85 n-associated molecular patterns (PAMPs), and damage-associated molecular patterns (DAMPs) activate fa

86 eds are carried out through the detection of damage-associated molecular patterns (DAMPs) and a respo

87 the cell content and subsequent exposure of damage-associated molecular patterns (DAMPs) and cytokin

88 ammasome activation in response to metabolic damage-associated molecular patterns (DAMPs) and discuss

89 ors (PRRs) and related receptors that detect damage-associated molecular patterns (DAMPs) and initiat

90 ring infection, certain S100 proteins act as damage-associated molecular patterns (DAMPs) and interac

91 uents and metabolic products can function as damage-associated molecular patterns (DAMPs) and promote

92 ors) and NLRs (NOD-like receptors) recognize damage-associated molecular patterns (DAMPs) and transdu

93 Damage-associated molecular patterns (DAMPs) are conside

94 Damage-associated molecular patterns (DAMPs) are critica

95 Damage-associated molecular patterns (DAMPs) are elevate

96 Damage-associated molecular patterns (DAMPs) are endogen

97 Damage-associated molecular patterns (DAMPs) are endogen

98 Damage-associated molecular patterns (DAMPs) are intrace

99 Damage-associated molecular patterns (DAMPs) are molecul

100 Damage-associated molecular patterns (DAMPs) are release

101 ular hallmark of ICD features the release of damage-associated molecular patterns (DAMPs) by dying ca

102 illustrate how the sensing of extracellular damage-associated molecular patterns (DAMPs) can shape t

103 Although endogenous RNAs acting as damage-associated molecular patterns (DAMPs) for pattern

104 defined by the emission of immunomodulatory damage-associated molecular patterns (DAMPs) from dying

105 m cell (BMSC) proliferation via transporting damage-associated molecular patterns (DAMPs) in male rat

106 microbial counterparts, several self-encoded damage-associated molecular patterns (DAMPs) induce infl

107 crotic cell death with subsequent release of damage-associated molecular patterns (DAMPs) is a charac

108 ng TLR4-dependent cytokine storm mediated by damage-associated molecular patterns (DAMPs) like HMGB1.

109 Here, we hypothesized that damage-associated molecular patterns (DAMPs) may link ep

110 cells are activated via binding to cellular damage-associated molecular patterns (DAMPs) or pathogen

111 om dead and dying cells can be recognized as damage-associated molecular patterns (DAMPs) or pattern-

112 sociated molecular patterns (PAMPs/MAMPs) or damage-associated molecular patterns (DAMPs) recognized

113 TLR binding to damage-associated molecular patterns (DAMPs) released by

114 Herein we show that osteoclasts sense damage-associated molecular patterns (DAMPs) released by

115 LR4 (Ahmad et al., 2014) and ligands such as damage-associated molecular patterns (DAMPs) released fr

116 patterns (PAMPs) derived from pathogens and damage-associated molecular patterns (DAMPs) released fr

117 We propose that inflammatogenic damage-associated molecular patterns (DAMPs) released in

118 ctivated in response to structurally diverse damage-associated molecular patterns (DAMPs) such as ext

119 emission of immunostimulatory cytokines and damage-associated molecular patterns (DAMPs) that active

120 ell death and the release of factors such as damage-associated molecular patterns (DAMPs) that elicit

121 n (alloHCT), recipient conditioning releases damage-associated molecular patterns (DAMPs) that genera

122 MR releases intracellular molecules known as damage-associated molecular patterns (DAMPs) that propag

123 nition receptor (PRR)-mediated perception of damage-associated molecular patterns (DAMPs) triggers th

124 The concept of damage-associated molecular patterns (DAMPs) was propose

125 Damage-associated molecular patterns (DAMPs) were decrea

126 reflecting inflammation, tissue injury, and damage-associated molecular patterns (DAMPs) were measur

127 in cancer cells as characterized by multiple damage-associated molecular patterns (DAMPs) which inclu

128 re-associated molecular patterns (HAMPs) and damage-associated molecular patterns (DAMPs), activating

129 ttern-associated molecular patterns (PAMPs), damage-associated molecular patterns (DAMPs), and marker

130 cluding death of neural cells and release of damage-associated molecular patterns (DAMPs), and progre

131 Endogenous danger signals, or damage-associated molecular patterns (DAMPs), are genera

132 n-associated molecular patterns (PAMPs), and damage-associated molecular patterns (DAMPs), can activa

133 Signals of tissue necrosis, damage-associated molecular patterns (DAMPs), cause infl

134 of immunogenic proteins within these exo(S): damage-associated molecular patterns (DAMPs), chaperones

135 nt cell wall-derived oligosaccharides, i.e., damage-associated molecular patterns (DAMPs), could be g

136 ry molecules released by dying cells, termed damage-associated molecular patterns (DAMPs), have been

137 In addition, the dying cells emit damage-associated molecular patterns (DAMPs), indicating

138 ar compartment, they change roles and become damage-associated molecular patterns (DAMPs), promoting

139 two, which involves recognition of PAMPs or damage-associated molecular patterns (DAMPs), such as ur

140 y cytoplasmic molecules, collectively called damage-associated molecular patterns (DAMPs), that activ

141 atory process associated with the release of damage-associated molecular patterns (DAMPs), the occurr

142 In SI, endogenous damage-associated molecular patterns (DAMPS), which are

143 Neutrophils are also activated by damage-associated molecular patterns (DAMPs), which are

144 endogenous signals referred to as danger- or damage-associated molecular patterns (DAMPs), which are

145 plasmic-reticulum stress, and the release of damage-associated molecular patterns (DAMPs), which recr

146 ndritic cells and macrophages in response to damage-associated molecular patterns (DAMPs).

147 vate plant innate immunity by functioning as damage-associated molecular patterns (DAMPs).

148 endogenous triggers of this process are the damage-associated molecular patterns (DAMPs).

149 gen-associated molecular patterns (PAMPs) or damage-associated molecular patterns (DAMPs).

150 e also been implicated in the recognition of damage-associated molecular patterns (DAMPs).

151 f immunostimulatory molecules referred to as damage-associated molecular patterns (DAMPs).

152 o-IL-1beta to a mature form is stimulated by damage-associated molecular patterns (DAMPs).

153 cognition of markers of tissue injury termed damage-associated molecular patterns (DAMPs).

154 n respond to cellular perturbations, such as damage-associated molecular patterns (DAMPs).

155 alerted to cell death by molecules known as damage-associated molecular patterns (DAMPs).

156 mounts of intracellular components including damage-associated molecular patterns (DAMPs).

157 , a CREB inducer and sensor for host-derived damage-associated molecular patterns (DAMPs).

158 te interactions important for AIM binding to damage-associated molecular patterns (DAMPs).

159 adaptive immune response through release of damage-associated molecular patterns (DAMPs).

160 other receptors involved in the detection of damage-associated molecular patterns (DAMPs).

161 ers pyroptosis and the concurrent release of damage-associated molecular patterns (DAMPs).

162 s triggered by endogenous elicitors known as damage-associated molecular patterns (DAMPs).

163 grammed cell death, releasing large cellular damage-associated molecular patterns (DAMPs).

164 release of inflammatory molecules, including damage-associated molecular patterns (DAMPs).

165 nself or the damaged self, the latter termed damage-associated molecular patterns (DAMPs).

166 th by necroptosis, accompanied by release of damage-associated molecular patterns (DAMPs).

167 overwhelming immune response is regulated by damage-associated molecular patterns (DAMPs).

168 activated by TLR ligands, IL4, TGFbeta, and damage-associated molecular patterns (DAMPS).

169 en-associated molecular patterns (PAMPs) and damaged-associated molecular patterns (DAMPs), and they

170 s can be considered to represent "danger (or damage)-associated molecular patterns" (DAMPs).

171 molecular patterns" (PAMPs) or host-derived "damage-associated molecular patterns" (DAMPs), RAGE has

172 reas others bind endogenous plant compounds (damage-associated molecular patterns, DAMPs) such as pep

173 s emit immunostimulatory signals (so-called "damage-associated molecular patterns," DAMPs), as they d

174 ndogenous molecules released by dying cells (damage-associated molecular patterns; DAMPs) activate ce

175 exhibited reduced responses to the pathogen/damage-associated molecular patterns elf26 and AtPep1, w

176 otherapy is thought to induce the release of damage-associated molecular patterns, eliciting a pro-in

177 similar signaling pathways are activated by damage-associated molecular patterns, epigenetic plastic

178 chondrial perturbations, which function as a damage-associated molecular pattern, exacerbate NLRP3 in

179 sepsis, and extracellular CIRP (eCIRP) is a damage-associated molecular pattern, exaggerating inflam

180 However, most pathogen-or-damage-associated molecular patterns exist within intact

181 6 following stimulation of human AECs by the damage-associated molecular pattern extracellular ATP sh

182 Activation of the NLRP3 inflammasome by the damage-associated molecular pattern, extracellular ATP,

183 CIH-induced release of damage-associated molecular patterns from injured CNS ce

184 Intraperitoneal injection of damage-associated molecular patterns from necrotic hepat

185 Transcriptional hyper-activation of damage-associated molecular pattern genes occurs followi

186 Pathogen-associated molecular patterns and damage-associated molecular patterns have been found to

187 or blood transfusion releases the endogenous damage-associated molecular pattern, hemoglobin (Hb), in

188 ccur in response to pathogens, pathogen- and damage-associated molecular patterns, homeostatic altera

189 ntially generates new diffusible pathogen-or-damage-associated molecular patterns, however, our curre

190 m dying cancer cells, which is an inhibitory damage-associated molecular pattern (iDAMP) that hindere

191 nctions of the IL-1 superfamily cytokine and damage-associated molecular pattern IL-33 continues to e

192 ellular energy source, ATP can also act as a damage-associated molecular pattern in both animals and

193 adenosine triphosphate (eATP) functions as a damage-associated molecular pattern in plant immunity.

194 ve revealed that extracellular ATP acts as a damage-associated molecular pattern in plants, and its s

195 alidated the role of injured neurons-derived damage-associated molecular patterns in amplifying micro

196 We also review emerging work on the role of damage-associated molecular patterns in mediating the lo

197 onnections include responses to pathogen and damage-associated molecular patterns including alarmins

198 ained, containing precursors of microbe- and damage-associated molecular patterns, including Flagelli

199 -associated molecular patterns as well as by damage-associated molecular patterns, including microRNA

200 Both pathogen- and tissue damage-associated molecular patterns induce inflammation

201 efense responses and models for the study of damage-associated molecular pattern-induced immunity.

202 e plant cell wall plays an important role in damage-associated molecular pattern-induced resistance t

203 th, we hypothesized that cffDNA can act as a damage-associated molecular pattern inducing an inflamma

204 these cells, heme can act as a prototypical damage-associated molecular pattern, inducing TLR4-depen

205 This is associated with increased damage-associated molecular patterns, innate cytokine re

206 e airway microbiome, antimicrobial proteins, damage-associated molecular patterns, innate lymphoid ce

207 Recognition of pathogen-or-damage-associated molecular patterns is critical to infl

208 t enhancing mitochondrial resilience against damage-associated molecular patterns may play a pivotal

209 haracterize the pathophysiologic response to damage-associated molecular pattern mediated organ injur

210 ured cell mitochondria (mitochondria-derived damage-associated molecular patterns, mito-DAMPs) trigge

211 chondrial dynamics, autophagy, mitochondrial damage-associated molecular patterns, mitochondria-media

212 by treating healthy PBMCs with mitochondrial damage-associated molecular patterns (MitoDAMPs) isolate

213 n HMGB1 is released from cells and acts as a damage-associated molecular pattern molecule during many

214 The damage-associated molecular pattern molecule high-mobili

215 Thus, CIRP is a damage-associated molecular pattern molecule that promot

216 lucidated the mechanism by which the nuclear-damage-associated molecular pattern molecule, high-mobil

217 aim was to investigate the role of IL-33, a damage-associated molecular pattern molecule, in adenovi

218 associated nuclear protein and extracellular damage-associated molecular pattern molecule, is a criti

219 EDA(+) isoform of fibronectin (EDA(+)Fn), a damage-associated molecular pattern molecule, which prom

220 The damage-associated molecular patterns molecule HMGB1 was

221 show that reactive astrocytes can release a damage-associated molecular-pattern molecule called high

222 In contrast, damage-associated molecular pattern molecules (DAMPs) ar

223 mediates the cellular response to a range of damage-associated molecular pattern molecules (DAMPs) in

224 oids and a neutralizing antibody targeted at Damage-Associated Molecular Pattern molecules (DAMPs) su

225 e inflammatory response upon engagement with damage-associated molecular pattern molecules (DAMPs) su

226 extracellular compartment where they act as damage-associated molecular pattern molecules (or alarmi

227 ty group box-1 and S100A9, both of which are damage-associated molecular pattern molecules and are kn

228 y of RAGE is dictated by the accumulation of damage-associated molecular pattern molecules at sites o

229 either by bacterial lipopolysaccharide or by damage-associated molecular pattern molecules released f

230 ermore, it has been ascribed to the group of damage-associated molecular pattern molecules that promo

231 g, perturbations in calcium homeostasis, and damage-associated molecular pattern molecules, among oth

232 genous ligands from damaged cells, so-called damage-associated molecular pattern molecules, can activ

233 ) is a pattern recognition receptor for many damage-associated molecular pattern molecules.

234 he local inflammatory milieu by pathogen- or damage-associated molecular pattern, molecules, and acti

235 Mitochondrial damage-associated molecular patterns (mtDAMPs) include p

236 Mitochondrial damage-associated molecular patterns (mtDAMPs), such as

237 failure is characterized by the presence of damage-associated molecular patterns, myeloid cells, and

238 stem is primarily initiated by pathogen- and damage-associated molecular patterns on cellular surface

239 be triggered by endogenous mediators, called damage associated molecular patterns or alarmins.

240 d regulation of the haematopoietic system by damage-associated molecular patterns, or as a consequenc

241 h as pathogen-associated molecular patterns, damage-associated molecular patterns, or proinflammatory

242 tion receptors (PRRs) to detect pathogen- or damage-associated molecular patterns (PAMP/DAMPs) and in

243 Innate immune receptors for pathogen- and damage-associated molecular patterns (PAMPs and DAMPs) o

244 roach, and to assess pathogen-associated and damage-associated molecular patterns (PAMPs, DAMPs) leve

245 and injury, immune cells respond to pathogen/damage-associated molecular patterns (PAMPs/DAMPs) throu

246 of distinct molecules, known as pathogen- or damage-associated molecular patterns (PAMPs/DAMPs), or d

247 Molecules containing damage-associated molecular patterns play an important r

248 tinamide phosphoribosyltransferase), a novel damage-associated molecular pattern protein (DAMP) and T

249 The damage-associated molecular pattern protein HMGB1 has be

250 Inhibition of the damage-associated molecular pattern protein HMGB1 or sig

251 ng evidence that S100B acts as a cytokine or damage-associated molecular pattern protein not only in

252 However, it serves as a damage-associated molecular pattern protein that activat

253 Cardiac damage-associated molecular patterns provide continuous

254 Neuroinflammation is sterile, as damage-associated molecular patterns rather than microbi

255 d survival, and while they express MyD88 and damage-associated molecular pattern receptors, their rol

256 to pathogen-associated molecular pattern and damage-associated molecular pattern recognition by the i

257 platelets were decorated with histone H4, a damage-associated molecular pattern released in massive

258 eceptors for sensing microbial molecules and damage-associated molecular patterns released from host

259 glucan breakdown as well as diversifying the damage-associated molecular patterns released.

260 se microbe-associated molecular patterns and damage-associated molecular patterns represent a conserv

261 roptotic pulmonary arterial endothelial cell damage-associated molecular patterns restructured the tr

262 High concentrations of the damage-associated molecular patterns S100A8 and S100A9 a

263 echanism of cross-talk between pathogen- and damage-associated molecular pattern-sensing pathways, wh

264 des, they play an active role in calcium and damage-associated molecular patterns signaling, amino ac

265 ymphocytes infiltrate muscles in response to damage-associated molecular pattern signalling and the r

266 During infection, the release of damage-associated molecular patterns, so-called "alarmin

267 d time-dependent increases in the release of damage-associated molecular patterns such as high-mobili

268 oduced upon host recognition of pathogen- or damage-associated molecular patterns, such as nucleic ac

269 EI11 expression is controlled by PME-related damage-associated molecular patterns, such as oligogalac

270 triphosphate (ATP) is released and acts as a damage-associated molecular pattern that activates innat

271 lular high-mobility group box-1 (HMGB1) is a damage-associated molecular pattern that contributes to

272 ty group B1 (HMGB1) is a recently identified damage-associated molecular pattern that is released dur

273 nment generates versikine, a novel bioactive damage-associated molecular pattern that may facilitate

274 d-inducible RNA-binding protein (eCIRP) is a damage-associated molecular pattern that promotes inflam

275 is activated by extracellular UDP-glucose, a damage-associated molecular pattern that promotes inflam

276 e is detected by TLR3 and that self-RNA is a damage-associated molecular pattern that serves as an en

277 dies reveal that melanocyte stress generates damage-associated molecular patterns that activate innat

278 down products have been described as typical damage-associated molecular patterns that activate plant

279 ong-term necrotic cell death, and release of damage-associated molecular patterns that activated macr

280 terile inflammation), necrotic cells release damage-associated molecular patterns that bind to Toll-l

281 led tumor cells is one of the most important damage-associated molecular patterns that can activate t

282 ardiomyocyte death releases a broad range of damage-associated molecular patterns that initiate both

283 , necrotic, or apoptotic hepatocytes release damage-associated molecular patterns that initiate steri

284 embrane rupture, which allows the release of damage-associated molecular patterns that trigger an imm

285 Despite the clear role of eATP as a plant damage-associated molecular pattern, the function of its

286 However, upon stimulation with damage-associated molecular patterns, the activation of

287 ular triggers such as pathogen-associated or damage-associated molecular patterns, the NLRP1 inflamma

288 rs (TLRs), sense and respond to pathogen- or damage-associated molecular patterns, their communicatio

289 es bacteria for clearance and functions as a damage-associated molecular pattern to activate inflamma

290 mune cells or necrotic tissues and acts as a damage-associated molecular pattern to activate Toll-lik

291 cellularly released, galectin-3 can act as a damage-associated molecular pattern to facilitate early

292 igand of LvLGBP similar to LPS and acts as a damage-associated molecular pattern to modulate the shri

293 ve microbe-associated molecular patterns and damage-associated molecular patterns to activate innate

294 ciated molecular patterns and cell-intrinsic damage-associated molecular patterns to contextualize th

295 hCD14(+) ) cells could produce IL-33 through damage-associated molecular pattern/Toll-like receptor 4

296 ansduction not only during MTI but also upon damage-associated molecular pattern-triggered immunity,

297 erapy, intestinal commensal bacteria and the damage-associated molecular pattern uric acid contribute

298 One of these damage-associated molecular patterns, uric acid, is incr

299 The alarmins S100A8 and S100A9 are damage-associated molecular patterns, which play a pivot

300 rtant oxidation-derived Th2 immunomodulatory damage-associated molecular pattern with potentially imp