ライフサイエンスコーパス: dark period

1 recorded for 20 h (8-hour light and 12-hour dark period).

2 ) mRNA, and oscillations continued in a 48 h dark period.

3 fferent phase and remained low during a 48 h dark period.

4 Sleep was encouraged in the dark period.

5 nd the peak appeared at the beginning of the dark period.

6 ed with a 16-hour light period and an 8-hour dark period.

7 nd the peak appeared at the beginning of the dark period.

8 d but only in the supine position during the dark period.

9 ite light (W) night break given 8 h into the dark period.

10 the light-wake period and supine during the dark period.

11 adults, with the peaks occurring in the late dark period.

12 ight-induced processes separated by a slower dark period.

13 re they were all but undetectable during the dark period.

14 nes were negatively affected after the first dark period.

15 and had low values (165-574 ppm) during the dark period.

16 nificantly higher delta power throughout the dark period.

17 ng the SD procedure was decreased during the dark period.

18 rom late afternoon into the first 2 h of the dark period.

19 uring both wakefulness and NREM sleep in the dark period.

20 period but more time in SWS and REMS in the dark period.

21 f leaves to export sugars during a prolonged dark period.

22 rease of active wake was observed during the dark period.

23 sured delta18O discrimination throughout the dark period.

24 midday and the low point midway through the dark period.

25 except for an increase in the middle of the dark period.

26 day, and modest decrease at the start of the dark period.

27 -type plants, especially at the start of the dark period.

28 nd of the light period and a peak in the mid-dark period.

29 ificantly inhibited food intake in the early dark period.

30 ce also had less non-REMS (NREMS) during the dark period.

31 nsufficient carbohydrate reserves during the dark period.

32 in the kinetics of response during the 5-day dark period.

33 malto-oligosaccharides generated during the dark period.

34 s) light pulses, interrupted by longer (9 s) dark periods.

35 be thoroughly degraded toward the end of the dark periods.

36 ursts of light and propagation during longer dark periods.

37 synthetic electron transfer chain during the dark periods.

38 the early light period (2-6 h) and the late dark period (4-6 h).

39 ant loss of photosynthetic efficiency during dark periods, a greater level of oxidative stress, and r

40 d PsaB was maximal in the dark or subjective-dark periods, a period during which PSI was primarily in

41 endent behavioral effects, including reduced dark-period activity, impaired acoustic startle response

42 exhibited reductions in REM during the 12-h dark period after contextual fear, whereas mice receivin

43 PIF1 reaccumulates in the subsequent dark period after light-induced degradation, signifying

44 upted baseline and during 8-h light and 12-h dark periods after three sessions of 5-min manual restra

45 o basal forebrain during the normally active dark period also increases ATP.

46 nviable in cycling conditions when light and dark periods alternate.

47 operiod, reaches a peak in the middle of the dark period and correlates with kinase activity.

48 during wakefulness in both the light and the dark period and during both wakefulness and NREM sleep i

49 ff followed by recording throughout the 12-h dark period and following 12-h light period.

50 prevented substrate availability during the dark period and increased the PNSB abundance from 50-67

51 in the marmosets: IOP was higher during the dark period and lower during the light period (mean chan

52 n concentrations were the highest during the dark period and lowest in the light period.

53 nation and appressoria formation, requires a dark period and moisture.

54 1 levels then gradually increase through the dark period and remain high following movement of Aaop1

55 ep-to-wake transitions are affected by light/dark period and sleep pressure.

56 creased total sleep, NREM and REM during the dark period and total sleep and NREM during light period

57 e approximately 1.6 times more active during dark periods and approximately 4 times more active durin

58 was dependent on both the ratio of light to dark periods and JH III.

59 d total sleep and NREM during both light and dark periods and significantly increased dark period REM

60 d, as demonstrated by intermittent light and dark periods and thus allowing access to spatiotemporal

61 level at 4 to 10 h in the dark or subjective-dark periods and were shown by Western blotting and elec

62 of the human light-dark cycle, but the mouse dark period) and the rest phase (the human dark period,

63 depth, was less in PKR(-/-) mice during the dark period, and core body temperatures were lower durin

64 (NREM) sleep by approximately 43% during the dark period, and increased delta power in the EEG during

65 arch is degraded progressively during a 12-h dark period, and then accumulates during the following 1

66 h glucans and lipids was observed during the dark period, and transcription profile data indicated th

67 At the end of this dark period, animals were exposed to constant light of 2

68 Transitions between light and dark periods appear to be major environmental events tha

69 and increased NREM and REM sleep during the dark period, as previously reported, and unexpectedly de

70 d to determine if light intensity (including dark periods) at time of harvest impacts concentrations

71 ference is projected to be higher during the dark period because of the change in IOP.

72 awakening event throughout the entire light/dark period but that this effect was diminished with sle

73 e dark period) and the rest phase (the human dark period, but the mouse light period), but also synch

74 When we extended the dark period by 4 h, mice remained active, but [Cl(-)](i)

75 em promotes wakefulness throughout the light/dark period by activating multiple downstream targets, w

76 PC1-B lacked PPC1 transcripts, PPC activity, dark period CO(2) fixation, and nocturnal malate accumul

77 PPC displayed up to a 66% reduction in total dark period CO2 fixation.

78 hen a chilling stress was applied during the dark period, concomitant with an increase in ABA levels.

79 od was either interrupted midway by a 2-hour dark period (controls in 0 lx dark night; experiment 1)

80 or FeCl(3), the depolymerization during the dark periods could be completely eliminated, thus enabli

81 sands of genes at the end of the reoccurring dark periods (dawn), including those involved in photosy

82 ght stimulation of both genotypes during the dark period did not change the Avp expression in the SCN

83 r SCN samples collected during the light and dark periods did show differences in expression and as s

84 e is readily lost during protracted (1-10 s) dark periods during photoactivation of Synechocystis cel

85 duced removal of Pfr at the beginning of the dark period (End-of-Day-FR (EOD-FR) treatment) results i

86 -)mfERG stimulates with flashes separated by dark periods, facilitating interpretation of late first-

87 The dark period had a differential effect on (13)C incorpora

88 imals had increased food intake in light and dark periods, higher weight gain per day, and more body

89 copic lights at the beginning and end of the dark period; (iii) wearing either +6 D lenses, -6 D lens

90 However, when the dark period in a normal diurnal cycle was cut short arti

91 d increase in ambulatory activity during the dark period in comparison to the light period and a 'W-s

92 prolongs the activity of PPC throughout the dark period in K. fedtschenkoi, optimizing CAM-associate

93 The changes in fru-2,6-P2 at the start of dark period in leaves and in the cell experiments genera

94 r) neurons during the light period and early dark period in photostimulated vs. control animals.

95 with increased oxidative stress late in the dark period in the mutant.

96 Prolonged light treatment followed by a dark period induced stress and cell death marker genes w

97 ormone release, especially at the end of the dark period, maintains high temperature.

98 was projected to be 5.26 mm Hg higher in the dark period (mean, 17.10 mm Hg) than in the light period

99 tle light exposure before entering the night/dark period, MeSA and its metabolizing enzymes were esse

100 and PEPC carboxylations alone, such that the dark period mesophyll conductance, g(i), was 0.044 mol m

101 nd even a brief exposure to light during the dark period (night-break) is sufficient to delay floweri

102 he mean swimming speed is greater during the dark period of a diurnal cycle.

103 sponse by 26% only in wakefulness during the dark period of the diurnal cycle to a level observed dur

104 and less time awake at the beginning of the dark period of the light:dark cycle.

105 noparticles, the distributions of bright and dark periods ('on' and 'off' times) follow Levy statisti

106 activity was significantly higher during the dark period (P < 0.01).

107 The midnight 2-hour dark period partially restored, but the evening melatoni

108 hotoperiod (phase IV of CAM), throughout the dark period (phase I), and into the light (phase II).

109 PPCK phosphorylates PPC during the dark period, reducing its sensitivity to feedback inhibi

110 NPY, injected at the start of the dark period, reliably increased 2 h food intake.

111 and dark periods and significantly increased dark period REM.

112 55 (36.9%) of the 149 neurones tested in the dark period responded to optic nerve stimulation while o

113 ulation of this truncated GluTR is higher in dark periods, resulting in increased protochlorophyllide

114 ements of CO2 response curves throughout the dark period revealed changing phosphoenolpyruvate carbox

115 e flash experiments, with a 10 s intervening dark period, reveals a faster, 15 ms phase that is accen

116 avenous infusion of ghrelin during the early dark period stimulates food intake in freely feeding rat

117 the plants displayed reduced but detectable dark period stomatal conductance and arrhythmia of the C

118 Average IOP was significantly higher in the dark period than in the light-wake period in both groups

119 y, while the abundance of HMG1 mRNA during a dark period that induced photoperiodically controlled fl

120 MSR activity occurred at the end of the 16-h dark period that was absent in pmsr2-1 plants.

121 display high levels of basal activity in the dark period (the rodent's awake/active time) that are at

122 During the dark period, the mean spontaneous firing rate (5.00 +/-

123 13C signal; all declined at the start of the dark period, then increased to a maximum 2 h before dawn

124 nes exhausted their carbohydrates during the dark period to a greater extent than the wild type and a

125 respiration in the early dark or subjective-dark periods to permit nitrogenase activity.

126 Both NE and mNE increased dark period total sleep, NREM and REM; however, mNE also

127 he nifHDK operon during the first 4 h of the dark period under light-dark conditions or during the fi

128 increases linearly with the duration of the dark period up to the longest period we could examine (1

129 as high after a 14-h night, but low when the dark period was 12 h or less.

130 Mean IOP in the dark period was significantly higher than mean IOP in th

131 ning of the light period or beginning of the dark period, we sought to determine whether the muscarin

132 for the 12-hour light period and the 12-hour dark period were compared.

133 wake period and the supine IOP data from the dark period were considered, elevation and reduction of

134 ings from the 10-h light period and the 12-h dark period were examined separately.

135 ns or during the first 6 h of the subjective-dark period when grown in continuous light.

136 tion of CNA on sleep and activity during the dark period when rats show higher arousal and less sleep

137 During the dark period, when these animals are normally active, 6 h

138 e-movement sleep (REMS) increased during the dark period, whereas during the light both NREMS and REM

139 -h intervals early in the dark or subjective-dark period, whereas photosynthesis was approximately 12

140 were injected systemically during the early dark period with melatonin (0.6 mg) or 2% ethanol vehicl

141 creases in slow-wave sleep (SWS) only in the dark period with no changes in rapid-eye-movement sleep

142 nd day periods and low movement rates during dark periods with highest nighttime rates at 10-<50% lun