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1 neurones obtained from older cultures (14-27 days in vitro).
2 ension and survival in PC-12 cells (up to 25 days in vitro).
3 d with human dental pulp stem cells up to 14 days in vitro.
4 on was significantly suppressed for up to 21 days in vitro.
5 the bladder and ileocecum and persisted for days in vitro.
6 nt failed to inhibit BrdU incorporation at 9 days in vitro.
7 thin minutes and is sustained for at least 3 days in vitro.
8 colonies generated new neurons as late as 7 days in vitro.
9 e and the experiments were carried out at 21 days in vitro.
10 s completely released from the tips within 4 days in vitro.
11 njectables, was demonstrated for at least 28 days in vitro.
12 h either avidin or streptavidin for up to 14 days in vitro.
13 orks of primary hippocampal neurons until 29 days in vitro.
14 HUVECs) and enhancing tube stability up to 6 days in vitro.
15 (0N3R), even at extended time points of 100 days in vitro.
16 nd can extend the release of chemokines to 4 days in vitro.
17 d highly viable and could be cultured for 10 days in vitro.
18 patch-clamp recordings were made after 7-28 days in vitro.
19 sed to 3.5 mM glutamate for 35 minutes at 21 days in vitro.
20 bled, CheA remains associated and active for days in vitro.
21 with 0-50 ng/mL rhVEGF(165) or rhFGF-2 for 7 days in vitro.
22 lls when mixed with irradiated T cells for 6 days in vitro.
23 eath between culture media over the first 14 days in vitro.
24 undergo apoptosis beginning approximately 4 days in vitro.
25 180), 0.1 microg/ml to 10.0 microg/ml, for 8 days in vitro.
26 e, and these differences were enhanced by 10 days in vitro.
27 ed PEG-N1000G-ChABC-SH3 release over several days in vitro.
28 priate layer-specific projections during 5-7 days in vitro.
29 fibers had degenerated during the first few days in vitro.
30 oped layer-specific axonal arbors during 5-7 days in vitro.
31 robustly rhythmic and persisted for up to 32 days in vitro.
32 10-day-old rats and maintained for 10 to 12 days in vitro.
33 sed substantially when cells were cultured 3 days in vitro.
34 duction is not sustained for more than a few days in vitro.
35 ed strongly stained presynaptic boutons by 3 days in vitro.
36 en GH3 and GHRH-CL1 cells were treated for 7 days in vitro.
37 tch clamp recordings were obtained after 6-8 days in vitro.
38 urons were synaptically coupled after 3 or 4 days in vitro.
39 creased during the next few days to 39% at 4 days in vitro.
40 t begun also developed taste buds after 9-12 days in vitro.
42 l cultures containing both neurons and glia (days in vitro 13-15) were exposed to periods of oxygen-g
44 ntly (-55.7+/-6.2%) in slices cultured for 2 days in vitro (2DIV) as compared to slices placed in cul
46 ainst induced apoptosis and mature cells (5+ days in vitro) against glutamate toxicity, but its preci
47 tects immature cerebellar granule cells (1-3 days in vitro) against induced apoptosis and mature cell
48 tes a Th0 population that is evident after 3 days in vitro and becomes prevalent after successive enc
49 blood monocytes were differentiated for 1-7 days in vitro and examined with respect to 1) uptake of
50 cultures from brains of 2-day-old rats at 7 days in vitro and plating them (0 days post-shake, DPS).
51 CCL21 and beta-cell antigens for at least 28 days in vitro and recapitulate properties associated wit
52 ed marrow of the long bones were cultured 14 days in vitro and subsequent colonies were assessed by l
53 120 Cameroonian newborns were cultured for 7 days in vitro and supernatants were assessed by ELISA fo
54 nd continuously released peptide for over 56 days in vitro and suppressed testosterone production in
55 ith either BMP4 or with various FGFs for two days in vitro and then grown under the kidney capsule of
56 stromal fibroblasts were pre-cultured for 3 days in vitro and then injected within a fibrin matrix i
57 tions increased markedly during the first 14 days in vitro and were inversely related to response lat
58 p loaded in the nanofibers was released in 5 days in vitro, and by applying the Mp-loaded nanofibers,
59 lation was constant at 64.2% for at least 30 days in vitro, and contractile activity was observed for
60 t after 100 days or more, stored again for 4 days in vitro, and retransplanted into 5 other diabetic
61 tnatal day 8 mice, cultured for 2-4 or 15-30 days in vitro, and studied by electron and fluorescence
62 vector and then exposed to 5-FC either for 4 days in vitro before transplantation or for 14 days imme
64 in all cells taken from young cultures (4-7 days in vitro) but the latter was absent in 55% of the n
66 lengths and fewer branches between 7 and 21 days in vitro compared with wild-type (WT) littermates.
67 medium from PS1 null neuronal cultures at 8 days in vitro contained higher levels of glutamate than
68 GAD67-labeled somata, whereas those grown 4 days in vitro contained primarily terminal-like varicosi
73 th time in all ages during the initial three days in vitro (DIV) but the proportion of Schwann cells
74 d a lower potency for GABA between 11 and 12 days in vitro (DIV) resulting in a shift of the EC50 fro
76 100 microM glutamate/10 microM glycine at 5 days in vitro (DIV), but became vulnerable to the same s
81 mouse cortical neurons were cultured 8 or 9 days in vitro (DIV), loaded with the Ca(2+) indicator Fl
83 GABAergic synapses appeared between 3 and 8 days in vitro (DIV), with GABAA receptor subunit cluster
91 from studies on immature cultures [< or = 6 days in vitro (DIV)] and cytoarchitectural analyses of m
92 rat hippocampal neurons increases over age [days in vitro (DIV)] in long-term primary cultures, appa
94 d cultured hippocampal neurons with TTX at 7 days in vitro, during rapid synaptogenesis, and at 14 da
96 reated chronically with 30 mM (140 mg%; 3-11 days in vitro) ethanol to study the actions of prolonged
97 Networks obtained from young cultures (14 days in vitro) exhibited a random topology, which evolve
99 we compared neural network activity (over 30 days in vitro) from primary neurons co-cultured with gli
100 te presynaptic release occurred after 3 or 4 days in vitro in high-density cultures but was absent in
102 ced to differentiate into osteoclasts for 21 days in-vitro in the presence of macrophage colony stimu
103 n cortical neurons grown for 5, 8, 11, or 14 days in vitro indicate that sequences between -1253 and
104 24-h exposure to NMDA (100 mum) or K25 at 2 days in vitro induced long term survival of CGNs in K5 c
105 ltured rat cortical neurons from day 4 (four days in vitro or DIV 4) to day 20 (DIV 20), we observed
107 -1 (KLS) and CD150CD48-KLS cells for up to 5 days in vitro prevented the culture-induced loss of Inte
109 nduced whole cell responses in mature (22-29 days in vitro) rat cortical neurons were potentiated nea
112 hen isolated E14-E16 slices were grown for 4 days in vitro, the width of the GAD65-labeled ventral ma
113 hippocampal neuron cultures between 6 and 35 days in vitro to investigate how the topology evolves du
114 were imaged every other day, beginning at 10 days in vitro, to assess how mitochondrial length and co
116 ium in organ culture is compromised after 14 days in vitro using an immersion system of tissue cultur
117 alysis revealed that the period from 6 to 12 days in vitro was the critical time for exposure to GDNF
118 Further, at extended time points of 365 days in vitro, we observe a switch in tau splicing to in
119 postnatal day 0 or 1 and examined after 3-4 days in vitro, we observed differences in the amplitude
120 specific antibodies (OS-2 and JH455) after 5 days in vitro, which corresponds to a time course simila
121 mation of a confluent endothelium within 3-4 days in vitro with human endothelial colony-forming cell
122 to release PRO from 3D printed IVRs for >100 days in vitro, with release rate and duration driven by