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1 e associated with dendrite and synapse loss (deafferentation).
2 y to the hypertension caused by baroreceptor deafferentation.
3 ntial parameters were observed after chronic deafferentation.
4 ainstem circuitry selectively in response to deafferentation.
5 ells exhibit markedly different responses to deafferentation.
6 vagal complex were increased following vagal deafferentation.
7 CN neurons to either death or survival after deafferentation.
8 uring swimming and jumping, before and after deafferentation.
9 l neurons leaves the terminals in a state of deafferentation.
10 loss of iGluR4 immunoreactivity by 24 h post-deafferentation.
11 ain was further investigated after olfactory deafferentation.
12 ion of Purkinje cells to episodic functional deafferentation.
13 with previous subdiaphragmatic vagal sensory deafferentation.
14 s which can be observed within 1 h following deafferentation.
15 y : fibre ratio at 7 days) was unaffected by deafferentation.
16 any kinds of injuries, including axotomy and deafferentation.
17 r remodeling and synaptogenesis initiated by deafferentation.
18 omplexity of responses to different forms of deafferentation.
19 n, and is balanced in the setting of chronic deafferentation.
20 cortex are quickly reduced within minutes of deafferentation.
21 reduction of amyloid plaques on the side of deafferentation.
22 s in Alzheimer's disease, namely hippocampal deafferentation.
23 ceptors, within 4 h of unilateral vestibular deafferentation.
24 g differences between central and peripheral deafferentation.
25 This facilitation was blocked by vagal deafferentation.
26 the IPSC equilibrium potential within 1 d of deafferentation.
27 ng animals are also more affected by sensory deafferentation.
28 mice during and after recovery from chemical deafferentation.
29 the mitotic behavior of astrocytes following deafferentation.
30 be produced by the brain related to auditory deafferentation.
31 n the OB can be reconstituted after chemical deafferentation.
32 inished at 1 week and absent by 3 weeks post-deafferentation.
33 lse facilitation was nearly eliminated after deafferentation.
34 was examined in zebrafish, using peripheral deafferentation.
35 e its target, the olfactory bulb (OB), after deafferentation.
36 urons following lesions, target loss, and/or deafferentation.
37 zation that immediately follows a peripheral deafferentation.
38 ssentially the same regardless of the age of deafferentation.
39 ion resulting from the reversible peripheral deafferentation.
40 his disease may be the result of cholinergic deafferentation.
41 recovery that follows unilateral vestibular deafferentation.
42 -NA) following neonatal unilateral vibrissae deafferentation.
43 ge from piston impact, brain displacement or deafferentation.
44 ular changes in response to brain injury and deafferentation.
45 otion of topographic plasticity after visual deafferentation.
46 l hippocampus indicating relatively complete deafferentation.
47 ceps brachii muscle proximal to the level of deafferentation.
48 or rats that received subdiaphragmatic vagal deafferentation.
49 l cuneate nucleus within three months of the deafferentation.
50 ton are responsible for cell death following deafferentation.
51 a-actin mRNAs were not noticeably altered by deafferentation.
52 atory response to nigrostriatal dopaminergic deafferentation.
53 tional protein synthesis apparatus following deafferentation.
54 in the human following peripheral or central deafferentation.
55 memory deficits associated with hippocampal deafferentation.
56 ty exercises, both before and after surgical deafferentation.
57 experiment performed with a person (IW) with deafferentation.
58 the normal inhibitory balance following ACC deafferentation.
59 gh the dendritic synapses are at the site of deafferentation.
60 strength was not observed following partial deafferentation.
61 ease of central gain attributable to sensory deafferentation.
62 d transient hypersensitivity consistent with deafferentation.
63 s that had earlier received a surgical vagal deafferentation.
64 vasion by ectopic visual inputs and not from deafferentation.
65 eath and are thought to occur in response to deafferentation.
66 llowing lithium administration and following deafferentation.
67 the substantia nigra and associated striatal deafferentation.
68 determinant of Bcl-2 upregulation following deafferentation.
69 pregulated in 20-30% of NM neurons following deafferentation.
70 uch as tinnitus are associated with auditory deafferentation.
71 -2 and increases neuronal survival following deafferentation.
72 e over an approximately 14-week period after deafferentation.
73 mulation, in contrast, were unaffected after deafferentation.
74 s in normally innervated cells but not after deafferentation.
75 on of somatosensory cortex that follows such deafferentations.
76 imals injected with PHA-L after intracranial deafferentations.
77 nsory input was reduced by either a complete deafferentation, a genetic impairment of neurotransmitte
84 amputees during the first 12 weeks following deafferentation and at 26 and 30 weeks post-amputation.
85 N-gamma blocking antibody prevented neuronal deafferentation and clinical disease without reducing CT
87 mates could be induced by premorbid ability, deafferentation and diaschisis, and neuroplasticity foll
88 with NFT densities respectively suggest that deafferentation and intrinsic neurofibrillary degenerati
89 ecovery are dependent both on the vestibular deafferentation and on the activation of glucocorticoid
91 ct persisted throughout the entire period of deafferentation and returned to baseline values afterwar
92 lfactory system during development and after deafferentation and suggest that the expression of Na be
93 ble and resilient retinal synapse types upon deafferentation and uncover how afferent input different
94 dorsal mPFC was enhanced after noradrenergic deafferentation and was negatively correlated with stres
96 inhibition of the ipsi-lesional cells after deafferentation, and thus promote the recovery of restin
99 rse and nature of development are altered by deafferentation at birth; 2) reorganization of terminals
100 vation but, surprisingly, did increase after deafferentation at P21, when all neurons ultimately surv
102 lar topology and broadcast the presence of a deafferentation-based bottom-up prediction error as a re
103 , beneficial effects on cortical cholinergic deafferentation-based impairments in attention may remai
104 m limbs-whether resulting from amputation or deafferentation-became illustrated, and some reasons for
105 ytoskeleton is altered or degraded following deafferentation but that this process is not regulated a
107 tions correlates well with the extent of MGN deafferentation, but not with extent of removal of norma
108 it in recognition of target loss and partial deafferentation by aged granule cells and/or an impaired
112 ing rule to show that loss of input (partial deafferentation) can trigger network reorganization that
113 he degradation of the cytoskeleton following deafferentation could potentially lead to either atrophy
118 to prevent microglial activation, olfactory deafferentation did not reduce adult neurogenesis, showi
119 ere altered in a BC-type specific manner and deafferentation differentially impacted expression of sy
121 r volume of HG, which suggests that auditory deafferentation does not lead to cell loss within primar
122 nt and form of reorganization resulting from deafferentation early in life vs. adulthood are not the
124 duced redox imbalance in the cochlea and the deafferentation effects upstream the acoustic pathway.
127 nput, with axonal synapses reacting first to deafferentation even though the dendritic synapses are a
129 d cells), but there was a critical degree of deafferentation for pathological network reorganization.
130 etwork target firing rate for all degrees of deafferentation (fraction of deafferented cells), but th
131 in the control situation and after selective deafferentation from cats with unilateral transection of
137 function following correction of congenital deafferentation has fired the imagination of scientists
138 rmine if NPY-containing neurons that survive deafferentation have any distinguishing morphological an
141 , nucleus magnocellularis (NM) die following deafferentation (i.e., deafness produced by cochlea remo
142 Bergmann gliosis, and signs of Purkinje cell deafferentation; (iii) selective striatal cholinergic in
143 behaviour of networks in the brain, we model deafferentation in a biologically realistic balanced net
145 the capacity of this map to reorganize after deafferentation in adults and animals late in developmen
148 radation of responses to surgical vestibular deafferentation in humans, skew deviation only occurring
150 n this study we delayed the onset of retinal deafferentation in rats and mice in order to determine t
153 of the inhibitory synaptic conductance after deafferentation, indicating that driving force was not s
154 -based transfer to new cell populations, and deafferentation induced degeneration are part of a proce
156 Inhibition of NFAT significantly attenuates deafferentation-induced apoptosis of AVCN neurons and ab
157 FAT-mediated gene expression plays a role in deafferentation-induced apoptosis of cochlear nucleus ne
158 functional auditory-limbic connectivity, and deafferentation-induced atrophy in frontal brain regions
159 d whether the chloramphenicol enhancement of deafferentation-induced cell death reveals the same ultr
165 in regulating programmed neuronal death and deafferentation-induced neuronal death in the brainstem
168 al dendrites is a significant contributor to deafferentation-induced reactive hyperexcitability.
170 of MMP inhibition during the early phases of deafferentation-induced sprouting, characterizing compon
172 l elements accounts for at least part of the deafferentation-induced volume decrease in the zebrafish
176 The loss of sensory input following a spinal deafferentation injury can be debilitating, and this is
178 Although the process can be triggered by deafferentation, intense activation of glutamate recepto
179 n of Bcl-2 gene expression by lithium and by deafferentation involves different molecular pathways.
183 that is seen immediately after a peripheral deafferentation is dependent upon both descending cortic
185 ccupancy of the vacant synaptic space due to deafferentation is the base for the mechanism of compens
188 recovery post-injury, we used an established deafferentation lesion model (dorsal root/dorsal column)
191 pothesis that broad functional (or anatomic) deafferentation may combine to reduce central thalamus a
192 (SDA), the most complete and selective vagal deafferentation method existing to date, to study the co
194 l experiments revealed that, following vagal deafferentation, mu-opioid receptors were colocalized on
195 ng cortical reorganization that follows limb deafferentation, neurons in deafferented forelimb SI bec
196 ted pathological features of neuronal system deafferentation, NFT progression and propagation, and ne
197 ter 2 weeks in all animals, neither striatal deafferentation nor in vitro induction of differentiatio
198 s in neuronal latency induced by the sensory deafferentation occurred as often in the thalamus as in
200 us studies have indicated that amputation or deafferentation of a limb induces functional changes in
202 citatory perforant path results in the acute deafferentation of a segregated zone on the distal dendr
204 In a control group of animals with neonatal deafferentation of auditory thalamus but without redirec
206 S lesions derive from the resulting combined deafferentation of dorsal and ventral hippocampal region
207 viously been suggested to indicate a partial deafferentation of epileptic neurons, but this interpret
210 e show that only during this critical period deafferentation of mouse AVCN neurons by in vivo cochlea
214 ytochrome oxidase (CO) histochemistry, after deafferentation of one eye or even by leaving afferent i
215 tion', the behavioural recovery that follows deafferentation of one inner ear, are largely unknown.
216 The current data suggest that cholinergic deafferentation of prefrontal cortex alters top-down and
220 erent models of chronic synaptic inhibition: deafferentation of the barrel cortex and administration
223 se phenotype in HD mice, we examined whether deafferentation of the corticostriatal and nigrostriatal
224 f mice, suggesting that the ensuing state of deafferentation of the DA terminals may contribute to th
226 in these nuclei during development and after deafferentation of the excitatory auditory nerve (nVIII)
227 otoxin, which leads to selective cholinergic deafferentation of the infused cortex, yielded recogniti
230 activity was still observed even after total deafferentation of the laryngeal and pharyngeal areas in
231 e essential for odor discrimination and that deafferentation of the majority of bulbar glomeruli may
232 To this end, the effects of either surgical deafferentation of the mediobasal hypothalamus or admini
233 in DOPAC in the nucleus accumbens; surgical deafferentation of the mediobasal hypothalamus prevented
234 duction of normal retinal targets along with deafferentation of the MGN are two concurrent factors re
239 the cerebellar hemispheres reorganizes after deafferentation of the upper lip in neonatal rats (postn
242 tween brain and body and the effects of this deafferentation on body representation are poorly unders
243 e interactive effects of age and cholinergic deafferentation on synaptic connectivity in frontal cort
245 amined both normal growth and the effects of deafferentation on the bulb from hatching to adulthood.
246 investigate the effects of very early visual deafferentation on the functional organization of the br
247 study evaluated the effects of total cardiac deafferentation on the reflex control of efferent renal
248 in cortical topography in primates following deafferentation or amputation, and this review will atte
249 e expression induced by peripheral olfactory deafferentation or naris blockade confirms that function
251 hway, instigated by more profound peripheral deafferentation or reflecting a preexisting marker of ti
253 enervation of the bladder, selective C-fiber deafferentation, or bladder sympathectomy prevented cyst
254 electroporation, live cell imaging, in vitro deafferentation, pharmacology, and electrophysiological
258 ted that less extensive cortical cholinergic deafferentation, produced by intracortical infusions of
262 on is comparable with that induced by direct deafferentation resulting from transection of NM axons.
264 ohistochemistry of the mouse CN to show that deafferentation results in strikingly different sets of
265 arlier study suggest that septal cholinergic deafferentation results in: (a) the loss of a distinct s
266 e used a rat model of subdiaphragmatic vagal deafferentation (SDA), the most complete and selective v
269 t shared synergies, however, were altered by deafferentation, suggesting that sensory inflow modulate
270 pression of these glial factors in fields of deafferentation suggests the possibility of additive or
272 rd section (approximately T8) and unilateral deafferentation (T12-S2) to answer the following questio
273 al studies suggest that temporary functional deafferentation (TFD) of parts of the stroke-affected up
274 the characteristics of network repair after deafferentation that are reported in experiments to stud
277 clearly demonstrate that peripheral sensory deafferentation triggers a system-wide reorganization, a
278 vity before and after unilateral cholinergic deafferentation using intracortical infusion of the immu
279 r the expression of GATs changes after nerve deafferentation using the rat superior colliculus (SC) a
280 subjects and six with unilateral vestibular deafferentation (UVD) underwent binocular eye and head m
283 and then 6 weeks after unilateral vestibular deafferentation via surgical resection of the tumor.
284 ase in average firing rate in the network by deafferentation was compensated by homeostatic synaptic
286 in [3H]CP55,940 binding contralateral to the deafferentation were largely absent at all post-lesion d
287 changes in the motor cortex contralateral to deafferentation were probed with TMS, measuring motor th
288 ical cholinergic and/or caudate dopaminergic deafferentation were produced by bilateral infusions of
290 he somatosensory hand area of primates after deafferentation where cortex can become activated by a m
291 amina ganglion mother cells ceased following deafferentation, whereas cell death in the lamina cortex
294 phenotype was produced by chronic "chemical deafferentation" with glutamate receptor antagonists.