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1 present in high copy, the chromosomes do not decondense.
2 curs primarily in early G1 after chromosomes decondense.
3 rallel, heterochromatin at chromocenters was decondensed.
4 chromosome is severely distorted, appearing decondensed.
5 e site of integration, the chromatin remains decondensed.
6 function of G3BP proteins, hijacking the RNA decondensing activity of eIF4A, and/or maintaining effic
9 main forms: euchromatin, which is relatively decondensed and enriched in transcriptionally active gen
10 to either trypsin or proteinase K gradually decondensed and softened chromosomes but without entirel
11 primitive streak region: Hoxb chromatin was decondensed and the Hoxb1 locus looped out from its chro
12 n the presence of nocodazole-the chromosomes decondensed and the nuclear envelope re-formed-whereas c
13 entered an interphase-like state (chromatin decondensed, and an interphase-like microtubule array an
14 plicating mitotic chromosomes became visibly decondensed, and, after DNA replication was complete, th
15 We here show that stimulated lymphocytes decondense chromatin by three differentially regulated s
17 neutrophils, PAD4 citrullinates histones to decondense chromatin that gets released as NETs in a man
18 the capacity of neutrophils to release their decondensed chromatin and form large extracellular DNA n
19 e chromatin spreads revealed the presence of decondensed chromatin as gap structures along the spread
20 in contact to particulate agents to extrude decondensed chromatin as neutrophil extracellular traps
23 eed surrounded by or adjacent to a domain of decondensed chromatin composed of sequences from the gen
27 scs' are boundary elements that delimit this decondensed chromatin domain, reflecting the mechanism b
29 se findings suggest that increased levels of decondensed chromatin in both normal progenitor cells an
30 ne hypercitrullination is detected on highly decondensed chromatin in HL-60 granulocytes and blood ne
31 form extracellular traps (NETs) by releasing decondensed chromatin lined with cytotoxic proteins.
32 il extracellular traps (NETs), a meshwork of decondensed chromatin lined with histones and neutrophil
33 26) modification colocalizes with ERalpha at decondensed chromatin loci surrounding the estrogen-resp
34 Neutrophil extracellular traps (NETs) are decondensed chromatin networks released by neutrophils t
37 il extracellular traps (NETs) originate from decondensed chromatin released to immobilize pathogens,
38 ng sites, a transition from a condensed to a decondensed chromatin structure appears to take place.
39 layed after DNA replication, indicative of a decondensed chromatin structure in all regions of the re
42 ochemical and bioimaging experiments suggest decondensed chromatin structures are associated with tra
43 ils trap and kill bacteria by forming highly decondensed chromatin structures, termed neutrophil extr
44 Peripheral blood neutrophils form highly decondensed chromatin structures, termed neutrophil extr
45 ocytes (PMNs), extrude molecular lattices of decondensed chromatin studded with histones, granule enz
46 tive genes may result from their location on decondensed chromatin that enables clustering around com
47 with probe penetration, (2) have relatively decondensed chromatin that is highly accessible to probe
48 on of neutrophil extracellular traps (NETs), decondensed chromatin threads decorated with cytoplasmic
49 ar traps (NETs) are structures consisting of decondensed chromatin with associated proteins, includin
50 cytosolic and granule proteins assembled on decondensed chromatin, kill pathogens and cause tissue d
51 r traps (NETs) resulting from the release of decondensed chromatin, were found to be part of the thro
52 ly, MEL-28 recruits PP1c to the periphery of decondensed chromatin, where it directs formation of a f
57 ced by recruitment of an acidic peptide that decondenses chromatin without affecting transcription, i
58 CTD charge reduction unfolds the domain and decondenses chromatin, a mechanism in consonance with re
60 elope components and successful expansion of decondensing chromatin compared with those induced by no
62 s depleted of Ca2+ and Mg2+ showed partially decondensed chromosomes and a loss of Topo II and ScII,
63 e viable only during slow growth and display decondensed chromosomes, suggesting that SMC complexes f
65 n daughter cells and rapidly associates with decondensing chromosomes in telophase, suggesting a role
67 mosome movement suggests that the surface of decondensing chromosomes, and by extension those of inta
69 a cells infected by the hepatitis C virus is decondensed compared to uninfected cells, which correlat
70 , large-scale chromatin structures on Xa are decondensed compared with the Xi and transcription inhib
71 escribed in neutrophils wherein chromatin is decondensed, decorated with cytotoxic granule enzymes, a
73 IL-1 dependent, and release of proteases and decondensed DNA from recruited neutrophils in the brain
74 were discovered as extracellular strands of decondensed DNA in complex with histones and granule pro
75 toxicity (i.e., the release of proteases and decondensed DNA triggered by phenotypic transformation d
76 arrest, appearance of convoluted nuclei with decondensed DNA, and formation of multinucleated cells.
80 ain specific regions of chromatin can become decondensed, even at physiological salt concentration, t
83 ytes arrest in a G(2) state based on uniform decondensed GV chromatin, interphase microtubule arrays,
84 g and growth-inhibitory genes.In conclusion, decondensing H3K27me3-marked chromatin by EZH2 inhibitio
85 lent methylated genes and transposons and to decondense heterochromatic chromocenters, despite only m
86 of multinucleated giant cells and cells with decondensed, highly convoluted and lobulated nuclei that
87 nstrate that camphor causes the nucleoids to decondense in vivo and when the three genes are present
90 that facilitate chromosome segregation, and decondensed interphase structures that accommodate trans
92 with [3H]uridine autoradiography, shows that decondensed micronuclear chromatin undergoing active tra
94 conserved antimicrobial strategy comprising decondensed nuclear DNA and associated histones that are
95 , a signature neutrophil pathway that expels decondensed nuclear DNA into extracellular compartments
96 These mutant embryos exhibit fragmented or decondensed nuclei and accumulate higher levels of SUMO-
97 ion of reactive oxygen species (ROS) and the decondensing of the nuclear DNA catalyzed by peptidyl ar
98 over, the free C-terminal domain can rapidly decondense ParB networks independently of its ability to
99 tability in MM may be associated with highly decondensed pericentromeric heterochromatin, which may p
101 The XNP focus corresponds to an unusual decondensed satellite DNA block, and both active genes a
102 rified hyperphosphorylated egg nucleoplasmin decondense sperm chromatin and remove sperm basic protei
104 iction-enzyme-mediated integration (REMI) on decondensed sperm nuclei followed by nuclear transplanta
108 onversion from a heavily compacted form into decondensed, spherical pronuclei, accompanied by rapid n
110 one-containing chromatin fiber to exist in a decondensed state under conditions that normally would p
113 exibility in vivo, maintaining a compact yet decondensed template that permits polymerase accessibili
114 the H3K27 histone methyltransferase EZH2 to decondense the H3K27me3-marked chromatin of AML cells en
120 tering mitosis, causing cells in prophase to decondense their chromosomes and return to G2 phase.
121 ay induces mid-prophase cells to transiently decondense their chromosomes, it is likely that it downr
122 In response to certain stimuli, neutrophils decondense their lobulated nucleus and release chromatin
123 treated with nocodazole, the majority (70%) decondensed their chromosomes and returned to G(2) befor
125 DNA fluorescent in situ hybridization (FISH) decondensed to varying degrees, in the most pronounced c
126 an immobilized polymerase molecule extrudes decondensed, transcribed sequences into its surroundings
127 ng mitosis, the genome is transformed from a decondensed, transcriptionally active state to a highly