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1 eration, we find no evidence to suggest that dedifferentiated cells acquire a ductal fate during this
3 , the proliferation and the migration of the dedifferentiated cells are dependent on Hh signaling.
5 transcription factors and that diseased and dedifferentiated cells can be returned to normal functio
6 transition, and exogenous PHD2 expression in dedifferentiated cells can restore an epithelial phenoty
7 lular matrix, and proliferation of partially dedifferentiated cells, evidence strongly supports the l
8 and three IR-resistant clones isolated from dedifferentiated cells have acquired the ability to prol
10 could be detected in similar assays with the dedifferentiated cell line HepG2.1 or the nonhepatic cel
13 n organized structure of lineage restricted, dedifferentiated cells that cooperate to regenerate the
14 erogeneous population of lineage-restricted, dedifferentiated cells that ultimately orchestrates rege
18 order to perform genetic lineage tracing of dedifferentiated cells while measuring the cellular tran
19 val may be manifested by the production of a dedifferentiated cell with broader potential and that th