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1 e Tg mice exhibited hepcidin excess and iron deficiency anemia.
2 elevated hepcidin levels and consequent iron deficiency anemia.
3 inguishes thalassemia-trait anemia from iron-deficiency anemia.
4 aining regimens with iron deficiency or iron deficiency anemia.
5 tion of abnormal hemoglobin to classify iron deficiency anemia.
6 s, 20%, p = 4e-7, OR = 5), particularly iron-deficiency anemia.
7 children, including iron deficiency and iron deficiency anemia.
8 05) in volunteers reporting to BCT with iron deficiency anemia.
9 causing gastrointestinal blood loss and iron deficiency anemia.
10 owing: osteoporosis or low bone mass or iron-deficiency anemia.
11 further characterization of this novel SOD2-deficiency anemia.
12 ted with a 40% reduction in the risk of iron deficiency anemia.
13 he risk of anemia, iron deficiency, and iron-deficiency anemia.
14 the benefits of successful treatment of iron deficiency anemia.
15 ause of gastrointestinal hemorrhage and iron deficiency anemia.
16 ues, while they continue to have severe iron deficiency anemia.
17 s cause gastrointestinal hemorrhage and iron deficiency anemia.
18 males and significantly associated with iron deficiency anemia.
19 ary hemochromatosis and iron-refractory iron deficiency anemia.
20 and education were not associated with iron deficiency anemia.
21 HFE mutations had a lower prevalence of iron deficiency anemia.
22 ding of thrombocytosis in patients with iron deficiency anemia.
23 %) were iron deficient; 24 of these had iron deficiency anemia.
24 r patients with rheumatoid arthritis or iron-deficiency anemia.
25 000 toddlers and 3.3 million women have iron deficiency anemia.
26 ency progresses from low iron stores to iron-deficiency anemia.
27 s have increased hematocrit and risk of iron deficiency anemia.
28 non-dialysis chronic kidney disease and iron deficiency anemia.
29 iency without anemia and about 13% have iron-deficiency anemia.
30 nd it predisposes Tfrc(Alb-Cre) mice to iron-deficiency anemia.
31 micronutrient intakes and reduction in iron deficiency anemia.
32 worldwide whose inadequate diet causes iron deficiency anemia.
33 ion in a mouse model of iron-refractory iron-deficiency anemia.
34 ncer mortality/morbidity and changes to iron-deficiency anemia.
35 nths of age in infants at high risk for iron deficiency anemia.
36 atients with unexplained, mild, chronic iron-deficiency anemia.
37 onsidered for children with nutritional iron-deficiency anemia.
38 be a safe and efficacious treatment for iron deficiency anemia.
39 , whose mutations cause iron refractory iron deficiency anemia.
40 of iron in macrophages and symptoms of iron deficiency anemia.
41 llenge the entrenched oral treatment of iron deficiency anemia.
42 ited States who do not have symptoms of iron deficiency anemia.
43 orders had been treated for concomitant iron deficiency anemia.
44 ary hemochromatosis and iron-refractory iron-deficiency anemia.
45 /L; P<0.05) and low in one patient with iron deficiency anemia.
46 appeared to be effective in correcting iron deficiency anemia.
47 important biomarker in diseases such as iron-deficiency anemia.
48 underlies erythropoietic repression in iron deficiency anemia.
49 His blood count shows signs of mild iron deficiency anemia.
50 re), and laboratory tests suggestive of iron deficiency anemia (0.5-percentage point reduction [95% C
52 34.7%] vs 225 women [13.6%]; P < .001), iron deficiency anemia (347 women [21.0%] vs 135 women [8.2%]
53 6 with secondary erythrocytosis, 2 with iron-deficiency anemia, 4 with hemochromatosis, or 5 normal s
54 tical improvements in the management of iron deficiency anemia, a disorder that may affect as many as
55 Consuming wildmeat may protect against iron-deficiency anemia, a serious public health problem globa
56 ons in predictors of iron deficiency or iron deficiency anemia according to iron supplementation sugg
57 tensity infection had a greater risk of iron deficiency anemia (adjusted prevalence odds ratio: 6.6;
62 evelopment, risk of iron deficiency, or iron deficiency anemia among breastfed infants in a setting o
64 for successful interventions to reduce iron deficiency anemia among food-insecure children and to im
65 study compared 96 female patients with iron-deficiency anemia and 60 healthy female control subjects
66 rther evaluation, from which those with iron-deficiency anemia and active bleeding had been excluded,
67 e of the tendency of infants to develop iron deficiency anemia and because of the documented adverse
68 ctive was to estimate the prevalence of iron deficiency anemia and examine potential reasons for this
69 els were used to identify predictors of iron deficiency anemia and iron deficiency without anemia.
70 ation of the potential contributions of iron deficiency anemia and iron treatment to this differentia
71 Hookworm infection is a major cause of iron deficiency anemia and malnutrition in developing countri
72 ifestation is epistaxis that results in iron-deficiency anemia and reduced health-related quality of
73 he association of lice infestation with iron-deficiency anemia and risk factors such as homelessness
74 n this cohort study of 24 patients with iron deficiency anemia and severe aortic stenosis, angiodyspl
76 ockout (Tmprss6-/-) mice, which exhibit iron deficiency anemia and thrombocytosis, we observed a Mk b
79 trolled trial focusing on patients with iron deficiency (+ /- anemia) and ND-CKD (serum ferritin < 20
80 ointestinal blood loss, and unexplained iron-deficiency anemia), and the performance of selected test
82 Fifty-two percent of the subjects had iron deficiency anemia, and 30% had serum 25-hydroxyvitamin D
83 enty-five percent of all anemia, 35% of iron deficiency anemia, and 73% of severe anemia were attribu
84 y lower prevalences of iron deficiency, iron deficiency anemia, and low vitamin B-12 and, in Kenya, a
86 f iron fortification of food to improve iron deficiency, anemia, and biological outcomes is not prove
89 erythematosus; infections; splenectomy; iron deficiency anemia; and solid tumors such as lung cancer.
90 tanding type II diabetes, hypertension, iron deficiency anemia, aortic stenosis, and prior bariatric
96 fect in iron absorption that results in iron-deficiency anemia, as revealed by an N-ethyl-N-nitrosour
97 ficiency among children worldwide, with iron-deficiency anemia associated with long-term adverse neur
100 ociated with decreased risk of maternal iron deficiency anemia at term (4 trials, n = 2230; 8.6% vs 1
101 comes that are associated with maternal iron deficiency anemia, both Canada and the United States rec
102 venous iron enables rapid correction of iron-deficiency anemia, but certain formulations induce fibro
103 is approved for use in the treatment of iron deficiency anemia, but it can serve as an alternative to
105 .48; 95% CI, 0.32-0.70) and the risk of iron deficiency anemia by 66% (RR, 0.34; 95% CI, 0.19-0.62).
107 mia of chronic disease, iron refractory iron deficiency anemia, cancer, hereditary hemochromatosis, a
108 ss, studies of a murine iron-refractory iron-deficiency anemia-causing mutant (Mt2I286F) in the compl
109 emia; and had a 64% lower prevalence of iron-deficiency anemia compared with control group children.
110 Four of 8 of the deprived monkeys had iron deficiency anemia compared with none of the controls and
112 re shared with conditions such as haematinic deficiency anemias. Definitive diagnosis of MDS requires
113 is the most accurate animal model of folate deficiency anemia described to date that closely capture
114 6; CI: 0.4, 0.7), but the prevalence of iron deficiency anemia did not change significantly in childr
115 mice and Belgrade (b) rats have severe iron deficiency anemia due to defects in intestinal iron tran
117 es the incidence of iron deficiency and iron deficiency anemia during pregnancy, but evidence on heal
122 iron metabolism, leading to functional iron deficiency, anemia, erythropoietic protoporphyria, and a
123 re undetectable or low in patients with iron deficiency anemia (ferritin < 10 ng/mL), iron-depleted H
124 en aged 9 to 48 months with nutritional iron-deficiency anemia, ferrous sulfate compared with iron po
125 n and its great value in distinguishing iron deficiency anemia from the anemia of chronic disease.
126 deficiency (from 60% to 6%; P<0.0001), iron deficiency anemia (from 35% to 15%; P<0.007), and abnorm
128 wildtype MDS, megaloblastic anemia, and iron deficiency anemia), Haemorasis detected over half a mill
130 5 patients aged 18 years and older with iron-deficiency anemia (hemoglobin level <=11 g/dL; serum fer
131 (HR per 1 SD, 0.85; 95% CI, 0.76-0.95), iron deficiency anemia (HR per 1 SD, 0.91; 95% CI, 0.84-0.98)
132 l that genetic variants associated with iron deficiency anemia (ICD10), obesity, type 2 diabetes, syn
135 s are to investigate the association of iron deficiency anemia (IDA) and FM and to find the effects o
136 s was to assess the association between iron deficiency anemia (IDA) and retinal changes via optical
137 oscopy or colonoscopy among adults with iron-deficiency anemia (IDA) and/or hematochezia have not bee
138 theast Asia, Thalassemia trait (TT) and iron deficiency anemia (IDA) are the two most common anemia t
139 Although the hazards associated with iron deficiency anemia (IDA) are well known, concerns about r
142 workup fails to establish the cause of iron deficiency anemia (IDA) in a substantial proportion of p
143 utine screening and supplementation for iron deficiency anemia (IDA) in asymptomatic, nonanemic pregn
144 he actual prevalence of anemia, ID, and iron deficiency anemia (IDA) in California remains unclear.
147 the standard approach for investigating iron deficiency anemia (IDA) in men older than 45 and postmen
148 al biomarkers used for the diagnosis of iron-deficiency anemia (IDA) in patients with CKD have limita
154 d young children in Sub-Saharan Africa, iron-deficiency anemia (IDA) is common, and many complementar
159 prevalence of iron deficiency (ID) and iron deficiency anemia (IDA) was 10-32% and 2-5%, respectivel
160 was defined as hemoglobin < 11 g/d and iron-deficiency anemia (IDA) was defined as low ferritin, hig
161 on strategy is unclear in patients with iron deficiency anemia (IDA) who have either normal kidney fu
162 1 billion people worldwide suffer from iron deficiency anemia (IDA), a state of systemic iron insuff
163 the prevalence of iron deficiency (ID), iron deficiency anemia (IDA), and other measures of iron nutr
164 d on iron biomarker concentrations were iron deficiency anemia (IDA), anemia of inflammation (AI), an
165 tin, which was suitable for identifying iron-deficiency anemia (IDA), but less reliable than serum as
168 (plasma ferritin < 12 ug/L) by 56%, and iron deficiency anemia (IDA; Hb < 110 g/L and plasma ferritin
169 ciency (ID; diminished iron stores) and iron deficiency anemia (IDA; poor iron stores and diminished
170 evere complications that may arise from iron deficiency anemia if it is not recognized and treated ea
171 ion at the locus, impacting the risk of iron deficiency anemia in African Americans with specific gen
178 ravenous iron is commonly used to treat iron deficiency anemia in non-dialysis chronic kidney disease
181 priate for estimating the prevalence of iron deficiency anemia in poor Jamaican girls 13-14 y of age.
183 pregnant women or routine screening for iron deficiency anemia in pregnant women or young children.
184 of benefits and harms of screening for iron deficiency anemia in pregnant women to prevent adverse m
185 disease is among the leading causes of iron-deficiency anemia in the developing world and is associa
187 imate prevalence of iron deficiency and iron deficiency anemia in the Jamaican girls: 7.6% and 4.3%,
190 alences of anemia, iron deficiency, and iron deficiency anemia increased in all three trial groups bu
191 ine protease mutated in iron-refractory iron deficiency anemia, inhibits hepcidin expression by dampe
194 iciency (serum ferritin <12 ng/mL), and iron deficiency anemia (iron deficiency and hemoglobin <10.5
200 acid fortification, the prevalence of folate-deficiency anemia is nearly nonexistent in a community-d
205 ncer, and 6 to 13 percentage points for iron deficiency anemia laboratory tests suggestive of colorec
209 n blood in the small intestine, causing iron-deficiency anemia, malnutrition, growth and development
210 ic therapy, antiangiogenic therapy, and iron deficiency anemia management across the HHT disease seve
211 duals with nonanemic iron deficiency or iron-deficiency anemia may be asymptomatic or experience fati
213 in level, iron-deficiency (IDA) and non-iron-deficiency anemia (NIDA), and inflammatory markers.
215 yroidism, liver disease, AIDS, coagulopathy, deficiency anemia, obesity, alcohol abuse, or drug abuse
216 asing risk of traits related to anemia (iron deficiency anemia: odds ratio [OR] scaled to a standard
217 ngle-center cohort study, patients with iron deficiency anemia on the TAVI waiting list from Septembe
219 who are malnourished, have symptoms of iron deficiency anemia, or have special hematologic condition
220 e other major modes of presentation are iron-deficiency anemia, osteoporosis, screening of family mem
222 ng early development in iron-refractory iron deficiency anemia patients, who present with microcytic
226 our findings from the murine models of iron deficiency anemia, primary human MEPs exhibit decreased
229 lthough much of the ethnic disparity in iron deficiency anemia remains unexplained, factors associate
230 sk [RR], 0.46; 95% CI, 0.16 to 1.30) or iron deficiency anemia (RR, 0.78; 95% CI, 0.05 to 12.46) at 1
231 woman has heavy menstrual bleeding and iron-deficiency anemia.She reports nocturia and urinary frequ
232 ly the microcytic hypochromic anemia or iron deficiency anemia that is the clinical hallmark of hookw
234 ut diagnosis and management of absolute iron deficiency anemia, the anemia of inflammation with funct
235 kness (CT) in the eyes of patients with iron-deficiency anemia, the most common form of the anemia, v
236 ost effective therapy of IBD-associated iron deficiency anemia, the perception of risk related to int
237 ory evaluation was notable for profound iron deficiency anemia, thrombocytosis, and hyperhomocysteine
239 inical trial in adults with NDD-CKD and iron deficiency anemia to compare the safety and efficacy of
240 n levels for diagnosing iron-refractory iron deficiency anemia to global health applications such as
241 n status across the full continuum from iron deficiency anemia to iron deficiency to iron replete to
242 n symptoms of irritable bowel syndrome, iron deficiency anemia, unexplained arthritis, and even chron
243 is a familial disorder characterized by iron deficiency anemia unresponsive to oral iron treatment bu
245 iron supplement use, the prevalence of iron deficiency anemia was 2.3 times higher in Mexican Americ
246 ncome) >3.0, however, the prevalence of iron deficiency anemia was 2.6 +/- 0.9% in Mexican American a
252 ring age were iron deficient; of these, iron deficiency anemia was found in 3% and 2% to 5%, respecti
255 st administration of FCM, correction of iron deficiency anemia was observed with improved mean hemogl
257 nia, sustained neutropenia, anemia, and iron deficiency anemia were infrequent and similar across all
258 e rates of anemia, iron deficiency, and iron deficiency anemia were significantly lower after 24 wk i
260 rasitic loads increase the risk of iron (Fe) deficiency anemia, which remains prevalent globally.
261 2.7%) to 6.8% (95% CI: 4.2%, 9.3%), and iron deficiency anemia, which was 6.2% (95% CI: 3.0%, 9.3%) a
262 In 2 randomized trials of patients with iron-deficiency anemia who were intolerant of or unresponsive
263 hat newborn homozygous Pcm mice exhibit iron deficiency anemia with increased duodenal Fpn1 expressio
265 mice (characterized by iron-refractory iron deficiency anemia) with and without adenine diet-induced
266 e prevalence of folate deficiency and folate-deficiency anemia within a sample of the Reasons for Geo
267 ith reduced risk of iron deficiency and iron deficiency anemia without a concomitant increase in the