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1 loss caused by thrombosis and a high risk of delayed graft function.
2 donor, post-kidney transplantation SSIs, and delayed graft function.
3 antation renal biopsies (PIB) as markers for delayed graft function.
4 unction and the second functioned well after delayed graft function.
5 ansplant/de novo donor-specific antibody and delayed graft function.
6 ascular thrombosis, especially when there is delayed graft function.
7 index higher than 27 kg/m, and occurrence of delayed graft function.
8 terial occlusion in transplanted grafts with delayed graft function.
9 ents receiving renal transplants at risk for delayed graft function.
10 lomerular filtration rate, and occurrence of delayed graft function.
11 help in understanding the pathophysiology of delayed graft function.
12 ar ejection fraction, low serum albumin, and delayed graft function.
13 tomy was associated with no mortality and no delayed graft function.
14 r sensitization, paucity of live donors, and delayed graft function.
15 There was no delayed graft function.
16 f hospital stay, time to return to work, and delayed graft function.
17 There were no surgical complications or delayed graft function.
18 of acute rejection but not the incidence of delayed graft function.
19 n from older donors and deceased donors, and delayed graft function.
20 with 100% 2-year graft survival and without delayed graft function.
21 reperfusion injury (IRI) is a major cause of delayed graft function.
22 g for registry-based risk factors, including delayed graft function.
23 ocesses could lead to decreased incidence of delayed graft function.
24 is (with a functioning graft) and 2 cases of delayed graft function.
25 stricture, urine leak, hernia formation, and delayed graft function.
26 GP1 was associated with early graft loss and delayed graft function.
27 s are frequently associated with a period of delayed graft function.
28 r 6-month eGFR only among recipients without delayed graft function.
30 ces between induction groups for outcomes of delayed graft function, 1-year acute rejection, 1-year B
32 7% SIR-NLD, P=0.04) and a lower incidence of delayed graft function (21% SIR-LD vs. 39% SIR-NLD, P<0.
33 ients of a living donor had a higher rate of delayed graft function (23.6% versus 18.7%; odds ratio,
35 ilure/rejection (16.7% vs 16.8%; P = 0.897), delayed graft function (29.97% vs 29.36%; P = 0.457) or
37 f graft loss (9.2% and 10.2%, respectively), delayed graft function (40.4% and 44.5%), and death (4.3
39 R and other causes of allograft dysfunction; delayed graft function (54+/-7.8 micromol/L), urinary tr
41 .85 kg), but there was a higher incidence of delayed graft function (7 of 11 vs. 1 of 16; P=0.002).
44 cyte antigens mismatches, immunosuppression, delayed graft function, acute rejection [AR]), previous
46 Donor pretreatment with NAC does not improve delayed graft function after kidney transplantation.
47 onors significantly reduced the incidence of delayed graft function after kidney transplantation.
49 , donor age more than 50 years (HR=1.86) and delayed graft function after retransplant (HR=1.95).
51 s before function returned and two developed delayed graft function; all transplanted livers and panc
55 ents of elderly DCD kidneys experienced more delayed graft function and acute rejection than did elde
56 associated with adverse outcomes, including delayed graft function and biopsy-proven acute rejection
57 Machine perfusion techniques have decreased delayed graft function and could improve graft survival.
58 iod could help to ameliorate the severity of delayed graft function and could provide a path to using
62 on injury (IRI) significantly contributes to delayed graft function and inflammation, leading to graf
63 ieu in high-acuity CLKT recipients increases delayed graft function and kidney allograft failure.
64 ieu in high acuity CLKT recipients increases delayed graft function and kidney allograft failure.
65 ning a mechanism underlying the link between delayed graft function and long-term allograft failure.
66 (null) cell frequencies were associated with delayed graft function and lower estimated glomerular fi
67 tch (RXM) demonstrated a higher incidence of delayed graft function and of acute rejection and graft
69 The 3- and 12-month treatment failure rates, delayed graft function and renal function, and patient a
71 ferior outcome that was not significant, and delayed graft function and warm ischaemic time had no ef
72 GFR), and incidence of acute rejection (AR), delayed graft function and/or graft loss at 2 years post
73 t graft dysfunction (primary nonfunction and delayed graft function) and were an independent risk fac
75 e than 65 years, five to six HLA mismatches, delayed graft function, and acute rejection were indepen
76 Ischemic damage is the most common cause of delayed graft function, and although it is known that ti
77 afts is associated with tubular cell injury, delayed graft function, and an increased incidence of ac
78 We found that the degree of HLA mismatch, delayed graft function, and AR were the only significant
79 ultivariate analysis revealed recipient age, delayed graft function, and BMI >30 to be independent ri
81 ass index, waiting time, cold ischemic time, delayed graft function, and coronary risk factors showed
82 5- and 10-year graft survival, incidence of delayed graft function, and estimated glomerular filtrat
84 iteria donors, race, cytomegalovirus status, delayed graft function, and immunologic risks were simil
85 ded patient and graft survival, incidence of delayed graft function, and incidence and severity of bi
86 idney transplant recipients, 31% experienced delayed graft function, and mean+/-SD 6-month eGFR was 5
87 included 1-year graft and patient survival, delayed graft function, and need for posttransplant dial
88 splantation), a significantly higher rate of delayed graft function, and significantly higher levels
89 g public insurance, panel reactive antibody, delayed graft function, and steroid withdrawal; in these
90 owing variables were recorded: demographics; delayed graft function; AR at 3, 6, and 12 months; time
91 of Foley catheter, ureteral stent, age, and delayed graft function are independent risk factors for
92 lly important in ischemia-reperfusion injury/delayed graft function as well as in acute and chronic a
94 comorbidities (OR, 2.01; 95% CI, 1.04-3.86), delayed graft function at the time of discharge (OR, 1.6
96 ath (DCD) kidneys suffer a high incidence of delayed graft function attributable to warm ischemia and
97 ted that C1-INH treatment may reduce IRI and delayed graft function, based on decreased requirements
98 sirolimus permits a window of recovery from delayed graft function before the introduction of reduce
99 There was no difference in the incidence of delayed graft function between CS and MP (32/51 (62.8%)
100 DCD kidneys (vs. static storage) may reduce delayed graft function but has no effect on long-term or
101 Donors with AKI are more likely to undergo delayed graft function but have similar long-term outcom
102 eptance in the match-run was associated with delayed graft function but not all-cause allograft failu
103 th donor thrombi were more likely to exhibit delayed graft function, but graft function at 1 and 2 ye
104 s were younger and less likely to experience delayed graft function compared with recipient of ECD ki
105 s: donor AKI (stage 2 or greater), recipient delayed graft function (defined as dialysis in first wee
109 In addition to providing overall results for delayed graft function (DGF) (requirement for dialysis i
110 RL) may increase the incidence of or prolong delayed graft function (DGF) after cadaveric renal trans
112 me studies have found an association between delayed graft function (DGF) after kidney transplantatio
115 idneys, which contribute to a higher risk of delayed graft function (DGF) after transplantation.
117 dered to contribute to the occurrence of the delayed graft function (DGF) and chronic graft failure.
118 response gene (MYD) 88, are associated with delayed graft function (DGF) and could be used as biomar
119 njury (IRI) to renal grafts, contributing to delayed graft function (DGF) and episodes of acute immun
120 determine the effect of recipient obesity on delayed graft function (DGF) and graft survival after re
121 nd cause of death influence the incidence of delayed graft function (DGF) and graft survival; however
122 inators between the profiles of kidneys with delayed graft function (DGF) and immediate graft functio
123 ed in renal allograft recipients at risk for delayed graft function (DGF) and immunologic rejection.
124 rolonged ischemia is a known risk factor for delayed graft function (DGF) and its interaction with do
128 nction (IGF), slow graft function (SGF), and delayed graft function (DGF) and the drop in estimated g
129 elial damage in the renal graft and leads to delayed graft function (DGF) and to an early loss of per
130 hemia time (CIT) with resulting increases in delayed graft function (DGF) and transplant-related cost
132 atric kidney transplant recipients developed delayed graft function (DGF) between 2000 and 2010.
134 ns experienced nearly twice the incidence of delayed graft function (DGF) compared with heart-beating
136 ted nomogram designed to predict the risk of delayed graft function (DGF) in a given transplant.
137 is study examined the association of PP with delayed graft function (DGF) in all (n=94,709) deceased
138 luate the efficacy of Mirococept in reducing delayed graft function (DGF) in deceased donor renal tra
139 tributing to ischemia-reperfusion injury and delayed graft function (DGF) in human kidney transplant
140 ther there is an association between sex and delayed graft function (DGF) in patients who received de
152 ee regimens benefited patients regardless of delayed graft function (DGF) or early acute rejection st
156 of the Kidney Donor Risk Index (KDRI) versus delayed graft function (DGF) to predict graft survival i
157 l using Cox regression, acute rejection, and delayed graft function (DGF) using logistic regression,
158 al using Cox regression, acute rejection and delayed graft function (DGF) using logistic regression,
161 , inflammation, and MHC II expression, while delayed graft function (DGF) was therefore reduced.
166 ysis can aid in predicting the occurrence of delayed graft function (DGF), acute rejection (AR), and
167 antation and evaluated one of these outcomes-delayed graft function (DGF), acute rejection, graft or
169 We examined the association between CIT and delayed graft function (DGF), allograft survival, and pa
170 cardiac death (DCD) exhibit higher rates of delayed graft function (DGF), and DCD livers demonstrate
172 erm postoperative outcome, the occurrence of delayed graft function (DGF), and long-term graft surviv
173 of this policy on cold ischemia time (CIT), delayed graft function (DGF), and transplant survival wa
175 Ischemia-reperfusion injury (IRI) leading to delayed graft function (DGF), defined by the United Netw
177 Topics included the development of IRI and delayed graft function (DGF), histology and biomarkers,
179 eceiving renal transplant centers focused on delayed graft function (DGF), patient and allograft surv
180 utcomes included graft loss, renal function, delayed graft function (DGF), patient death, and the inc
182 expression changes in kidney allografts with delayed graft function (DGF), which often follows ischem
193 ts with serum biomarker measurements, 26 had delayed graft function (DGF; hemodialysis within 1 week
194 nd to assess their performance in predicting delayed graft function (DGF=dialysis requirement during
195 days, early events (acute rejection [AR] and delayed graft function [DGF] before day 90) were recorde
196 eased donor kidney transplant [DDKT] without delayed graft function [DGF] hazard ratio: 24.634.447.9,
197 e higher rates of primary graft nonfunction, delayed graft function, discard, and retrieval associate
198 y ischemia detection in porcine kidneys with delayed graft function early after transplantation.
199 tive predictor of poor KT outcomes including delayed graft function, early hospital readmission, immu
200 in the DKT group, although the incidence of delayed graft function, early rejection treatment, and g
202 ve, deceased donor, expanded donor criteria, delayed graft function, elevated panel reactive antibody
203 " was compared with the following endpoints: delayed graft function, estimated glomerular filtration
204 , there was no effect of arteriosclerosis on delayed graft function, estimated glomerular filtration
205 urrence and duration of functionally defined delayed graft function (fDGF) in donation after circulat
206 nt of six microRNAs had predictive value for delayed graft function following kidney transplantation.
209 Renal transplant recipients who experience delayed graft function have increased risks of rejection
210 tes, being on a ventilator, hospitalization, delayed graft function, hepatocellular carcinoma, and in
211 y obese patients may be at increased risk of delayed graft function, higher postoperative complicatio
212 GAL concentrations associated with recipient delayed graft function (highest versus lowest NGAL terti
214 ents is associated with an increased risk of delayed graft function; however, this does not compromis
215 age (HR, 1.1; 95% CI, 1.0-1.2; P=0.03), and delayed graft function (HR, 1.4; 95% CI, 1.0-1.9; P=0.06
216 cute rejection (HR=1.47; 95% CI, 1.23-1.76), delayed graft function (HR=1.46; 95% CI, 1.25-1.71), and
217 human leukocyte antigen match, occurrence of delayed graft function, immunosuppressive regimen, weigh
219 that the use of MP reduces the incidence of delayed graft function in donation after circulatory dea
221 rmia in the donor safely reduced the rate of delayed graft function in kidney transplant recipients w
223 alondialdehyde (MDA) levels, correlates with delayed graft function in renal transplant recipients.
229 , cytomegalovirus D+/R-, cold ischemia time, delayed graft function, induction with antithymocyte glo
230 ury leading to acute renal failure (ARF) and delayed graft function is an important problem in organ
231 ugh the incidence of primary nonfunction and delayed graft function is higher with organs obtained fr
234 plantation was associated with an absence of delayed graft function, low acute rejection rates, and h
235 r a significant increase in those developing delayed Graft Function (miR-9: P = 0.068, mIR-10a: P = 0
236 as consistently elevated in those developing Delayed Graft Function (n = 165 samples from 33 patients
237 (O.R. 0.15, 95% C.I. 0.03-0.91, P=0.04) and delayed graft function (O.R. 4.49, 95% C.I. 1.67-36.56,
242 /m were associated with an increased risk of delayed graft function (odds ratio [95% confidence inter
243 ion) and were an independent risk factor for delayed graft function (odds ratio, 2.152; 95% confidenc
244 n in renal transplant recipients at risk for delayed graft function or acute rejection (n=278), TMG w
246 AKI but provide limited value in predicting delayed graft function or early allograft function after
247 presented with TE, which was not related to delayed graft function or estimated glomerular filtratio
249 s at engraftment and 3 and 6 months, without delayed graft function or interval rejection, and we con
250 nth (OR 0.62 per 10 ml/min/1.73 m, P<0.001), delayed graft function (OR 11.5, P = 0.02), and a SRL-ba
251 egree) was not significantly associated with delayed graft function (OR, 1.16; 95% CI, 0.94-1.43; P =
252 ce interval [CI]: 1.09-90.58; p = 0.041) and delayed graft function (OR: 3.40; 95% CI: 1.08-10.73; p
253 dysfunction, variably referred to as slow or delayed graft function, or in the most extreme cases, pr
254 ensitized, receive public insurance, develop delayed graft function, or undergo steroid withdrawal.
255 ents not receiving steroids experienced less delayed graft function (p = 0.01) and pretransplant dial
258 ne (P=0.0001), 1-year creatinine (P=0.0015), delayed graft function (P=0.007), total human leukocyte
260 oven acute rejection, graft loss, or death), delayed graft function, patient and graft survival rates
261 tly higher body mass index and older donors, delayed graft function, prevalence of metabolic syndrome
262 ement for mechanical circulatory support for delayed graft function, primarily in recipients with ven
269 fusion might help to decrease posttransplant delayed graft function rates and to increase the donor p
271 warm ischemia time and other donor outcomes, delayed graft function rates, recipient creatinine at 1
273 for age, gender, deceased donor transplant, delayed graft function, tacrolimus and exposure to antib
274 sufficient peri-transplant ischemia to cause delayed graft function than from allografts with slow or
275 matched, though the 12-week cohort had more delayed graft function than their 24-week counterparts (
276 (CI) is a risk factor for the development of delayed graft function that predicts reduced 5-year kidn
277 Although there was a higher incidence of delayed graft function, there was no significant differe
288 For death-censored kidney graft survival, delayed graft function was the strongest negative predic
290 ger pretransplantation dialysis vintage, and delayed graft function were associated with higher ED us
291 only recipients with both a positive RXM and delayed graft function were at significantly higher risk
293 longer in DKT (22.2+/-9.7 hr), but rates of delayed graft function were lower (29.3%) compared to EC
294 and living donor kidney transplants without delayed graft function were randomized to receive predni
295 h after transplantation and the incidence of delayed graft function were similar in both groups.
298 patients at high risk for acute rejection or delayed graft function who received a renal transplant f
299 patients at high risk for acute rejection or delayed graft function who received a renal transplant f
300 ltivariate model of CCL2, recipient age, and delayed graft function yielded an AUC 0.87 for predictio