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1 minds (denoted as "threat/control override" delusions).
2 ve neuropsychiatric model of the persecutory delusion.
3 in worry accounted for 66% of the change in delusion.
4 tive behaviours, largely linked to patients' delusions.
5 at are believed to constitute the genesis of delusions.
6 ng aberrant perceptions and the formation of delusions.
7 Children's self-reported hallucinations and delusions.
8 n-related processes are involved in paranoid delusions.
9 ded to understand the emergence of grandiose delusions.
10 sistent visual hallucinations and persistent delusions.
11 its association with psychotic disorders and delusions.
12 olume positively correlated with severity of delusions.
13 ciation with positive symptoms, specifically delusions.
14 tion between tangle pathology and persistent delusions.
15 predict individual variation in severity of delusions.
16 ed: behavioral disturbances, depression, and delusions.
17 egression models correlated with severity of delusions.
18 s of disorganization and hallucinations plus delusions.
19 ness or guilt and suicidal thoughts also had delusions.
20 which generates scores on six dimensions of delusions.
21 eviewing the literature on the assessment of delusions.
22 ymptoms manifested as paranoid and grandiose delusions.
23 ) and lower ratings for thought disorder and delusions.
24 ferences in information seeking unrelated to delusions.
25 ly in participants without hallucinations or delusions.
26 ychotic symptoms, such as hallucinations and delusions.
27 participants with reported hallucinations or delusions.
28 ntinuum ranging from conventional beliefs to delusions.
29 for their effects on decisions, beliefs, and delusions.
30 volume and false recognition or presence of delusions.
31 chanism may underwrite persecutory and other delusions.
32 ext of social learning to target persecutory delusions.
33 rtion (115 [63.2%]) reported threshold-level delusions.
34 nd be over-reinforced, with implications for delusions.
35 ssing about a more chaotic world relevant to delusions.
36 functions were accentuated in patients with delusions.
37 bates about the formation and maintenance of delusions.
38 may contribute to erroneous beliefs, such as delusions.
39 in the disorder, such as hallucinations and delusions.
40 viour therapy (CBT) would reduce persecutory delusions.
41 the first large trial focused on persecutory delusions.
42 his framework may explain hallucinations and delusions.
43 itive biases involved in the pathogenesis of delusions.
44 often explain their violence as being due to delusions.
45 ed headaches, seizures, and rarely olfactory delusions.
46 I treatment was shown in the AD subgroup for delusions (-0.08; 95% CI, -0.14 to -0.03; P = .006) and
47 -0.04; P = .003) and in the PD subgroup for delusions (-0.14; 95% CI, -0.26 to -0.01; P = .04) and h
48 , 95% CI 3.30-9.40; p<0.001) and persecutory delusions (2.08 [SE 0.73] PSYRATS units, 95% CI 0.64-3.5
51 dd beliefs or delusions (87.6%), persecutory delusions (75.2%), ideas of reference (55.8%), and visua
53 ode(s), participants reported odd beliefs or delusions (87.6%), persecutory delusions (75.2%), ideas
55 nistered seven-item scale designed to assess delusions across a wide range of psychiatric disorders.
56 he stability of the dimensional structure of delusions across diagnoses and delusion types suggests t
57 ed a range from reducing but also increasing delusions (adjusted mean difference 0.3, 95% CI -2.0 to
58 ies were not associated with the presence of delusions after accounting for confounding variables, an
59 were found to exhibit a higher frequency of delusions, agitation, and depression (delusions: odds ra
62 the frequency of irritability, anxiety, and delusions; among patients who had these symptoms at week
69 ychosis, typically characterized by paranoid delusions and auditory hallucinations and often associat
70 araphrenia presenting with extensive bizarre delusions and auditory hallucinations but no prominent n
71 ting), two positive symptom factors (bizarre delusions and auditory hallucinations), and a disorganiz
72 uggestive of psychotic illness and transient delusions and auditory or visual hallucinations occurred
73 sed by the Insomnia Severity Index (ISI) and delusions and hallucinations assessed by the Psychotic S
74 nd pimavanserin is approved for treatment of delusions and hallucinations associated with psychosis o
78 e of these agents, has been found to improve delusions and hallucinations in patients who fail to res
79 years) in the majority of patients, paranoid delusions and hallucinations were intermediate in their
80 ly less apathetic and anxious, and had fewer delusions and hallucinations while on treatment than con
81 at positive symptoms of schizophrenia (e.g., delusions and hallucinations) and have been approved by
82 orresponding to positive (i.e., subthreshold delusions and hallucinations) and negative (i.e., subthr
84 ffective at reducing positive symptoms (i.e. delusions and hallucinations), they do little to improve
85 ioral disturbance, intermediate for paranoid delusions and hallucinations, and least for depressed mo
86 t fits better with the work in patients with delusions and hallucinations, how they behave, and what
90 not find evidence of a relationship between delusions and history of suicidal ideation or suicide at
95 ognitive mechanisms that explain persecutory delusions and provide testable, phenomenologically relev
96 positive symptoms (including hallucinations, delusions and thought disorder), negative symptoms (such
97 This study explores the relationship between delusions and violence among patients recently discharge
100 ency than the normal subjects, patients with delusions and/or hallucinations exhibited significantly
101 Psychotic symptoms, i.e., the presence of delusions and/or hallucinations, is a frequent complicat
102 , including positive (eg, hallucinations and delusions) and negative (eg, avolition and alogia) sympt
105 D-TDP were significantly more likely to have delusions, and for the delusions to occur in the first 3
106 hese 20 symptoms/signs, 3 (thought disorder, delusions, and hallucinations) were included in all diag
109 rs are particularly prone to hallucinations, delusions, and rapid eye movement sleep behavior disorde
112 suffer from symptoms including psychosis and delusions, apathy, anhedonia, and cognitive deficits.
116 us model of the data indicated that paranoid delusions are associated with a combination of pessimist
120 n types suggests that even seemingly diverse delusions are more like than unlike each other; this is
126 ings on dimensions of the MacArthur-Maudsley Delusions Assessment Schedule were significantly but mod
127 delusions were given the MacArthur-Maudsley Delusions Assessment Schedule, which generates scores on
128 associated with the emergence of persecutory delusions at follow-up (odds ratio=3.52, 95% CI=1.18-10.
129 ed measure of persecutory delusions (PSYRATS delusion) at 12 weeks (Cohen d, 0.47; 95% CI, 0.17-0.78;
131 aged 18-65 years with persistent persecutory delusions but non-affective psychosis from two centres:
135 t persistent positive symptoms, particularly delusions, can be improved by cognitive behaviour therap
136 e most clearly articulated the ways in which delusions cannot, whereas phenomenologists have best art
137 to present with only attenuated/subthreshold delusions compared with only attenuated/subthreshold hal
138 these 115, 104 patients (90.4%) had thematic delusion content consistent with that reported during th
140 ty, a number of authors suggested that manic delusions could arise directly from a euphoric mood.
141 ic manifestations such as hallucinations and delusions could provide a window into the biology of psy
143 psychiatric symptoms (visual hallucinations, delusions, depression) to plaques (Consortium to Establi
144 oss 10 BPSDs (agitation, apathy, depression, delusions, disinhibition, auditory hallucinations, visua
145 es or syndromes consisting of hallucinations/delusions, disorganization of thought and behavior, and
146 0.66) and positive symptoms (hallucinations, delusions, disorganization) (adjusted mean [SE], 9.4 [3.
147 ological Evaluation Scale and hallucinations/delusions, disorganization, and the deficit syndrome wer
148 ippocampus and other brain regions, paranoid delusions, disorganized speech, deficits in auditory gat
149 y did not find evidence that the presence of delusions distinguished persons with or without a histor
150 e palsy, gait instability and the absence of delusions distinguished PSP from diffuse Lewy body disea
151 chotic symptoms-defined as hallucinations or delusions-during affective episodes were compared in fam
152 that could have theoretically explained the delusion effects, such as those related to subjective va
153 strong evidence of association by using the "delusion" factor as the quantitative trait at three SNPs
154 om-specific networks for impaired memory and delusions, finding that our results matched symptom netw
155 on, this study examined the possibility that delusions form because of disrupted prediction--error pr
156 The major cognitive theories of persecutory delusion formation and maintenance are critically examin
159 results support a neurobiological theory of delusion formation that implicates aberrant prediction-e
162 tive symptoms (g = 0.50; 95% CI, 0.34-0.67), delusions (g = 0.69; 95% CI, 0.45-0.93), hallucinations
163 ing a current persecutory delusion; that the delusion had persisted for at least 3 months; a clinical
165 levant response (>25% reduction in scores on delusions, hallucinations and agitation domains of the N
166 thought disorder; yet the severity of their delusions, hallucinations, and bizarre behavior did not
167 The 9 factors include the often-reported delusions, hallucinations, disorganization, negative, an
168 s of daily living, even after adjustment for delusions, hallucinations, sleep disturbance, and severi
172 Two positive symptoms (suspiciousness and delusions), however, were more severe among successful s
173 k of most BPSDs (HR range, 0.98-0.99) except delusions (HR, 1.00 [95% CI, 1.00-1.01]) and auditory ha
174 ks, we sought a mechanistic understanding of delusions in a sample of medicated and unmedicated patie
175 oral data were compared for individuals with delusions in AD and those without using independent-samp
177 ngs for a specific symptom within a disease (delusions in Alzheimer's disease) localize to a symptom-
179 sions in general nor threat/control override delusions in particular were associated with a higher ri
181 ite growing interest in the phenomenology of delusions in psychosis, at present little is known about
182 he use of non-content-related descriptors of delusions in revealing differences across diagnostic cat
183 al INT alterations for hallucinations versus delusions in the auditory and somatosensory systems, thu
184 s indicate that the emergence of persecutory delusions in untreated schizophrenia explains violent be
185 with a higher risk of violence, particularly delusions in which patients believe that people are seek
189 with the degree of conviction on the Peters Delusions Inventory (original sample: r = 0.32 [P = .05]
192 ministered instruments to assess persecutory delusions is the Persecutory Ideation Questionnaire (PIQ
193 psychosis, which includes hallucinations and delusions, is frequent and debilitating in people with P
194 he Agitation/Aggression, Hallucinations, and Delusions items (Core Total) of the Neuropsychiatric Inv
199 a significant contribution to the idea that delusions may involve both executive and emotional dysfu
200 he Penn State Worry Questionnaire (PSWQ) and delusions measured by the PSYRATS-delusion scale; we did
202 ropsychiatric symptoms such as agitation and delusions occur commonly in elderly patients with dement
203 cantly less likely to be reported as showing delusions (odds ratio=0.40), anxiety (odds ratio=0.43),
204 ncy of delusions, agitation, and depression (delusions: odds ratio [OR], 2.18; 95% CI, 1.15-3.93; P =
206 n suggested that auditory hallucinations and delusions of control in persons with schizophrenia could
209 m experienced auditory hallucinations and/or delusions of control) and 19 normal subjects were studie
210 d consequent sensory events-here captured by delusions of control-should lead to lesser intentional b
212 in addition, by the ability to characterize delusions of different types and diagnoses so as to high
216 thought insertion, strange experiences, and delusions of persecution, were measured before and after
217 , melancholia was often defined primarily by delusions or as the initial phase of a unitary psychosis
219 eep disturbance occurs in most patients with delusions or hallucinations and should be treated as a c
220 ged 18-65 years) with persistent distressing delusions or hallucinations in the context of insomnia a
221 hotic symptoms, defined as the occurrence of delusions or hallucinations, are frequent in Alzheimer d
225 sional symptoms may result in mislabeling as delusions other phenomena that can contribute to violenc
226 d Negative Syndrome Scale (PANSS) items: P1 (delusions), P2 (conceptual disorganisation), P3 (halluci
227 s/signs were added but then dropped: bizarre delusions, passivity symptoms, and mood incongruity.
228 omplications, more severe hallucinations and delusions, poorer attention at baseline, and the develop
229 report the onset of attenuated/subthreshold delusions prior to attenuated/subthreshold hallucination
231 effectiveness in treating hallucinations and delusions, prolonged exposure to antipsychotic medicatio
232 alytic strength increased; however, for more delusion-prone participants, analytic strength predicted
233 timulation to probe the relationship between delusion-proneness and the effect of learned predictions
234 in an observer-rated measure of persecutory delusions (PSYRATS delusion) at 12 weeks (Cohen d, 0.47;
235 ce, other paranoid ideation, quasi-psychotic delusions, quasi-psychotic hallucinations, and true psyc
236 ded psychopathologists continue to urge that delusion reflects a deeper disturbance of mindedness tha
237 ations of people/animals/objects, as well as delusions regarding a place and delusions of misidentifi
240 (PSWQ) and delusions measured by the PSYRATS-delusion scale; we did the analyses in the intention-to-
241 uropsychiatry and the Maudsley Assessment of Delusions Schedule) and interviewed about their displayi
244 icantly greater reductions in the core PANSS delusion score, after 3 years compared with the control
245 xcluded from the analysis, hallucination and delusion scores predicted 22% of the variance in the agg
246 ncluded the thematic content and severity of delusions (scores >=3 using the Scale for the Assessment
248 genome-wide significant association between delusion severity and polymorphisms in intron 1 of NRG3
253 ificantly more likely to have self-elevating delusions such as grandiosity and erotomania compared to
254 ic processes in anomalous beliefs, including delusions, such that cognitive resources may be rallied
255 s hypoactivation correlated with severity of delusions, suggesting a role for abnormal semantic proce
256 hierarchy may explain why hallucinations and delusions tend to cluster together yet sometimes manifes
258 ith schizophrenia generally had more intense delusions than those in other diagnostic categories.
260 etimes lead to bizarre symptoms, such as the delusion that a family member has been replaced by an im
263 ale (PSYRATS) denoting a current persecutory delusion; that the delusion had persisted for at least 3
264 on and maintenance and thence to persecutory delusions - the fixed false beliefs that others intend h
265 at, prior to the onset of hallucinations and delusions, the Noise Pareidolia Task can easily be used
269 controlled mean levels of hallucinations and delusions throughout follow-up (beta = -0.30; t68 = -2.6
270 y more likely to have delusions, and for the delusions to occur in the first 3 years of the disease,
273 structure of delusions across diagnoses and delusion types suggests that even seemingly diverse delu
275 om dimension, which includes suspiciousness, delusions, unusual thought content, and hallucinations (
277 Thus, the population attributable risk for delusion was only 2.62% compared with 14.60% for apathy.
278 relation analysis indicated that severity of delusions was associated with hypometabolism in addition
281 ensional approach to the characterization of delusions was used to examine the use of non-content-rel
285 Finally, 15 lesions causing other types of delusions were connected to expectation violation (P < 0
288 the greatest conviction, whereas persecutory delusions were marked by strong negative affect and a pr
290 rly emergence of comorbid hallucinations and delusions were observed in 57% of individuals with ADAD
292 ostility, suspiciousness, hallucinations, or delusions) were randomly assigned to receive either cita
293 psychotic symptoms (e.g., hallucinations and delusions), were significantly associated with decisiona
294 hiatric features, such as hallucinations and delusions, were scored and compared across pathological
295 tion was not associated with the presence of delusions when confounding variables were included in bi
296 trated sustained effects in the reduction of delusions, which were over and above the effects of anti
297 this study, the minority of individuals with delusions who later relapsed experienced similar delusio
300 symptoms (depressed mood, guilt feelings and delusion, work and activities and psychic anxiety) and s